ライフサイエンスコーパス: macrophage progenitor

1 her megakaryocyte/erythrocyte or granulocyte/macrophage progenitors.

2 aneous appearance of primitive erythroid and macrophage progenitors.

3 CSF, demonstrating that MIP-3 beta attracts macrophage progenitors.

4 teristics of colony-forming unit-granulocyte-macrophage progenitors.

5 rimed multipotent progenitors or granulocyte/macrophage progenitors.

6 sistent with the immunophenotypic profile of macrophage progenitors.

7 genitors but relative sparing of granulocyte-macrophage progenitors.

8 renewing hematopoietic stem cells (HSCs) and macrophage progenitors.

9 oid progenitors and depletion of granulocyte-macrophage progenitors.

10 e very similar to that of normal granulocyte macrophage progenitors.

11 m common myeloid progenitors and granulocyte/macrophage progenitors.

12 d, common myeloid progenitors or granulocyte/macrophage progenitors.

13 264.7 cells and bone marrow-derived monocyte/macrophage progenitors.

14 CML hematopoietic stem cells and granulocyte-macrophage progenitors.

15 differentiation of murine 32Dcl3 granulocyte/macrophage progenitors.

16 s a severe reduction in myeloid (granulocyte/macrophage) progenitors.

17 production prevented cancer-induced HSC and macrophage progenitor amplification and thus restrained

18 iption reduces both the leukemic granulocyte-macrophage progenitor and leukemia-initiating cell (LIC)

19 ith a hematopoietic shift toward granulocyte macrophage progenitor and myeloid cells.

20 progenitors committed to become granulocyte-macrophage progenitors and as megakaryocyte-erythroid pr

21 e common myeloid progenitors and granulocyte-macrophage progenitors and down-regulation of proerythro

22 TPIP2 deficiency causes both an expansion of macrophage progenitors and increased responsiveness of m

23 ed with their normal counterpart granulocyte-macrophage progenitors and myeloblast precursors.

24 tumor-bearing mice induces the expansion of macrophage progenitors and the supply of macrophages.

25 Splenic granulocyte and macrophage progenitors and their descendants were likewi

26 o differentiate into bipotential granulocyte/macrophage progenitors and their progeny.

27 rting increased proliferation by granulocyte/macrophage progenitors and, surprisingly, multipotent pr

28 g common myeloid progenitors and granulocyte-macrophage progenitors, and 4-1BB was inducible on activ

29 lls, common myeloid progenitors, granulocyte-macrophage progenitors, and megakaryocyte-erythroid prog

30 oattracts T cells, B cells, dendritic cells, macrophage progenitors, and NK cells and facilitates den

31 ctivation of beta-catenin in CML granulocyte-macrophage progenitors appears to enhance the self-renew

32 ge lineage outputs from a common granulocyte-macrophage progenitor are still not completely understoo

33 Runx1-deficient granulocyte-macrophage progenitors are characterized by increased cl

34 are markedly increased, whereas granulocyte-macrophage progenitors are reduced.

35 tion with cytomegalovirus, human granulocyte-macrophage progenitors carry the viral genome but fail t

36 -regulation in MDS is related to granulocyte-macrophage progenitor cell sensitivity to TRAIL-induced

37 in bone marrow-derived bipotent granulocyte macrophage progenitor cells (GM-colony forming cell [CFC

38 n myeloid progenitors (CMPs) and granulocyte-macrophage progenitor cells (GMPs) differentiate into PA

39 a vascular adventitial population containing macrophage progenitor cells and investigated their origi

40 ATI-2341-mediated release of granulocyte/macrophage progenitor cells from the bone marrow was con

41 dramatic increase in numbers of granulocyte-macrophage progenitor cells in the marrow and spleen.

42 cterization of resident vascular adventitial macrophage progenitor cells provides new insight into ad

43 es revealed that Sca-1(+)CD45(+) adventitial macrophage progenitor cells were not replenished via the

44 Rather adventitial macrophage progenitor cells were upregulated in hyperlip

45 by C/EBPalpha-p42, and in normal granulocyte/macrophage progenitor cells, we detect C/EBPalpha bound

46 iferation of colony-forming unit-granulocyte-macrophage progenitor cells.

47 oid nodes, bone marrow cells and granulocyte-macrophage progenitor cells.

48 of common myeloid progenitor and granulocyte macrophage progenitor cells.

49 immunomodulatory cells (dendritic cells and macrophages), progenitor cells, vasa vasorum endothelial

50 Granulocyte-macrophage progenitors (CFU-GM) were present only during

51 Unlike normal granulocyte-macrophage progenitors, CML granulocyte-macrophage proge

52 atopoietic stem cells (HSCs) and granulocyte-macrophage progenitors compared with wild-type controls.

53 Granulocyte/macrophage progenitors displayed a relatively normal pro

54 gadd45b-/- colony forming units granulocyte/macrophage progenitors displayed prolonged proliferation

55 Macrophage progenitors expressing the Shp-2 mutants disp

56 cyte-macrophage progenitors, CML granulocyte-macrophage progenitors formed self-renewing, replatable

57 deficient mice display increased bone marrow macrophage progenitor frequency and decreased tissue mac

58 ary leukemic colony-forming unit granulocyte/macrophage progenitors from patients with CML.

59 nt megakaryocyte-erythrocyte and granulocyte-macrophage progenitors give rise to unipotent progenitor

60 Here we use imaging to track granulocyte/macrophage progenitor (GMP) behaviour in mice during eme

61 -erythroid progenitor (MEP), and granulocyte-macrophage progenitor (GMP) cells, accompanied by increa

62 , common myeloid progenitor, and granulocyte/macrophage progenitor (GMP) cells.

63 f the lin-/Sca-1/c-kit (LSK) and granulocyte macrophage progenitor (GMP) compartments at the expense

64 mon myeloid progenitor (CMP) and granulocyte/macrophage progenitor (GMP) populations, and decreased t

65 ased numbers of DCs, even in the granulocyte-macrophage progenitor (GMP), which does not normally pos

66 ion protein selectively expanded granulocyte/macrophage progenitors (GMP) and enhanced their self-ren

67 factor critical for formation of granulocyte-macrophage progenitors (GMP) and leukemic GMP.

68 f splenic cells that derive from granulocyte/macrophage progenitors (GMP) compared with wild-type mic

69 xpands the numbers of LT-HSC and granulocyte/macrophage progenitors (GMP) resulting in chronic MPD.

70 ogenous leukemia (CML), abnormal granulocyte macrophage progenitors (GMP) with nuclear beta-catenin a

71 lls (HSC) or more differentiated granulocyte-macrophage progenitors (GMP).

72 ctivation of beta-catenin within granulocyte-macrophage progenitors (GMP).

73 i-potential progenitors (LMPPs), granulocyte-macrophage progenitors (GMPs) and multi-lymphoid progeni

74 n myeloid progenitors (CMPs) and granulocyte-macrophage progenitors (GMPs) but retained megakaryocyte

75 LPs]) and CMPs and their progeny granulocyte-macrophage progenitors (GMPs) can give rise to functiona

76 ique spatiotemporal mechanism of granulocyte-macrophage progenitors (GMPs) employed in emergency hema

77 We identify a recurrent loss of granulocyte-macrophage progenitors (GMPs) in the bone marrow of low

78 that myeloid differentiation to granulocyte macrophage progenitors (GMPs) is critical for LSC genera

79 ag1(-/-) mice, lineage-committed granulocyte-macrophage progenitors (GMPs) or bone marrow-derived mac

80 n myeloid progenitors (CMPs) and granulocyte-macrophage progenitors (GMPs) preferentially differentia

81 m wild-type and Dicer1-deficient granulocyte-macrophage progenitors (GMPs) revealed that 20 miRNA fam

82 have identified a population of granulocyte-macrophage progenitors (GMPs) that were highly enriched

83 xpression in MLL-AF9-transformed granulocyte macrophage progenitors (GMPs) that were initially Evi1(n

84 generation of granulocytes from granulocyte-macrophage progenitors (GMPs) were markedly reduced in S

85 mmon myeloid progenitors (CMPs), granulocyte/macrophage progenitors (GMPs), and thymocyte progenitors

86 e differentiation in bipotential granulocyte-macrophage progenitors (GMPs), its role in regulating ce

87 mmon myeloid progenitors (CMPs), granulocyte-macrophage progenitors (GMPs), megakaryocyte-erythrocyte

88 mon lymphoid progenitors (CLPs), granulocyte-macrophage progenitors (GMPs), or early thymic progenito

89 rogenitor differentiation toward granulocyte-macrophage progenitors (GMPs), resulting in a myeloproli

90 evated beta-catenin signaling in granulocyte macrophage progenitors (GMPs), which enables this popula

91 genitors partially overlaps with granulocyte-macrophage progenitors (GMPs).

92 HSCs/multipotent progenitors and granulocyte/macrophage progenitors have self-renewal capability, res

93 d LPS, and may play a role in trafficking of macrophage progenitors in and out of the bone marrow in

94 Osteoclasts arise from macrophage progenitors in bone marrow (BMMs) as a conseq

95 ed numbers of peripheral-blood monocytes and macrophage progenitors in bone marrow.

96 phage depletion is associated with increased macrophage progenitors in bone marrow.

97 numbers of colonies derived from granulocyte-macrophage progenitors in cultures supplemented with low

98 sideration of the broader existence of local macrophage progenitors in other tissues.

99 osure to erythemal UV radiation can modulate macrophage progenitors in the BM such that their differe

100 bited some degree of toxicity to granulocyte/macrophage progenitors in the bone marrow of mice.

101 common myeloid progenitors, and granulocyte/macrophage progenitors in the spleen.

102 FU-E), mast cell and bipotential granulocyte/macrophage progenitors in the yolk sac.

103 C/EBPalpha(-/-) FL lacked macrophage progenitors in vitro and had impaired ability

104 rogenitor lineage skewing toward granulocyte-macrophage progenitors, increased colony-forming unit gr

105 logy reported in TTP (-/-) animals, and that macrophage progenitors may be involved in the transplant

106 Steady-state numbers of granulocyte-macrophage progenitors, myeloid-lineage cells and mature

107 Clonogenic macrophage progenitors of fetal origin were present in t

108 The granulocyte-macrophage progenitor pool from patients with CML in bla

109 the same myeloid-restricted pre-granulocyte-macrophage progenitor (pre-GM) (Lin(-)Sca-1(-)c-Kit(+)CD

110 HSC, multipotent progenitor, and granulocyte/macrophage progenitor proliferation and reactive neutrop

111 rophage colony-forming unit (CFU-GM) growth, macrophage progenitor proliferation, and activation of t

112 During latency in granulocyte-macrophage progenitors, RC2710 DNA was detected at level

113 begins to be up-regulated at the granulocyte-macrophage progenitor stage and continues throughout mye

114 leukemia evolving from committed granulocyte-macrophage progenitors that have acquired the self-renew

115 tal latent infection of cultured granulocyte-macrophage progenitors, the viral genome was detected in

116 eukaemias initiated in committed granulocyte macrophage progenitors through introduction of the MLL-A

117 Consistently, macrophage progenitors transduced with the Shp-2 mutants

118 but differentiation from CMP to granulocyte/macrophage progenitor was decreased, and the mature gran

119 nd progenitor cells, notably granulocyte and macrophage progenitors, which produced CD11b(+) Ly-6C(hi

120 ique in that it is initiated by pericyte and macrophage progenitors, with endothelial cell recruitmen