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2 alpha M (ITGAM; CD11b) is a component of the macrophage-1 antigen complex, which mediates leukocyte a
4 ), protease-activated receptor 3 (PAR3), and macrophage-1 antigen (Mac-1) blocked APC inhibition of N
5 ffects were reversed with antibodies against Macrophage-1 antigen (Mac-1) or intercellular adhesion m
7 of traditional circulating cytokines, where macrophage-1 antigen-mediated microglia proinflammatory
9 t of the alphaM/beta2 integrin (CD11b/CD18), macrophage-1 antigen, or complement receptor 3, as a cel
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