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1                          This confirmed that macrophage-derived 15-PGDH was responsible for catalyzin
2 y, stress markedly increased the contents of macrophage-derived [(3)H]cholesterol in the intestinal l
3 n of vascular and cardiac CB(1) receptors by macrophage-derived and platelet-derived endocannabinoids
4  responsible for the increased expression of macrophage-derived angiogenic activity.
5                     To determine the role of macrophage-derived angiotensin in the development of ath
6                                              Macrophage-derived AnxA1 plays a functional role in modu
7 ata provide in vivo evidence suggesting that macrophage-derived apoE delays development of atheroscle
8                                              Macrophage-derived apoE in the vessel wall has important
9  we evaluated whether the effect produced by macrophage-derived apoE3 is related to its ability to bi
10                                              Macrophage-derived apolipoprotein E (apoE) in the vessel
11                                        Thus, macrophage-derived Arg I protects hosts against excessiv
12                            The monocyte- and macrophage-derived beta-chemokines were sufficient to bl
13         HA challenge resulted in cleavage of macrophage-derived caspase1 and IL-1beta, suggesting a r
14 d by Ly6C(high)CCR2(+) inflammatory monocyte/macrophage-derived CCL11.
15 s in the liver and highlight a rationale for macrophage derived cell therapy in regenerative medicine
16 LAL and chLAL distributed to macrophages and macrophage-derived cells of various organs.
17 localize to the actin-rich phagocytic cup of macrophage-derived cells, suggesting the complex may reg
18 ts: nitrosoperoxycarbonate (ONOOCO(2)(-)), a macrophage-derived chemical mediator of inflammation, an
19 sion of macrophage inflammatory proteins and macrophage-derived chemokine (CCL22) in the synovial tis
20 p-regulation of CCR4 and one of its ligands, macrophage-derived chemokine (CCL22), and that tolerance
21 s toxin, neutralization of the CC chemokine, macrophage-derived chemokine (CCL22), or by desensitizat
22                     One anti-CXCR4 factor is macrophage-derived chemokine (chemokine ligand 22, CCL22
23                                              Macrophage-derived chemokine (MDC) is a potent chemoattr
24             Interestingly, neutralization of macrophage-derived chemokine (MDC) with Ab caused a sign
25 nd activation-regulated chemokine (TARC) and macrophage-derived chemokine (MDC)), CD (10 proteins), a
26           We analyzed the functional role of macrophage-derived chemokine (MDC), a potent mononuclear
27  (IL)-12, tumor necrosis factor (TNF)-alpha, macrophage-derived chemokine (MDC), and C10, known to en
28 crophage colony-stimulating factor (GM-CSF), macrophage-derived chemokine (MDC), and macrophage infla
29 induce the release of CC chemokines, RANTES, macrophage-derived chemokine (MDC), macrophage inflammat
30                  It is now reported that the macrophage-derived chemokine (MDC), thymus activation-re
31 arily as a mixture of three beta chemokines [macrophage-derived chemokine (MDC), thymus and activatio
32  and I-309 were induced by LPS; in addition, macrophage-derived chemokine (MDC), thymus and activatio
33 nd activation-regulated chemokine (TARC) and macrophage-derived chemokine (MDC).
34 induced protein of 10-kDa (IP-10)/CXCL10 and macrophage-derived chemokine (MDC)/CCL22 production were
35 activation-regulated chemokine (TARC)/CCL17, macrophage-derived chemokine (MDC)/CCL22, I-309/CCL1) an
36 rombin, or others like epinephrine (EPI) and macrophage-derived chemokine (MDC, CCL22).
37 te chemoattractant protein-3 (MCP-3/CCL7) or macrophage-derived chemokine (MDC/CCL22), elicited anti-
38 kine/CC chemokine ligand 17 (TARC/CCL17) and macrophage-derived chemokine (MDC/CCL22), which preferen
39 on regulated chemokine (TARC; or CCL17), and macrophage-derived chemokine (MDC; or CCL22).
40 CP-4, IL-8, interferon-inducible protein-10, macrophage-derived chemokine [MDC], and platelet factor-
41            TSLP-activated human pDCs secrete macrophage-derived chemokine CCL-22 and thymus- and acti
42 ed CD169(+)marginal zone macrophages and the macrophage-derived chemokine CCL22 to increase splenic C
43        Alcohol increased IL-10 and decreased macrophage-derived chemokine concentrations, whereas the
44 d other inflammatory diseases by suppressing macrophage-derived chemokine production via the EP4 rece
45 ial paracrine effect of endogenous PGE(2) on macrophage-derived chemokine production, we co-cultured
46                                              Macrophage-derived chemokine was present in all samples.
47  (thymus and activation-regulated chemokine, macrophage-derived chemokine) correlated with airway eos
48 d activation-regulated chemokine) and CCL22 (macrophage-derived chemokine).
49 ated chemokines CCL11 (eotaxin-1) and CCL22 (macrophage-derived chemokine).
50                  Enhanced secretion of IL-8, macrophage-derived chemokine, and MIP-1alpha was also ob
51 L-6, IL-8, CD38, and CD69 and down-regulated macrophage-derived chemokine, human leukocyte antigen DR
52 38, and CD69 were reduced, whereas levels of macrophage-derived chemokine, human leukocyte antigen DR
53 ymus- and activation-regulated chemokine and macrophage-derived chemokine, ligands for CCR4, were mea
54 ruiting chemotactic factors, including IL-8, macrophage-derived chemokine, macrophage inflammatory pr
55  platelet aggregation induced by SDF-1alpha, macrophage-derived chemokine, or thymus and activation-r
56 A-specific IgE responses, but have defective macrophage-derived chemokine-mediated CD4+ T cell migrat
57 a) but lower levels of the anti-inflammatory macrophage-derived chemokine.
58 and activation-regulated chemokine/CCL17 and macrophage-derived chemokine/CCL22 in the lung after all
59 L, the production of the Th2-chemokines MDC (macrophage-derived chemokine/CCL22) and TARC (thymus and
60 ted chemokine; also known as CCL17) and MDC (macrophage-derived chemokine; CCL22).
61 ion regulated chemokine; TARC) and CCL22 (or macrophage-derived chemokine; MDC), in Th2-type cytokine
62 ct the entire lung and induce high levels of macrophage-derived chemokines and cytokines, which resul
63 A)R expression was increased in macrophages, macrophage-derived chemokines were reduced in response t
64 mus-expressed chemokine, eotaxin, eotaxin 2, macrophage-derived chemokines, and C10 were also induced
65 m, i.e., the inhibition of the production of macrophage-derived chemokines.
66  LA-apoA-I-/- mice had significantly reduced macrophage-derived cholesterol esterification and revers
67 ch led to a significant increase in LDL- and macrophage-derived cholesterol fecal excretion (both P <
68  absorption, which in turn promotes LDL- and macrophage-derived cholesterol fecal excretion.
69      These data identify Cat-S as a monocyte/macrophage-derived circulating PAR2 agonist and mediator
70    Taken together, our results indicate that macrophage-derived CO permits efficient and coordinated
71  VIP and PACAP inhibit the expression of the macrophage-derived CXC chemokines macrophage inflammator
72 esity is associated with accumulation of the macrophage-derived cytokine osteopontin (OPN) in adipose
73                                     However, macrophage-derived cytokine production is diminished by
74 anied by changes in plasma concentrations of macrophage-derived cytokine, eotaxin, IL10, TIMP1, VEGF,
75 -1beta, and TNF-alpha) and chemokine (MCP-1, macrophage-derived cytokine, monokine-induced IFN-gamma,
76 oinflammatory and anti-inflammatory monocyte/macrophage derived cytokines in serum/plasma), reduced T
77                              Proinflammatory macrophage-derived cytokines may sustain and/or enhance
78  conditioning of tissue microenvironments by macrophage-derived cytokines may therefore lead to defec
79  mouse colons identified the neutrophil- and macrophage-derived damage products 3-chlorotyrosine (Cl-
80               We now show that tumor ascites macrophage-derived dendritic cells induced tumor-associa
81                     Furthermore, exposure of macrophage-derived dendritic cells to apoptotic non-T ce
82 urther cytokine stimulation, and, similar to macrophages derived during EAU recovery, behave operatio
83 ion of IL-12 and IFN-gamma, and induction of macrophage-derived effector molecules like NO.
84 cates macrophage metalloelastase (MMP-12), a macrophage-derived elastinolytic protease in inflammatio
85                                              Macrophage-derived endocannabinoids have been implicated
86 and contained remarkably few macrophages and macrophage-derived epithelioid cells and giant cells.
87                                    Therefore macrophage-derived EV-packaged WNTs are essential for re
88 s, would impact on ERalpha and we found that macrophage-derived factors caused loss of ERalpha expres
89          Importantly, a combined blocking M2 macrophage-derived factors TGF-beta, VEGF and SDF-1 abol
90                             If stimulated by macrophage-derived factors, however, RGCs can regenerate
91  macrophage infiltration but also exploiting macrophage-derived fibroblast growth factors (FGFs).
92 the exact relationship between ER stress and macrophage-derived foam cell formation and whether ER st
93 here they differentiate into macrophages and macrophage-derived foam cells and cause atherosclerotic
94                                              Macrophage-derived foam cells are thought to play a majo
95                                              Macrophage-derived foam cells in atherosclerotic lesions
96                                              Macrophage-derived foam cells in developing atherosclero
97                     These data indicate that macrophage-derived foam cells in the lesion derive mainl
98                Co-culture of large SMCs with macrophage-derived foam cells induced a transition to th
99                                              Macrophage-derived foam cells promote selective migratio
100  consistent with multiple roles for Prx I in macrophage-derived foam cells that include functionality
101 plaque tissues of the diseased portion, only macrophage-derived foam cells were retrieved.
102 hanism of action, we treated macrophages and macrophage-derived foam cells with exogenous TIMP-2 in v
103  and diseased portions after co-culture with macrophage-derived foam cells).
104 rnatively activated alveolar macrophages and macrophage-derived foam cells, both cell types relevant
105 f ritonavir on cholesterol efflux from human macrophage-derived foam cells, which is a critical facto
106 rosclerotic lesions through the formation of macrophage-derived foam cells.
107 ic properties and biological activity to the macrophage-derived FOG(7).
108 ucive to increase in motility and control of macrophage-derived free radicals provides survival and p
109 o anti-inflammatory effects on LPS-activated macrophages (derived from THP-1 cell line) and using the
110           LPS-stimulated elicited peritoneal macrophages derived from 12/15-LO-deficient (Alox15) mic
111 extended to other TLR ligands and to primary macrophages derived from a healthy donor.
112 B6) restricts L. pneumophila growth, whereas macrophages derived from A/J mice allow >10(3)-fold bact
113 whereas the capacity for bone marrow-derived macrophages derived from A7(-/-) mice to take up oligome
114                Adoptive transfer of alveolar macrophages derived from Abx-treated mice was sufficient
115 ys than their conventional counterparts, and macrophages derived from aged germ-free mice maintain an
116                                   Peritoneal macrophages derived from all three genotypes overexpress
117 cal hypotheses, we performed WGBS of primary macrophages derived from an experimental rat system of g
118                                              Macrophages derived from animals lacking SLAT show an el
119            Complement C5-deficient (C5(-/-)) macrophages derived from B.10 congenic mice were found t
120                                              Macrophages derived from bone marrow of MyD88-deficient
121                                              Macrophages derived from bone marrow produced sufficient
122 l-like receptor 4, as each was diminished in macrophages derived from C3H/HeJ mice.
123  translocation were discerned in bone marrow macrophages derived from C57BL/6 mice, which are primary
124                        The profiles classify macrophages derived from CBA/Ca mice as M1-like (pro-inf
125  LPS-stimulated cytokine responses of mature macrophages derived from CD117 and ER-MP12 bone marrow p
126 ivation of HCMV, since virus was observed in macrophages derived from CD14(+) monocytes stimulated by
127 IL-1beta release was inhibited in peritoneal macrophages derived from CD44-deficient mice, in an MH-S
128 thesis was supported by the observation that macrophages derived from chronic proliferative dermatiti
129 ed AKI, there was an early increase in renal macrophages derived from circulating inflammatory (M1) m
130 ne corresponded with an increasing number of macrophages derived from circulating monocytes (bromodeo
131     Similarly, the addition of hyaluronan to macrophages derived from cryopyrin-deficient mice increa
132                                We found that macrophages derived from donor hematopoietic precursors
133 induced SOCS-1 expression in comparison with macrophages derived from Egr-1(+/+) littermate controls.
134 and inhibition of TLR4 expression is lost in macrophages derived from Enos(-/-) mice.
135                                              Macrophages derived from Fpr2-KO mice showed a more pote
136 tudies were performed using peripheral blood macrophages derived from healthy donors and treated with
137 cell line THP-1, and extend these results to macrophages derived from healthy individuals and HH pati
138 uring development and throughout adult life, macrophages derived from hematopoietic progenitors are s
139          Here, we describe an approach using macrophages derived from human induced pluripotent stem
140 cts of conditioned media from CCL5-activated macrophages derived from human-CCR5ki mice on PA-SMCs fr
141 VS intracellular growth by as much as 95% in macrophages derived from IFN-gamma receptor knockout (IF
142                                    Moreover, macrophages derived from IL-10(-/-) mice exhibit enhance
143                                  Bone marrow macrophages derived from IL-23p19(-/-) mice showed a slo
144 ated with iNOS inhibitor, and primary murine macrophages derived from iNOS knockout mice.
145 o 2.5 times as efficient as that observed in macrophages derived from littermate controls.
146  similar findings were observed with primary macrophages derived from lung, peritoneum, and blood but
147 ctivation of the inflammasome is enhanced in macrophages derived from lupus patients.
148      LXRbeta selectivity was confirmed using macrophages derived from LXR mutant mice.
149 r mRNAs was greatly attenuated in peritoneal macrophages derived from LXRalpha/beta null mice.
150  replication in cultures of CD4+ T cells and macrophages derived from macaques.
151 was prevented by SB 203580 and suppressed in macrophages derived from MAPKAP-K2-deficient mice.
152 utively phosphorylated on residue Thr(72) in macrophages derived from mice homozygous for the motheat
153                                        Using macrophages derived from mice unable to make TNF-alpha,
154        Also, in vitro studies with polarized macrophages derived from mice with macrophage-specific l
155 lysis to interrogate the transcriptome of M1 macrophages derived from mice with macrophage-specific l
156                Furthermore, we observed that macrophages derived from Mmp28(-/-) mice migrated faster
157 ually been considered to represent activated macrophages derived from monocytes entering the lesions
158                                           In macrophages derived from most mouse strains, the LCP is
159                           The utilization of macrophages derived from MyD88-, TRIF-, Toll-like recept
160           Moreover, IL-1beta production from macrophages derived from Nlrp3(A350V) knockin mice, whic
161                           Furthermore, using macrophages derived from NOS(-/-) and Phox(-/-) mice, we
162 es in EAE, an adoptive transfer of activated macrophages derived from Notch1(fl/fl) x Mx1cre(+/-) (No
163 production of proinflammatory cytokines from macrophages derived from overweight/obese subjects with
164       Ex vivo studies indicate that alveolar macrophages derived from overweight/obese subjects with
165 nd treated with TNF and using synovial fluid macrophages derived from patients with RA.
166                 It is unknown to what extent macrophages derived from peripheral blood adopt the phen
167                                        Human macrophages derived from peripheral blood monocytes expr
168 ethods were applied to NAD biosynthesized by macrophages derived from peripheral blood monocytes.
169          The six mutants also were tested in macrophages derived from peripheral blood mononuclear ce
170                                 In contrast, macrophages derived from premorbid rats do not exhibit a
171                            Here we have used macrophages derived from primary explants of bone marrow
172 on using a clinical isolate of HCMV (TR) and macrophages derived from primary human monocytes.
173 monary epithelial cells (A549) as well as in macrophages derived from primary human peripheral blood
174 2(-) and chemokine (C-C motif) receptor 2(+) macrophages derived from primitive yolk sac, recombinati
175 ed from mouse bone marrow and synovial fluid macrophages derived from RA patients were also tested fo
176 y of Rac2 to Rac1 (92% amino acid identity), macrophages derived from Rac2-/- mice, which continue to
177 high-throughput sequencing of RNA in primary macrophages derived from rodents and humans, we establis
178 essing cells were MCMV-infected RPE cells or macrophages derived from RPE cells.
179 -induced events were compared in bone marrow macrophages derived from SHIP(+/+) and SHIP(-/-) mice.
180                 CR3-mediated phagocytosis in macrophages derived from SHIP(-/-) mice was up to 2.5 ti
181 o this end, we used immortalized bone marrow macrophages derived from SHP-1-deficient motheaten mice
182 nduced TNF and IL-1 production are normal in macrophages derived from spin mice.
183                                 In addition, macrophages derived from SR-A-/- mice were substantially
184 olded protein response (UPR) is activated in macrophages derived from the bone marrow of HLA-B27 tran
185                                Consistently, macrophages derived from the bone marrow of Sirt6(-/-) m
186 e lipophilic antioxidant drug, probucol, and macrophages derived from the human monocyte cell line, T
187 nd multimer formation also occurred in human macrophages derived from the monocyte cell line THP-1.
188 induce an IL-1beta response in primary human macrophages derived from the same blood donors as the mo
189 ppeared normal, but cultured fibroblasts and macrophages derived from them exhibited increased stabil
190                       In bone marrow-derived macrophages derived from these mice LTbetaR-induced cros
191 itro studies with bone marrow and peritoneal macrophages derived from these mice showed that treatmen
192                                              Macrophages derived from these recruited monocytes parti
193                     It is still not clear if macrophages derived from these two populations differ in
194                     LPS-activated peritoneal macrophages derived from TIA-1-/- mice produced signific
195              In this study, we have utilized macrophages derived from TRAF6 knock-out mice and myeloi
196                           IL-10 expressed by macrophages derived from transduced BMCs inhibited ather
197                                              Macrophages derived from volunteers homozygous for the r
198 tion by, and TNF messenger RNA expression of macrophages derived from volunteers with known TNF (-308
199                      In this report, we used macrophages derived from wild type (wt) mice and from mi
200                                        Using macrophages derived from wild type and Cd36(-/-) mice to
201  In this study, we analyzed the responses of macrophages derived from wild-type (IFN-beta(+/+)) mice
202                                   Peritoneal macrophages derived from wild-type and TIA-1-/- mice wer
203                     Microarray analysis with macrophages derived from wild-type and TLR4-deficient mi
204 lar processing of CpG-siRNA, we used primary macrophages derived from wild-type and Tlr9-deficient mi
205 t of B7-1 and B7-2 expression on B cells and macrophages derived from wild-type BALB/c mice after in
206                     In this report, by using macrophages derived from wild-type mice (having both rec
207 dysregulated cytokine production compared to macrophages derived from wild-type mice.
208 igration in response to sFasL compared to M1 macrophages derived from young animals.
209 nt differences between the responsiveness of macrophages derived from younger donors and that from ol
210 nhanced the engulfment of necrotic debris by macrophages derived from zymosan-induced peritonitis, M1
211 espective global gene expression profiles of macrophages, derived from human monocytes by GM-CSF or M
212 ferences between the respective profiles for macrophages, derived from murine bone marrow cells by ea
213 SAP triggers a potent, complement-dependent, macrophage-derived giant cell reaction that swiftly remo
214 2+))-binding protein oncomodulin as a potent macrophage-derived growth factor for RGCs and other neur
215 ts of inflammation were shown to require the macrophage-derived growth factor Oncomodulin (Ocm).
216 monstrate that human monocytes, by releasing macrophage-derived HB-EGF, enhance DDR in neighboring ce
217 cate that strategies to reduce activation of macrophage derived HIF-1alpha may be used as a target to
218         These combined data demonstrate that macrophage-derived HL significantly contributes to early
219 with macrophages they did respond to LPS via macrophage-derived IFN-beta; (2) macrophages required ty
220 ediated by an extremely low concentration of macrophage-derived IFNbeta.
221 ugh the induction of astrocyte and microglia-macrophage-derived IGF-1.
222 eation; and 4) increasing levels of infected macrophage derived IL-10 promotes bacterial persistence
223                           In response to Ad, macrophage-derived IL-1 alpha triggered IL-1RI-dependent
224 ing reinforced an autocrine feedback loop of macrophage-derived IL-10 and this synergized with inhibi
225              We previously demonstrated that macrophage-derived IL-10 could contribute to disease exa
226                                     We found macrophage-derived IL-10 dispensable for gut homeostasis
227           Further, following mucosal injury, macrophage-derived IL-10 resulted in epithelial cAMP res
228 ine effect of IFN-gamma on the production of macrophage-derived IL-10 with lipoprotein as a stimulant
229 h a cytokine-signaling network that involves macrophage-derived IL-1beta and fibroblast-derived CXCR2
230  both peripheral blood monocyte- and uterine macrophage-derived IL-1beta induce secretion of antimicr
231                                              Macrophage-derived IL-1beta production was induced by HF
232 culture experiments identified the effect of macrophage-derived IL-1beta to promote IL-22 and IL-17 p
233 in the intestine by inhibiting expression of macrophage-derived IL-23.
234                         A novel role for the macrophage-derived immunoregulatory cytokine IL-27 was i
235                             MMP-12 is solely macrophage derived in this model, being expressed by tum
236 adipose tissue and muscle, and it suppresses macrophage-derived inflammation.
237                However, most of the analyzed macrophage-derived inflammatory and regulatory cytokines
238 ity was elevated in recurrent GBM, driven by macrophage-derived insulin-like growth factor-1 (IGF-1)
239                                              Macrophage-derived insulin-like growth factor-I (IGF-I)
240 s) in a mouse hepatitis virus (MHV)-infected macrophage-derived J774.1 cell line showed activation of
241  by the abnormal clonal proliferation of the macrophage-derived Langerhans cell.
242  functioning as anti-inflammatory cells, and macrophage-derived matrix metalloproteinases regulate fi
243                                        Wound macrophage-derived MFG-E8 was recognized as a critical d
244                       Here, we characterized macrophage-derived microvesicles and explored their role
245 F) circulates in plasma, largely on monocyte/macrophage-derived microvesicles that can bind activated
246 We found that RNA molecules contained in the macrophage-derived microvesicles were transported to tar
247  We also identified the miRNA content of the macrophage-derived microvesicles.
248 ng antimelanoma immune responses reveal that macrophage-derived MIF participates in macrophage altern
249 lymerase chain reaction array, we found that macrophage-derived miR-342-5p and miR-155 are selectivel
250                                              Macrophage-derived miR-342-5p promotes atherosclerosis a
251                                              Macrophage-derived MMP-12 regulates elastin degradation
252                  These findings suggest that macrophage-derived MMP-9 and mesenchymal cell MMP-2 are
253                       Although monocyte- and macrophage-derived molecules are known to promote extrac
254  mouse model of AAA, we now demonstrate that macrophage-derived MT1-MMP plays a dominant role in dise
255 network during AAA formation is dependent on macrophage-derived MT1-MMP, which unexpectedly serves as
256 d triggers the rapid clearance of amyloid by macrophage-derived multinucleated giant cells.
257 litates hydroxyapatite nucleation within the macrophage-derived MV membrane.
258                We tested the hypothesis that macrophage-derived MVs contribute directly to microcalci
259                                     Alveolar macrophage-derived MVs were fully internalized by alveol
260 neutrophil-specific CXC chemokines (alveolar macrophage-derived neutrophil chemotactic factor [AMCF]-
261 pes demonstrated that neural progenitor- and macrophage-derived NRP1 were dispensable, whereas endoth
262                          We demonstrate that macrophage-derived ODC is a critical regulator of M1 mac
263 on to its role in inducing IL-22 production, macrophage-derived or CD103(-) CD11b(+) DC-derived IL-23
264 s-stimulated macrophages induced bone marrow macrophage-derived osteoclastogenesis.
265     Dihydroethidium (DHE) was used to detect macrophage-derived oxidants generated during phagocytosi
266 ctly affect IL-2 production by Thp cells via macrophage-derived PPARgamma ligands.
267                F4/80(+)CD11b(+)CD11c(dull/-) macrophage-derived proinflammatory cytokines significant
268           We recently identified a family of macrophage-derived proresolving and tissue regenerative
269 found to mediate pro-inflammatory effects of macrophage-derived prostaglandin E2 (PGE2) on Th17 cells
270 acrophages in inflammation, the functions of macrophage-derived proteinases are typically relegated t
271                             We proposed that macrophage-derived proteinases may contribute to the ant
272 trix metalloproteinases (MMPs), particularly macrophage-derived proteinases, in COPD pathogenesis.
273         After gel-filtration chromatography, macrophage-derived proteins > or =30 kDa were found to b
274 ivation and increased expression of monocyte/macrophage-derived proteins fostering wound healing.
275 sent in the vitreous, acting in concert with macrophage-derived proteins, stimulates mature rat RGCs
276  cAMP-dependent and was augmented further by macrophage-derived proteins.
277  in mouse primary astrocytes, microglia, and macrophage-derived RAW264.7 cells induced by interferon-
278 fine for the first time, to our knowledge, a macrophage-derived regulator of placental growth during
279 baseline, demonstrating a regulatory role of macrophage-derived ROS in autoimmunity.
280 study points to the utility of monocyte- and macrophage-derived sCD163 as a marker of HIV activity th
281  ferritin, we hypothesize that ferritin is a macrophage-derived signal that promotes oligodendrogenes
282                         Here, we showed that macrophage-derived soluble factors induce canonical Wnt
283 ssion of IgA class switching in B cells by a macrophage-derived sterol in response to TLR activation
284 nhancement of apoptotic neutrophil uptake by macrophage-derived TG2 restrains gout-like neutrophilic
285 -permissive environment by identification of macrophage-derived therapeutic molecules may therefore a
286  invasion, these results suggest that excess macrophage-derived TNF-alpha augments expression of MMP-
287 ed with NK cell-derived IFN-gamma and either macrophage-derived TNF-alpha or IL-1beta synergistically
288 iting TNF in pathologies primarily driven by macrophage-derived TNF.
289 of sepsis support the central role played by macrophage-derived TNFalpha in sepsis.
290 ls produced an angiogenic response driven by macrophage-derived TNFalpha.
291                             The potential of macrophage-derived transforming growth factor beta1 to p
292 subjects with dAIH, indicating that monocyte/macrophage-derived triggers might play a central role in
293                                              Macrophage-derived tumor necrosis factor (TNF)-alpha has
294                            We tested whether macrophage-derived uPA plays essential roles in macropha
295 ental role in macrophage chemotaxis and that macrophage-derived uPA promotes efficient muscle regener
296 al (ECs) and/or perivascular cells (PVCs) (a macrophage-derived vascular cell type) is implicated in
297                                The switch of macrophage-derived VEGF-A during the early stage of tiss
298 repair phases, and the complementary role of macrophage-derived VEGF-A in coordinating effective tiss
299                 Here, we analyse the role of macrophage-derived WNT in intestinal repair in mice by i
300                Previously, it was shown that macrophage-derived Wnt molecules promote vascular remode

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