1                             CD11b+ monocytic/macrophagic cells (Mo/Mph), which infiltrate into skin a

2 int, small numbers of infiltrating monocytic/macrophagic cells are already apparent.

3 n of class II MHC+CD11b+Gr-1+ and -monocytic/macrophagic cells in the dermis and epidermis.

4                             CD11b+ monocytic/macrophagic cells that infiltrate human skin after in vi

5  while the activities in P388D1 and RAW264.7 macrophagic cells were considerably higher than the basa

6                    Taken together, monocytic/macrophagic cells with high IL-10 and low IL-12 expressi

7 ocytes or class II MHC+CD11b+Gr-1+ monocytic/macrophagic cells.

8 ng the bacteria to gain direct access to the macrophagic cytoplasm.

9 vigate the 48-h "viability gate" and undergo macrophagic differentiation has remained elusive.

10 w levels in progenitor cells of granulocytic-macrophagic, erythroid, or megakaryocytic lineage detect

11 oach, we showed that muscles did not require macrophagic hif1a or hif2a to regenerate.

12                  These results indicate that macrophagic HIFs do not play a crucial role during skele

13 surface receptors of IL-18-responsive NK and macrophagic KG-1 cells.

14 ain metabolic abnormalities in patients with macrophagic myofascitis (MMF) and the relationship with

15 tently downregulated in the CD11b+ monocytic/macrophagic population (p < 0.0002).

16 increased to 70%, the infiltrating monocytic/macrophagic population had risen to 1.2% of the total de

17 lative to 0 h by the CD11b+ dermal monocytic/macrophagic population peaked at 24-48 h and tapered the

18 dermal CD11b+ (CD1-, 3-, 24-, 56-) monocytic/macrophagic population.

19 studies, HIV production was increased in the macrophagic U1 cell line expressing the HIV genome after