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1 e, with implications for the pathogenesis of macrosomia.
2 th SGA(2SD) and did not decrease the odds of macrosomia.
3 d with SGA(2SD) but did decrease the odds of macrosomia.
4 yperinsulinemic hypoglycemia associated with macrosomia.
5 yperinsulinemic hypoglycemia associated with macrosomia.
6 t outcomes, including preeclampsia and fetal macrosomia.
7 ted fetal development with increased risk of macrosomia.
8 ia appears not to be the sole cause of fetal macrosomia.
9 ood glucose concentrations may contribute to macrosomia, adiposity, and poorer vascular health in the
10 ic status) and the effects of exclusion (for macrosomia and maternal diabetes) to be examined.
11                 Three infants presented with macrosomia and severe hypoglycemia with a positive famil
12 comes: placental weight, head circumference, macrosomia, Apgar score, small for gestational age, larg
13 .14, 0.78 per 1-mmol/L increase) and risk of macrosomia (birth weight >4000 g) (RR = 1.21; 95% CI: 1.
14 e-ethnicity-specific mean BW (SGA(2SD)), and macrosomia (BW ge 4500 g).
15 s of women with deflation had a high risk of macrosomia compared with controls (adjusted RR 0.40, p=0
16 rcent and 13 percent, respectively); who had macrosomia, defined as a birth weight of 4000 g or more
17 ciated with embryonal cancers, macroglossia, macrosomia, ear pits or ear creases, and midline abdomin
18  weight, participants who had a sibling with macrosomia had a lower mean IQ score.
19 in less preeclampsia, shoulder dystocia, and macrosomia; however, current evidence does not show an e
20                    Seven percent of neonatal macrosomia in all the population, and 13% in Black mothe
21 ases of preeclampsia, shoulder dystocia, and macrosomia in the treated group.
22 ery for ultrasonographically diagnosed fetal macrosomia is medically and economically unsound.
23 luding admission to neonatal intensive care, macrosomia, low Apgar scores, and perinatal death.
24                     (ii) Perinatal outcomes: macrosomia, low birth weight, admission to neonatal inte
25 and surrounding genes increases the risk for macrosomia, mild developmental delay and pervasive devel
26 low birth weight, P < .05; 7.7% vs 14.6% for macrosomia, P < .05).
27  reported [1 study]; and 0% vs 2.6%-4.3% for macrosomia, P = not reported [1 study] and P = .28 [1 st
28                             Late gestational macrosomia was absent, apparently requiring a different
29 ents with midline abdominal-wall defects and macrosomia was significantly higher, 65% (41/63) and 60%
30 elivered at term via cesarean section due to macrosomia, with a reported birth weight of 11 lb 8.7 oz

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