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1 e, with implications for the pathogenesis of macrosomia.
2 th SGA(2SD) and did not decrease the odds of macrosomia.
3 d with SGA(2SD) but did decrease the odds of macrosomia.
4 yperinsulinemic hypoglycemia associated with macrosomia.
5 yperinsulinemic hypoglycemia associated with macrosomia.
6 t outcomes, including preeclampsia and fetal macrosomia.
7 ted fetal development with increased risk of macrosomia.
8 ia appears not to be the sole cause of fetal macrosomia.
9 ood glucose concentrations may contribute to macrosomia, adiposity, and poorer vascular health in the
12 comes: placental weight, head circumference, macrosomia, Apgar score, small for gestational age, larg
13 .14, 0.78 per 1-mmol/L increase) and risk of macrosomia (birth weight >4000 g) (RR = 1.21; 95% CI: 1.
15 s of women with deflation had a high risk of macrosomia compared with controls (adjusted RR 0.40, p=0
16 rcent and 13 percent, respectively); who had macrosomia, defined as a birth weight of 4000 g or more
17 ciated with embryonal cancers, macroglossia, macrosomia, ear pits or ear creases, and midline abdomin
19 in less preeclampsia, shoulder dystocia, and macrosomia; however, current evidence does not show an e
25 and surrounding genes increases the risk for macrosomia, mild developmental delay and pervasive devel
27 reported [1 study]; and 0% vs 2.6%-4.3% for macrosomia, P = not reported [1 study] and P = .28 [1 st
29 ents with midline abdominal-wall defects and macrosomia was significantly higher, 65% (41/63) and 60%
30 elivered at term via cesarean section due to macrosomia, with a reported birth weight of 11 lb 8.7 oz
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