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1 ients with subfoveal neovascular age-related macular degeneration).
2 isual outcomes for patients with age-related macular degeneration.
3 n eye diseases like glaucoma and age-related macular degeneration.
4 tis pigmentosa (RP) and atrophic age-related macular degeneration.
5 liferative vitreoretinopathy and age-related macular degeneration.
6 ve effect of FHR-1 deficiency in age-related macular degeneration.
7 lassification of Age-related Maculopathy and Macular Degeneration.
8 olamine elimination in a cell-based model of macular degeneration.
9 in eyes with advanced stages of age-related macular degeneration.
10 f Toll-like receptor 2 (TLR2) in age-related macular degeneration.
11 macular degeneration (AMD) and 2 with myopic macular degeneration.
12 egenerative disorders, including age-related macular degeneration.
13 signaling in the pathogenesis of age-related macular degeneration.
14 ome and C3 glomerulopathies, and age-related macular degeneration.
15 ve relevance for the etiology of age-related macular degeneration.
16 n many patients with neovascular age-related macular degeneration.
17 loss in diabetic retinopathy and age-related macular degeneration.
18 tis pigmentosa (RP) and atrophic age-related macular degeneration.
19 on and shows symptoms similar to age-related macular degeneration.
20 erative diabetic retinopathy and age-related macular degeneration.
21 gene commonly cause retinal dysfunction and macular degeneration.
22 ve diabetic retinopathy, and wet age-related macular degeneration.
23 ity, but patients still progressed to severe macular degeneration.
24 omatopsia, cone dystrophies, and early onset macular degeneration.
25 refractive error, cataracts, and age-related macular degeneration.
26 ed retinoschisis (XLRS), a monogenic form of macular degeneration.
27 cently reported association with age-related macular degeneration.
28 rs such as Stargardt disease and age-related macular degeneration.
29 he assimilation of lutein in humans to avoid macular degeneration.
30 t disease and the ocular disease age-related macular degeneration.
31 rosis development in neovascular age-related macular degeneration.
32 ambiguity about cone survival in age-related macular degeneration.
33 eatment regimens for neovascular age-related macular degeneration.
34 in the management of neovascular age-related macular degeneration.
35 's membrane alterations, such as age-related macular degeneration.
36 asked how these are compromised in models of macular degeneration.
37 patients (n = 14) showed early and extensive macular degeneration.
38 , including Behcet's disease and age-related macular degeneration.
39 inal dystrophy that is accompanied by severe macular degeneration.
40 apeutic approach for neovascular age-related macular degeneration.
41 her macular disease including AMD and myopic macular degeneration.
42 mes and drives the pathogenesis of Stargardt macular degeneration.
43 tients with advanced neovascular age-related macular degeneration.
44 ive diabetic retinopathy and wet age-related macular degeneration.
45 ould be effective therapeutic approaches for macular degenerations.
46 site of injury in inherited and age-related macular degenerations.
49 served in 55.6 % of photographs (age-related macular degeneration: 34.2 %; diabetic retinopathy: 4.2
50 or glaucoma (63.4%); only half were aware of macular degeneration; 37.3% were aware of diabetic retin
53 ve participants with neovascular age-related macular degeneration, 7 of whom were treatment naive.
55 18.2 million to 109.6 million]), age-related macular degeneration (8.4 million [0.9 million to 29.5 m
57 either persistent amblyopia and age-related macular degeneration (AMB + AMD), or with bilateral age-
61 for the treatment of neovascular age-related macular degeneration (AMD) among Medicare beneficiaries.
62 geographic atrophy secondary to age-related macular degeneration (AMD) and 2 eyes (5%) had geographi
63 s were diagnosed with coincident age-related macular degeneration (AMD) and 2 with myopic macular deg
64 tients with AVLs associated with age-related macular degeneration (AMD) and adult-onset foveomacular
65 aHUS), also confers high risk of age-related macular degeneration (AMD) and associates with C3 glomer
66 tic reviews of interventions for age-related macular degeneration (AMD) and described the main findin
67 ociations between early and late age-related macular degeneration (AMD) and neovascular AMD (nvAMD) w
70 ges of patients with neovascular age-related macular degeneration (AMD) and to demonstrate its use to
71 non-neovascular and neovascular age-related macular degeneration (AMD) and to provide recommendation
72 scularization (CNV) secondary to age-related macular degeneration (AMD) and VA between 20/25 and 20/3
73 icity on the association between age-related macular degeneration (AMD) and vision-specific functioni
74 available treatment options for age-related macular degeneration (AMD) are limited, particularly for
75 ecline in the risk of developing age-related macular degeneration (AMD) continued for people born dur
77 g cardinal features of human dry age-related macular degeneration (AMD) in 12-month-old male and fema
78 year incidence of early and late age-related macular degeneration (AMD) in a Singaporean Malay popula
79 ve eyes diagnosed with exudative age-related macular degeneration (AMD) in comparison with eyes with
80 terranean diet and prevalence of age-related macular degeneration (AMD) in countries ranging from Sou
81 incidence of intermediate-stage age-related macular degeneration (AMD) in patients with acquired imm
102 on-based prevalence estimates of age-related macular degeneration (AMD) need to be determined to asse
104 used by persistent amblyopia and age-related macular degeneration (AMD) or by bilateral AMD, had an a
105 d (TREX) regimen for neovascular age-related macular degeneration (AMD) or fellow control eyes, as we
107 uated for protection against wet age-related macular degeneration (AMD) over a 6month period followin
108 , 1 from a 78-year-old exudative age-related macular degeneration (AMD) patient, 1 from a 58-year-old
109 ascularization (NV) in eyes with age-related macular degeneration (AMD) receiving anti-vascular endot
111 -function (pLoF) variants within age-related macular degeneration (AMD) risk loci and AMD sub-phenoty
113 LLQ) in patients with a range of age-related macular degeneration (AMD) severity are associated with
114 subset of eyes with neovascular age-related macular degeneration (AMD) that have persistent exudatio
115 ophy (GA) is an advanced form of age-related macular degeneration (AMD) that leads to progressive and
116 y (GA) secondary to nonexudative age-related macular degeneration (AMD) through multimodal imaging.
117 ome in patients with neovascular age-related macular degeneration (AMD) treated initially with bevaci
118 anatomic outcome in neovascular age-related macular degeneration (AMD) treated with as-needed ranibi
119 al evaluating progression of dry age-related macular degeneration (AMD) using color photographs at an
120 bolomic profile of patients with age-related macular degeneration (AMD) using mass spectrometry (MS).
121 y choriocapillaris blood flow in age-related macular degeneration (AMD) using optical coherence tomog
123 Most classification systems for age-related macular degeneration (AMD) were developed from patients
124 standard care for patients with age-related macular degeneration (AMD) who are being considered for
125 ls for the study of nonexudative age-related macular degeneration (AMD) with an emphasis on a novel c
126 y SD OCT study participants with age-related macular degeneration (AMD) with bilateral large drusen o
127 pants with at least intermediate age-related macular degeneration (AMD) with control subjects without
128 t; 62.7% were performed to treat age-related macular degeneration (AMD), 16.1% to treat diabetic reti
129 iking phenotypic similarities to age-related macular degeneration (AMD), a common and genetically com
130 capillaris are characteristic of age-related macular degeneration (AMD), a common vision-threatening
133 H), show strong association with age-related macular degeneration (AMD), a major cause of blindness.
135 nt factor in the pathogenesis of age-related macular degeneration (AMD), although direct evidence for
136 have implicated AP activation in age-related macular degeneration (AMD), and AP dysfunction predispos
138 f the importance of nutrition in age-related macular degeneration (AMD), but few studies have explore
139 zation (CNV) among patients with age-related macular degeneration (AMD), but no economic evaluation h
140 To test potential treatments for age-related macular degeneration (AMD), clinical trials need standar
141 opathy (PCV), a subtype of 'wet' age-related macular degeneration (AMD), constitutes up to 55% of cas
143 imer's disease, hemochromatosis, age-related macular degeneration (AMD), diabetes mellitus, and cysti
144 ls for management of neovascular age-related macular degeneration (AMD), diabetic macular edema (DME)
145 rse range of diseases, including age-related macular degeneration (AMD), glaucoma and refractive erro
148 es including dry and neovascular age-related macular degeneration (AMD), retinitis pigmentosa, and di
149 ular pathologies in the eye like age-related macular degeneration (AMD), the diabetic retinopathie (D
150 eration, a subset of neovascular age-related macular degeneration (AMD), which is associated with hig
151 uited patients with intermediate age-related macular degeneration (AMD), without other vitreoretinal
152 examine their associations with age-related macular degeneration (AMD)-related features and AMD prog
153 lation, of physical activity and age-related macular degeneration (AMD)-the main cause of irreversibl
181 and C2 genes are associated with age-related macular degeneration (AMD); however, the association of
182 und to be highly associated with age-related macular degeneration (AMD); however, the effect on clini
183 mes of patients with neovascular age-related macular degeneration (AMD, n = 400), diabetic macular ed
184 e 4 most prevalent eye diseases (age-related macular degeneration [AMD], cataract, diabetic retinopat
186 refractive error, cataracts, and age-related macular degeneration among adults 65 years or older in t
187 years or older with neovascular age-related macular degeneration and a baseline best-corrected visua
191 es for retinal diseases, such as age-related macular degeneration and inherited retinal dystrophies,
192 in both eyes of adult mammals is a model for macular degeneration and leads to retinotopic map reorga
194 rse clinical disorders including age-related macular degeneration and paroxysmal nocturnal hemoglobin
196 nant I62-CFH (protective against age-related macular degeneration) and V62-CFH functioned equivalentl
197 dema, 32 (25.8%) had neovascular age-related macular degeneration, and 32 (25.8%) had other causes of
198 es such as diabetic retinopathy, age-related macular degeneration, and central retinal vein occlusion
200 ematurity, diabetic retinopathy, age-related macular degeneration, and glaucoma, as well as anticipat
201 uded aphakia, pseudophakia, late age-related macular degeneration, and vision impairment due to catar
202 tion of pure geographic atrophy or exudative macular degeneration, any type of drusen with pigmentary
203 regimen with ranibizumab for wet age-related macular degeneration (ARMD) in real life clinical settin
204 inal pigment epithelium (RPE) of age-related macular degeneration (ARMD) patients and therefore could
205 d with neovascularization in wet age-related macular degeneration (ARMD), choriocapillaris degenerati
207 urity, diabetic retinopathy, and age-related macular degeneration, as well as corneal diseases with a
210 cizumab injections for exudative age-related macular degeneration between January 1, 2009, and Decemb
212 f ophthalmic diseases, including age-related macular degeneration, cataracts, diabetic retinopathy, g
213 ic atrophy is a blinding form of age-related macular degeneration characterized by retinal pigmented
214 s is an end stage of neovascular age-related macular degeneration, characterized by fibrous membrane
216 dry eye syndrome) and posterior (age-related macular degeneration, diabetic retinopathy and glaucoma)
217 common, but serious, conditions: age-related macular degeneration (Dr. Fine), diabetic retinopathy (D
218 ould be of clinical and research interest in macular degeneration, for example in estimating visual p
220 Ophthalmic diseases, such as age-related macular degeneration, glaucoma, and diabetic retinopathy
221 irment) and a high prevalence of age-related macular degeneration (>14% of blindness) as causes in th
222 eases associated with NAION were age-related macular degeneration (HR = 1.29; 95% CI: 1.08-1.54) and
224 etween plasma HDL-C and risk for age-related macular degeneration; (iii) only some mechanisms of lowe
225 tinoschisis (XLRS) is one of the most common macular degenerations in young males, with a worldwide p
226 ystallin plays multiple roles in age-related macular degeneration, including cytoprotection and angio
227 tic reviews of interventions for age-related macular degeneration incorporated into clinical practice
230 oschisis (XLRS), a leading cause of juvenile macular degeneration, is characterized by a spoke-wheel
236 el, Switzerland) for neovascular age-related macular degeneration (nAMD) after 2 years when using a t
237 monthly visits for patients with neovascular macular degeneration (nAMD) compared with monthly pro re
238 F-A in patients with neovascular age-related macular degeneration (nAMD) demonstrated dramatic benefi
239 mes in patients with neovascular age-related macular degeneration (nAMD) during anti-vascular endothe
240 ces of patients with neovascular age-related macular degeneration (nAMD) for different anti-vascular
241 ens in patients with neovascular age-related macular degeneration (nAMD) from the TReat and extEND (T
242 real aflibercept for neovascular age-related macular degeneration (nAMD) in routine clinical practice
243 he natural course of neovascular age-related macular degeneration (nAMD) is essential in discussing p
244 ment-naive eyes with neovascular age-related macular degeneration (nAMD) tracked by the Fight Retinal
245 VA) in patients with neovascular age-related macular degeneration (nAMD) treated with a single anti-v
246 hy (GA) in eyes with neovascular age-related macular degeneration (nAMD) treated with ranibizumab.
247 among patients with neovascular age-related macular degeneration (nAMD) who participated in a large-
248 ths in patients with neovascular age-related macular degeneration (nAMD) with insufficient response t
249 patients treated for neovascular age-related macular degeneration (nAMD), diabetic macular edema (DME
250 with ranibizumab for neovascular age-related macular degeneration (nAMD), diabetic macular oedema (DM
251 for the treatment of neovascular age-related macular degeneration (nAMD), ophthalmologists have shift
253 oidal neovascularisation akin to age-related macular degeneration, NCD loss attenuated vessel leakage
255 guiding therapy for neovascular age-related macular degeneration (nvAMD) to the research investments
256 among patients with neovascular age-related macular degeneration (NVAMD) treated with anti-vascular
260 Differences in progression and severity of macular degeneration, optic nerve pallor, and vascular a
261 When a patient with neovascular age-related macular degeneration or diabetic macular edema does not
262 When a patient with neovascular age-related macular degeneration or diabetic macular edema does not
263 mg injections of aflibercept for age-related macular degeneration or diabetic macular edema in a 9-me
268 antly with sets found by GWAS of age-related macular degeneration (P=1.4 x 10(-12)), ulcerative colit
269 mer's disease (P=4.4 x 10(-15)), age-related macular degeneration (P=6.4 x 10(-6)), and Parkinson's d
270 that iPSC-derived RPE cells from age-related macular degeneration patients express increased levels o
271 emplary search for patients with age-related macular degeneration, performed cataract surgery, and at
273 ovascularization and neovascular age related macular degeneration presenting with hemorrhagic and exu
274 ATEMENT Central retinal lesions, a model for macular degeneration, result in functional reorganizatio
275 cclusion, macular hole, epiretinal membrane, macular degeneration, retinal detachment repair, and pro
276 diseases, of which the main are age-related macular degeneration, retinal vein occlusion and diabeti
277 ictions for proteins involved in age-related macular degeneration, retinitis pigmentosa, and Leber's
279 urity, diabetic retinopathy, and age-related macular degeneration, threaten the visual health of chil
280 .9 million to 124.1 million), by age-related macular degeneration to 8.8 million (0.8 million to 32.1
281 diseases, including stroke, AD, age-related macular degeneration, traumatic brain injury, Parkinson'
282 e of blindness for patients with age-related macular degeneration, treat symptoms but not the underly
283 inical trials, the Comparison of Age-Related Macular Degeneration Treatments Trials (CATT) and the Di
284 inical trials, the Comparison of Age-Related Macular Degeneration Treatments Trials (CATT) and the Di
285 ts enrolled in the Comparison of Age-Related Macular Degeneration Treatments Trials (CATT), a randomi
288 etic retinopathy and neovascular age-related macular degeneration, uncontrolled angiogenesis can lead
289 r inhibitors (anti-VEGF) for wet age-related macular degeneration (wAMD), and to acquire a snapshot o
290 final vision (P = .007), whereas age-related macular degeneration was associated with poorer vision (
293 tients with advanced neovascular age-related macular degeneration were enrolled in the study between
294 f 1097 patients with neovascular age-related macular degeneration were randomized to intravitreal ran
296 rusenoid deposits, a hallmark of age-related macular degeneration, which is a common blinding disease
297 erent metrics for cataract surgery and 2 for macular degeneration, which showed only limited overlap
298 ur method to an in-depth GWAS of age-related macular degeneration with 33,976 individuals and 9,857,2
299 ented by patients with bilateral age-related macular degeneration with similar decrease of vision.
300 n oral treatment for neovascular age-related macular degeneration would be less burdensome than repea
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