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1 with missing central peak of photopigment or macular pigment.
2 phyll levels, and the optical density of the macular pigment.
3 ong intake of xanthophylls and no detectable macular pigment.
4 ion because of their photo-oxidative role as macular pigment.
5 Lutein is a component of macular pigment.
6 m lutein supplementation can raise levels of macular pigment.
7 s the precursor of RSZ, a major component of macular pigment.
8 e consistent with the absorption spectrum of macular pigment.
9 RPE cells are sensitive to the absence of macular pigment.
10 eso-zeaxanthin are the major constituents of macular pigment, a compound concentrated in retinal area
12 ge-related decline in the optical density of macular pigment among volunteers with no ocular disease
13 n cone photopigment distribution to those of macular pigment and examine those loci for subretinal ch
15 n of the role for SCARB1 in the transport of macular pigment and the possible modulation of age-relat
17 ns, determine absorption and distribution of macular pigment, and assess retinal health and visual fu
18 sity, spatial profile, and lateral extent of macular pigment, and it has been suggested that foveal a
19 ze contributions of extraneous fluorophores, macular pigment, and melanin, all measurements used exci
21 anthin, the major carotenoids comprising the macular pigment, are present in rod outer segment (ROS)
25 s yellow in color due to the presence of the macular pigment, composed of two dietary xanthophylls, l
30 umber of individuals in this sample with low macular pigment density motivates the need for populatio
38 sensitivity across the central field follows macular pigment density; (iii) polarization patterns are
42 udy, macular pigment optical density (MPOD), macular pigment distributions, and skin carotenoid level
43 s (FA) (44.4% vs 12.5%, P = .03), absence of macular pigment epithelium atrophy on FA (88.9% vs 62.5%
44 mparable to that for L and M cones, and that macular pigment has no significant function in improving
45 patterns follows the spectral sensitivity of macular pigment; (ii) the change in sensitivity across t
47 ids, and vitamins to increase the density of macular pigment in first-generation offspring of parents
48 We used blue light reflectance to image the macular pigment in premature babies at the time of retin
52 plasma as well as the optical density of the macular pigment increased significantly in the groups ra
53 epidemiological evidence that the amount of macular pigment is inversely associated with the inciden
54 aternal carotenoid status and newborn infant macular pigment levels and systemic carotenoid status.
55 in prenatal supplementation can raise infant macular pigment levels and/or improve ocular function.
60 r understanding why some clinical methods of macular pigment measurement have had difficulty detectin
61 lature, small yellow macular deposits and/or macular pigment mottling, and abnormal electroretinogram
63 as designed to determine the heritability of macular pigment (MP) augmentation in response to supplem
64 Schultze, in 1866, originally proposed that macular pigment (MP) could improve acuity by reducing th
65 s investigated how individual differences in macular pigment (MP) density are related to loss of visu
67 n these variables and the optical density of macular pigment (MP) in a group of subjects from a north
73 c flicker photometry was used to measure the macular pigment (MP) levels of 169 healthy volunteers, o
74 lutein (L) and zeaxanthin (Z) that form the macular pigment (MP) may help to prevent neovascular age
78 of fluorescent lipofuscin, light-attenuating macular pigment (MP), cone photopigment, and retinal pig
86 lutein, zeaxanthin, and omega-3 LCPUFAs and macular pigment optical densities were measured at basel
87 ital video fundus camera (RetCam) to measure macular pigment optical density (MPOD) and distributions
88 a-3 polyunsaturated fatty acids may increase macular pigment optical density (MPOD) and thereby prote
89 supplementation with lutein (L) capsules on macular pigment optical density (MPOD) and visual acuity
93 tudy evaluated serum lutein, zeaxanthin, and macular pigment optical density (MPOD) responses at 0.25
97 retinal circulation in three dimensions, and macular pigment optical density (MPOD), which quantifies
100 a significant relation between variation in macular pigment optical density and immediate effects on
104 on on concentrations of retinal carotenoids (macular pigment, or MP) is of particular interest becaus
105 ees ) eccentricity, which is adjacent to the macular pigment peak, and parafoveally at 1.5 mm ( appro
108 tributions: central peak of photopigment and macular pigment, small foveal alterations, and broad dis
109 nrolled at the Moran Eye Center had MPOD and macular pigment spatial distributions measured by dual-w
110 er clinical factors (ocular media opacities, macular pigment, statistical interpretation, and related
112 fore might represent a more potent source of macular pigments than green leafy vegetables like spinac
116 distributions of foveal cone photopigment or macular pigment were found that varied among the subject
117 been proposed to explain the function of the macular pigment, which selectively absorbs short-wavelen
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