ライフサイエンスコーパス: macule

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1 typically appearing as a solitary pigmented macule.

2 ired in the melanocytes of the hypomelanotic macule.

3 On pathologic examination, 96% of miners had macules, 70% micronodules, 45% macronodules, 15% silicos

4 patients with 63 ambiguous pigmented facial macules and 12 control photodamaged facial areas were in

5 microscopy (RCM), ambiguous pigmented facial macules and establish a correlation between RCM, histopa

6 erall, q-type opacities were associated with macules and micronodules, whereas the large r-type opaci

7 20s presented with asymptomatic red to brown macules and papules.

8 cused on a single skin feature- cafe-au-lait macules-and partitioned the disease space into hierarchi

9 "Cafe-au-lait" macules (CALMs) and overall skin hyperpigmentation are e

10 Response to laser treatment for cafe au lait macules (CALMs) is inconsistent and difficult to predict

11 ividuals presenting mainly with cafe au lait macules (CALMs), axillary freckling, and macrocephaly.

12 ly characterised by the development of white macules due to the loss of functioning melanocytes in th

13 en presented with eruptions of hypopigmented macules following coryzal symptoms.

14 mary melanocytes isolated from hypomelanotic macules from 6 patients with TSC all exhibited reduced T

15 development of white, genetically revertant macules in red, diseased skin.

16 Benign melanotic macules (MAC) are the most frequent cause of lip pigment

17 sions of coal workers' pneumoconiosis (CWP): macules, micro- and macronodules (small and large fibrot

18 tegory 0/0 was often reported for cases with macules of mild to moderate grade and mild levels of mic

19 Pigmented macules of the glans penis, delayed motor development an

20 the gastrointestinal tract and by pigmented macules of the lips, buccal mucosa, and digits.

21 gressively sclerotic and presented pigmented macules on a background of hypopigmentation and teleangi

22 ure glaucoma following the appearance of new macules on her upper extremities.

23 Pigmented facial macules on photodamaged skin are a clinical, dermoscopic

24 spective study of ambiguous pigmented facial macules on photodamaged skin was conducted in a tertiary

25 Six patients had pigmented macules on sun-exposed skin, but none developed a skin n

26 ents presented with hypo- and hyperpigmented macules over the body, resembling dyschromatosis univers

27 presentations were characterized by purpuric macules, papules, and confluent plaques predominantly on

28 r characterized by circumscribed depigmented macules resulting from the loss of cutaneous melanocytes

29 Patients with TSC develop hypomelanotic macules (white spots), but the molecular mechanisms unde

30 y enhanced the diagnosis of pigmented facial macules with 91.7% sensitivity and 86.8% specificity.

31 immunodeficiency mice led to black-pigmented macules within 3 weeks of treatment.

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