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1 onic catheter introduced through the foramen magnum.
2  We performed a follow-up study based on the Magnum 12+12 Cohort of patients who received a kidney tr
3  the location and orientation of the foramen magnum, and changes in the breadth of the basioccipital.
4  interpreting results where both I(CRAC) and MagNuM are activated.
5 ivated more rapidly and more completely than MagNuM carried by monovalent ions.
6 ll intracranial structures above the foramen magnum), cerebral death (all supratentorial structures)
7 agnesium-nucleotide-regulated metal current (MagNuM) channel that is regulated by serum Mg(2+) concen
8 overexpression of TRPM7 increased endogenous MagNuM currents, suggesting that these currents are depe
9 ernal 2-aminoethoxydiphenyl borate inhibited MagNuM currents.
10 ow (CBF) and ventriculostomy defect, foramen magnum (FM), and cerebral aqueduct CSF flow.
11 BL (rat basophilic leukemia) cells and named MagNuM (for Mg(2+)-nucleotide-inhibited metal) or MIC (f
12        Besides a relatively anterior foramen magnum, humans differ from apes in the lateral shift of
13 ve generations, and expression levels in the magnum of the oviduct were constant over at least 16 mon
14 argely confined to the tubular glands of the magnum of the oviduct, where egg white synthesis occurs,
15 ulminated in 1950 with the appearance of his magnum opus, Variation and Evolution in Plants.
16 ertebrae, loss of synchondroses, and foramen-magnum shape anomalies.
17 In these disorders, spinal canal and foramen magnum stenosis can cause serious neurologic complicatio
18                     Spinal canal and foramen magnum stenosis in heterozygous achondroplasia patients,
19                Tonsillar herniation, foramen magnum stenosis, and severe osteomyelitis of the jaw wer
20 w that OVAX is specifically expressed by the magnum tissue, which is responsible for egg white format
21  tumor extending from just above the foramen magnum to C6.
22 ions would have favoured the contribution of MagNuM to monovalent conductance and call for caution in
23  but the time to half-maximal activation for MagNuM was about two to three times slower than that of
24 2+)](i) to 90 nM and selective inhibition of MagNuM was accomplished by intracellular solutions conta
25 s extending more than 5 mm below the foramen magnum were classified by the neurosurgeon as symptomati
26 m-nucleotide-regulated metal cation current (MagNuM) (which is conducted by the LTRPC7 channel).
27 ium-nucleotide-regulated metal ion currents (MagNuM) with regulation and permeation properties essent

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