ライフサイエンスコーパス: major blood vessel

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1 act form and splay open just as they reach a major blood vessel.

2 nd cavitation, and disease location close to major blood vessels.

3 vascular basement membranes and the walls of major blood vessels.

4 lung, tongue, tendons, salivary glands, and major blood vessels.

5 through the umbilical, vitelline, and other major blood vessels.

6 es are present in the mesenchyme surrounding major blood vessels and developing bone and in lung.

7 ntact nondiseased human lungs (including the major blood vessels and draining lymph nodes) obtained e

8 vascular imaging, providing visualization of major blood vessels and microvasculature and providing i

9 mans could enable the clinical assessment of major blood vessels and microvasculature.

10 cs were disorganized, and the endothelium of major blood vessels and of the heart was frequently disc

11 treatment of tissue immediately adjacent to major blood vessels and other connective tissue structur

12 sue functions without surgical connection to major blood vessels and sets the stage for the transplan

13 Major blood vessels appeared normal at embryonic day 9.5

14 omas) and for the alpha4 chain of laminin, a major blood vessel basement membrane component.

15 At major blood vessel crossings, a focal alteration in nerv

16 However, somites and major blood vessels do form.

17 secondary to heat-sink effects and damage to major blood vessels; however, needle tract seeding is ob

18 oma (NHL) patients are often proximal to the major blood vessels in the abdomen or neck.

19 registration relies on the identification of major blood vessels in the CT and 99mTc SPECT images.

20 cells differentiate to smooth muscle of all major blood vessels in the mesenteries and gut.

21 Image quality and venous overlay of the major blood vessels of the abdomen, thigh, and calf were

22 ce exhibit skeletal defects and apoptosis in major blood vessels proximal to the heart.

23 The examination of the major blood vessels revealed that the aortas of Rgs5(-/-

24 e of death of mutant mice was rupture of the major blood vessels, similar to patients with type IV Eh

25 ute to adrenergic vasodilation only in a few major blood vessels, suggesting that differential distri

26 Adjusting for radioactivity in major blood vessels that were in the ROI led to lower ma

27 he nerve fiber pathway obviates the need for major blood vessels to bend or kink to achieve a crossin

28 rtial or complete surrounding of one or more major blood vessels with little or no luminal narrowing

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