ライフサイエンスコーパス: malabsorption

1 2-hydroxybutyrate (a known marker of glucose malabsorption).

2 unting), and abnormal function (for example, malabsorption).

3 resulting in villous degeneration and lipid malabsorption.

4 osomal recessive disorder, hereditary folate malabsorption.

5 re akin to that of starvation and intestinal malabsorption.

6 cose to the body and avoidance of intestinal malabsorption.

7 asal ion secretion and corrected the glucose malabsorption.

8 by increased basal ion secretion and glucose malabsorption.

9 ns, which indicated appreciable carbohydrate malabsorption.

10 ing in chronic diarrhea and life-threatening malabsorption.

11 to be a poor test for diagnosing pancreatic malabsorption.

12 vels > or =30 ng/mL, suggesting selective Ca malabsorption.

13 bypass (RYGB) is generally attributed to fat malabsorption.

14 oblastic anemia (MGA1), owing to vitamin B12 malabsorption.

15 ntion studies suggest supplemental vitamin E malabsorption.

16 ssociated with three severe disorders of fat malabsorption.

17 m bile acid concentrations, itching, and fat malabsorption.

18 bile and pancreatic secretions to induce fat malabsorption.

19 deficiencies may develop because BPD causes malabsorption.

20 ormally, were fertile and showed no signs of malabsorption.

21 t on parenteral nutrition because of chronic malabsorption.

22 es small bowel transit, which may exacerbate malabsorption.

23 tive intestinal vitamin B12 (B12, cobalamin) malabsorption.

24 and human diseases such as primary bile acid malabsorption.

25 um absorption and reversed water and glucose malabsorption.

26 tic-enzyme supplements to control intestinal malabsorption.

27 c cycling of bilirubin occurs with bile salt malabsorption.

28 1 was identified in the subject with global malabsorption.

29 ity of some stressed individuals to fructose malabsorption.

30 over 8 d in healthy adults without fructose malabsorption.

31 ted after procedures that result in the most malabsorption.

32 supplementation, and enteral causes such as malabsorption.

33 sion but no improvement of symptoms/signs of malabsorption.

34 re the molecular basis for hereditary folate malabsorption.

35 functional hormones resulted in generalized malabsorption.

36 uodenum, avoiding blind loops and minimizing malabsorption.

37 You find no evidence of malabsorption.

38 , narrow anastomoses, excluded segments, and malabsorption.

39 150 cm, the procedure is believed to induce malabsorption.

40 t of vitamin A under conditions of bile acid malabsorption.

41 utations are the basis for hereditary folate malabsorption.

42 osomal recessive disorder, hereditary folate malabsorption.

43 ase did not appear to have a role in the fat malabsorption.

44 d of Pakistani origin with hereditary folate malabsorption.

45 e 1 is a poor test for diagnosing pancreatic malabsorption.

46 astrointestinal inflammatory disease, 15.4%; malabsorption, 5.9%; granulomatous disease, 9.7%; liver

47 peripheral neuropathy and ataxia, is usually malabsorption-a result of fat malabsorption or genetic a

48 On average, malabsorption accounted for approximately 6% and 11% of

49 Rapid weight loss and malabsorption after bariatric surgery in patients with N

50 eficiency as a result of iatrogenic maternal malabsorption after BPD.

51 the caecum and colon, consistent with severe malabsorption, along with a unique adaptive up-regulatio

52 transporter is mutated in hereditary folate malabsorption, an autosomal recessive disorder, the mole

53 c insufficiency, dietary lactose or fructose malabsorption, anal sphincter dysfunction causing fecal

54 essive disorders characterized by severe fat malabsorption and a failure to thrive in infancy.

55 essive disorder characterized by generalized malabsorption and a paucity of enteroendocrine cells.

56 patients frequently sustain severe nutrient malabsorption and are dependent on PN for life.

57 demonstrate the necessity of coordinated Na+ malabsorption and barrier dysfunction in TNF-induced dia

58 TR transgenic animals ameliorated intestinal malabsorption and concomitantly led to an increase in CF

59 shift in intestinal function leads to lipid malabsorption and decreased deposition of body fat.

60 mmalian small intestine, leading to nutrient malabsorption and diarrhea but rarely causing inflammati

61 ice) or 2 weeks (rats), indices of bile salt malabsorption and enterohepatic cycling of bilirubin wer

62 virus (HIV)-infected patients often develop malabsorption and increased intestinal permeability with

63 d correlated with fecal measures of nutrient malabsorption and inflammation, suggesting that E. coli

64 s gastrointestinal disease includes nutrient malabsorption and intestinal inflammation.

65 Malabsorption and malnutrition are prevalent in survivor

66 These conditions include intestinal malabsorption and primary neural cell neoplasia.

67 kindreds with selective intestinal cobalamin malabsorption and proteinuria and that normal brush-bord

68 ized by intestinal cobalamin (vitamin B(12)) malabsorption and proteinuria.

69 h age in the pancreas to such an extent that malabsorption and rapid weight loss occurred in a subset

70 epithelial cell dysplasia leading to severe malabsorption and significant morbidity and mortality.

71 icotropin-releasing factor (CRF) on fructose malabsorption and the resulting volume of water in the s

72 it in arising only infrequently from severe malabsorption and thus being less likely to progress, co

73 PCFT gene in subjects with hereditary folate malabsorption and, more recently, by the Pcft-null mouse

74 amylase activity (which causes carbohydrate malabsorption), and (3) if the inhibitor alters pancreat

75 tal/malabsorptive RYGB, 12 restapled without malabsorption, and 5 loop bypasses revised to standard R

76 elated to folate uptake in hereditary folate malabsorption, and delivery of PCFT-targeted chemotherap

77 hibition prevented NHE3 internalization, Na+ malabsorption, and diarrhea despite continued barrier dy

78 Bone loss is influenced by diet, malabsorption, and disease-related imbalances in bone tu

79 cts observed in mutant mice caused diarrhea, malabsorption, and early death.

80 of the small bowel mucosa, villous atrophy, malabsorption, and increased intestinal permeability, is

81 phangiectasia, edema due to hypoproteinemia, malabsorption, and less frequently, bowel inflammation,

82 n result in intestinal mucosal inflammation, malabsorption, and numerous secondary symptoms and autoi

83 Reduced food intake, impaired digestion, malabsorption, and poor growth rate are frequently obser

84 atory bowel disease, pancreatitis, motility, malabsorption, and radiation enteropathy.

85 c steatosis, despite impaired intestinal fat malabsorption, and why very severe hypocholesterolemia d

86 hol-dependent individuals, and patients with malabsorption are at higher risk of inadequate intake or

87 e common, data on gut and pancreatic-related malabsorption are scant.

88 The survivors had fat malabsorption as newborns and as adults, but only when f

89 with a long thread of interest in folic acid malabsorption as one of the determinants of nutritional

90 risk for vitamin A deficiency because of fat malabsorption as well as for the inflammatory stresses o

91 ents with IFM and in one subject with global malabsorption, as compared with 15 healthy parents of su

92 ricts the small bowel and increases fructose malabsorption, as shown by increased ascending colon vol

93 not be used clinically to diagnose cobalamin malabsorption because of overlap with normal values.

94 In older persons, food-bound cobalamin malabsorption becomes the predominant cause of deficienc

95 mbination of osmotic sensitivities, nutrient malabsorption, bowel dilatation and dysmotility, and cha

96 and gastrointestinal disease with or without malabsorption, but not with bronchiectasis, autoimmunity

97 Severe malabsorption by the gastrointestinal (GI) tract was the

98 viduals with presumed lactose intolerance or malabsorption can tolerate 12 to 15 g of lactose.

99 Intestinal damage and malabsorption caused by chronic environmental enteropath

100 The impact of nutrient malabsorption caused from untreated celiac disease is we

101 sociated with the disorder glucose-galactose malabsorption, characterized by severe diarrhea.

102 USP U of pancreatin with meals decreased fat malabsorption compared with placebo.

103 ins has been implicated in glucose/galactose malabsorption, congenital hypothyroidism, Bartter's synd

104 By causing bile salt malabsorption, dietary UDCA and cholesterol induce enter

105 Short bowel syndrome (SBS) is a serious malabsorption disorder, and dietetic management of patie

106 Those with fat malabsorption disorders malabsorb vitamin D and thus mus

107 A bile acid malabsorption-driven increase in cholesterol synthesis u

108 Decreased nutrient intake, malabsorption, drug-nutrient interactions, anorexia, and

109 ealthy infants and is common in infants with malabsorption due to cystic fibrosis (CF).

110 ion of cobalamin than does the more complete malabsorption engendered by disruption of intrinsic fact

111 trast, suckling PLRP2-deficient mice had fat malabsorption evidenced by increased fecal weight, incre

112 reath hydrogen (an indicator of carbohydrate malabsorption), flatus frequency, and abdominal symptoms

113 a course of parenteral ivermectin because of malabsorption from severe gastrointestinal strongyloidia

114 Glucose-Galactose Malabsorption (GGM) is caused by a defect in SGLT1.

115 ause or are associated with gastrointestinal malabsorption has led to extensive investigation into th

116 Hereditary folate malabsorption (HFM) is a rare autosomal recessive disord

117 osomal recessive disorder, hereditary folate malabsorption (HFM).

118 Children were considered to have malabsorption if they had chronic diarrhea, weight loss

119 with clinical evidence of isolated fructose malabsorption (IFM) has stimulated interest in possible

120 is the molecular basis for hereditary folate malabsorption in a family with this disease.

121 plementation does not completely correct fat malabsorption in CF patients.

122 small-intestinal mucosal injury and nutrient malabsorption in genetically susceptible individuals in

123 r fructose may explain the high incidence of malabsorption in infants and cause problems in adults un

124 capillariasis is one of the common causes of malabsorption in the East.

125 Although fat malabsorption in the short-bowel syndrome is caused in p

126 l pathophysiological changes such as glucose malabsorption, increased chloride ion (Cl(-)) secretion,

127 L-thyroxine (L-T4) malabsorption is a potential concern in patients with au

128 udies support previous reports that fructose malabsorption is associated with unexplained gastrointes

129 Thus, calcium malabsorption is inherent to gastric bypass.

130 Ileal bile acid malabsorption is present in Crohn's ileitis.

131 lial cell dysfunction occur in the pancreas (malabsorption), liver (biliary cirrhosis), sweat glands

132 least commonly performed because of greater malabsorption, longer operative duration, and higher tec

133 If hyperoxaluria is indeed caused by fat malabsorption, magnitudes of hyperoxaluria and steatorrh

134 Fructose absorption and malabsorption may alter the microbiota and could be medi

135 Vitamin D deficiency resulting from malabsorption may be a factor in the etiology of low bon

136 pancreatitis (8) Early treatment of pain and malabsorption may improve life quality (9) Antifibrogene

137 deficiency is associated with food-cobalamin malabsorption more often than with pernicious anemia.

138 In patients with primary bile acid malabsorption, mutations in the ileal bile acid transpor

139 Malabsorption not responding to antibiotic therapy shoul

140 ulose suggested that the tea extract induced malabsorption of 25% of the carbohydrate.

141 ealthy subjects, examining the effect on the malabsorption of a 40-g fructose test meal and its trans

142 ounts of vitamin D can normalize the calcium malabsorption of aging.

143 HIV infection may lead to malabsorption of anti-TB drugs and acquired rifamycin re

144 Ileal malabsorption of bile salts is observed in Crohn's ileit

145 Although calcitriol corrects the malabsorption of calcium, it remains to be seen whether

146 lem of osteomalacia, but it does not correct malabsorption of calcium.

147 ck, green, and mulberry tea leaves to induce malabsorption of carbohydrate and triacylglycerol in hea

148 sorder characterized by selective intestinal malabsorption of cobalamin (vitamin B12) and urinary los

149 obalamin) deficiency is caused by intestinal malabsorption of cobalamin.

150 ng celiac sprue is usually attributed to the malabsorption of dietary iron or the loss of iron from t

151 Malabsorption of fat-soluble vitamins is a major complic

152 In contrast to the malabsorption of fructose, Glut5(-/-) mice did not exhib

153 hamnose ratio were used to test for possible malabsorption of glucose after surgery.

154 his was due to restriction of food intake or malabsorption of ingested macronutrients.

155 Anemia secondary to malabsorption of iron, folic acid, and/or vitamin B12 is

156 Conditions associated with malabsorption of macronutrients and gastrointestinal dis

157 Bile acid (BA) malabsorption of moderate severity is reported in 32% of

158 lorhydric patients were found to have severe malabsorption of nonheme iron, which persisted after the

159 ually due to the large mass of the bezoar or malabsorption of nutrients.

160 markers for colonic fermentation, because of malabsorption of oligosaccharides (e.g., lactose or fruc

161 Malabsorption of partially digested food causes bloating

162 idered, because such individuals suffer from malabsorption of vitamin B-12 rather than from an inadeq

163 ing exons 1-4 of human AMN lead to selective malabsorption of vitamin B12 (a phenotype associated wit

164 duction of gastric acid and thus may lead to malabsorption of vitamin B12.

165 Hereditary folate malabsorption (OMIM 229050) is a rare autosomal recessiv

166 ced persistent alteration of bowel habits or malabsorption; only 1 minor wound complication has occur

167 he tea extract did not cause triacylglycerol malabsorption or any significant increase in symptoms.

168 neonatal cholestasis and fat-soluble vitamin malabsorption or as late onset chronic liver disease.

169 ia, is usually malabsorption-a result of fat malabsorption or genetic abnormalities in lipoprotein me

170 onutrients that were not absorbed because of malabsorption or restricted food intake.

171 of nutritional dyshomeostasis (oral failure, malabsorption, or both), and to quantify the effects of

172 e to discernable changes in food intake, fat malabsorption, or heat production, although intestinal l

173 ases of cholestasis and other forms of lipid malabsorption, oral administration of TPGS is the treatm

174 Primary bile acid malabsorption (PBAM) is an idiopathic intestinal disorde

175 The partial nature of this form of malabsorption produces a more slowly progressive depleti

176 Features of FPE are nonbloody diarrhea, malabsorption, protein-losing enteropathy, hypoalbuminem

177 that diarrhea in IBD is due to TNF-mediated malabsorption rather than to secretory processes.

178 Five months after bypass, malabsorption reduced absorption of combustible energy b

179 Fourteen months after bypass, malabsorption reduced energy absorption by 172 +/- 60 kc

180 ated intrahepatic cholestasis and intestinal malabsorption, reduced C27-bile acid intermediate produc

181 transplant diarrhea may lead to dehydration, malabsorption, rehospitalization, immunosuppression, non

182 ctrolyte disturbances with regard to enteral malabsorption, renal compensation, and the influence of

183 ty of <55% of predicted; 4) in the GI tract, malabsorption, repeated episodes of pseudoobstruction, o

184 Severe malabsorption required parenteral nutrition support for

185 Treatment of fat malabsorption requires 90,000 USP U of lipase with meals

186 limb edema, serosal effusions, and vitamin D malabsorption resulting in osteoporosis.

187 , early stage Crohn's disease, and bile salt malabsorption should be excluded, as should colon cancer

188 on of the enteric nervous system, as well as malabsorption, suggesting that common mechanisms of path

189 activation of the enteric nervous system and malabsorption, suggesting that common mechanisms of path

190 was 100.00 (95% CI, 98.68-100.00) when only malabsorption symptoms were used instead of any symptom

191 These include the typical malabsorption syndrome (classic symptoms) and a spectrum

192 serves as a model for the hereditary folate malabsorption syndrome and is the most accurate animal m

193 SIBO was added to the list of causes of the malabsorption syndrome and the pathophysiology of its co

194 n has become less common, with diarrhea or a malabsorption syndrome as the mode of presentation in fe

195 glt1(-/-) mice developed a glucose-galactose malabsorption syndrome but thrive normally when fed a gl

196 a new murine model of the hereditary folate malabsorption syndrome that we developed through targete

197 phenotypes were classified as: 1) classical (malabsorption syndrome); 2) non-classical (extraintestin

198 d in patients with steatorrhea caused by the malabsorption syndrome.

199 refore lead to an erroneous diagnosis of the malabsorption syndrome.

200 patient include inadequate dietary intakes, malabsorption syndromes (especially owing to cholestatic

201 tions for the prevention and/or treatment of malabsorption syndromes and diet-related disorders inclu

202 tamin D supplementation to patients with fat malabsorption syndromes as well as patients with other m

203 nadequate copper intakes, in prematurity, in malabsorption syndromes, and in conditions predisposing

204 novel treatments for obesity, diabetes, and malabsorption syndromes.

205 al intestine causes unremitting diarrhea and malabsorption that can lead to chronic and sometimes fat

206 re small-bowel villus atrophy, diarrhea, and malabsorption that is reversible with drug discontinuati

207 Because of the malabsorption that results from BPD, patients need lifel

208 acid (UDCA) and cholesterol causes bile salt malabsorption; the former by competition for and the lat

209 clinical manifestations, ranging from severe malabsorption to minimally symptomatic or non-symptomati

210 orptive disorders, especially food-cobalamin malabsorption, underlie about half of all cases of precl

211 s with a history of TB, five had evidence of malabsorption (vomiting and/or diarrhea), versus none of

212 Fat malabsorption was a prominent feature.

213 Therefore, glucose-galactose-malabsorption was assumed.

214 A new mechanism for carbohydrate malabsorption was discovered: in sucrase-isomaltase defi

215 Malabsorption was evident in 73% of patients, of whom 44

216 No evidence of glucose malabsorption was found.

217 Lactose malabsorption was not an important factor.

218 rations, such as Cl(-) secretion and glucose malabsorption, was studied using tissues derived from ma

219 Signs of malabsorption were rare.

220 iciency or with HIV infection and intestinal malabsorption were very similar to those of the B cell-d

221 ntestinal graft absorption capacity with fat malabsorption, which necessitates energy intakes of at l

222 A more frequent problem is food-cobalamin malabsorption, which usually arises from atrophic gastri

223 d on a unique proband with glucose-galactose malabsorption who was investigated 30 years ago, and the

224 cted to present with fat/fat soluble vitamin malabsorption with minimal cholestasis.

225 etic basis of selective intestinal cobalamin malabsorption with proteinuria was investigated in a can

226 y and effectively in patients in whom severe malabsorption would preclude the effective use of oral f