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1 d in patients with steatorrhea caused by the malabsorption syndrome.
2 refore lead to an erroneous diagnosis of the malabsorption syndrome.
3 novel treatments for obesity, diabetes, and malabsorption syndromes.
4 phenotypes were classified as: 1) classical (malabsorption syndrome); 2) non-classical (extraintestin
5 serves as a model for the hereditary folate malabsorption syndrome and is the most accurate animal m
6 SIBO was added to the list of causes of the malabsorption syndrome and the pathophysiology of its co
7 tions for the prevention and/or treatment of malabsorption syndromes and diet-related disorders inclu
8 nadequate copper intakes, in prematurity, in malabsorption syndromes, and in conditions predisposing
9 n has become less common, with diarrhea or a malabsorption syndrome as the mode of presentation in fe
10 tamin D supplementation to patients with fat malabsorption syndromes as well as patients with other m
11 glt1(-/-) mice developed a glucose-galactose malabsorption syndrome but thrive normally when fed a gl
13 patient include inadequate dietary intakes, malabsorption syndromes (especially owing to cholestatic
14 a new murine model of the hereditary folate malabsorption syndrome that we developed through targete
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