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1 with valgus thrust were disease severity and malalignment.
2 femoral cartilage score, with adjustment for malalignment.
3 n lead to growth abnormalities and potential malalignment.
4 ield development, resulting in arterial pole malalignment.
5 (OA) differs depending on the degree of limb malalignment.
6 some contribution from slightly more severe malalignment.
7 nship is reduced after controlling for varus malalignment.
12 blation of the CNCC results in arterial pole malalignment and failure of outflow septation, resulting
13 y because of the combined focus of load from malalignment and the excess load from increased weight.
14 BMI is correlated with the severity of varus malalignment, and 4) the BMI-medial tibiofemoral OA seve
17 whether meniscal damage, meniscal extrusion, malalignment, and laxity each predicted tibiofemoral car
18 cal characteristics such as joint laxity and malalignment, and radiographic severity are discussed.
20 tibia angle, and examine the relationship of malalignment by each approach with osteoarthritis (OA) t
21 ee of 75 knees with lateral PF OA had valgus malalignment compared with only 5 of 21 patients with me
22 increases overall loading of the knee, limb malalignment concentrates that loading on a focal area,
23 on of the truncus arteriosus, leading to OFT malalignment defects including double-outlet right ventr
24 stly in malaligned limbs, on the side of the malalignment (e.g., new medial BMLs in varus-aligned kne
25 d joint of mild OA may be less vulnerable to malalignment effects than the more-damaged joint of mode
26 of BMI is limited to knees in which moderate malalignment exists, presumably because of the combined
27 gression (OR 1.00), and in those with severe malalignment (> or =7 degrees ), the effect was similarl
29 ic predisposition, aging, obesity, and joint malalignment; however have been unable to conclusively d
31 ve percent and 22% of persons had zero mm of malalignment in maxillary and mandibular incisors, respe
34 unknown, lesions on bone scan and mechanical malalignment increase risk for radiographic deterioratio
37 ographic OA at baseline to determine whether malalignment is a risk factor for incident disease or si
38 ore common than medial PF OA, whether valgus malalignment is more frequent in lateral PF OA than in m
40 cedes or follows the onset of disease, varus malalignment is one local factor that may contribute to
43 Initiative (OAI) to define limbs with valgus malalignment (mechanical axis of >/=1.1 degrees valgus)
44 ss included medial meniscal damage and varus malalignment (medially) and lateral meniscal damage (lat
46 um from the secondary heart field leading to malalignment of the arterial pole with the ventricles.
47 tion to expand to the right, with subsequent malalignment of the atrioventricular endocardial cushion
53 e OA examined, the impact of varus or valgus malalignment on the odds of OA progression over the ensu
54 vely, in certain joint environments, such as malalignment or laxity, greater strength may translate i
56 limited to knees in which there was moderate malalignment (OR per 2-unit increase in BMI 1.23, 95% CI
57 d femoral denuded bone increase, while varus malalignment predicted medial tibial cartilage volume an
58 sk of lateral OA progression, that burden of malalignment predicts decline in physical function, and
59 neuromuscular factors (varus-valgus laxity, malalignment, proprioceptive inaccuracy, quadriceps stre
61 knee pain severity, physical activity, varus malalignment severity, hip OA presence, and hip OA sympt
62 olated PF OA were more likely to have valgus malalignment than those with isolated TF OA (P = 0.0002)
63 ciations of severity of meniscal tears, knee malalignment, tibiofemoral cartilage damage, knee effusi
71 3]).We found a strong relationship of valgus malalignment with progressive lateral meniscal damage.
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