ライフサイエンスコーパス: malarial parasite

1 ms are a result of selective pressure by the malarial parasite.

2 n of pathogens such as Plasmodium falciparum malarial parasite.

3 stems-wide biological view of this important malarial parasite.

4 as been demonstrated to be essential for the malarial parasite.

5 he outer membrane of the mitochondria of the malarial parasite.

6 idate antigen against the blood stage of the malarial parasite.

7 o protection against infection by this human malarial parasite.

8 stant to the growth of Plasmodium falciparum malarial parasites.

9 ely on the detection of antigens specific to malarial parasites.

10 tein synthesis in Gram-positive bacteria and malarial parasites.

11 ned model for altered CQ accumulation in CQR malarial parasites.

12 for lipid biosynthesis of intraerythrocytic malarial parasites.

13 s in humans and purine auxotrophs, including malarial parasites.

14 as HIV/SIV, Mycobacterium tuberculosis, and malarial parasites.

15 quine sensitive (CQS) versus resistant (CQR) malarial parasites.

16 versus time data for live, intraerythrocytic malarial parasites.

17 zed, intraerythrocytic Plasmodium falciparum malarial parasites.

18 en species (ROS) are widely believed to kill malarial parasites.

19 and dissimilar to those of plasmepsins from malarial parasites.

20 ant in carbohydrate and energy metabolism in malarial parasites.

21 as the erythrocyte receptor for invasion by malarial parasites.

22 in digestive vacuolar pH for drug resistant malarial parasites.

23 ts virulence, as the most malignant of human malarial parasites.

24 ated to the initiation of drug resistance in malarial parasites.

25 ween the blood stage trophozoite form of the malarial parasite and the sexual stage gametocyte form.

26 DH) is a key enzyme for energy generation of malarial parasites and is a potential antimalarial chemo

27 D DIC transmittance "z stack" images of live malarial parasites and use those to quantify hemozoin (H

28 f activities against human tumor cell lines, malarial parasites, and bacterial pathogens including lo

29 gnostic/triaging kits for early detection of malarial parasites are critical for prevention of malari

30 se, yet it is still not clear how bloodstage malarial parasites are killed.

31 aches to studying mitochondrial functions in malarial parasites are quite limited because of the tech

32 d2) (CQR via transfection with mutant pfcrt) malarial parasites as they develop within the human red

33 The malarial parasites assemble flagella exclusively during

34 hypothesized that the killing of liver-stage malarial parasites by IFN-gamma involves autophagy induc

35 host red blood cell hemoglobin is toxic, so malarial parasites crystallize heme to nontoxic hemozoin

36 This probe localizes to the malarial parasite digestive vacuole (DV) during initial

37 s has been heme biomineralization within the malarial parasite digestive vacuole.

38 The malarial parasite dramatically alters its host cell by e

39 t cell-mediated immunity against blood-stage malarial parasites during chronic malaria (i) requires t

40 The malarial parasite encodes two homologous aspartic protea

41 Tryptophan-rich antigens of malarial parasites have been proposed to be the potentia

42 Malarial parasites have evolved resistance to all previo

43 Its mode of action against malarial parasites, however, has remained undefined.

44 Dietary supplementation of malarial-parasite-infected mice with L-arginine or L-cit

45 Our findings reveal that malarial-parasite-infected mice, like humans, develop L-

46 Data on confirmed malarial parasite infections from health facilities in i

47 Women with >/=2 malarial parasite infections tended to have lower z scor

48 with this device: 1) BSDF-based detection of Malarial parasites inside unstained human erythrocytes;

49 first successful expression of a full-length malarial parasite integral membrane protein in yeast.

50 When malarial parasites invade and develop within the bloodst

51 Human infection by malarial parasites of the genus Plasmodium begins with t

52 highly A+T rich genomes of human and rodent malarial parasites offer unprecedented glimpses of a lin

53 s had no effect on erythrocytic infection by malarial parasite or movement of raft markers into the p

54 he mosquito resists infection with the human malarial parasite P. falciparum by engaging the NF-kappa

55 Plasmodium vivax and the related simian malarial parasite P. knowlesi use the Duffy blood group

56 ations and resistance to CQ in the important malarial parasite P. vivax.

57 resistance protein homologues found in this malarial parasite (PfMDR1) may further modify or tailor

58 e Gulu case, ebolavirus antigen localized to malarial parasite pigment-laden macrophages.

59 ecognition and killing of ookinetes from the malarial parasite Plasmodium berghei, a model for the hu

60 wild-type pfcrt allele into the rodent model malarial parasite Plasmodium berghei.

61 luorescent protein-based fluorescence on the malarial parasite Plasmodium berghei.

62 he structurally similar SSB protein from the malarial parasite Plasmodium falciparum (Pf-SSB) also bi

63 emozoin during metabolism of heme within the malarial parasite Plasmodium falciparum and assist ongoi

64 nt function in hemoglobin degradation in the malarial parasite Plasmodium falciparum and have generat

65 expression data set on the life cycle of the malarial parasite Plasmodium falciparum and systematical

66 activated by peroxynitrite, with GR from the malarial parasite Plasmodium falciparum being more sensi

67 Mitochondrial DNA of the malarial parasite Plasmodium falciparum comprises approx

68 Our in vitro investigations with the human malarial parasite Plasmodium falciparum document a remar

69 The malarial parasite Plasmodium falciparum has acted as a p

70 The malarial parasite Plasmodium falciparum is known to be s

71 aerythrocytic development cycle of the human malarial parasite Plasmodium falciparum is subject to ti

72 of gene expression (SAGE) was applied to the malarial parasite Plasmodium falciparum to characterize

73 Resistance of the human malarial parasite Plasmodium falciparum to the antimalar

74 In the malarial parasite Plasmodium falciparum, a multifunction

75 he prokaryote Vibrio harveyi, the eukaryotic malarial parasite Plasmodium falciparum, the parasitic A

76 complexity regions (LCRs) in proteins of the malarial parasite Plasmodium falciparum.

77 te Plasmodium berghei, a model for the human malarial parasite Plasmodium falciparum.

78 to cause chloroquine resistance (CQR) in the malarial parasite Plasmodium falciparum.

79 ut also in apicomplexan protists such as the malarial parasite Plasmodium falciparum.

80 The malarial parasite Plasmodium must complete a complex lif

81 urface that is the known entry point for the malarial parasite Plasmodium vivax.

82 te surface protein-1 (MSP-1) from the rodent malarial parasite Plasmodium yoelii yoelii 17XL, express

83 ative genomics of apicomplexans, such as the malarial parasite Plasmodium, the cattle parasite Theile

84 se (HG(X)PRT) is crucial for the survival of malarial parasites Plasmodium falciparum (Pf) and Plasmo

85 The complete genome sequence of the malarial parasite, Plasmodium falciparum, has provided r

86 enetically encoding this sensor in the human malarial parasite, Plasmodium falciparum, we have quanti

87 ibosylation factor (ARF) gene from the human malarial parasite, Plasmodium falciparum.

88 charomyces cerevisae and the non-model human malarial parasite, Plasmodium falciparum.

89 ility of the mosquito, Aedes aegypti, to the malarial parasite, Plasmodium gallinaceum, was investiga

90 lexa (intracellular parasites, including the malarial parasite, Plasmodium) share two short insertion

91 unctional analysis of essential genes in the malarial parasite, Plasmodium, is hindered by lack of ef

92 Continual exposure of malarial parasite populations to different drugs may hav

93 the superfamily of chemokine receptors and a malarial parasite receptor.

94 The malarial parasite relies on de novo pyrimidine biosynthe

95 iosensor shows the lowest detection limit of malarial parasites reported in the literature spanning d

96 Despite increasing prevalence of malarial parasite resistance to sulfadoxine-pyrimethamin

97 34-residue insertion specific for the GRs of malarial parasites shows no density, implying that it is

98 ediates signal transduction processes in the malarial parasite that regulate host erythrocyte invasio

99 for addressing the problem of resistance in malarial parasites that are solidly based in evolutionar

100 family of protozoa including Plasmodium sp (malarial parasite), Toxoplasma gondii, Cryptosporidium s

101 OH-inducible expression of the P. falciparum malarial parasite transporter PfCRT in P. pastoris yeast

102 The pLDH from all four species of human malarial parasites were cloned, expressed, and analyzed

103 n is highly effective against drug-resistant malarial parasites, which affects nearly half of the glo

104 Understanding the biology of malarial parasites will be facilitated by DNA microarray

105 It is likely that hypnozoites of relapsing malarial parasites will prove to be directly sporozoite-

106 ines (TBV), which prevent the development of malarial parasites within their mosquito vector, thereby