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1 cs of ARS, including corneal and iris stroma maldevelopment.
2 Delayed repair causes permanent motion VEP maldevelopment.
3 deficiency, a disease with potential retinal maldevelopment.
4 atterns that are a consequence of visuomotor maldevelopment.
5 here are no means of directly assessing such maldevelopment.
6 focal cortical dysgenesis and may be due to maldevelopment.
7 entation, or a mixture of these two modes of maldevelopment.
8 d open defects (i.e., exencephaly) and gross maldevelopment.
9 somatic postzygotic genetic lesions in brain maldevelopment.
11 ocephaly complex with associated features of maldevelopment and examine the roles of OTX2 and PRRX1.
12 n the fetal genome and the risk of placental maldevelopment and preeclampsia, possibly due to impairm
13 ide the first detailed description of the V1 maldevelopments associated with unrepaired natural, infa
15 er overactive BG, and a frontostriatoinsular maldevelopment in ASD with reduced structure and functio
17 gs thus far suggest subtle prenatal neuronal maldevelopment in the cerebellum and certain limbic stru
24 n of the two factors is necessary to trigger maldevelopment of prefrontal cortical and ventral striat
27 t is due to an absence of the hippocampus, a maldevelopment of the dorsolateral prefrontal cortex, or
28 velopmental disorder most commonly involving maldevelopment of the fingernails, kneecaps and elbow jo
29 ate that these disorders may result from the maldevelopment of the oculomotor (nIII), trochlear (nIV)
31 esis syndrome (TDS) hypothesis proposes that maldevelopment of the testis, irrespective of cause, lea
32 in humans and the monkey is associated with maldevelopment of visual motion responsiveness, one mani
33 arge-angle infantile esotropia have striking maldevelopments of binocular (disparity-driven) converge
34 exosc3 in zebrafish embryos caused embryonic maldevelopment, resulting in small brain size and poor m
36 nically relevant imaging markers of cerebral maldevelopment, which offer new insights into the nature
37 vivo evidence in humans for possible retinal maldevelopment with a predilection for retinal GCL loss
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