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1 sis, prevents spermiogenesis, and results in male sterility.
2 koencephalopathy in multiple brain areas and male sterility.
3 position and subsequently collapsed, causing male sterility.
4 tin degeneration of germ cells, resulting in male sterility.
5 ci for both behavioural isolation and hybrid male sterility.
6 sis, spermatid individualization, and causes male sterility.
7 hich displayed maternal-effect lethality and male sterility.
8 s the most likely mechanism producing hybrid male sterility.
9 M. musculus X chromosome involved in hybrid male sterility.
10 e phenotype, producing stable, nontransgenic male sterility.
11 on between species by contributing to hybrid male sterility.
12 ric ORFs are candidate genes for cytoplasmic male sterility.
13 Taf1 contribute about equally to HMS1 hybrid male sterility.
14 (hms2) locus to cause nearly complete hybrid male sterility.
15 nding to nematode proteins, 37% (7/19) cause male sterility.
16 rescues sDMA modification of Sm proteins and male sterility.
17 at targeted mutation of the Tenr gene causes male sterility.
18 ich accounts for the microspore abortion and male sterility.
19 stamen development that results in complete male sterility.
20 sive gene expression, delayed flowering, and male sterility.
21 togonia to differentiate, resulting in adult male sterility.
22 ble and the only observed defect is complete male sterility.
23 m for genetic factors contributing to hybrid male sterility.
24 al structure and functions, hypotension, and male sterility.
25 , almost there (amo), caused nearly complete male sterility.
26 and block sperm maturation in vivo, causing male sterility.
27 bit reduced lifespan, locomotor defects, and male sterility.
28 One of the most common HIs is male sterility.
29 lock the pathway of JA synthesis result into male sterility.
30 s, resulting in failed microsporogenesis and male sterility.
31 and the restoration of Texas cytoplasm-based male sterility.
32 l known selfish genetic element, cytoplasmic male sterility.
33 , which is necessary for T cytoplasm-induced male sterility.
34 express novel mitochondrial genes that cause male sterility.
35 contradictory observations exist for hybrid male sterility.
36 al protein necessary for T cytoplasm-induced male sterility.
37 ural defects and functions, hypotension, and male sterility.
38 nd genetic mechanisms underlying t haplotype male sterility.
39 0 Mb of DNA but has only six loci mutable to male sterility.
40 ption causes renal atrophy, hypotension, and male sterility.
41 ecause homozygosity of the distorters causes male sterility.
42 levant to the meiotic drive theory of hybrid male sterility.
43 ntional myosin, 95F myosin, which results in male sterility.
44 unctions in the point mutants, that leads to male sterility.
45 me translocations (T(Y;A)) to cause complete male sterility.
46 us thymidine kinase, which is known to cause male sterility.
47 n and testosterone production and results in male sterility.
48 utant flies exhibit complete azoospermia and male sterility.
49 ent stages are affected, leading to complete male sterility.
50 l for the nuclear restoration of cytoplasmic male sterility.
51 ordination, a defective escape response, and male sterility.
52 ich also causes behavioral abnormalities and male sterility.
53 nts exhibit defective spermatogenesis and/or male sterility.
54 6 in mice leads to acephalic spermatozoa and male sterility.
55 d flower development, delayed flowering, and male sterility.
56 ion of a phytotoxic barnase and provides for male sterility.
57 al differentiation and resulted in increased male sterility.
58 Gnat3(-/-) doubly null mice led to inducible male sterility.
59 plants overexpressing PAE1 exhibited severe male sterility.
60 irst cycles of spermatogenesis, resulting in male sterility.
61 )) and observed disrupted spermiogenesis and male sterility.
62 susceptible to perturbations that result in male sterility.
63 and reduction of its levels correlates with male sterility.
64 anther-specific Ms2 activation that confers male sterility.
65 ellular bridges in all germ cells and causes male sterility.
66 with pollen production and results in plant male sterility.
67 ependent incompatibilities that cause hybrid male sterility.
68 NbSACPD-C expression caused female, but not male, sterility.
69 effect: the M. guttatus allele at the hybrid male sterility 1 (hms1) locus acts dominantly in combina
70 stream of the transcriptional activator MS1 (MALE STERILITY 1), which contains a PHD domain associate
71 h recessive M. nasutus alleles at the hybrid male sterility 2 (hms2) locus to cause nearly complete h
72 gical processes, including immunity, cancer, male sterility, adaptive evolution, and non-Mendelian in
73 c) that depends strongly on the dominance of male sterility alleles, while N(en) remains equal to the
74 pecies carries any autosomal dominant hybrid male sterility alleles: reciprocal F(1) hybrid males are
77 e restorer loci with complementary recessive male-sterility alleles, as well as a locus with duplicat
78 osely related populations isolated by hybrid male sterility also show fixation of alternative neo-Y h
80 ceptor alpha (RARalpha) function resulted in male sterility and aberrant spermatogenesis, which resem
84 a strong association between X-linked hybrid male sterility and disruption of MSCI and suggest that t
85 hibited JA-insensitive phenotypes, including male sterility and enhanced resistance to P. syringae in
89 ster and to show that the insertion leads to male sterility and male mating behavior defects that inc
90 er, we discover at least six genes of hybrid male sterility and none for female sterility by deficien
91 tic basis of association between cytonuclear male sterility and other floral traits in Mimulus hybrid
93 f the genetic architecture underlying hybrid male sterility and segregation distortion between the Bo
94 to be necessary but not sufficient for both male sterility and segregation distortion in F(1) hybrid
95 w that the same gene, Overdrive, causes both male sterility and segregation distortion in F1 hybrids
96 al a complex genetic architecture for hybrid male sterility and suggest a prominent role for reproduc
97 pleiotropic phenotypes, including dwarfism, male sterility and the development of swellings at branc
100 with diseases such as cystic kidney disease, male sterility, and hydrocephalus in humans and model ve
102 ifferent from most described cases of hybrid male sterility, and may represent an earlier stage of hy
103 ice, which display behavioral abnormalities, male sterility, and perturbed breast development during
106 the molecular and functional basis of hybrid male sterility, and strongly reinforce the role of DNA-b
107 ficant reductions in DAZL protein levels and male sterility, and the knockdown was stable over multip
109 ects including facial dysmorphism, dwarfing, male sterility, anemia, and progressive polycystic kidne
110 fects in mitochondrial function resulting in male sterility, apoptotic muscle degeneration, and minor
113 enetic screens for modifiers of dfxr-induced male sterility, as a means to efficiently dissect FMRP-m
114 ow that knockdown of Gld2 transcripts causes male sterility, as GLD2-deficient males do not produce m
115 d female levels of sterility was great, with male sterility being up to 23 times greater than female
116 later generation (backcross and F(2)) hybrid male sterility between D. virilis and D. americana is no
117 hromosome has only a modest effect on hybrid male sterility between D. virilis and D. americana.
119 Here, I examine the genetic basis of hybrid male sterility between two species of Drosophila, Drosop
120 monocot rice (Oryza sativa) causes complete male sterility, but not in the dicot model Arabidopsis (
121 ines segregating for the restorer region and male sterility, but with unique flanking introgressions.
122 eriments revealed that the slcer6 mutant has male sterility caused by (1) hampered pollen dispersal a
123 It provides the first in vivo model for male sterility caused by a discrete signalling pathway d
124 exhibit the meiotic arrest, DNA damage, and male sterility characteristic of mice lacking piRNAs.
125 d perception mutants is profound sporophytic male sterility characterized by failure of stamen filame
126 plex sex determination involving cytoplasmic male sterility (CMS) alleles interacting with nuclear re
127 e CMS-92 mitochondria that cause cytoplasmic male sterility (CMS) by homeotic transformation of the s
128 al to asexual continuum, whether cytoplasmic male sterility (CMS) facilitates the evolution of patern
129 tima, sex determination involves cytoplasmic male sterility (CMS) genes and nuclear restorers of male
130 olus spp and the degree to which cytoplasmic male sterility (cms) has been characterized in the commo
132 hondrial-encoded genes can cause cytoplasmic male sterility (CMS), resulting in the coexistence of fe
133 These results suggest that the cytoplasmic male sterility (CMS)-PPR interaction is highly conserved
137 ertility (Rf) alleles for S-type cytoplasmic male sterility (CMS-S) are prevalent in Mexican races of
138 f maize plants exhibiting S-type cytoplasmic male sterility (cms-S) contain a repeated DNA region des
141 undulata cytoplasm that confers cytoplasmic male sterility (CMS92) or (ii) normal, with the fertile
143 viability, with surviving adults displaying male sterility, decreased female fertility, wing pattern
144 triple knockout mutants suffer from a strong male sterility defect as a consequence of pollen tubes t
146 nt and cell-wall properties, and resulted in male sterility due to complete disruption of formation o
148 defects, while Loxl2 overexpression triggers male sterility due to epididymal dysfunction caused by e
150 eletion of the SSTK gene in mice resulted in male sterility due to profound impairment in motility an
151 asymmetric genetic basis to X-linked hybrid male sterility during the early stages of speciation in
153 pecific genetic divergence underlying hybrid male sterility, especially in contrast with the low degr
154 ity can be highly polygenic and complex, and male sterility evolves substantially faster than female
156 etermination region and included a candidate male sterility factor and additional genes with sex-spec
158 This observation strongly indicates that male sterility factors have evolved more rapidly than ei
160 a confirm that the X is a hotspot for hybrid male sterility factors, providing a proximate explanatio
161 d in the general location of the two major t male sterility factors, S1 and S2, within inversions 1 a
162 andard models often find an excess of hybrid male sterility factors, we found no QTL for sterility an
163 inary screen to find additional small-effect male sterility factors, we identified one additional loc
165 that balancing selection acts on cytoplasmic male-sterility factors in several gynodioecious species
166 cy-dependent selection on linked cytoplasmic male-sterility factors, the putative molecular basis of
167 results clearly suggest that the customized male-sterility & fertility-restoration system can be exp
168 g this gene and its promoter for engineering male sterility for hybrid production of various plant sp
174 iations between marker loci and the inferred male-sterility genes can be maintained only with very lo
175 istence of nuclear restorers and cytoplasmic male-sterility genes in a population where females are v
176 ovide evidence that evolution of cytoplasmic male sterility has been characterized by frequent turnov
177 osphinothricin (5 mg/l), confirming that the male sterility has been successfully engineered in rice.
179 hile X-linked loci that contribute to hybrid male sterility have been precisely localized in many ani
180 ry reproductive barrier in house mice-hybrid male sterility-have been restricted to a single subspeci
182 in regulatory pathways may result in hybrid male sterility (HMS) when dominance and epistatic intera
184 le germ cells, but alpha 85E causes dominant male sterility if it makes up more than one-half of the
185 that dominantly interact with piwi2 to cause male sterility, implying that dosage-sensitive regulatio
186 . musculus domesticus Y chromosome to hybrid male sterility in a cross between wild-derived strains i
192 n, the D. virilis Y chromosome causes hybrid male sterility in combination with recessive D. american
193 cally, we show that the occurrence of hybrid male sterility in crosses between Drosophila mojavensis
194 TL do not contribute significantly to hybrid male sterility in crosses between the sympatric species
195 t site of the Odysseus (Ods) locus of hybrid male sterility in Drosophila contains such a homeobox ge
198 important clues about the genetics of hybrid male sterility in house mice, they have been restricted
199 lved in genetic incompatibilities leading to male sterility in hybrids between Drosophila simulans an
201 , we demonstrate that nearly complete hybrid male sterility in Mimulus results from a simple genetic
205 potential method for generating maintainable male sterility in plants by using existing agrochemicals
206 ociated with naturally occurring cytoplasmic male sterility in plants, a transgenic approach for RNAi
208 asmic types, one of which appears to produce male sterility in progeny from any hermaphrodite pollen
210 ant autosomal factors contributing to hybrid male sterility in the allopatric species pair Drosophila
211 of the CMS cytotypes has been sequenced, and male sterility in the cms-S and cms-T cytotypes is linke
217 -linked QTL that underlie measures of hybrid male sterility, including testis weight, sperm density,
219 through chemical mutagenesis showed that the male sterility is a distinctive feature of the qk(v) all
223 the mitochondrial genes encoding cytoplasmic male sterility is altered in the presence of one or more
224 estorer alleles on Linkage Group 7, and that male sterility is associated with reduced corolla size.
225 hat, in a population of hermaphrodites where male sterility is caused by a dominant allele in a nucle
226 reduction in cell size and fewer cells, and male sterility is caused by loss of the pollen coat and
227 ila melanogaster-D. simulans hybrids, hybrid male sterility is caused by the lack of a single-copy ge
230 ous autosomal factors contributing to hybrid male sterility is comparable to the density of X chromos
231 particular interest in plants as cytoplasmic male sterility is controlled by mitochondrial genotypes,
233 l phenotype but a strong influence on hybrid male sterility is discussed in light of Haldane's rule o
238 process of species divergence and why hybrid male sterility is often the first sign of speciation, we
241 monstrating that the genetic basis of hybrid male sterility largely differs between these closely rel
244 l tremor indicative of neurological defects, male-sterility, low female fertility, but near normal li
247 three of the four QTL associated with hybrid male sterility occur in collinear (uninverted) regions o
249 on of ACA12 rescues the phenotype of partial male sterility of a null mutant of the plasma membrane i
250 bundance of piRNAs in germline cells and the male sterility of Miwi mutants suggest a role in gametog
253 in the relative frequencies of mutations to male sterility or in the frequencies of genes with male-
254 While biocontainment might be achieved using male sterility or transgenic mitigation tools, we believ
255 (SCs) leads to severe testicular atrophy and male sterility owing to rapid depletion of both SCs and
258 linked QTL associated with a range of hybrid male sterility phenotypes, including testis weight, sper
260 However, homozygous mutant mice exhibit male sterility, probably because homologous recombinatio
261 arfed stature; dark green, thickened leaves; males sterility; reduced apical dominance; and de-etiola
262 rental species, however, the map location of male sterility reflected the maternal donor in one cross
265 ain (axDHC) gene, Dnahc8, has been linked to male sterility resulting from aberrant sperm motility.
266 f Dnahc8 expression has been associated with male sterility resulting from an early breakdown in sper
267 ize, with its excellent forward genetics and male sterility screens, was used to identify >50 meiotic
268 similar defects as pink1 and parkin mutants: male sterility, shortened lifespan, and reduced climbing
270 e mapping of two crosses showed dominance of male sterility similar to the parental species, however,
271 ction in Arabidopsis resulted in conditional male sterility, since pollen coat lipids are responsible
273 nction has been used to develop a reversible male sterility system applicable to hybrid crop producti
274 results in the first engineered cytoplasmic male-sterility system in plants, offers a new tool for t
275 n the X Chromosome is more likely to produce male sterility than on autosome (so-called large-X theor
277 an nonsense germline variant associated with male sterility that results in loss of NLRP14 function a
280 extensive recombination, tentatively linking male sterility to orf293, a mitochondrial gene causing h
283 We demonstrate that multiple cytoplasmic male-sterility types are present in a gynodioecious popu
285 with duplicate action, which cannot produce male sterility unless the plant is also homozygous for t
286 Results are given of genetic studies of male sterility using plants from two natural populations
287 The contribution of each gene to hybrid male sterility was assessed by means of germ-line transf
290 36H), which extended more proximally, caused male sterility when heterozygous with a complete t haplo
291 , which results in virtually complete hybrid male sterility when homozygous in the genetic background
293 class of mutants termed CMS (for cytoplasmic male sterility), which is associated with mutations in t
294 osome of the hybrid reflects the location of male sterility within the maternal donor species and (3)
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