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1 B, TUBB4A, and TUBB2A) have been linked with malformations of cortical development.
2 y the discovery of rare mutations that cause malformations of cortical development.
3 CLN gene) is involved in the pathogenesis of malformations of cortical development.
4 n shows promise for localizing epileptogenic malformations of cortical development.
5 me or early myoclonic encephalopathy without malformations of cortical development.
6 e (1Hz, 1,200 pulses), focally targeting the malformations of cortical development.
7 ects in the brain, clinically referred to as malformations of cortical development.
8 ts with localization-related epilepsy due to malformations of cortical development.
9 genitors and nascent neurons produces severe malformations of cortical development.
17 disease loci in putative Mendelian forms of malformations of cortical development has been hindered
18 ionship to structural lesions, in particular malformations of cortical development, has been further
19 analysis of over 2,000 children with complex malformations of cortical development identified five in
20 tumors (mainly ganglioglioma) in 23.6%, and malformations of cortical development in 19.8% (focal co
22 ognitive effects of seizures on rodents with malformations of cortical development induced with methy
28 increasingly recognized as a major cause of malformations of cortical development (MCD), typically l
30 or partial volume effect in 10 patients with malformations of cortical development (MCDs) and partial
36 x development and in the number and types of malformations of cortical development that have been rep
37 ients with localization-related epilepsy and malformations of cortical development, we have now appli
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