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1 nded as a therapy for patients with advanced malignant pleural mesothelioma.
2 VAT-PP and talc pleurodesis in patients with malignant pleural mesothelioma.
3 ) identified 68 with advanced ASS1-deficient malignant pleural mesothelioma.
4 idered the best available serum biomarker of malignant pleural mesothelioma.
5 veral control groups and 1,026 patients with malignant pleural mesothelioma.
6 acy of trimodality therapy in stage I to III malignant pleural mesothelioma.
7 rging therapeutic option in the treatment of malignant pleural mesothelioma.
8 atment with cisplatin alone in patients with malignant pleural mesothelioma.
9 our activity in patients with PD-L1-positive malignant pleural mesothelioma.
10 e extent of disease in patients with diffuse malignant pleural mesothelioma.
11 s promise in the palliation of patients with malignant pleural mesothelioma.
12 ne depletion in patients with ASS1-deficient malignant pleural mesothelioma.
15 y of trimodality therapy in the treatment of malignant pleural mesothelioma and identify prognostic f
16 val in patients with pleural effusion due to malignant pleural mesothelioma, and talc pleurodesis mig
17 am of the type I IGF receptor in a subset of malignant pleural mesothelioma cell lines and determined
20 urvival and quality of life in patients with malignant pleural mesothelioma have, to our knowledge, n
22 published literature of clinical studies in malignant pleural mesothelioma, including phase II trial
28 ic cell-based immunotherapy in patients with malignant pleural mesothelioma is feasible, well-tolerat
32 gulated in malignant mesothelial tissues and malignant pleural mesothelioma (MPM) cell lines as compa
47 d pemetrexed in patients with ASS1-deficient malignant pleural mesothelioma (MPM) or non-small-cell l
50 ated receptor-1 (PAR1, F2R) on the growth of malignant pleural mesothelioma (MPM), using human MPM ce
56 ge to undergoing cancer-directed surgery for malignant pleural mesothelioma (MPM); however, it is unc
58 ich has been shown to be highly expressed in malignant pleural mesotheliomas (MPM), was detected in s
60 NA mutations and expression levels unique to malignant pleural mesotheliomas (MPMs) and not present i
62 ough SV40 oncoproteins have been detected in malignant pleural mesotheliomas (MPMs), their role in th
64 prevent procedure-tract metastases (PTMs) in malignant pleural mesothelioma remains controversial, an
67 efore, IGF system components represent novel malignant pleural mesothelioma therapeutic targets for i
68 e clinical benefit reported in patients with malignant pleural mesothelioma treated in a phase 1 stud
70 fulness of (18)F-FDG-CI in the assessment of malignant pleural mesothelioma using histopathology as t
71 nty-nine patients with histologically proven malignant pleural mesothelioma were enrolled (26 male pa
72 viously treated patients with PD-L1-positive malignant pleural mesothelioma were enrolled from 13 cen
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