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1 Staphylococcus aureus, and Stenotrophomonas maltophilia).
2 al microbiology laboratories were in fact S. maltophilia.
3 Escherichia hermannii, and Stenotrophomonas maltophilia.
4 reus, A. xylosoxidans, D. acidovorans and S. maltophilia.
5 ident among other respiratory isolates of S. maltophilia.
6 from the emerging pathogen Stenotrophomonas maltophilia.
7 osa and the human pathogen, Stenotrophomonas maltophilia.
8 zinc L1 beta-lactamase from Stenotrophomonas maltophilia.
9 ug resistant human pathogen Stenotrophomonas maltophilia.
10 negative bacilli, including Stenotrophomonas maltophilia.
11 ygenase gene to a megaplasmid in cells of P. maltophilia.
12 d at least one sputum sample positive for S. maltophilia.
13 lin-resistant S. aureus], 2 Stenotrophomonas maltophilia, 1 Klebsiella pneumoniae) and resulted in an
15 erobacter aerogenes (4.4%), Stenotrophomonas maltophilia (4.3%), Proteus mirabilis (4.0%), Klebsiella
16 dans (100%) followed by MDR Stenotrophomonas maltophilia (46%), MDR Achromobacter xylosoxidans (33%),
17 reus, Burkholderia cepacia, Stenotrophomonas maltophilia, Achromobacter xylosoxidans and atypical myc
18 552 genomes of the pathogen Stenotrophomonas maltophilia across 23 sites of the lungs from a patient
19 Pseudomonas aeruginosa, and Stenotrophomonas maltophilia--all major threats to our cancer patients.
20 ring in 2 patients with MDR Stenotrophomonas maltophilia and 2 patients with MDR Achromobacter xyloso
21 To examine the molecular epidemiology of S. maltophilia and A. xylosoxidans in CF, isolates from pat
22 ing can distinguish unique CF isolates of S. maltophilia and A. xylosoxidans, person-to-person transm
25 thogen interaction between C. elegans and S. maltophilia and established a new animal model with whic
26 the opportunistic pathogens Stenotrophomonas maltophilia and Ochrobactrum anthropi were detected in m
27 romobacter xylosoxidans and Stenotrophomonas maltophilia and their antibiotic susceptibility patterns
28 in-resistant P. aeruginosa, Stenotrophomonas maltophilia, and Achromobacter xylosoxidans but was less
30 niae, Enterobacter cloacae, Stenotrophomonas maltophilia, and the Burkholderia cepacia complex (BCC)
31 epresents the first examination of T2S in S. maltophilia, and the data obtained indicate that Xps T2S
32 biology references describe Stenotrophomonas maltophilia as oxidase negative and variable with respec
33 ibitors reverse ceftazidime resistance in S. maltophilia because, unlike clavulanic acid, they do not
34 f CF patients with moderate lung disease, S. maltophilia can be cultured from respiratory tract secre
36 eruginosa, 14 for A. baumannii, and 2 for S. maltophilia Categorical agreement (CA) was assessed usin
37 ions in the dinuclear active site of the S. maltophilia Class B3 MbetaL move away from each other, b
41 xty-one of 69 CF centers screened had 183 S. maltophilia culture-positive patients, and 46 centers ha
42 ients with > or =10 positive cultures (12 S. maltophilia cultures, 15 A. xylosoxidans cultures) had s
43 sequencing for identification and, unlike S. maltophilia, demonstrated susceptibility to most antibio
44 uginosa, the hazard ratio associated with S. maltophilia detection was 0.89 (95% confidence interval,
48 maltophilia, those patients positive for S. maltophilia had the following baseline characteristics b
49 the opportunistic pathogen Stenotrophomonas maltophilia has been determined at 1.7 A resolution by t
50 ic sarcosine oxidase (TSOX) from Pseudomonas maltophilia has been determined at 1.85 A resolution.
54 Although patients with CF who acquire S. maltophilia have more advanced disease than those who do
55 romobacter xylosoxidans and Stenotrophomonas maltophilia have similar posttransplant survival as comp
58 rs to be important in the pathogenesis of S. maltophilia infection as less than 20% of TNFR1 null mic
60 plus aztreonam as combination therapy for S. maltophilia infections and confirm that aztreonam-like b
61 allo-beta-lactamase L1 from Stenotrophomonas maltophilia is a dinuclear Zn(II) enzyme that contains a
69 The Gram-negative bacterium Stenotrophomonas maltophilia is increasingly identified as a multidrug-re
70 metallo-beta-lactamase from Stenotrophomonas maltophilia is unique among this class of enzymes becaus
72 ngosepticum isolates, and 1 Stenotrophomonas maltophilia isolate) producing IMP-1, IMP-1-like, IMP-18
75 ulted in amplification of a band from all S. maltophilia isolates and was uniformly negative for all
76 that can rapidly and accurately identify S. maltophilia isolates and which can be used for the direc
77 veloped and tested against a panel of 112 S. maltophilia isolates collected from diverse geographic l
78 s of suspected small-colony-variant (SCV) S. maltophilia isolates from the sputa of five CF patients
79 SCV S. maltophilia isolates were the only S. maltophilia isolates in these cultures, and none were cl
86 ficantly differentially expressed between S. maltophilia JCMS and avirulent bacteria (Escherichia col
87 ken together, these findings suggest that S. maltophilia JCMS evades the pathogen resistance conferre
90 aride lyase (Smlt1473) from Stenotrophomonas maltophilia k279a, which exhibited significant activity
92 be P. aeruginosa (n = 10), Stenotrophomonas maltophilia (n = 1), and Burkholderia cepacia (n = 1).
93 as maltophilia (n = 5), MDR Stenotrophomonas maltophilia (n = 26), and CF patients without Achromobac
94 3), Serratia spp. (n = 10), Stenotrophomonas maltophilia (n = 43), Sphingobacterium spp. (n = 3), and
95 ans (n = 15), pan-resistant Stenotrophomonas maltophilia (n = 5), MDR Stenotrophomonas maltophilia (n
96 who were older than 6 years of age, were S. maltophilia negative in the first year of enrollment, an
100 on of Burkholderia cepacia, Stenotrophomonas maltophilia, or Alcaligenes xylosoxidans; however, isola
102 aminodeoxychorismate synthase activity of S. maltophilia PabB alone revealed that it is virtually ina
103 ss spectral analysis further suggest that S. maltophilia PabB, like Escherichia coli PabB, binds tryp
104 ies exhibited by StmPr1 may contribute to S. maltophilia pathogenesis in the lung by inducing tissue
106 o antibiotics may select for both the SCV S. maltophilia phenotype and SXT resistance by interference
111 ata from this study, we hypothesized that S. maltophilia strain ZL1 was able to convert E1 to amino a
112 To achieve the objective, Stenotrophomonas maltophilia strain ZL1 was used as a model estrogen degr
113 is a multicomponent enzyme from Pseudomonas maltophilia, strain DI-6, that catalyzes the conversion
116 nes xylosoxidans strains, 5 Stenotrophomonas maltophilia strains, and 5 Pseudomonas aeruginosa strain
117 monas aeruginosa strains, 8 Stenotrophomonas maltophilia strains, and 9 isolates belonging to nine ot
119 edicated pabA is evident in the genome of S. maltophilia, suggesting that another cellular amidotrans
123 rain K279a, the first clinical isolate of S. maltophilia to be sequenced, encodes a functional type I
124 penemases are restricted to Stenotrophomonas maltophilia, to a few Bacteroides fragilis, and to rare
125 overall virulence of clinical isolates of S. maltophilia using the well-characterized opportunistic p
126 la, Pseudomonas aeruginosa, Stenotrophomonas maltophilia, Vibrio cholerae, and Yersinia enterocolitic
128 A putative alginate lyase (Smlt1473) from S. maltophilia was heterologously expressed in Escherichia
129 35 home-use nebulizers, and Stenotrophomonas maltophilia was isolated from 4 of 35 home-use nebulizer
131 e phenotypic switch from wild-type to SCV S. maltophilia was reproducible in vitro by exposure to SXT
132 allo-beta-lactamase L1 from Stenotrophomonas maltophilia was studied using rapid-scan and stopped-flo
133 lues for P. aeruginosa, A. baumannii, and S. maltophilia were 94.1%, 92.7%, and 95.5%, respectively,
134 lues for P. aeruginosa, A. baumannii, and S. maltophilia were 99.5%, 99.2%, and 100%, respectively.
139 sight into the virulence potential of the S. maltophilia Xps type II secretion system and its StmPr1
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