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1 lial-mesenchymal signaling in the developing mammary bud.
2 e examined IRS-1/2 double knockout embryonic mammary buds.
3 that BMP4 can rescue outgrowth of PTHrP(-/-) mammary buds.
4 in signaling, is expressed in the developing mammary bud and in luminal epithelial cells in the adult
5 ization in the developing E14.5day embryonic mammary bud and within the ducts, terminal end buds (TEB
6 PTHrP is made in the epithelial cells of the mammary bud and, during embryonic mammary development, i
8 actions of BMP4, triggering outgrowth of the mammary buds and inducing MSX2 expression, which, in tur
9 elays formation of Eda-induced supernumerary mammary buds and normalizes the embryonic and postnatal
10 gnaling is important for outgrowth of normal mammary buds and that BMP4 can rescue outgrowth of PTHrP
11 trast, PPR1 expression is not limited to the mammary bud, but is found generally within the subepider
14 mary marker expression and lack two pairs of mammary buds, demonstrating that Gli3 is essential for m
19 ttern, first in the epithelium overlying the mammary bud during embryogenesis and then in the myoepit
20 ough p190-B and IRS proteins is critical for mammary bud formation and ensuing epithelial-mesenchymal
21 ds, demonstrating that Gli3 is essential for mammary bud formation and preceding patterning events.
23 e mammary gland development, we transplanted mammary buds from genetically rescued ErbB2(-/-) embryos
24 ede mammary gland induction, but compromises mammary bud growth, as well as TEB formation, ductal out
25 wth that is responsible for transforming the mammary bud into the rudimentary mammary duct system.
32 s of homozygous deletion of p190A, embryonic mammary buds were rescued by transplantation into the cl
33 ion of the TOPGAL-C reporter and resulted in mammary buds with reduced expression of mammary mesenchy
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