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1 -1 signaling, thereby inducing expression of manganese superoxide dismutase.
2 1, catalase, Cu/Zn superoxide dismutase, and manganese superoxide dismutase.
3 stimates of the metal binding affinities for manganese superoxide dismutase.
4 an adenoviral vector containing the cDNA for manganese superoxide dismutase.
5 y transfection with an expression vector for manganese superoxide dismutase.
6 tem and is specific to cells totally lacking manganese superoxide dismutase.
7 sphorylation, by uncoupling protein-1 and by manganese superoxide dismutase.
8 with no difference in protein expression of manganese superoxide dismutase.
9 s downstream anti-oxidant genes catalase and manganese superoxide dismutase.
10 nse of the cell against superoxide radicals, manganese superoxide dismutase.
11 idative stress response enzymes catalase and manganese superoxide dismutase.
12 n of the anti-apoptotic factors Survivin and manganese superoxide dismutase.
13 mitochondrial targeting sequence (MTS) from manganese superoxide dismutase.
14 the mitochondrial localization sequence from manganese-superoxide dismutase.
16 Expressions of the antioxidants catalase and manganese superoxide dismutase-2 (SOD2) and the autophag
18 arkers of mitochondrial dysfunction, such as manganese superoxide dismutase (9-fold), and reduced rat
21 of transcription 3-mediated upregulation of manganese superoxide dismutase, a primary mitochondrial
22 hione reductase, glutathione peroxidase, and manganese superoxide dismutase activities were slightly
23 onylation in the myocardium despite elevated manganese superoxide dismutase activity during endotoxem
24 oups had hippocampal reduced glutathione and manganese superoxide dismutase activity levels comparabl
28 in, glutathione-S-transferases, and possibly manganese superoxide dismutase, affect ascorbate levels
29 theta-1 [T-1], and pi-1 [P-1: Ile-105Val]), manganese superoxide dismutase (Ala-9Val), and catalase
30 cytochrome c, subunit IV of complex IV, and manganese superoxide dismutase, all encoded in the nucle
32 vels of c-IAP1, c-IAP2, and Bcl-2 as well as manganese superoxide dismutase and c-Jun NH2-terminal ki
33 cies (peroxides) and to the induction of the manganese superoxide dismutase and catalase activities b
35 endothelial cells has identified two genes, manganese superoxide dismutase and cyclooxygenase-2, tha
36 synthase (eNOS) and the antioxidant enzymes manganese superoxide dismutase and cytochrome c oxidase.
37 ation and (2) the effect of gene transfer of manganese superoxide dismutase and endothelial nitric ox
38 d upregulated the expression and activity of manganese superoxide dismutase and glutathione levels, s
39 e there is an up-regulation of mitochondrial manganese superoxide dismutase and glutathione peroxidas
40 as well as induction of antioxidant enzymes manganese superoxide dismutase and heme oxygenase 1 in i
41 lternative oxidase, dynamin related protein, manganese superoxide dismutase and Lon protease, respect
43 h down-regulation of GR and up-regulation of manganese superoxide dismutase and reduced glutathione l
44 e in mediating Sirt1-induced upregulation of manganese superoxide dismutase and suppression of oxidat
47 performed to differentiate polymorphisms for manganese superoxide dismutase and three classes of glut
48 in a concentration-dependent manner by both manganese-superoxide dismutase and cytochrome c proteins
49 expression of either uncoupling protein 1 or manganese superoxide dismutase, and azaserine, an inhibi
50 utathione system (e.g. ascorbate peroxidase, manganese superoxide dismutase, and dehydroascorbate red
51 As much as 40% of the total Mn(II) was in manganese superoxide dismutase, and it is this protein a
52 thione synthetase genes, increased levels of manganese superoxide dismutase, and NADPH oxidase-comple
53 RNA expression of the glucose 6-phosphatase, manganese superoxide dismutase, and sterol carrier prote
54 iron can outcompete manganese for binding to manganese superoxide dismutase, and through Fenton chemi
55 ression of catalase, glutathione peroxidase, manganese superoxide dismutase, and VDUP1, a protein tha
56 by vitamin E treatment or transfection with manganese superoxide dismutase, and was further increase
57 ondrial topoisomerase I, peroxiredoxin 3 and manganese superoxide dismutase--are elevated in A-T cell
59 id leader peptide of a new human recombinant manganese superoxide dismutase can enter cells and carry
60 ng enzymes copper/zinc superoxide dismutase, manganese superoxide dismutase, catalase, and glutathion
64 e utility of this methodology in examining a manganese superoxide dismutase cDNA fragment which we de
65 PLP animals expressed increased (P<0.01) manganese superoxide-dismutase, copper-zinc superoxide-d
67 total superoxide dismutase and mitochondrial manganese superoxide dismutase (cytochrome c reduction m
68 oxic environment increases the resistance of manganese superoxide dismutase-deficient astrocytes to s
69 ty, and MC fibronectin accumulation, whereas manganese superoxide dismutase depletion increases mitoc
71 vascular cell adhesion molecule-1 (VCAM-1), manganese superoxide dismutase, endothelial-specific mol
72 e dismutase (cytochrome c reduction method), manganese superoxide dismutase expression (Western blot)
74 tablish a novel nutrient-sensing pathway for manganese superoxide dismutase expression mediated throu
75 ns with 8Br-cGMP also activated catalase and manganese superoxide dismutase expression, indicating th
79 talloproteinase 1, ferritin light chain, and manganese superoxide dismutase genes were identified as
80 ntified: tropomyosin, heat shock protein 27, manganese superoxide dismutase, glutathione S-transferas
82 e replacement Tyr 34 --> Phe (Y34F) in human manganese superoxide dismutase (hMnSOD) and crystallized
83 parent full-length cDNA sequences coding for manganese superoxide dismutase (HvMnSOD) and extracellul
85 dothelial growth factor, p21(WAF1/CIP1), and manganese superoxide dismutase in MCF7 breast cancer cel
86 fic biological oxidants on recombinant human manganese superoxide dismutase in vitro have been evalua
87 conserved residue in the active site of the manganese superoxide dismutases in organisms from bacter
88 umarase, upstream of the sodA gene, encoding manganese superoxide dismutase, in Pseudomonas aeruginos
90 dependent absorption) for anion complexes of manganese superoxide dismutase indicating a change in co
91 ese results, a mechanism for metal uptake by manganese superoxide dismutase involving reorientation o
93 demonstrated that the mitochondrial protein manganese superoxide dismutase is inactivated, tyrosine
94 ssion of both Cu/Zn superoxide dismutase and manganese superoxide dismutase, leading to increased oxi
95 ncoding the mitochondrial antioxidant enzyme manganese superoxide dismutase, led to the identificatio
96 rotein locus (chromosome 16) (P=.01) and the manganese superoxide dismutase locus (chromosome 6) (P=.
97 diation (MCF+FIR cells) or overexpression of manganese superoxide dismutase (MCF+SOD cells) demonstra
98 g mitochondrial-targeted MPG using the human manganese superoxide dismutase mitochondrial-targeting s
99 ochondrial adaptive or stress proteins (e.g. manganese superoxide dismutase, mitochondrial KATP chann
100 opper/zinc superoxide dismutase (Cu/Zn SOD), manganese superoxide dismutase (Mn SOD), catalase, and g
103 ential role of the mitochondrial antioxidant manganese superoxide dismutase (Mn-SOD) as a potential e
104 adjacent subunits in the homotetramer human manganese superoxide dismutase (Mn-SOD) form a hydrogen
106 enes c-inhibitor of apoptosis 2 (c-IAP2) and manganese superoxide dismutase (Mn-SOD) in breast cancer
114 in, albumin, total antioxidant status (TAS), manganese superoxide dismutase (Mn-SOD), aconitase, glut
120 the six-coordinate azide adduct of oxidized manganese superoxide dismutase (Mn3+ SOD) that is formed
122 es, which expressed a range of mitochondrial manganese superoxide dismutase (MnSOD) activities, with
123 n part, this derived from TAM downregulating manganese superoxide dismutase (MnSOD) activity by causi
126 re not well understood, reports suggest that manganese superoxide dismutase (MnSOD) and calpain may b
127 oxo3a-dependent), antioxidant-encoding genes manganese superoxide dismutase (MnSOD) and catalase (Cat
128 dentified the p53-dependent up-regulation of manganese superoxide dismutase (MnSOD) and glutathione p
129 DNA despite eight- and fourfold increases in manganese superoxide dismutase (MnSOD) and mitochondrial
130 ve oxygen species, in part, by deacetylating manganese superoxide dismutase (MnSOD) and mitochondrial
131 mitochondrial targeting sequence (MTS) from manganese superoxide dismutase (MnSOD) and placed them u
132 phosphorylated eNOS (p-eNOS) (ser 1177), and manganese superoxide dismutase (MnSOD) and reduced serum
133 totic role via the induction of target genes manganese superoxide dismutase (MnSOD) and/or calbindin.
134 complex on the promoters of the catalase and manganese superoxide dismutase (MnSOD) antioxidant genes
135 +)-induced dephosphorylation was observed in manganese superoxide dismutase (MnSOD) as well as the pr
139 ency of the mitochondrial antioxidant enzyme manganese superoxide dismutase (MnSOD) exacerbated amylo
140 /CS-like individuals, 74 (20%) had increased manganese superoxide dismutase (MnSOD) expression, a man
141 -dependent genes heme oxygenase-1 (Ho-1) and manganese superoxide dismutase (Mnsod) following DHB pre
144 Bacterial lipopolysaccharide can induce manganese superoxide dismutase (MnSOD) gene expression i
145 luating the tumor suppressor activity of the manganese superoxide dismutase (MnSOD) gene on establish
147 n, concentration-dependent p53 effect on the manganese superoxide dismutase (MnSOD) gene was investig
154 ncoupling protein-1 (UCP1) HVJ-liposomes, or manganese superoxide dismutase (MnSOD) HVJ-liposomes com
155 estigates the role of the antioxidant enzyme manganese superoxide dismutase (MnSOD) in androgen-indep
156 Here we report that decreased expression of manganese superoxide dismutase (MnSOD) in EPCs contribut
159 related with a higher level of expression of manganese superoxide dismutase (MnSOD) in the latter cel
160 ased expression of the NF-kappaB target gene manganese superoxide dismutase (MnSOD) in the pulmonary
170 he disproportionation of superoxide by human manganese superoxide dismutase (MnSOD) is characterized
172 alysis of the interfaces between subunits in manganese superoxide dismutase (MnSOD) is currently unde
173 rial analysis of gene expression (SAGE) that manganese superoxide dismutase (MnSOD) is enriched in th
177 group: liver pathology, lipid peroxidation, manganese superoxide dismutase (MnSOD) levels, copper-zi
178 ersed by free radical scavengers such as the manganese superoxide dismutase (MnSOD) mimetic TBAP and
180 o induce transcriptional activation of human manganese superoxide dismutase (MnSOD) mRNA in human lun
182 5) but there was no change in mRNA levels of manganese superoxide dismutase (MnSOD) or glutamate cyst
184 has a reduced ability to activate the human manganese superoxide dismutase (MnSOD) promoter in A293
185 m Sed females exhibited significantly higher manganese superoxide dismutase (MnSOD) protein expressio
186 r laboratories showed that overexpression of manganese superoxide dismutase (MnSOD) reduced tumor inc
187 This study investigates the hypothesis that manganese superoxide dismutase (MnSOD) regulates cellula
188 regimen of cutaneous gene therapy of eNOS or manganese superoxide dismutase (MnSOD) restored such hea
189 binds to the 3' untranslated region (UTR) of manganese superoxide dismutase (MnSOD) RNA has been desc
190 ined region in the 3' untranslated region of manganese superoxide dismutase (MnSOD) RNA; both the MnS
191 ratories demonstrated that overexpression of manganese superoxide dismutase (MnSOD) suppressed both t
192 ion of the mitochondrial antioxidant protein manganese superoxide dismutase (MnSOD) to the heart to p
193 ss was assessed using aconitase activity and manganese superoxide dismutase (MnSOD) transcript levels
194 ribozyme that targets the protective enzyme manganese superoxide dismutase (MnSOD) was expressed in
195 e depletion of superoxide catalyzed by human manganese superoxide dismutase (MnSOD) was observed spec
196 AC5 Tg hearts, whereas protein expression of manganese superoxide dismutase (MnSOD) was reduced by 38
197 month-old mice showed that protein levels of manganese superoxide dismutase (MnSOD) were unchanged in
198 e lines expressing increased levels of human manganese superoxide dismutase (MnSOD), a mitochondrial
202 ppaB-dependent anti-apoptotic genes A20, A1, manganese superoxide dismutase (MnSOD), and cellular inh
203 chondrial levels of the anti-oxidant enzyme, manganese superoxide dismutase (MnSOD), are dramatically
204 tified sod2, encoding the antioxidant enzyme manganese superoxide dismutase (MnSOD), as a RelA target
206 dants (enzymatic), including the polymorphic manganese superoxide dismutase (MnSOD), can act to reduc
207 teracts with the primary antioxidant enzyme, manganese superoxide dismutase (MnSOD), consistent with
208 and enzyme activity gels were performed for manganese superoxide dismutase (MnSOD), copper/zinc, cat
209 side chain of Gln143, a conserved residue in manganese superoxide dismutase (MnSOD), forms a hydrogen
211 the cytokine-inducible cytoprotective genes manganese superoxide dismutase (MnSOD), interleukin-6 (I
213 up of NF-kappaB-related proteins [i.e., p65, manganese superoxide dismutase (MnSOD), phosphorylated e
216 ncrease the activity, but not the levels, of manganese superoxide dismutase (MnSOD), the mitochondria
217 polymorphism [valine (V) --> alanine (A)] of manganese superoxide dismutase (MnSOD), the primary anti
219 on of either uncoupling protein-1 (UCP-1) or manganese superoxide dismutase (MnSOD), which decrease h
221 ydrophobic side of the active site cavity of manganese superoxide dismutase (MnSOD), with its indole
236 and downregulates the ROS-scavenging protein manganese superoxide dismutase (MnSOD); the alpha(1)-AR-
237 ncreased mitochondrial antioxidant defenses [manganese superoxide dismutase (MnSOD)P< 0.05; copper/zi
241 from Pseudomonas putida (Pp), sodA, encoding manganese-superoxide dismutase (MnSOD) and, sodB, iron-s
243 the present study, we show that VEGF induces manganese-superoxide dismutase (MnSOD) mRNA and protein
244 We recently reported that wild-type (WT) manganese superoxide dismutases (MnSODs) from Saccharomy
246 proteins play roles in antioxidant defense (manganese superoxide dismutase), neuronal communication
247 etic mice by either transgenic expression of manganese superoxide dismutase or by administration of a
250 Transgenic mice overexpressing mitochondrial manganese superoxide dismutase or extracellular superoxi
252 hereas there was no change in c-IAP2 levels, manganese superoxide dismutase, or c-Jun NH2-terminal ki
255 are elevated in the retina in diabetes, and manganese superoxide dismutase overexpression prevents t
256 erived oxidative stress by overexpression of manganese-superoxide dismutase prevented insulin resista
258 ransient transfection with catalase (but not manganese-superoxide dismutase) produced a decrease in G
264 copper-zinc superoxide dismutase (CuZnSOD), manganese superoxide dismutase (SOD), catalase T, or met
266 expression array analysis revealed increased manganese superoxide dismutase (SOD-2) expression in the
267 significant elevations in the expression of manganese superoxide dismutase (Sod2) are associated wit
268 In this study, we identified mitochondrial manganese superoxide dismutase (SOD2) as one of the key
270 cytes deficient in the mitochondrial form of manganese superoxide dismutase (SOD2) do not survive in
271 interaction between plasma selenium and the manganese superoxide dismutase (SOD2) gene and incident
273 f mitochondrial derived H2O2, as a result of manganese superoxide dismutase (Sod2) overexpression, le
274 a mechanism whereby Saccharomyces cerevisiae manganese superoxide dismutase (SOD2) preferentially bin
275 transgenic mice with a partial depletion of manganese superoxide dismutase (SOD2) would affect the d
276 copper, zinc superoxide dismutase (SOD1), or manganese superoxide dismutase (SOD2), and against marke
278 ic increase in the ROS detoxification enzyme manganese superoxide dismutase (Sod2, also known as MnSo
281 of 27- and 70-kD stress proteins as well as manganese superoxide dismutase, suggesting that TNF-alph
282 he nigrostriatal system by overexpression of manganese superoxide dismutase supports a role for mitoc
283 amino acid sequencing techniques identified manganese superoxide dismutase, the major antioxidant en
284 pression of prosurvival molecules, including manganese superoxide dismutase, thioredoxin-1, and Bcl-x
287 quence, TGACGTCT, which we identified as the manganese superoxide dismutase TPA-responsive element, M
288 e anti-apoptotic genes for Bcl-2, Bcl-x, and manganese superoxide dismutase was observed in PEDF-trea
289 In particular, the SOD2 promoter, encoding manganese superoxide dismutase, was activated less stron
290 Y193) of the nine total tyrosine residues in manganese superoxide dismutase were nitrated by peroxyni
291 oxidation was demonstrated by overexpressing manganese superoxide dismutase, which improved HRas palm
292 we examined whether overexpression of SOD2, manganese superoxide dismutase, would prevent the hypoxi
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