ライフサイエンスコーパス: mansoni

1 ity (IC(5)(0): 0.3 muM) on adult Schistosoma mansoni .

2 granulomas in mice infected with Schistosoma mansoni.

3 the digenetic trematode parasite Schistosoma mansoni.

4 last-like cells in the trematode Schistosoma mansoni.

5 tions, as revealed by genetic analyses of S. mansoni.

6 ng in response to challenge with Schistosoma mansoni.

7 ff, were naturally infected with Schistosoma mansoni.

8 fibrosis in mice following infection with S mansoni.

9 ely by the eggs of the trematode Schistosoma mansoni.

10 ction with the helminth parasite Schistosoma mansoni.

11 om liver fibrosis following infection with S mansoni.

12 this species as a suitable snail host for S. mansoni.

13 fter infection with the helminth parasite S. mansoni.

14 tions with the helminth parasite Schistosoma mansoni.

15 e against the parasitic helminth Schistosoma mansoni.

16 blood-feeding trematode parasite Schistosoma mansoni.

17 iate Th1 response to the parasite Shistosoma mansoni.

18 ed eIF4E from the human parasite Schistosoma mansoni.

19 mission of the human blood fluke Schistosoma mansoni.

20 te host of the human blood fluke Schistosoma mansoni.

21 Ixodes ricinus, and the flatworm Schistosoma mansoni.

22 omoides polygyrus prior to infection with S. mansoni.

23 polygyrus, Trichuris muris, and Schistosoma mansoni.

24 mice infected with the trematode Schistosoma mansoni.

25 en these mice were infected with Schistosoma mansoni.

26 in the Platyhelminth trematode, Schistosoma mansoni.

27 ity against pathogenic trematode Schistosoma mansoni.

28 in Batf3(-/-) mice infected with Schistosoma mansoni.

29 pression or after infection with Schistosoma mansoni.

30 by the cercarial larvae stage of Schistosoma mansoni.

31 (SULT) in the parasitic flatworm Schistosoma mansoni.

32 TC, TT (P = 0.03; OR = 11]) infected with S. mansoni.

33 ere infected percutaneously with Schistosoma mansoni.

34 th muscles of the human parasite Schistosoma mansoni.

35 45%), Mansonella perstans (21%), Schistosoma mansoni (18%), and Plasmodium falciparum (11%).

36 f information obtained through RNA-Seq in S. mansoni (88 libraries).

37 dontis, a filarial nematode, and Schistosoma mansoni, a blood fluke.

38 Schistosoma mansoni, a causative agent of schistosomiasis, resides i

39 bits male-specific expression in Schistosoma mansoni, a derived, dioecious flatworm.

40 The micro-exon genes (MEG) of Schistosoma mansoni, a parasite responsible for the second most wide

41 ction with the helminth parasite Schistosoma mansoni, Ab regulates hepatic inflammation, and local pr

42 aused by the parasitic trematode Schistosoma mansoni after deposition of eggs in the liver and intest

43 A-DRB1) and posttreatment Ig responses to S. mansoni Ags were assessed in 199 individuals aged 7-50 y

44 the posttreatment Th2 immune response to S. mansoni Ags, as well as a downstream association with lo

45 Th2-mediated immune response to Schistosoma mansoni Ags.

46 n (VSVG) for the transduction of Schistosoma mansoni and delivery of reporter transgenes into schisto

47 results show that the genomes of Schistosoma mansoni and Drosophila melanogaster lack detectable DNA

48 arasitological examination for diagnosing S. mansoni and flow cytometry for lymphocyte (CD3, CD4, CD8

49 n schoolchildren coinfected with Schistosoma mansoni and hookworm.

50 f schoolchildren coinfected with Schistosoma mansoni and hookworm.

51 e results may have implications for human S. mansoni and L. donovani co-infections and also demonstra

52 model in C57BL/6 mice involving Schistosoma mansoni and Leishmania donovani, two important human pat

53 s mortality after infection with Schistosoma mansoni and much more susceptibility to Nippostrongylus

54 with the Th2-inducing pathogens Schistosoma mansoni and Nippostrongylus brasiliensis and examined th

55 ected with the helminth parasite Schistosoma mansoni and observed an upregulation of CD14 expression

56 domain from Sm-TSP-2, a TSP from Schistosoma mansoni and one of the better prospects for the developm

57 hey could be applied for the detection of S. mansoni and other parasites in settings without reliable

58 re there is transmission of both Schistosoma mansoni and Plasmodium falciparum.

59 After adjusting for Schistosoma mansoni and Plasmodium infection, we estimated a signifi

60 se-causing agents, Schistosoma japonicum, S. mansoni and S. haematobium, are blood flukes that have c

61 of oxamniquine derivatives that kill both S. mansoni and S. haematobium, the two species responsible

62 sive searching of the genomes of Schistosoma mansoni and S. japonicum.

63 scence modeling reveals the speciation of S. mansoni and S. rodhaini as 107.5-147.6KYA, a period whic

64 fluence both the distribution of Schistosoma mansoni and Schistosoma haematobium and the incidence of

65 and mature earlier compared with Schistosoma mansoni and Schistosoma haematobium.

66 as a vaccine candidate for both Schistosoma mansoni and Schistosoma japonicum.

67 Collectively, S. mansoni and several other schistosomes are responsible f

68 Schistosoma mansoni (and rarely Schistosoma haematobium) intestinal

69 luble egg Ag (SEA) obtained from Schistosoma mansoni, and by RA.

70 vatives was tested against adult Schistosoma mansoni, and values in the micromolar range (26-68 muM)

71 The differences between Schistosoma mansoni- and Schistosoma japonicum-induced hepatic granu

72 anded RNA interference (RNAi) in Schistosoma mansoni, appraises delivery systems for transgenesis and

73 trate that somatic stem cells in Schistosoma mansoni are biased towards generating a population of ce

74 Hepatitis C virus (HCV) and Schistosoma mansoni are major causes of chronic liver disease (CLD)

75 flukes Schistosoma japonicum and Schistosoma mansoni are the first major human platyhelminth pathogen

76 arasitic helminth worms, such as Schistosoma mansoni, are endemic in regions with a high prevalence o

77 with helminth parasites, such as Schistosoma mansoni, are often chronic and characterized by the deve

78 n, using the parasitic helminth, Schistosoma mansoni as an experimental model.

79 These results indicate that chronic S. mansoni attenuates the severity of P. knowlesi coinfecti

80 rst application of population genomics to S. mansoni based on high-coverage resequencing data from 10

81 ta subunit of the human parasite Schistosoma mansoni (beta(Sm)), a motif that does not occur in other

82 va during natural infection with Schistosoma mansoni, but the role of TGF-beta is less clear.

83 s can be made resistant to infection with S. mansoni by first inducing hemocyte proliferation with Bg

84 Sm-p80, the large subunit of Schistosoma mansoni calpain, is a leading antigen candidate for a sc

85 We sought to determine whether Schistosoma mansoni causes experimental PH associated with pulmonary

86 tion with the parasitic helminth Schistosoma mansoni causes significant liver fibrosis and extracellu

87 Schistosoma mansoni cercariae display specific behavioral responses

88 y/secretory products released by Schistosoma mansoni cercariae rapidly produce IL-10 as a result of M

89 ifiable occupational exposure to Schistosoma mansoni cercariae revealed that some individuals develop

90 f mice to repeated doses (4x) of Schistosoma mansoni cercariae, compared to a single dose (1x), resul

91 6J mice were percutaneously infected with S. mansoni cercariae, followed by i.v. injection of eggs.

92 Eight rhesus macaques were exposed to S. mansoni cercariae.

93 , TSLPR(-/-) mice were also infected with S. mansoni cercariae.

94 s; eight animals were exposed to Schistosoma mansoni cercariae.

95 p (n = 3) were infected with 500 Schistosoma mansoni cercariae.

96 ce IL-10 in vivo early postinfection with S. mansoni cercariae.

97 el of Mtb infection, we demonstrated that S. mansoni coinfection or immunization with S. mansoni egg

98 Thus, S. mansoni coinfection significantly increased viral replic

99 ve previously been observed with Schistosoma mansoni coinfection.

100 hammerhead ribozyme derived from Schistosoma mansoni containing the rate-enhancing peripheral domain

101 e whether children infected with Schistosoma mansoni develop protection-related immune responses afte

102 mice infected with the helminth Schistosoma mansoni develop severe CD4 T cell-mediated hepatic granu

103 mice infected with the helminth Schistosoma mansoni develop small hepatic granulomas around parasite

104 ough a recent treatment trial in Schistosoma mansoni did not detect this association.

105 In contrast to other helminth infections, S. mansoni did not elicit a Foxp3(+) Treg cell response dur

106 y product of eggs from the parasitic worm S. mansoni, efficiently triggers basophils to release the i

107 n-derived Ag (soluble extract of Schistosoma mansoni egg [SEA]) that induces type 2 immunity.

108 ated Schistosoma mansoni eggs followed by S. mansoni egg Ag challenge directly in the airways and Th1

109 response to the helminth soluble Schistosoma mansoni egg Ag, which conditions DCs to induce Th2 respo

110 Last, S. mansoni egg Ag-pulsed NF-kappaB1(-/-) DC failed to prime

111 purified protein derivative and Schistosoma mansoni egg antigen challenge indicating an unbiased eff

112 purified protein derivative- or Schistosoma mansoni egg antigen-coated beads.

113 mansoni coinfection or immunization with S. mansoni egg antigens can reversibly impair Mtb-specific

114 om postseptic mice at days 8 and 16 after S. mansoni egg challenge exhibited defective IL-12 synthesi

115 We collected up to 12 S. mansoni egg counts from 414 children aged 6-12 years bef

116 nti-leishmanial immunity fails within the S. mansoni egg granuloma, consistent with a lack of L. dono

117 Persisting amastigote replication in the S. mansoni egg granulomas may thus explain the increased L.

118 significantly associated with increasing S. mansoni egg IgG1 titers and RF titers of >or=80 (adjuste

119 irmed by in vivo studies using a Schistosoma mansoni egg-challenged mouse model, a well-studied syste

120 ith recombinant helminth-derived Schistosoma mansoni egg-derived omega1 (omega1), a potent inducer of

121 rticipates in the development of Schistosoma mansoni egg-induced CD4(+) Th2 responses, it plays only

122 basophils for protective immunity against S. mansoni egg-induced pathology during the patent stage of

123 The IL-4-inducing principle from Schistosoma mansoni eggs (IPSE/alpha-1), the major secretory product

124 CFA, aluminum salts (Alum), and Schistosoma mansoni eggs (Sm Egg).

125 ribonuclease (RNase) secreted by Schistosoma mansoni eggs and abundantly present in soluble egg antig

126 Schistosoma mansoni eggs contain factors that trigger potent Th2 res

127 and subsequently challenged with Schistosoma mansoni eggs developed pulmonary hypertension associated

128 gainst Trichuris muris worms and Schistosoma mansoni eggs do not develop in mice with IRF-4-deficient

129 were sensitized with inactivated Schistosoma mansoni eggs followed by S. mansoni egg Ag challenge dir

130 Pulmonary granulomas induced by S. mansoni eggs in cecal ligation and puncture survivors we

131 The immune response to S. mansoni eggs is characterized by increased Th2 cells, eo

132 S. mansoni eggs lodge in the hepatic sinusoids of infected

133 f the variation in the number of Schistosoma mansoni eggs per gram of fecal matter.

134 sitized and intravenously challenged with S. mansoni eggs to induce experimental PH.

135 ens (SEA; a soluble extract from Schistosoma mansoni eggs) inhibit the activation of DCs in response

136 Following exposure to Schistosoma mansoni eggs, a model of Th2 cytokine-mediated disease t

137 re M-IPSE), a major protein secreted from S. mansoni eggs, can infiltrate host cells.

138 uced in the liver and lung in response to S. mansoni eggs, confirmed by both DNA microarray and real-

139 sure to bleomycin (BLM), but not Schistosoma mansoni eggs, IL-17A produced by CD4(+) and gammadelta(+

140 After intravenous injection of Schistosoma mansoni eggs, IL-31Ralpha(-/-) mice developed severe pul

141 production when challenged with Schistosoma mansoni eggs.

142 ged with lung granuloma-inducing Schistosoma mansoni eggs.

143 People in regions of Schistosoma mansoni endemicity slowly acquire immunity, but why this

144 Children (8-10 years old) were tested for S. mansoni every 4 months and treated with praziquantel whe

145 ident tissue macrophages, which encounter S. mansoni excretory/secretory products during infection, a

146 The human pathogen Schistosoma mansoni exhibits a highly evolved and intricate relation

147 ning differential gene expression between S. mansoni-exposed schistosome-resistant and susceptible sn

148 Schistosoma mansoni exposure results in prototypical type-2 inflamma

149 examining children infected with Schistosoma mansoni from 6 schools in Uganda that had previously rec

150 cent release of version 3 of the Schistosoma mansoni genome assembly has made a wealth of information

151 d structural annotation of lncRNAs in the S. mansoni genome.

152 evidence for cytosine methylation in the S. mansoni genome.

153 sis, coupled to inspection of the current S. mansoni genomic assembly, revealed that many of the SmVA

154 l host for the human blood fluke Schistosoma mansoni Granulins are growth factors that drive prolifer

155 oup-I: patients with chronic schistosomiasis mansoni, group-II: HCV patients without cirrhosis, group

156 Vitamin A-deficient mice infected with S. mansoni had disrupted liver granuloma architecture and i

157 ystal structure of a full-length Schistosoma mansoni hammerhead ribozyme that permits us to explain t

158 o systematically explore this hypothesis, S. mansoni hemozoin was purified and added to in vitro bone

159 VSVG-pseudotyped MLV for transgenesis of S. mansoni, herald a tractable pathway forward toward germl

160 nity to the intestinal trematode Schistosoma mansoni Here, we report that abrogation of IL-4 receptor

161 prepared as potent inhibitors of Schistosoma mansoni histone deacetylase 8 (smHDAC8).

162 xplore selected protein-encoding genes of S. mansoni implicated in the disease process.

163 ome IgE are associated with resistance to S. mansoni in children, and these immunological parameters

164 ociated with resistance to reinfection by S. mansoni in HIV-1-seronegative persons.

165 ina, are the major intermediate hosts for S. mansoni in sub-Saharan Africa, where more than 90% of gl

166 dysmic ratios of 1 and 2 against Schistosoma mansoni in vitro.

167 lyze the reproductive biology of Schistosoma mansoni in-depth we isolated complete ovaries and testes

168 volved in the human blood fluke (Schistosoma mansoni) in Brazil in the 1970s.

169 tes activation markers in chronic HCV and S. mansoni induced CLD that may have a role in disease prog

170 -4Ralpha signaling on B cells exacerbated S. mansoni-induced mortality and pathology in BALB/c mice,

171 Experimental S mansoni-induced pulmonary vascular disease relies on can

172 ammals by the parasitic helminth Schistosoma mansoni induces antibodies to glycan antigens in worms a

173 S. mansoni-infected Arg I-deficient bone marrow chimeras de

174 S. mansoni-infected Cd14(-/-) mice also presented with smal

175 blood, spleen, and hepatic granulomas of S. mansoni-infected high-pathology CBA mice and low-patholo

176 er fibrosis were significantly reduced in S. mansoni-infected IL-21R-/- mice and in IL-21R+/+ mice tr

177 essary and sufficient to prevent Schistosoma mansoni-infected mice from developing severe inflammatio

178 d eosinophils from the livers of Schistosoma mansoni-infected mice.

179 n was analyzed in offspring from Schistosoma mansoni-infected mothers mated during the TH1, TH2, or r

180 Previously, in a group of S. mansoni-infected Ugandan males, we showed that IgE respo

181 uracy of 3 different diagnostic tests for S. mansoni infection (stool microscopy [samples prepared by

182 t inflammation is controlled during acute S. mansoni infection by two distinct, organ-specific mechan

183 Praziquantel treatment for Schistosoma mansoni infection enhances Th2 responsiveness against pa

184 S. mansoni infection had no consistent association with chi

185 Children with > or =2 repeat diagnoses of S. mansoni infection had significantly increased levels of

186 There was no evidence for S. mansoni infection having a similar suppressive effect on

187 ponses during the first 3 weeks of murine S. mansoni infection in C57BL/6 mice, a time when larval pa

188 nsitive screening option for asymptomatic S. mansoni infection in Eritrean refugees, compared with st

189 gical characteristics, caused by Schistosoma mansoni infection in IL-4 receptor alpha-deficient mice

190 he levels were negatively correlated with S. mansoni infection intensities and were lower among child

191 parasites using the established Schistosoma mansoni infection model in 2 novel mouse models of eosin

192 on traditional measures of disease in the S mansoni infection model in mice.

193 pot, not associated with study arm, where S. mansoni infection prevalence and intensity did not decre

194 Instead, S. mansoni infection resulted in accumulation of high argin

195 over three consecutive days for Schistosoma mansoni infection simultaneously by age group at baselin

196 We used a murine model of Schistosoma mansoni infection to further investigate whether the per

197 ubstantial heritability for the burden of S. mansoni infection was confirmed in these Brazilian famil

198 otal of 81% of baboons exposed to chronic S. mansoni infection with or without praziquantel treatment

199 rch as well as CCA for mapping surveys of S. mansoni infection, although additional diagnostic tools

200 During the patent phase of Schistosoma mansoni infection, Foxp3(+) Treg cells are activated and

201 paired Th2 cell responses during Schistosoma mansoni infection, Schistosoma egg antigen (SEA) immuniz

202 (AST) increased in all mice in response to S mansoni infection, with no eosinophil-dependent differen

203 then studied in mice harboring a chronic S. mansoni infection.

204 single-day double KK for the diagnosis of S. mansoni infection.

205 ty and mortality of the TKO mice following S mansoni infection.

206 children with > or =2 repeat diagnoses of S. mansoni infection.

207 igrants from endemic regions for Schistosoma mansoni infection.

208 develops in response to chronic Schistosoma mansoni infection.

209 We show that mice with established S. mansoni infections fail to control L. donovani growth in

210 As with Schistosoma mansoni infections, the pathology of urogenital schistos

211 concomitant chronic HCV and schistosomiasis mansoni infections.

212 The larvae of Schistosoma mansoni invade their mammalian host by utilizing a serin

213 The intravascular trematode Schistosoma mansoni is a causative agent of schistosomiasis, a disea

214 Schistosoma mansoni is a parasitic fluke that infects millions of pe

215 The trematode Schistosoma mansoni is one of the etiological agents of schistosomia

216 Schistosoma mansoni is responsible for the neglected tropical diseas

217 hree main human schistosome species, only S. mansoni is sensitive to oxamniquine therapy despite the

218 The hammerhead ribozyme from Schistosoma mansoni is the best characterized of the natural hammerh

219 The blood fluke Schistosoma mansoni is the causative agent of the intestinal form of

220 prominent role in regulating immunity to S. mansoni larvae and that the character of the initial imm

221 Induction of inflammation by S mansoni, liver fibrosis, and mortality increased greatly

222 In addition, we find strong evidence that S. mansoni migrated to the New World with the 16-19th Centu

223 al regions that are commonly the sites of S. mansoni miracidium penetration.

224 that the transition from the free-living S. mansoni miracidium to parasitic mother sporocyst depends

225 ctivity in the juvenile (WBR: 18.9-43.1%) S. mansoni mouse model.

226 ine (DA) are reduced during the course of S. mansoni multiplication and transformation within the sna

227 S. mansoni, N. americanus, S. stercoralis, T. trichiura, M.

228 eening approach to identify inhibitors of S. mansoni NAD(+) catabolizing enzyme (SmNACE), a receptor

229 arative genomic analyses demonstrate that S. mansoni NRs share an evolutionary lineage with that of a

230 analysis shows that more than half of the S. mansoni nuclear receptors evolved from a second gene dup

231 re very similar to those of its congener, S. mansoni, offering the prospect of designing chemicals th

232 sed on this evidence, we hypothesize that S. mansoni omega-1 acts by limiting the interaction of DCs

233 elpful tool for understanding the role of S. mansoni on malaria parasitemia and antimalarial immune r

234 s an intermediate snail host for Schistosoma mansoni, one of the important schistosomes infecting man

235 of B. glabrata to infection with Schistosoma mansoni or Echinostoma paraensei, and functions as an op

236 ther hepatosplenomegaly or infection with S. mansoni or P. falciparum.

237 th P. falciparum that are coinfected with S. mansoni or S. haematobium.

238 ts from a chronic infection with Schistosoma mansoni or Schistosoma japonicum.

239 n DCs responding to the helminth Schistosoma mansoni or the allergen house dust mite (HDM).

240 Our results indicate that S. mansoni originated in East Africa and experienced a decl

241 determined the responsiveness to Schistosoma mansoni over a 2-year period, when reinfection was restr

242 ium-Plasmodium falciparum versus Schistosoma mansoni-P. falciparum) has produced conflicting results.

243 In schistosomiasis mansoni, parasite eggs cause hepatointestinal granulomat

244 of the initial immune response invoked by S. mansoni parasites contrasts with the responses to other

245 Schistosoma mansoni parasites of both sexes recovered from infected

246 er transgenes into the genome of Schistosoma mansoni parasites.

247 was significantly decreased in HCV and/or S. mansoni patients.

248 ress SMDR2, a Pgp homologue from Schistosoma mansoni (Platyhelminthes), in Chinese hamster ovary (CHO

249 Here, we characterize two Schistosoma mansoni products, tyrosinase 1 and tyrosinase 2 (SmTYR1/

250 B. glabrata/S. mansoni provides a useful model system for investigating

251 igens were associated with lower Schistosoma mansoni reinfection intensity, while no associations bet

252 ombinant antigen vaccine against Schistosoma mansoni remains elusive, in part because the parasite de

253 he case of the human blood fluke Schistosoma mansoni, responsible for intestinal bilharzia, the pheno

254 tion with the trematode parasite Schistosoma mansoni results in a distinct heterogeneity of disease s

255 tion with the trematode helminth Schistosoma mansoni results in a parasite egg-induced, CD4 T-cell-me

256 In the mouse, infection with Schistosoma mansoni results in an egg-producing infection and associ

257 he eggs of the helminth parasite Schistosoma mansoni (schistosome egg Ag (SEA)) leads to the inductio

258 In murine schistosomiasis mansoni, schistosome worms produce ova that incite focal

259 ctures of the GTPase domain of a Schistosoma mansoni septin (SmSEPT10), one bound to GDP and the othe

260 SmSrpQ, one of two S. mansoni serpins found in larval secretions, is only expr

261 After challenge with Schistosoma mansoni (Sm) eggs, Retnla(-/-) mice developed exacerbate

262 (legumains) from the bloodfluke, Schistosoma mansoni (SmAE), and the hard tick, Ixodes ricinus (IrAE)

263 mine kinase from the blood fluke Schistosoma mansoni (SmTK) belongs to the phosphagen kinase (PK) fam

264 regation mediated by HIVgp120 or Schistosoma mansoni soluble egg Ag accelerated maximal CXCR4 express

265 whole-blood cultures were stimulated with S. mansoni soluble egg antigen (SEA) or soluble worm antige

266 tive chemotherapy strategies for Schistosoma mansoni, spatial scan statistics were used to find infec

267 rom the intestine, is sufficient to prime S. mansoni-specific Th2 responses.

268 RF-4-deficient DCs also effectively prime S. mansoni-specific Th2 responses.

269 Despite enhanced posttreatment S. mansoni-specific type 2 responses, no increase in eosino

270 ly binds to hemocytes and the tegument of S. mansoni sporocysts in a sugar-inhibitable fashion sugges

271 fied Schistosoma haematobium and Schistosoma mansoni surveys done in, respectively, 9318 and 9140 uni

272 as been hampered by the lack of validated S. mansoni targets.

273 nd SmCalp2) are expressed in the Schistosoma mansoni tegument.

274 uding the allergen-like proteins Schistosoma mansoni tegumental-allergen-like 1 protein (SmTAL1), SmT

275 The digenetic trematode Schistosoma mansoni that causes the form of schistosomiasis found in

276 er directional and balancing selection in S. mansoni that may facilitate adaptation to the human host

277 We have targeted a protein of Schistosoma mansoni that plays an important role in the surface memb

278 ealth problem and infection with Schistosoma mansoni the major cause of liver disease.

279 sessed the lncRNAs complement of Schistosoma mansoni, the blood fluke that causes schistosomiasis, ra

280 tion with the trematode helminth Schistosoma mansoni, the severity of CD4 T cell-mediated hepatic gra

281 -based virtual screening (VS) of Schistosoma mansoni thioredoxin glutathione reductase (SmTGR) inhibi

282 infected with the human parasite Schistosoma mansoni through mechanisms that are currently unclear.

283 ings the total number of identified NR in S. mansoni to 21.

284 7 Ugandans (aged 7-50) in a high Schistosoma mansoni transmission area, both before and 7 wk posttrea

285 The site of S. mansoni transmission was molluscicided throughout.

286 illages with different levels of Schistosoma mansoni transmission.

287 Using data from a study of Schistosoma mansoni (trematode) infections in Biomphalaria glabrata

288 observed between infection with Schistosoma mansoni, Trichuris, or Strongyloides species and P. falc

289 hammerhead ribozyme derived from Schistosoma mansoni under conditions that permit detailed observatio

290 lar to other metazoan pathogens, Schistosoma mansoni undergoes transcriptional and developmental regu

291 In vitro studies with adult Schistosoma mansoni using several substrates suggest that the excret

292 tion of an SCP/TAPS gene family (Schistosoma mansoni venom allergen-like (SmVALs)) in the medically i

293 of the hammerhead ribozyme from Schistosoma mansoni was monitored with double electron-electron reso

294 S. mansoni was positively associated with Dermatophagoides-

295 ptible snails, 2 to 24 h post-exposure to S. mansoni, was hybridized to the custom made cDNA microarr

296 vely recent empirical studies on Schistosoma mansoni, we use a mathematical model to investigate the

297 died extensively for the related organism S. mansoni, which is more amenable to laboratory studies.

298 in sera from mice infected with Schistosoma mansoni, which revealed the presence of both IgM and IgG

299 Emergence of Schistosoma mansoni with reduced sensitivity to praziquantel, the dr

300 assay false positivity and the levels of S. mansoni worm IgG1 and IgG2 and Plasmodium falciparum IgG