ライフサイエンスコーパス: marcescens

1 example is serratin, isolated from Serratia marcescens.

2 by a Gram-negative entomopathogen, Serratia marcescens.

3 e positive for P. aeruginosa also yielded S. marcescens.

4 undii group, Enterobacter spp., and Serratia marcescens.

5 ors from Pseudomonas aeruginosa and Serratia marcescens.

6 anism for the second step of secretion in S. marcescens.

7 the processive chitinase ChiA from Serratia marcescens.

8 gy-dependent and Sec-dependent pathway in S. marcescens.

9 y any environmental or staff reservoir of S. marcescens.

10 ecorina and the chitinase ChiA from Serratia marcescens.

11 uce SlpB as a new cytotoxic protease from S. marcescens.

12 onized with provisionally matching GBS or S. marcescens.

13 tance test using the live bacterium Serratia marcescens.

14 , including Clostridium species and Serratia marcescens.

15 m the opportunistic human pathogen, Serratia marcescens.

16 insect survival after oral infection with S. marcescens.

17 rocessive endochitinase, ChiC, from Serratia marcescens.

18 a TagJ homologue as shown here with Serratia marcescens.

19 ia burhodogranaria, is gram negative like S. marcescens.

20 fection with the bacterial pathogen Serratia marcescens.

21 rance of a bacterial infection with Serratia marcescens, 3 Acps significantly reduced the bacterial c

22 (9.1%), Acinetobacter spp. (6.2%), Serratia marcescens (5.5%), Enterobacter aerogenes (4.4%), Stenot

23 th Acinetobacter spp., P. aeruginosa, and S. marcescens, 5/6 with Citrobacter spp., 13/14 with Entero

24 Finally, recombinant expression of the S. marcescens 56-kDa metalloprotease conferred a cytotoxic

25 Secretomes from 95% of Serratia marcescens, 71% of Pseudomonas aeruginosa, 29% of Staphy

26 Serratia marcescens, a member of the carbapenem-resistant Enterob

27 Serratia marcescens, a member of the Enterobacteriaceae family, i

28 of anthranilate synthase (AS) from Serratia marcescens, a mesophilic bacterium, has been solved in t

29 lity of the tigecycline Etest for testing S. marcescens, Acinetobacter spp., and S. pneumoniae is war

30 one acetyltransferase 1) and SmAAT (Serratia marcescens aminoglycoside 3-N-acetyltransferase), sugges

31 related N-acetyltransferase (GNAT), Serratia marcescens aminoglycoside 3-N-acetyltransferase, bound t

32 ast histone acetyltransferase 1 and Serratia marcescens aminoglycoside 3-N-acetyltransferase.

33 ive means to control the dissemination of S. marcescens, an in-depth analysis of the population struc

34 (R)-(-)-mellein and micromolide, against S. marcescens and a Gram-positive bacterium, Staphylococcus

35 An outbreak of S. marcescens and E. cloacae bacteremia in a surgical inten

36 infusions from two case patients yielded S. marcescens and E. cloacae.

37 s of the cheA loci from isolates of Serratia marcescens and Enterobacter cloacae, demonstrating the p

38 tate transcarbamoylases (ATCase) of Serratia marcescens and Escherichia coli differ in both regulator

39 tate transcarbamoylases (ATCase) of Serratia marcescens and Escherichia coli have distinct allosteric

40 o the natural product isolated from Serratia marcescens and from overexpression of the biosynthetic g

41 hit operons previously reported for Serratia marcescens and Haemophilus influenzae, respectively, and

42 of TLM on the ecFabB homologues in Serratia marcescens and Klebsiella pneumonia is an important fact

43 n of bronchoscopes with P. aeruginosa and S. marcescens and possible infection of patients at a commu

44 late in midgut cells in response to Serratia marcescens and Sindbis virus or when the native microbio

45 noted among Acinetobacter spp. and Serratia marcescens and, to a lesser extent, with Streptococcus p

46 s spp., Pseudomonas aeruginosa, and Serratia marcescens) and 6 antimicrobial resistance determinants

47 R-flagellins from Serratia marcescens (S. marcescens) and Salmonella muenchen (S. muenchen) do not

48 xidase activity, resistance against Serratia marcescens), and for the life history traits, age and si

49 homologues, a homologue of OmpF of Serratia marcescens, and a locus (designated rscBAC) with similar

50 eradication of Pseudomonas species, Serratia marcescens, and Enterobacter aerogenes in most of the tr

51 freundii, Yersinia enterocolitica, Serratia marcescens, and Morganella morganii) and two nonenteric

52 a gonorrhoeae and N. meningitidis), Serratia marcescens, and other gram-negative bacteria utilize a p

53 egative bacteria (Escherichia coli, Serratia marcescens, and Pseudomonas aeruginosa).

54 faciens, Agrobacterium radiobacter, Serratia marcescens, and Pseudomonas aureofaciens) and fungi (Phy

55 ent was observed, excluding A. baumannii, S. marcescens, and S. pneumoniae, for which >/=4-fold diffe

56 r to biotin synthases from E. coli, Serratia marcescens, and Saccharomyces cerevisiae (about 50% sequ

57 holderia cepacia, Escherichia coli, Serratia marcescens, and Stenotrophomonas maltophilia isolates.

58 rst reports applying PFGE to the study of S. marcescens, and this method was a useful marker of strai

59 Using Serratia marcescens as a model organism, we identify here a stage

60 ify a common fecal enterobacterium, Serratia marcescens, as the causal agent of white pox.

61 tic collection of antimicrobial-resistant S. marcescens associated with bloodstream infections in hos

62 stimulates the catalytic activity of the S. marcescens ATCase and CTP/UTP inhibitory synergism has b

63 s, the temperature response of the native S. marcescens ATCase suggests a strong entropic effect that

64 A case was defined as the occurrence of S. marcescens bacteremia in any patient in the surgical int

65 ing to univariate analysis, patients with S. marcescens bacteremia stayed in the surgical intensive c

66 Twenty-six patients with S. marcescens bacteremia were identified; eight (31 percent

67 y risk factors, we compared patients with S. marcescens bacteremia with randomly selected controls.

68 ve care unit of a hospital acquired Serratia marcescens bacteremia.

69 roswimmer system driven by multiple Serratia marcescens bacteria, we quantify the chemotactic drift o

70 ogenes, K. pneumoniae, P. aeruginosa, and S. marcescens) became more susceptible.

71 which OxyR contributes to early stages of S. marcescens biofilm formation by influencing fimbrial gen

72 In March 2011, Serratia marcescens bloodstream infections (BSIs) were identified

73 Cases were defined as S. marcescens BSIs in patients receiving PN from the pharma

74 g standards contributed to an outbreak of S. marcescens BSIs.

75 ter they fed on the insect pathogen Serratia marcescens but not after feeding on the Leishmania that

76 Purified LPS from S. marcescens, but not from Escherichia coli or S. marcesce

77 observed with another CGD pathogen, Serratia marcescens, but not with Escherichia coli.

78 l antibiotic resistance enzyme from Serratia marcescens catalyzes adenylation and acetylation of amin

79 acterium and opportunistic pathogen Serratia marcescens causes ocular infections in healthy individua

80 It has been reported that a S. marcescens chimera, SM : rS5'ec, in which five divergent

81 ha + beta domain similar to that of Serratia marcescens chitinases A and B.

82 asured differences in processivity of the S. marcescens chitinases.

83 of E. coli have been replaced with their S. marcescens counterpart, lost both heterotrophic and homo

84 Serratia marcescens culture filtrates have been reported to be cy

85 treatments abrogated the cytotoxicity of S. marcescens culture filtrates towards HeLa cells, suggest

86 o cytotoxic activity commonly observed in S. marcescens culture filtrates.

87 ve detection of Escherichia coli or Serratia marcescens cultures from 1 to 10(3) CFU mL(-1).

88 The hemophore protein HasA from Serratia marcescens cycles between two states as follows: the hem

89 Bioinformatic analysis of the S. marcescens Db11 genome revealed three additional open re

90 S. marcescens does not normally colonize human skin, but ar

91 and prodigiosin concentration changes for S. marcescens during cultivation in batch culture.

92 Fis are 100% identical in K. pneumoniae, S. marcescens, E. coli, and S. typhimurium and 96 to 98% id

93 One crystal form of the S. marcescens enzyme displays a bound pyruvate as well as a

94 r97) of the regulatory polypeptide of the S. marcescens enzyme have been replaced with their E. coli

95 etained 455 out of 460 amino acids of the S. marcescens enzyme, it possessed characteristics similar

96 ric bacteria Klebsiella pneumoniae, Serratia marcescens, Erwinia carotovora, and Proteus vulgaris but

97 terobacteria Klebsiella pneumoniae, Serratia marcescens, Erwinia carotovora, and Proteus vulgaris, st

98 thms, especially in differentiating Serratia marcescens, Escherichia coli, and Yersinia enterocolitic

99 S. marcescens expresses prodigiosin, a bright red and cell-

100 The Serratia marcescens extracellular nuclease gene, nucA, is positiv

101 amily of mutants overexpressing the Serratia marcescens extracellular nuclease has been known for dec

102 The Serratia marcescens extracellular nuclease is a secreted protein

103 reted by the Gram-negative bacteria Serratia marcescens, extracts heme from host hemoproteins and shu

104 -), and TLR4/5(-/-) corneas infected with S. marcescens had significantly increased CFU, indicating i

105 Hemophores from Serratia marcescens (HasA(sm)) and Pseudomonas aeruginosa (HasA(p

106 ven of these had Enterobacter cloacae and S. marcescens in the same culture.

107 pairs of the trpEDCBA operator from Serratia marcescens indicated an obligate hierarchy of site occup

108 Tobramycin-killed S. marcescens induced corneal inflammation in C57BL/6 mice,

109 S. marcescens induced neutrophil recruitment to the corneal

110 interleukin-1 receptor type 1 (IL-1R1) in S. marcescens-induced corneal inflammation and infection.

111 ial therapeutic targets for inhibition of S. marcescens-induced corneal inflammation.

112 S. marcescens induces corneal inflammation by activation of

113 Several risk factors for S. marcescens infection were identified, but hospital and h

114 reaks of Pseudomonas aeruginosa and Serratia marcescens infections associated with bronchoscopy have

115 ant clones suggests that the treatment of S. marcescens infections will become increasingly difficult

116 nia hospital acquired postoperative Serratia marcescens infections, and 1 died.

117 Our results show that S. marcescens is a diverse species with a high level of gen

118 Chitinase B (ChiB) from Serratia marcescens is a family 18 exo-chitinase whose catalytic

119 Serratia marcescens is a gram-negative environmental bacterium an

120 Serratia marcescens is a soil- and water-derived bacterium that s

121 Serratia marcescens is a well-known cause of nosocomial infection

122 Serratia marcescens is an extremely rare cause of necrotizing fas

123 The enteric bacterium Serratia marcescens is an opportunistic human pathogen.

124 Serratia marcescens is an opportunistic pathogen associated with

125 Serratia marcescens is frequently isolated from lenses of patient

126 The extracellular nuclease of Serratia marcescens is one of a wide variety of enzymes secreted

127 A Serratia marcescens isolate was particularly efficient in coloniz

128 Pseudomonas aeruginosa isolates, 1 Serratia marcescens isolate, 1 Aeromonas hydrophila isolate, 1 Ae

129 Three bla(SME)-carrying Serratia marcescens isolates and one bla(NDM-1) carrying Providen

130 ted an investigation of P. aeruginosa and S. marcescens isolates related to bronchoscopy at a communi

131 S. marcescens isolates were compared using restriction-endo

132 lates, 5 Enterobacter cloacae isolates, 2 S. marcescens isolates, 1 Proteus mirabilis isolate, and 2

133 Together these data suggest that S. marcescens LPS is sufficient for inhibition of epithelia

134 Literature pertinent to S. marcescens-mediated necrotizing fasciitis is also review

135 alpha-thrombin by only 10% whereas Serratia marcescens metalloprotease reduced the Ca2+ response by

136 in vitro cytotoxic activity revealed that S. marcescens mutant strains that are deficient in producti

137 ation in the opportunistic pathogen Serratia marcescens, mutations in an oxyR homolog and predicted f

138 Despite multiple clinical descriptions of S. marcescens nosocomial pneumonia, little is known regardi

139 tracellular nuclease (Nuc) from the Serratia marcescens nucA chromosomal locus is inhibited in cells

140 The Serratia marcescens NucC protein is structurally and functionally

141 Extracellular secretion of Serratia marcescens nuclease occurs as a two-step process via a p

142 NucC binding to the S. marcescens nuclease promoter P(nucA) and to the sequence

143 . coli for extracellular secretion of the S. marcescens nuclease.

144 birth, but gut colonization with GBS and S. marcescens occurred closer to time of bloodstream infect

145 iming during infections with either Serratia marcescens or with Escherichia coli.

146 tained group B Streptococcus (GBS), Serratia marcescens, or Escherichia coli before their sepsis epis

147 t is observed in other bacterial species, S. marcescens OxyR is required for oxidative stress resista

148 S. marcescens oxyR mutants were severely impaired in biofil

149 3 of 15), and Enterobacter cloacae, Serratia marcescens, Pneumocystis carinii pneumonia, and unknown

150 aspiration model of lethal and sublethal S. marcescens pneumonia in BALB/c mice and extensively char

151 This study describes a model of S. marcescens pneumonia that mimics known clinical features

152 uginosa PAO1, Proteus mirabilis and Serratia marcescens, possibly by interfering with their QS system

153 located upstream of NucC-dependent Serratia marcescens promoters and the late promoters of P2-relate

154 hat is located upstream of NucC-dependent S. marcescens promoters and the late promoters of P2-relate

155 herichia coli, Salmonella muenchen, Serratia marcescens, Proteus mirabilis, and Proteus vulgaris).

156 re in complex with chitinase B from Serratia marcescens provide further insight into the mechanism of

157 volution with a bacterial pathogen (Serratia marcescens) resulted in significantly more outcrossing i

158 R-flagellins from Serratia marcescens (S. marcescens) and Salmonella muenchen (S. m

159 oreskin fibroblasts was also inhibited by S. marcescens secretomes indicating that the effect is not

160 LPS depletion of S. marcescens secretomes with polymyxin B agarose rendered

161 nella enterica serovar Typhimurium, Serratia marcescens, Shigella flexneri, Enterobacter aerogenes, K

162 , which, based on their homology to Serratia marcescens shlA and shlB genes, are believed to encode t

163 pneumoniae, Pseudomonas aeruginosa, Serratia marcescens, Staphylococcus aureus, and Stenotrophomonas

164 owth-promoting rhizobacteria (PGPR) Serattia marcescens strain 90-166 and Bacillus pumilus strain SE3

165 nous bacteria (Enterobacter sp. and Serratia marcescens strain Db11) and parasitic African trypanosom

166 binding protein, RsmA, in Ecc71 and Serratia marcescens strain SM274.

167 s transporter ShlB resulted in attenuated S. marcescens strains that failed to cause profound weight

168 cescens, but not from Escherichia coli or S. marcescens strains with mutations in the waaG and waaC g

169 chromosome of carbapenem-resistant Serratia marcescens strains.

170 rthermophile Sulfolobus solfataricus, the S. marcescens structure shows similar subunit structures bu

171 y ingesting the pathogenic bacteria Serratia marcescens, suggesting that subdued has novel functions

172 oteins are the original substrates of the S. marcescens T6SS, before horizontal acquisition of other

173 s of the chest wall due to infection with S. marcescens that initially manifested as bilateral breast

174 e set of D. melanogaster lines with Serratia marcescens, the bacterium used in the previous study, an

175 m on prodigiosin (PG) production by Serratia marcescens TKU011 is examined.

176 We adsorbed swarmer cells of Serratia marcescens to polydimethylsiloxane or polystyrene.

177 a and experimental Tn-Seq data from Serratia marcescens transposon mutant library used to identify ge

178 udomonas spp., Salmonella enterica, Serratia marcescens, Vibrio vulnificus and Enterobacter nimipress

179 ection with Burkholderia cepacia or Serratia marcescens was caused by a new strain in 9 of 10 cases (

180 The activity of the fraction against S. marcescens was explained by (R)-(-)-mellein alone, and t

181 S. marcescens was identified from a pharmacy water faucet,

182 knockout mice was abraded, and 1 x 10(7) S. marcescens were added in the presence of a silicone hydr

183 0 nM), and Enterobacter cloacae and Serratia marcescens were highly resistant (IC(50), >10,000 nM).

184 ases produced by P. fluorescens and Serratia marcescens, which comprise a second sequence family, be

185 e the structure of chitinase B from Serratia marcescens, which consists of a catalytic domain with a

186 sis except for Escherichia coli and Serratia marcescens, which could not be interdifferentiated using

187 solated the Gram-negative bacterium Serratia marcescens, which is a potent entomopathogen that can ra