ライフサイエンスコーパス: marker chromosome

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します

1 ecular component, the size, and shape of the marker chromosome.

2 dings in AML is structurally highly abnormal marker chromosomes.

3 translocations, deletions, duplications, and marker chromosomes.

4 cations and triplications, and generation of marker chromosomes.

5 impact, and underlying biological origin of marker chromosomes.

6 At PMC meiosis, two marker chromosomes 1 and two marker chromosomes 2 formed

7 acentric 5S rDNA loci and were designated as marker chromosomes 1.

8 n greater detail: chromosome 6 (9 additional markers), chromosome 11 (8 additional markers), and chro

9 ve genomic hybridization, about one-third of marker chromosomes (18/49) had arisen from chromothripsi

10 MC meiosis, two marker chromosomes 1 and two marker chromosomes 2 formed bivalents, whereas the other

11 A loci and three of these were designated as marker chromosomes 2.

12 k karyotypes displayed a higher frequency of marker chromosomes (26.5% in adverse-risk, 40.3% in comp

13 .0 that were not found by the microsatellite markers: chromosome 8, with a maximum model-free LOD sco

14 mor suppressor gene responsible for the i12p marker chromosome abnormality and development of FISH pr

15 At least for such marker chromosomes, alpha-satellite DNA at levels detect

16 l breakpoints, characterize the add(14)(q32) marker chromosomes, and to identify other recurring tran

17 be filtered by: family, person, trait value, markers, chromosomes, and chromosome ranges.

18 In conclusion, marker chromosomes are indicative of chromothripsis and

19 riplications, and supernumerary isodicentric marker chromosomes, as well as the deletions that cause

20 s synergistically increased the loss rate of marker chromosomes carrying a centromere lacking the CP1

21 d highly rearranged karyotypes with numerous marker chromosomes, common in tumour cell preparations,

22 th a distinct phenotype, and a supernumerary marker chromosome, +der(8)(8p23.1pter), which is also a

23 and in addition, resolve the identity of all marker chromosomes from our initial karyotyping and G-ba

24 An add(14)(q32) marker chromosome has been reported to be the most frequ

25 earrangements and identify all of the clonal marker chromosomes in tumor cells.

26 tellite DNA, we have studied eight accessory marker chromosomes in which fluorescence in-situ hybridi

27 In multivariate analysis, marker chromosomes independently predicted poor prognosi

28 We show that the T(17(16))65Dn marker chromosome is inherited at expected frequency and

29 ations, inversions, isochromosomes and small marker chromosomes, may also involve susceptibility to r

30 virtually undetectable in a control group of marker chromosome-negative complex aberrant karyotypes (

31 consistently amplified in the ring and giant marker chromosomes of atypical lipomatous tumors (ALTs),

32 Supernumerary marker chromosomes (SMCs) are common, but their molecula

33 Supernumerary marker chromosomes (SMCs) of chromosome 15, designated "

34 The variability of a small supernumerary marker chromosome (sSMC)-related phenotype is determined

35 een spermatozoa with the small supernumerary marker chromosome (sSMC; sSMC(+)) and spermatozoa with n

36 Ts65Dn mice inherit a marker chromosome, T(17(16))65Dn, producing segmental tr

37 rovide insight into a newly defined class of marker chromosomes that lack detectable alpha-satellite

38 information, including the identification of marker chromosomes, the detection of subtle chromosomal

39 Marker chromosomes were associated with a poorer prognos

40 , AML2003) from the Study Alliance Leukemia, marker chromosomes were detectable in 165/1026 (16.1%) o

41 nslocations that generated the add (14)(q32) marker chromosomes were identified in all cases in which

42 All but one of the marker chromosomes were linear mirror image duplications

43 These accessory marker chromosomes were present in the majority of or al

44 Chromosomal origin of the marker chromosomes were successfully identified in seven

45 ALTS (85.0%), all of which had ring or giant marker chromosomes with amplification of 12q13-15, stron

WebLSDに未収録の専門用語（用法）は "新規対訳" から投稿できます。