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1 ultaneously removing the unwanted selectable marker gene.
2 ich could be fully characterised by a single marker gene.
3 g in the excision of the target-site flanked marker gene.
4 ection in each of the 7 introns of the EPSPS marker gene.
5 d a Ds element into each intron of the EPSPS marker gene.
6 sted from a natural environment with a viral marker gene.
7 , there is a need for alternative selectable marker genes.
8 esses and have elevated expression of stress marker genes.
9 ncluding previously characterized NF-induced marker genes.
10 howed enhanced expression of leaf senescence marker genes.
11 ribosome binding sites, and yeast selectable marker genes.
12 gen fixation - and could be visualised using marker genes.
13 on in the regulatory regions of pluripotency marker genes.
14 oxidative burst and expression of PTI early marker genes.
15 res required no tissue culture or selectable marker genes.
16 akage, and the expression of leaf senescence marker genes.
17 ced expression of osteoblast differentiation marker genes.
18 ns that show no detectable expression of SMC marker genes.
19 uction of a large portion of innate immunity marker genes.
20 Bacteroides human, ruminant, and gull source-marker genes.
21 runk marker genes and gain expression of tip marker genes.
22 s of the 16S rRNA gene or other phylogenetic marker genes.
23 bition and the expression of various (1)O(2) marker genes.
24 istinct types and for identifying associated marker genes.
25 natomy and expression levels of SMC-specific marker genes.
26 ent expression of established digit blastema marker genes.
27 he expression pattern of conserved molecular marker genes.
28 ctionally distinct cell types and associated marker genes.
29 r cells (RPCs) that express Rx and other RPC marker genes.
30 cript and protein level, including known ABA marker genes.
31 riant reconstruction within species-specific marker genes.
32 s and analysed expression of cardiac lineage marker genes.
33 rentiation and mRNA expression of osteoclast marker genes.
34 munity (MTI), inducing the expression of MTI marker genes.
35 eta) signaling pathway and enhances fibrosis marker genes.
36 nd expression of hormone related or meristem marker genes.
37 ltilocus sequence typing and strain-specific marker genes.
38 ogenic HPV18 genome was replaced by CpG free marker genes.
39 regulating the expression of major decidual marker genes.
40 e senescence-specific and pathogen-resistant marker genes.
41 damage and elevated expression of DNA damage marker genes.
42 chronous occurrences of preplacodal and otic marker genes.
43 fq transcriptomes, mainly on shade-avoidance marker genes.
44 troscopy and the induction of singlet oxygen marker genes.
45 nes that are co-regulated with characterized marker genes.
46 inar identity using canonical cortical layer marker genes.
47 PPP1R14a and other neuronal differentiation marker genes.
49 and specific Cre recombinase expression and marker gene activation in all hepatocytes of adult Rosa2
51 RKNs and investigated the expression of PTI marker genes after RKN infection using both quantitative
53 y, mineralization, and up-regulation of bone marker genes (alkaline phosphatase/ALPL, osteopontin/SPP
55 l tools, including highly specific stem cell marker genes along with more rigorous experimental metho
56 The expression level of early osteogenic marker genes, ALP, Runx2, and type I collagen, which pla
57 more, TSH increased expression of osteoblast marker genes ALPL (8.2+/-4.6-fold), RANKL (21+/-5.9-fold
58 tify WRKY factors that bind the promoters of marker genes (Alternative oxidase1a, NADH dehydrogenaseB
59 uence reads to identify error-prone reads in marker gene analyses and their contribution to spurious
62 of cyclooxygenase 2 (COX-2) expression as a marker gene and by using gene expression profiling to id
65 1-related tissue-specific changes of various marker genes and a significant transcriptomic overlap wi
67 cells, the Notch-suppressed cells lose trunk marker genes and gain expression of tip marker genes.
68 y expressed genes, including important tuber marker genes and genes involved in cell growth, transcri
69 wn also reduced the expression of osteoclast marker genes and inflammation-induced cytokine genes.
70 xpressed Gtpbp2 can induce ventral-posterior marker genes and localize to cell nuclei in Xenopus anim
71 nactive UC, however, was the mobilization of marker genes and proteins for the Epithelial Mesenchymal
72 sed expression of osteoblast differentiation marker genes and reduced expression of genes associated
75 performance, it is effective at identifying marker genes and simultaneously it detects mislabelled a
76 the expression of early and late odontoblast marker genes and stage-specific proteases involved in de
77 between the expression levels of these known marker genes and the expression of the coregulated genes
78 anti-Plasmodium falciparum effector genes, a marker gene, and the autonomous gene-drive components ar
79 hologic features, shift in expression of EMT marker genes, and decrease in mir-200 levels consistent
80 es results in up-regulation of mitochondrial marker genes, and increased mitochondrial activity and u
81 sing oxygen consumption rate, brown-specific marker genes, and miR-30b and 378, which were abrogated
82 we find that while many of the early muscle marker genes are reprogrammed, global gene expression an
83 lely responsible for transcript induction of marker genes around 3 to 6 h after chemical inhibition o
85 bstitutes for antibiotic/herbicide-dependent marker genes as well as surprisingly sensitive reporters
86 tified Cox7a1, well-known as brown adipocyte marker gene, as a cold-responsive protein of brown adipo
88 onucleotide down-regulated the expression of marker genes associated with early primitive hematopoiet
89 sis) and mRNA expression of the hypertrophic marker genes, atrial natriuretic factor, brain natriuret
91 t of genes, and recapitulating expression of marker genes by BAC transgenesis or knock-in has generat
92 fication of neurons; transcript profiles for marker gene candidates identify molecular handles for ma
93 dditions to the genome such as insertions of marker gene cassettes or functional elements, without th
95 s and down-regulation of the G2/M cell-cycle marker gene, CYCB1;1 TCP20 and NLP6&7 also support root
96 ical analyses and expression analyses of the marker genes demonstrated that spermatogenesis is arrest
97 silico method, for identifying subpopulation marker genes directly from the original mixed gene expre
98 is finding, expression of the auxin-response marker gene DR5::GUS did not increase in spa mutant seed
100 the expression of osteoclastogenesis-related marker genes during osteoclastogenesis, and prevented os
102 but we observed misexpression of left-sided marker genes early in development, suggesting that nodal
104 ng genes of the PHB pathway and a selectable marker gene encoding spectinomycin resistance, was flank
105 on of the PATHOGENESIS-RELATED GENE1 defense marker gene, enhanced reactive oxygen species (ROS) burs
107 aining a single 6-kb piggyBac element with a marker gene expressing EGFP had the vector in new genomi
108 3 +/- 18 to 1497 +/- 29 mm(2), P < 0.01) and marker gene expression (atrial natriuretic peptide incre
109 Knockdown of Safb1 inhibited skeletal muscle marker gene expression and differentiation in cultured C
111 naling is required for the maintenance of DP marker gene expression and feather regeneration, excessi
112 Th17 differentiation by digoxin lowered Th17 marker gene expression and IL-17 production and strongly
113 ntiate, as evidenced by increased osteoblast marker gene expression and increased mineralization.
114 ated with significant reduction in adipocyte marker gene expression and subcutaneous lipoatrophy.
115 oxygen consumption, but lipolysis and brown marker gene expression are increased; the latter is also
117 activation and PHI1, WRKY33, and FRK1 immune marker gene expression but have reduced reactive oxygen
118 its Sox9-dependent activation of chondrocyte marker gene expression by blocking Sox9-enhancer DNA ass
119 genously applied brassinolide and altered BR marker gene expression demonstrate that zmbri1-RNAi tran
120 ovel mechanism for epigenetic control of SMC-marker gene expression during development through intera
122 ryonic head morphogenesis, and developmental marker gene expression in combinatorial ey and toy knock
123 nockdown of WDR5 significantly decreased SMC-marker gene expression in cultured SMC differentiation s
125 the observed enrichment of lineage-specific marker gene expression in Nanog-negative cells is associ
126 R are unlikely to account for restoration of marker gene expression in the SHR lines when used with a
127 x (ECM) synthesis and increased pro-fibrotic marker gene expression including connective tissue growt
129 r morphological change, a lack of osteoblast marker gene expression, and an inability to mineralize m
131 sistance to virulent pathogens, high defence marker gene expression, and salicylic acid accumulation.
132 Current methods enable targeting based on marker gene expression, development, anatomical projecti
133 s with a deficit in dermomyotome and myotome marker gene expression, suggesting that Ripply1 promotes
134 terized by a decline of stem cell-associated marker gene expression, the appearance of large polyploi
135 romotes VSMC proliferation and enhances VSMC marker gene expression, which may contribute to the defe
142 and albumin expression, qPCR for hepatocyte markers gene expression, Periodic Acid-Schiff staining f
143 f which is characterized by specific surface markers, gene-expression patterns, and distinct function
145 ses, notably delayed upregulation of the PTI marker gene FLG22-INDUCED RECEPTOR-LIKE KINASE1, reduced
146 ings greatly expand the diversity of the key marker gene for anaerobic alkane cycling and outline the
151 nary relationships between amoA, a conserved marker gene for Thaumarchaeota, and soil characteristics
152 critical problem of the absence of validated marker genes for many (including novel) subpopulations,
154 xpression of posterior Hox genes and that of marker genes for presomitic mesoderm and the chordoneura
157 induction of DA1 and BB expression, several marker genes for the transition from proliferation to ex
158 curately captured differential expression of marker genes for type II (e.g. the Tas1r genes, Plcb2, T
159 ylinositol 3-phosphate] and up-regulation of marker genes from the synthesis phase of the cell cycle
160 The expression of the trichome development marker genes GLABRA2 (GL2) and Ethylene Receptor2 (ETR2)
162 expression of specific Mullerian epithelial marker genes, grow in soft agar and develop ectopic inva
166 2-1 mutants show reduced upregulation of PTI marker genes, impaired callose deposition, and defective
167 ngo system exhibit increased expression of a marker gene in the presence of dopamine and blue-light e
169 ole tumor expression of corresponding immune marker genes in a large gene expression microarray cohor
171 xpression of previously identified NP and NC marker genes in adult human NP cells from a range of age
172 A qPCR time course of (1)O2 induced systemic marker genes in directly and indirectly connected leaves
174 ormal responses of key ventral telencephalic marker genes in Foxg1(-/-) telencephalic tissue followin
175 gene expression of Alpl and other osteogenic marker genes in mouse osteoblasts and mesenchymal stem c
176 he cassette stably and efficiently expressed marker genes in progeny under either an artificial or an
177 accumulation and the expression of adipocyte marker genes in the cultured cells, and shRNA knockdown
179 tochrome B (eYHB) as plant-derived selection marker genes in the model plant Arabidopsis (Arabidopsis
181 evidenced by lower mRNA levels of macrophage marker genes including F4/80, Cd68, Cd11b, Cd11c, and ma
184 embryos and knockout VSMCs, differentiation marker genes, including alphaSMA, SM22, and CNN1, were s
188 to expressing elevated levels of pluripotent marker genes involved in proliferation and self-renewal.
190 criptional repressor activity of KLF4 on SMC marker genes is dependent on cooperative binding with pE
191 of the species present, PCR of virulence or marker genes is mainly focused on a handful of known spe
192 nstructing 16S ribosomal RNA, a phylogenetic marker gene, is usually required to analyze the composit
193 th reduced steady-state expression of immune marker genes, leading to increased susceptibility to the
194 S and higher expression levels of senescence marker genes, leading to premature senescence of KO sili
196 sponses, notably delayed upregulation of PTI marker genes, lower callose deposition, and mitogen-acti
197 curate novel method enabling reference-based marker gene matching (picking Operational Taxonomic Unit
198 Our objective was to test whether or not the marker genes may be lost by homologous recombination via
199 titative real time PCR (qRT-PCR) of fibrotic marker genes, measurement of hydroxyproline content and
200 e used to automatically detect cell-specific marker genes (MGs) located on the scatter radii of mixed
201 k for PERMANOVA power estimation tailored to marker-gene microbiome studies that will be analyzed by
202 1, and JAK2), and the alternative activation marker gene (MRC1) were analyzed on PBMCs and on CD14- a
203 to decode premature stop codons in metabolic marker gene mRNAs, that can be used as in vivo reporters
204 also restores the expression of osteoblastic marker genes, namely Ocn and bone sialoprotein, in Atf4(
206 playing decreased negative selection-related marker genes (Nur77 and CD5high) in CD4 single positive
207 analysis demonstrated reduced expression of marker genes of alternative activation in monocytes sort
208 most upregulated genes were Ms4a8a and Ym1, marker genes of alternatively activated myeloid cells.
209 ssion of proteins involved in malignancy and marker genes of epithelial-mesenchymal transition (EMT).
210 ons that develop chronological expression of marker genes of non-neural ectoderm, preplacodal ectoder
211 to investigate changes in DNA methylation in marker genes of T-cell subsets during allergen sensitiza
212 regulation of Hippo pathway target genes and marker genes of TGF-beta signaling, including biomarkers
217 proaches require a priori information on the marker genes or composition of known subpopulations.
218 no effect on the mRNA levels of hypertrophic marker genes or the proinflammatory gene cyclooxygenase
219 it upregulated expression of beige adipocyte marker genes, particularly during an HFD, in association
220 full induction of the JA/ET defense pathway marker gene PLANT DEFENSIN1.2 (PDF1.2) and for resistanc
221 ly, an elevated expression of the adipogenic marker genes PPARgamma and Cebpalpha with a concomitant
222 gulated the expression of an oligodendrocyte marker gene PPP1R14a and other neuronal differentiation
223 the systemic acquired resistance-associated marker genes PR1, PR2 and PR5, transgenic plants over-ex
225 wnregulation of neuronal and oligodendrocyte marker genes preceding clinical disease onset, whereas n
226 pression profiling did not identify a single marker gene predicting docetaxel response, despite an in
228 NA-seq reveals that MMPs express a number of marker genes previously assigned to mesenchymal stem/pro
232 stone H3K4me within smooth muscle cell (SMC)-marker gene promoters during the early stages of develop
233 4, LGR5, TP63 (p63), as well as neural crest marker genes PSIP1 (p75(NTR)), PAX3, SOX9, AP2B1 (AP-2be
236 driven by identification of novel stem cell marker genes, revealing the existence of quiescent, slow
237 duction of transgenic plants, the selectable marker gene(s) used in the process are redundant, and th
238 -based methods such as database alignment or marker gene search, which limits the set of detectable c
239 RE1 transcripts, induction of the senescence marker gene SENESCENCE-ASSOCIATED GENE12, and cell death
247 niques have seldom been applied to microbial marker gene sequencing studies, which have mostly relied
248 al windows, we used high-resolution 16S rRNA marker gene sequencing to examine outcomes in our mouse
251 d protein extracts induce the Mi-1.2 defense marker gene SlWRKY72b, indicating that both saliva and e
255 Concordantly, expression levels of brown marker genes, such as Prdm16, Cidea, Pgc1alpha, Pparalph
256 he expressions of the atrioventricular canal marker genes, such as tbx2b, hyaluronan synthase 2 (has2
257 a through enhanced activation of mesenchymal marker genes, such as Zeb1, Snail1, Pai1, and alphaSMA,
258 it sporadic expression of many of these same marker genes, suggesting the existence of a general prog
261 me was measured with a two-color fluorescent marker gene that converts from expressing tandem dimer T
262 I gene becomes a highly efficient selectable marker gene that facilitates the selection of transgenic
265 ion clock." Using hundreds of stage-enriched marker genes that compose this clock, we show that extre
266 adata about the specimen source analyzed and marker genes that serve as the most useful features in m
267 tissue, and yeast cell cycle, revealed novel marker genes that were otherwise undetectable using exis
268 to inhibit the expression of a Pi starvation marker gene (the high-affinity Pi transporter PHT1;4).
269 thus far have relied on a single selectable marker gene, the spectinomycin resistance gene aadA.
270 d for three specific inflammation-associated marker genes (TNF-alpha, IL-8, IP-10) using qRT-PCR.
271 Here we used high-throughput sequencing of marker genes to elucidate the responses of soil fungal,
272 f community fingerprinting and sequencing of marker genes to identify community changes associated wi
273 we adopt a selection system that allows for marker genes to integrate into the genome through homolo
274 ession measurements of a small subset of 100 marker genes to predict transcriptome-wide gene abundanc
275 S and 18S rRNA gene have been widely used as marker genes to profile diversity of microorganisms in e
276 present an approach that uses clade-specific marker genes to unambiguously assign reads to microbial
278 e promoters, resulting in suppression of SMC marker gene transcription and, consequently, in inhibiti
280 ted activation of ADH1 and activates hypoxia marker genes under both anoxic and normoxic conditions.
281 e generated MSTN-mutant Meishan pigs with no marker gene via zinc finger nucleases (ZFN) technology.
283 nt was detected when the modified rice EPSPS marker gene was inserted with the Ac transposon 5' end,
284 iescent center (QC) cells, expression of the marker gene was silenced specifically in the QC cells wi
287 nrichment of RNA polymerase II at primed PTI marker genes was observed in environmentally challenged
288 Escherichia coli (using blaNDM and mcr-1 as marker genes), we sampled poultry, dogs, sewage, wild bi
289 ations and analyses of core and phylogenetic marker genes, we show that patterns of diversity for the
290 capturing strain variation in clade-specific marker genes were able to distinguish among 100s of indi
292 , H2O2- and photooxidative stress-responsive marker genes were found to be dramatically up-regulated
293 eoclast numbers and expression of osteoclast marker genes were increased in parallel with reduced Erk
294 UCROSE NONFERMENTING-RELATED KINASE1 (SnRK1) marker genes were most affected in reproductive tissue.
296 ed strain were simultaneously substituted by marker genes, which were subsequently excised using Cre/
297 a dark period induced stress and cell death marker genes while reducing photosynthetic efficiency.
298 ss C. cellulans, which revealed 49 potential marker genes with known association to human infections,
299 By combining the detection of a pair of marker genes with operon prediction using intergenic dis
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