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1 ical threshold to noxious stimulation of the masseter.
2 he anatomy and muscle fiber phenotype of the masseter.
3 anatomical regions of male and female mouse masseters.
4 decrease to a non-detectable level in mouse masseter, (2) an increase to become the sole TnT in shee
5 an increase to become the sole TnT in sheep masseter, (3) an increase of the total level as well as
6 om contralateral (cMM) and ipsilateral (iMM) masseters, activated at 10% of maximal voluntary contrac
7 SupV BPNs is sufficient to induce bilateral masseter activation both during resting state and during
8 toxin light chain (TeNT) increases bilateral masseter activation during chewing, an effect driven by
9 ter and cutaneous inputs project to the MDH, masseter afferents provide an additional input to the Vi
10 rds were made of the electromyogram (EMG) in masseter and anterior digastric muscles and of the unloa
12 and slow skeletal muscle fibres such as the masseter and is an important source of reference work fo
16 rce calibrations determined subject-specific masseter and temporalis muscle activities per 20-N bite-
17 , on intramuscular hemodynamics in the human masseter and temporalis muscles following a sustained is
18 anesthesia during maximum stimulation of the masseter and temporalis muscles in each of five condylar
20 every other hour at the level of the thenar, masseter, and deltoid muscles along with central hemodyn
21 eft and right anterior digastric (LAD, RAD), masseter, buccinator, and genioglossus (GG) muscles with
22 es had distinct gene expression profiles and masseter cells usually proliferated more and differentia
25 erties was preserved in both EDL-derived and masseter-derived satellite cells from old mice, although
27 was masked by bilateral reflex depression of masseter EMG caused by auditory input from the coil disc
28 ack-test recordings of single, skinned human masseter fibers at 15 degrees C revealed maximum shorten
30 Analysis by gel electrophoresis found 63% of masseter fibers to contain pure type I MyHC and the rema
34 cid led to the elimination or attenuation of masseter hyperalgesia/allodynia developed after masseter
35 ne blue (MB), and ODQ, in the Vc, attenuates masseter hypersensitivity induced by intramuscular injec
36 in the subnucleus caudalis (Vc) in mediating masseter hypersensitivity under acute inflammatory condi
38 ein immunohistochemistry, we show that after masseter inflammation, a population of neurons in the do
42 alization of muscle fiber types in the mouse masseter is consistent with the functional compartmental
45 se, intracellular records were obtained from masseter motoneurons before and after carbachol-induced
46 nucleus pontis oralis (NPO) induced IPSPs in masseter motoneurons following, but never prior to, the
48 eurokinin 1 (NK1) receptor antagonist in one masseter muscle 15 min prior to the MO injection in the
49 erties of Vi neurons that receive input from masseter muscle afferents by characterizing their respon
50 agent, complete Freund's adjuvant, into the masseter muscle and perfused at 2 hours postinflammation
52 mapped premotor neurons for the jaw-closing masseter muscle and the tongue-protruding genioglossus m
56 these results suggest that the activation of masseter muscle nociceptor alters spindle afferent respo
57 MK-801 (0.3 mg/kg) preadministered in the masseter muscle significantly reduced the peak and overa
59 ically active neurons that function to lower masseter muscle tone, whereas unilateral optogenetic act
60 of complete Freund's adjuvant (CFA) into the masseter muscle under methohexital anesthesia after a sm
62 icantly (p < 0.01) greater body (by 18%) and masseter muscle weight (by 83%), compared with wild-type
63 Significant correlations were noted between masseter muscle weight and mandibular body length (r = 0
64 nd the resulting reflexes in the ipsilateral masseter muscle were examined electromyographically.
65 instem neurons following the inflammation of masseter muscle were examined in order to differentiate
66 challenge (hypertonic saline infused in the masseter muscle) with and without placebo administration
67 results suggest that the inflammation of the masseter muscle, an injury of orofacial deep tissue, res
74 to determine how trigeminal motoneurons and masseter muscles are switched off during REM sleep in ra
76 cal facilitation (ICF) were evaluated in the masseter muscles of 12 subjects and the cortical silent
77 ic (e.g., sternocleidomastoid, temporal, and masseter muscles) and artifactual (e.g., dental implants
81 we show that the affinity of ADP for bovine masseter myosin in the absence of actin (represented by
83 electrical stimulation of the caudal Vme or masseter nerve, mechanical stimulation of jaw muscles an
87 ) with cold pressor stimulation, both in the masseter (p < 0.001) and in the temporalis (p < 0.001) m
88 ake value were found in the control muscles (masseter [P = 0.38] and pterygoid [P = 0.70]) and subcut
89 , and IIb--was determined within the defined masseter partitions by means of Western blot analysis an
90 ng characteristic curve analysis showed that masseter tissue oxygen saturation (area under the curve
91 ygen saturation was consistently higher than masseter tissue oxygen saturation (p = .04) and deltoid
92 that in the early 6-hr resuscitation period, masseter tissue oxygen saturation accurately identified
94 g characteristic curves analyses showed that masseter tissue oxygen saturation was better predictor o
95 oid tissue oxygen saturation (p = .002), and masseter tissue oxygen saturation was consistently highe
97 after inflammation of the skin overlying the masseter was attenuated by injection of AP-5 into the MD
98 ent was unaffected in all muscles except the masseter, where its expression was extinguished in the I
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