ライフサイエンスコーパス: mastitis

1 cells and in a mouse model of staphylococcal mastitis.

2 3.3, 95% CI: 1.92, 5.62) strongly predicted mastitis.

3 uarters suffering recurrent cases of E. coli mastitis.

4 (CoNS) from bovine clinical and subclinical mastitis.

5 subspecies zooepidemicus, a cause of bovine mastitis.

6 f early inflammatory responses during bovine mastitis.

7 as up-regulated on leukocytes from cows with mastitis.

8 were studied during Escherichia coli-induced mastitis.

9 7, a serum-resistant isolate from a cow with mastitis.

10 ent approach to treat lactational infectious mastitis.

11 the development of chronic S. aureus-related mastitis.

12 the dairy industry are used to treat bovine mastitis.

13 ays relevant to bovine S. aureus subclinical mastitis.

14 hanistic studies on susceptibility of bovine mastitis.

15 lead to the development of vaccines against mastitis.

16 ysbiosis of the milk microbiome that permits mastitis.

17 m dairy cattle with transient and persistent mastitis.

18 ctiae, one of the causative agents of bovine mastitis.

19 ntributing to higher milk viral loads during mastitis.

20 ient previously diagnosed with granulomatous mastitis.

21 ads in breast milk were not increased during mastitis.

22 ical processes that occur during LPS-induced mastitis.

23 ontribute to milk composition changes during mastitis.

24 hanisms affected in cows more susceptible to mastitis.

25 lso be used in the virgin animal with little mastitis 3 days after transduction.

26 ancy to women who had experienced infectious mastitis after previous pregnancies.

27 -32 (IL-32) in Staphylococcus aureus-induced mastitis, an inflammation of the mammary gland, is uncle

28 microbiome of milk that are associated with mastitis and antimicrobial therapy.

29 Mastitis and breast milk HIV-1 load may increase the ris

30 ith the progression of S. aureus subclinical mastitis and could be used as powerful biomarkers for th

31 ein, we develop a murine model of autoimmune mastitis and provide a detailed characterization of its

32 s dysgalactiae isolated from cases of bovine mastitis and septic arthritis in lambs.

33 de causing diseases such as pleuropneumonia, mastitis and septicaemia.

34 understanding of the epidemiology of E. coli mastitis and suggest that pathogen adaptation and host s

35 ghest homology with a GBS strain causing cow mastitis and that the 1992 ST-1 strain differed from ser

36 es were collected from cows showing signs of mastitis and used for microbiological culture.

37 associated with symptomatic and asymptomatic mastitis and with the quantity of HIV-1 RNA and DNA in m

38 have been identified as causative agents in mastitis, and are traditionally diagnosed by bacterial c

39 ens but have so far not been associated with mastitis, and DNA of bacteria that are currently not kno

40 te immune responses, reduces the severity of mastitis, and facilitates clearance and neutralization o

41 Escherichia coli strains that caused bovine mastitis, and have since been implicated in many physiol

42 ing genital ulcer disease, chorioamnionitis, mastitis, and malnutrition in HIV-infected women, and of

43 ncluding hearing loss, orchitis, oophoritis, mastitis, and pancreatitis.

44 Protection against S. aureus mastitis appears to be achievable with as little as 3 mi

45 Cows diagnosed with clinical mastitis associated with Gram-negative pathogens or nega

46 notable differences in the genomes of bovine mastitis-associated and human clones of S. aureus and pr

47 omparative genomic analysis between a bovine mastitis-associated clone, RF122, and the recently seque

48 Cows diagnosed as negative for mastitis at dry off were randomly allocated to receive a

49 f 9.5% reported provider-diagnosed lactation mastitis at least once during the 12-week period, with 6

50 ss-sectional study, laboratory indicators of mastitis (breast milk sodium [Na(+)] concentration, sodi

51 Staphylococcus aureus commonly causes bovine mastitis, but bovine strains, unlike human isolates of S

52 e findings indicate that 38% of all clinical mastitis cases and 63% of the PTEs attributed to S. uber

53 PTE ST, represented 40% of all the clinical mastitis cases and occurred in 63% of the herds.

54 s sequences were the third most prevalent in mastitis cases diagnosed as Staphylococcus aureus by cul

55 were the second most prevalent sequences in mastitis cases diagnosed as Streptococcus dysgalactiae b

56 were the second most prevalent sequences in mastitis cases diagnosed as Trueperella pyogenes by cult

57 accounted for >50% of all S. uberis clinical mastitis cases in 33% of the herds.

58 olates of Streptococcus uberis from clinical mastitis cases in a study of 52 commercial dairy herds o

59 Forty-one percent of all clinical E. coli mastitis cases occurred in just 2.2% of the population.

60 S. uberis mastitis cases that occurred in different cows within th

61 acter, and Staphylococcus, often involved in mastitis cases, were the most abundant genera across tre

62 After inoculation of the MG with a mastitis-causing E. coli strain, the bacterial load incr

63 Staphylococcus aureus is a major mastitis-causing pathogen in cattle.

64 irst lysin that kills all four of the bovine mastitis-causing pathogens.

65 he E. coli P4 were submitted to a homologous mastitis challenge.

66 Bovine mastitis continues to be the most detrimental factor for

67 choice of the antibiotic to treat cows with mastitis could be determined based on the naked eye.

68 samples from women with laboratory-diagnosed mastitis (defined as elevated BM Na(+) levels) were 5.4-

69 catalase in milk samples, a key indicator of mastitis disease in milk.

70 fied DNA of bacteria that are known to cause mastitis, DNA of bacteria that are known pathogens but h

71 g a part in the changing pattern of clinical mastitis experienced in the modern dairy herd.

72 We hypothesized that mastitis facilitates the passage of HIV-1 from blood int

73 Current vaccines to Escherichia coli mastitis have shown some albeit limited efficacy.

74 I: 1.37, 8.54), and (for women with no prior mastitis history) using a manual breast pump (OR = 3.3,

75 Globally, 44 of 108 women (41%) developed mastitis; however, the percentage of women with mastitis

76 be an efficient method to prevent infectious mastitis in a susceptible population.

77 Staphylococcus aureus is a major cause of mastitis in bovine and other ruminant species.

78 cteriophages that may be used for therapy of mastitis in cattle.

79 causes severe invasive disease in humans and mastitis in cattle.

80 most important pathogens causing contagious mastitis in dairy cattle worldwide.

81 erichia coli is a leading cause of bacterial mastitis in dairy cattle.

82 . have become an important cause of clinical mastitis in dairy cows in New York State.

83 aureus is a common causative agent of bovine mastitis in dairy herds.

84 y pathogen causing pneumonia, arthritis, and mastitis in infected cattle.

85 litis, progressive pneumonia, arthritis, and mastitis in sheep and goats.

86 Clinical mastitis in six Somerset dairy herds was monitored over

87 titis; however, the percentage of women with mastitis in the probiotic group (25% [n = 14]) was signi

88 y important in the epidemiology of S. uberis mastitis in the United Kingdom, with cow-to-cow transmis

89 for approximately 33% of all cases of bovine mastitis in the United Kingdom.

90 f Corynebacterium accolens and granulomatous mastitis in this patient is discussed.

91 until they stopped breastfeeding to describe mastitis incidence, mastitis treatment, and any associat

92 ved in the host defense of the udder against mastitis infection and that selective recruitment of the

93 Many cases of mastitis involve no known infectious agent and may funda

94 S. aureus-related bovine mastitis is a common reason for therapeutic and/or proph

95 Mastitis is a substantial clinical problem in lactating

96 Subclinical mastitis is a widely spread disease of lactating cows.

97 ssible role of anaerobic pathogens in bovine mastitis is also suggested.

98 Dairy cow mastitis is an important disease in the dairy industry.

99 cell line (MAC-T) by a Staphylococcus aureus mastitis isolate to study the potential role of intracel

100 Like a previously characterized bovine mastitis isolate, the standard laboratory strain, RN6390

101 fied in a pathogenicity island from a bovine mastitis isolate.

102 Although a significant number of mastitis isolates produce SAgs, the effect of these mole

103 Escherichia coli infection by using a mouse mastitis model.

104 ed breast milk sodium levels consistent with mastitis occurred in 16.4% of HIV-1-infected women and w

105 When mastitis occurred, the milk bacterial counts in the prob

106 itis treatment, and any associations between mastitis occurrence and hypothesized host characteristic

107 h Bovine spongiform encephalopathy, clinical mastitis or somatic cell count.

108 ipple thrush) in the same 3-week interval as mastitis (OR = 3.4, 95% CI: 1.37, 8.54), and (for women

109 cracks and nipple sores in the same week as mastitis (OR = 3.4, 95% CI: 2.04, 5.51), using an antifu

110 Mastitis, or inflammation of the breast, is associated w

111 clinical presentations in cattle, including mastitis, otitis, arthritis, and reproductive disorders.

112 The first mastitis outbreak was caused by a single strain of Klebs

113 We describe the occurrence of two Klebsiella mastitis outbreaks on a single dairy farm.

114 e involved in the response of the udder to a mastitis pathogen and if the type of mastitis pathogen i

115 er to a mastitis pathogen and if the type of mastitis pathogen influenced the subset composition of t

116 Control of the bovine mastitis pathogen Streptococcus uberis requires sensitiv

117 taphylococcus aureus is the major contagious mastitis pathogen, accounting for approximately 15-30% o

118 The mastitis pathogens identified by culture were generally

119 reast-feeding (OR, 1.7; 95% CI, 1.0-2.9) and mastitis (relative risk [RR], 3.9; 95% CI, 1.2-12.7) wer

120 As a first step toward enhancing mastitis resistance of dairy animals, we report the gene

121 ful biomarkers for the improvement of bovine mastitis resistance.

122 Duration of feeding was not associated with mastitis risk.

123 ted with increased transmission overall, and mastitis (RR, 21.8; 95% CI, 2.3-211.0) and breast absces

124 mphocytes in cows with S. aureus subclinical mastitis (SA group) and healthy controls (CK) were gener

125 ifferences were observed in culture-negative mastitis samples when compared to healthy milk.

126 ide isolated from the cell envelop of bovine mastitis Streptococcus dysgalactiae 2023 is reported for

127 The chronic nature of bovine staphylococcal mastitis suggests that some products or components of S.

128 tic marker in the CXCR2 gene associated with mastitis susceptibility.

129 A second outbreak of Klebsiella mastitis that occurred several weeks later was caused by

130 li, Klebsiella spp. and Streptococcus uberis mastitis) the single most prevalent microorganism.

131 Mastitis, the most consequential disease in dairy cattle

132 85.7% of cases of recurrent quarter E. coli mastitis, the same genotype was implicated as the cause

133 althy cows and cows with naturally occurring mastitis to determine if distinct alphabeta and gammadel

134 reastfeeding to describe mastitis incidence, mastitis treatment, and any associations between mastiti

135 Mastitis was defined as an elevated milk sodium (Na(+))

136 numbers observed in cows with streptococcal mastitis was due to a parallel increase in both CD4(+) a

137 The occurrence of mastitis was evaluated during the first 3 months after d

138 ollected from cows in August, 1998, although mastitis was evident among cows on the suspected farm.

139 requency in the same week or the week before mastitis was included in the model (for the same week: 7

140 Mastitis was not associated with compartmentalization by

141 inflammatory response; however, significant mastitis was observed 12 days after transduction.

142 Mastitis was present in 63 (15%) of 407, 60 (15%) of 407

143 served in milk from cows with staphylococcal mastitis was primarily due to increased numbers of CD4(+

144 the acute host response to Escherichia coli mastitis, we analyzed gene expression patterns of approx

145 experiment modeling phage therapy for bovine mastitis, we observed pathogenicity island transfer betw

146 h confirmed staphylococcal and streptococcal mastitis were characterized by increased numbers of gamm

147 olved in recurrent cases of clinical E. coli mastitis were compared by DNA fingerprinting with entero

148 m cows with staphylococcal and streptococcal mastitis were due to a selective recruitment of a distin

149 y virus (HIV) type 1 load in breast milk and mastitis were examined as risk factors for vertical tran

150 very, HIV-1 load and sodium (an indicator of mastitis) were measured in breast milk from 334 HIV-1-in

151 t in a logistic regression model, history of mastitis with a previous child (odds ratio (OR) = 4.0, 9

152 f the nipple, previous treatment for Candida mastitis with oral or topical antifungals was ineffectiv