ライフサイエンスコーパス: maternal exposure

1 opportunity for conception that may minimize maternal exposure.

2 TB and LBW are dependent upon the periods of maternal exposure.

3 fants by both passive antibodies and reduced maternal exposure, and will provide valuable information

4 ubfoot, but studies of risks associated with maternal exposures are lacking.

5 r childhood germ-cell tumors associated with maternal exposures before pregnancy, during pregnancy, a

6 Three latent maternal exposure clusters were determined: a high-risk,

7 Prospective questionnaires provided maternal exposure data; case status was established from

8 Information on maternal exposure during pregnancy or direct exposure to

9 Here, we provide evidence that maternal exposure during pregnancy to environmentally re

10 We tested hypotheses that maternal exposure during the periconceptional period to

11 gs highlight the importance of understanding maternal exposures during pregnancy and factors influenc

12 isk among young children have suggested that maternal exposures during pregnancy, such as low-dose ra

13 aly has genetic causes, some associated with maternal exposures including radiation, tobacco smoke, a

14 Among those with available maternal exposure information, at age 20, 421 offspring

15 Following arsenite maternal exposure of the F0 generation, subsequent gener

16 (95% confidence interval [CI], 1.3-2.6) for maternal exposure only and 2.4 (CI, 1.6-3.6) for neonata

17 ong women of reproductive age as a proxy for maternal exposures, or estimating the impact of risk fac

18 ether the decreased risks observed for these maternal exposures reflected a true association or were

19 Maternal exposure to 0.2% lead acetate in drinking water

20 In the female offspring, the effects of maternal exposure to a high-fat diet during pregnancy on

21 Therefore, only 9 days of maternal exposure to a sodium-restricted diet is require

22 amine the epidemiological studies addressing maternal exposure to air pollutants and fetal growth dur

23 l study investigated the association between maternal exposure to air pollutants, carbon monoxide, ni

24 Although maternal exposure to air pollution can potentially have

25 Evidence supports an association between maternal exposure to air pollution during pregnancy and

26 Maternal exposure to ambient air pollution has been asso

27 s suggest some positive associations between maternal exposure to ambient PM10 during the first two m

28 Intrauterine inflammation and maternal exposure to ambient PM2.5 during preconception

29 Our goal was to determine whether maternal exposure to an AhR ligand directly alters CD4+

30 oral clefts, and urinary tract defects after maternal exposure to antiepileptic drugs were 2.2 (95 pe

31 these latter data revealed an association of maternal exposure to any pesticides during the first tri

32 ify an intergenerational association between maternal exposure to childhood abuse and risk for autism

33 Maternal exposure to cocaine increased gene expression o

34 However, it is unclear whether maternal exposure to common human allergens such as hous

35 Thus, maternal exposure to DEA reduces the number of neural pr

36 We created a mouse model that linked maternal exposure to DEPs with asthma susceptibility in

37 ort an association between neuroblastoma and maternal exposure to diuretic agents, antiinfective agen

38 Maternal exposure to dLAN dampened delayed type hypersen

39 We evaluated maternal exposure to drinking water brominated trihalome

40 Maternal exposure to each by-product was evaluated for e

41 programming and transgenerational effects of maternal exposure to early-life stress on several phenot

42 Atopic status of the mother and maternal exposure to environmental factors are associate

43 In the current study, we show that maternal exposure to estrogens, including 17alpha- and b

44 Our data demonstrate that maternal exposure to HDM during pregnancy increases asth

45 alyses showed a positive association between maternal exposure to herbicides during the postnatal per

46 The authors examined whether maternal exposure to herpes virus type 2 is associated w

47 Maternal exposure to high MF during pregnancy may be a n

48 We investigated the role of maternal exposure to human influenza virus [HI] in C57BL

49 as observed between serologically documented maternal exposure to influenza and bipolar disorder in o

50 lutamate antipsychotics are both affected by maternal exposure to influenza virus, with increased hea

51 ors examined whether serologically confirmed maternal exposure to influenza was associated with an in

52 Serological documentation of maternal exposure to influenza was determined using the

53 Maternal exposure to intranasal triamcinolone during pre

54 ns in juvenile nonhuman primate tissue after maternal exposure to intravenous gadoteridol during preg

55 lf-life of approximately 62 years), and even maternal exposure to lead decades before pregnancy can s

56 We assessed the effect of maternal exposure to low concentrations of ambient air p

57 ine model of bronchopulmonary aspergillosis, maternal exposure to mainstream CS increases AHR after a

58 Maternal exposure to mild daily stress during pregnancy

59 k of offspring ASD in relation to a combined maternal exposure to multiple (up to 42) common and rare

60 The synthetic effects of maternal exposure to multiple risk factors could increas

61 ) were used to estimate associations between maternal exposure to nitrogen dioxide (NO2), dietary int

62 We investigated the association between maternal exposure to nitrogen dioxide and fine particula

63 2-2005), we investigated the associations of maternal exposure to nitrogen dioxide, nitrogen oxides,

64 study was to evaluate whether high levels of maternal exposure to O3, SO2, NO2, CO are related to inc

65 There is evidence in animal models that maternal exposure to PAHs during pregnancy is associated

66 sistent with the results for the cell lines, maternal exposure to PAHs was associated with hypermethy

67 We explored the association between maternal exposure to particulate matter (PM) air polluti

68 Steps should be taken to decrease maternal exposure to phthalates during pregnancy.

69 aimed to investigate the association between maternal exposure to PM2.5 and IUI in the Boston Birth C

70 Higher maternal exposure to PM2.5 during pregnancy, particularl

71 emiological and animal studies indicate that maternal exposure to pollutants that bind the aryl hydro

72 owever, it has yet to be established whether maternal exposure to polycyclic aromatic hydrocarbons (P

73 und between acute lymphoblastic leukemia and maternal exposure to professional pest exterminations (o

74 serorevert at a younger age (P = .0052), and maternal exposure to protease inhibitors was associated

75 We found that maternal exposure to seasonal energy restriction in uter

76 It is well documented that maternal exposure to second-hand smoke (SHS) during preg

77 Maternal exposure to second-trimester fever was associat

78 In conclusion, maternal exposure to SHS during pregnancy may lead to LB

79 g identified two plausible pathways by which maternal exposure to SHS during pregnancy might cause LB

80 significantly and positively associated with maternal exposure to SHS during pregnancy, lower placent

81 determine the effects in adult offspring of maternal exposure to stress and alcohol during pregnancy

82 We tested the hypothesis that maternal exposure to stressful life events would be asso

83 Maternal exposure to TCDD resulted in durable changes in

84 intrauterine environment as conveyed by the maternal exposure to the general life environment, the s

85 Gallagher et al. (2013) show that maternal exposure to the infection-induced inflammatory

86 This study shows that maternal exposure to this endocrine-active compound shif

87 Adjustment for maternal exposure to tobacco smoke during pregnancy and

88 rmations to estimate the association between maternal exposure to VOCs adjusting for sex, mother's ag

89 We investigated maternal exposures to air pollutants during weeks 2-8 of

90 Maternal exposures to environmental factors during pregn

91 ase-control study of the association between maternal exposures to environmental sulfur dioxide and t

92 ariates, there were associations of TGA with maternal exposures to herbicides (odds ratio (OR) = 2.8;

93 tes suggests an association between VLBW and maternal exposures to high levels of air pollution.

94 Maternal exposures to most chemicals were not associated