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1 ggesting they were protected from suboptimal maternal nutrition.
2 g disease might be preventable by optimizing maternal nutrition.
3 nct from the proposed role of programming by maternal nutrition.
4 fluctuations that are naturally softened by maternal nutrition.
5 es; however, the mechanism(s) whereby excess maternal nutrition affects fetal development remain poor
7 es suggestive evidence that periconceptional maternal nutrition alters offspring methylation at impri
9 e nutrients that show extreme sensitivity to maternal nutrition and are implicated in neurological de
11 classified risk factors into five clusters: maternal nutrition and infection, teenage motherhood and
12 f these relations, however, is the effect of maternal nutrition and nutrient transport on healthy fet
13 gs contain presentations about the effect of maternal nutrition and the placental environment on feta
14 roles of these genes in uterine development, maternal nutrition, and vascular control support their m
15 and experimental studies have pointed toward maternal nutrition as a major player during prenatal dev
16 anthropometric indicators for childhood and maternal nutrition at a district level in Pakistan and a
17 from human and animal studies suggests that maternal nutrition can induce developmental programming
18 tional source for the infant; and for a few, maternal nutrition can lead to substantial variations in
19 rth size, often associated with insufficient maternal nutrition, contributes to a large share of glob
23 e was to investigate the association between maternal nutrition during pregnancy and intrauterine dev
24 logic studies suggest a relationship between maternal nutrition during pregnancy and the occurrence o
26 considering associations with birth weight, maternal nutrition during pregnancy, child growth patter
28 ascular system in utero including over/under maternal nutrition, excess glucocorticoid and chronic hy
31 l and geographical inequalities in child and maternal nutrition in Pakistan, masked by national and p
32 of adulthood, and it has been suggested that maternal nutrition is a major determinant of fetal growt
33 f maternal hypoxia independent of changes in maternal nutrition on maternal circulatory and placental
37 baboons, whose diets were either restricted [maternal nutrition restriction (MNR)] or who were fed ad
41 udy in 1001 healthy participants in the Pune Maternal Nutrition Study (PMNS), replication studies in
42 Mecp2(1lox) mice, a model of RTT, enhancing maternal nutrition through choline supplementation atten
43 suggest a potential pathway that could link maternal nutrition to reproductive development in the of
45 describe a life cycle approach to improving maternal nutrition, which goes beyond the traditional pr
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