ライフサイエンスコーパス: matrix protein

1 crystal structure of the Sudan virus (SUDV) matrix protein.

2 forming growth factor-beta1 or extracellular matrix protein.

3 sialoprotein (DSP) is a dentin extracellular matrix protein.

4 to myofibroblasts that secrete extracellular matrix proteins.

5 the Ser-x-Glu/pSer motifs in several enamel matrix proteins.

6 pread arthropod gene family, the peritrophic matrix proteins.

7 rin without altering adhesion to fibronectin matrix proteins.

8 otifs present in loops in many extracellular matrix proteins.

9 fibrin clots and cross-linking extracellular matrix proteins.

10 components, and proteases processing enamel matrix proteins.

11 function via interactions with extracellular matrix proteins.

12 ne-related gene regulators and extracellular matrix proteins.

13 which secrete large amounts of extracellular matrix proteins.

14 gan and aberrant deposition of extracellular matrix proteins.

15 as well as the expression pattern of enamel matrix proteins.

16 grity and the release of soluble peroxisomal matrix proteins.

17 aracterized by accumulation of extracellular matrix proteins.

18 and enhances the production of extracellular matrix proteins.

19 genesis, angiogenesis, and the deposition of matrix proteins.

20 ile genes and up-regulation of extracellular matrix proteins.

21 n with resulting deposition of extracellular matrix proteins.

22 mpedes adherence to endothelial cells and/or matrix proteins.

23 expression of cartilage tissue extracellular matrix proteins.

24 collagen I secretion without affecting other matrix proteins.

25 tegrin subtypes and sensing of extracellular matrix proteins.

26 in the degradation of various extracellular matrix proteins.

27 to collagen and other exposed subendothelial matrix proteins.

28 llagen type I (COL1) and other extracellular matrix proteins.

29 interacts with a multitude of extracellular matrix proteins.

30 Dentin matrix protein 1 (DMP1) is a non-collagenous calcium-bin

31 tes mineral homeostasis by repressing dentin matrix protein 1 (DMP1) via the vitamin D receptor pathw

32 s osteocalcin, bone sialoprotein, and dentin matrix protein 1 (DMP1) was also suppressed.

33 aled more abundant osteopontin (OPN), dentin matrix protein 1 (DMP1), and matrix extracellular phosph

34 en (procollagen I) and a reduction in dentin matrix protein 1 (DMP1), which is partially responsible

35 These mice were bred with a dentin matrix protein 1 (DMP1)-Cre line for overexpression of T

36 Here, we report that extracellular matrix protein 1 (ECM1) is a previously unrecognized reg

37 in EGF-containing fibulin-like extracellular matrix protein 1 (EFEMP1) to investigate the role of the

38 The matrix protein 1 (M1) of influenza A virus (IAV) exists

39 nd CD8+ peptides from the internal proteins (matrix protein 1 [M1], nucleoprotein [NP], polymerase ba

40 The altered levels of dentin matrix protein 1 and osteopontin in Fam20C-deficient bon

41 Dmp1 (dentin matrix protein 1), Dkk1 (Dickkopf WNT signaling pathway

42 Extracellular matrix protein 1, a direct targeting molecule of parathy

43 C-deficient bone had a lower level of dentin matrix protein 1, and higher levels of osteopontin and b

44 rylated full-length human recombinant dentin matrix protein-1 (17-513 AA), this bioinspired approach

45 osteopontin-, bone sialoprotein-, and dentin matrix protein-1-enriched fractions by anion-exchange ch

46 Here we identified the host factor Golgi matrix protein 130 (GM130) as a novel target of CVB3 dur

47 Knockdown of the influenza matrix protein 2 (M2) with siRNA, or inhibition of its a

48 inidase (NA) and the extracellular domain of matrix protein 2 (M2e) fused to a virus-like particle (V

49 s, including low-abundance proteins like the matrix protein 2 and nonstructural protein 1, with a sim

50 rins, osteopontin, and related extracellular matrix proteins; (2) clastic cell fusion and activation

51 nistration (FDA) approved the use of Nuclear matrix protein 22 (NMP22) as a monitoring tool for predi

52 tectable, but antibodies against Ebola viral matrix protein 40 (not in the vaccine) were not detected

53 duced NOX4 expression, oxidative stress, and matrix protein accumulation in renal epithelial cells; t

54 ls netrin-4 as a non-enzymatic extracellular matrix protein actively disrupting pre-existing BMs.

55 Within the mitochondrial matrix, protein aggregation activates the mitochondrial

56 ent of Trap1, an Hsp90-related mitochondrial matrix protein and a member of the mitochondrial unfolde

57 ripts: an unspliced mRNA that encodes the M1 matrix protein and a spliced transcript that encodes the

58 specific molecule targeting an extracellular matrix protein and delivering a TGF-beta mAb resulted in

59 high glucose-induced expression of NOX4 and matrix protein and whether H2S and NO pathways are integ

60 Virtually all mitochondrial matrix proteins and a considerable number of inner membr

61 osis is associated with an overproduction of matrix proteins and a pathological increase of liver sti

62 sed level of cell-junction and extracellular matrix proteins and an altered cytokine secretory profil

63 teoglycans interact with other extracellular matrix proteins and are important regulators of matrix a

64 titute the major portion of secretory enamel matrix proteins and are known to be highly alternative s

65 The main extracellular matrix proteins and associated cytokines were evaluated.

66 hat is responsible for binding extracellular matrix proteins and certain arenaviruses.

67 ts (ghosts) rapidly incorporated peroxisomal matrix proteins and developed into peroxisomes.

68 eceptor (CAR) T cells, reduced extracellular matrix proteins and glycosaminoglycans.

69 ed by cellular interactions with surrounding matrix proteins and growth factors that mediate cellular

70 contain dedicated machineries for import of matrix proteins and insertion of membrane proteins.

71 alpha-smooth muscle actin and extracellular matrix proteins and is dependent on metabolic reprogramm

72 ylation modulates its interaction with Golgi matrix proteins and is regulated by SIRT2.

73 ons as a receptor for multiple extracellular matrix proteins and its dysfunction leads to a form of m

74 c molecules, including several extracellular matrix proteins and myofibroblast and cell contractility

75 fferentiated ameloblasts synthesizing enamel matrix proteins and odontoblasts expressed the gene.

76 y cytokines, immune mediators, extracellular matrix proteins and oncogene expression.

77 per assembly of RGD-containing extracellular matrix proteins and the correct incorporation and activa

78 folds increase in secretion of extracellular matrix proteins and the reorganization of the matrix fib

79 ients by recruiting fibroblasts that produce matrix proteins and thicken the lamina reticularis.

80 collagen-I, fibronectin (major extracellular-matrix proteins), and alpha-SMA (well-characterized mark

81 g-protein 5, nidogen-1, cartilage oligomeric matrix protein, and insulin-like growth factor binding p

82 on of growth factor signals by extracellular matrix proteins, and activation of self-renewal pathways

83 lease correlates with rapid deacetylation of matrix proteins, and SIRT3 is required for recovery of m

84 Extracellular matrix proteins are biosynthesized in the rough endoplas

85 , where excessive collagen and extracellular matrix proteins are deposited within the wound area, res

86 of cingulins and the other insoluble organic matrix proteins are post-translationally modified by sho

87 ap junctions, soluble factors, extracellular matrix proteins, blood vessels and neural inputs.

88 is issue, Huang et al. show that the spindle matrix protein BuGZ is sufficient to form micron-scale c

89 ammation not only by degrading extracellular matrix proteins but also by controlling the influx of ch

90 ealed that these structures lack peroxisomal matrix proteins but are the sole sites of the major pero

91 duced depletion of Pex1 blocks the import of matrix proteins but does not affect membrane protein del

92 has been shown to bud basolaterally, and the matrix protein, but not glycoprotein, was proposed to me

93 his study, we demonstrate that extracellular matrix proteins can mediate interactions between gp120 a

94 Peroxisomal matrix proteins carry peroxisome-targeting signals that

95 The nuclear matrix protein Cip1-interacting zinc finger protein 1 (C

96 ADAMTS2, 3 and 14), the cartilage oligomeric matrix protein-cleaving enzymes (ADAMTS7 and 12), the vo

97 dependent on induction of the extracellular matrix protein collagen type V alpha 1 (col5a1).

98 rum caused by mutations in the extracellular matrix protein collagen VI.

99 , we show that, similar to the extracellular matrix protein collagen, amyloids of various proteins/pe

100 ed with gene expression of the extracellular matrix proteins, collagen (Col)1a1, Col1a2, and fibronec

101 Histologic analyses of four matrix proteins-collagen I and IV, laminin, and fibronec

102 eagues demonstrate that cartilage oligomeric matrix protein (COMP) acts as a major endogenous plasma-

103 Cartilage oligomeric matrix protein (COMP) functions as a structural componen

104 region of the gene for cartilage oligomeric matrix protein (COMP) leads to pseudoachondroplasia, a s

105 cause misfolding of the cartilage oligomeric matrix protein (COMP).

106 ys of what is likely the membrane-associated matrix protein, contain multiple cores that can be captu

107 The matrix protein contains a motif termed a "late domain" t

108 and they rely exclusively on import of their matrix protein content from the cytosol.

109 Import of peroxisomal matrix proteins, crucial for peroxisome biogenesis, is m

110 The mitochondrial matrix protein cyclophilin D (CypD) is an essential comp

111 nsible for A549 cell rounding, extracellular matrix protein degradation, and IL-8 degradation, additi

112 re, we identified the secreted extracellular matrix protein Del-1 as a component and regulator of the

113 mined the interactions of wild-type (WT) and matrix protein-deleted (DeltaMA) HIV-1 precursor Gag (Pr

114 n to decrease TGF-beta-induced extracellular matrix protein deposition in the kidney in Ang-II induce

115 plied to in vitro cell-based models in which matrix protein deposition reflects the underlying biolog

116 te molecules revealed that the extracellular matrix protein dermatopontin (Dpt) is involved in HSC ma

117 In this study, we show that mitochondrial matrix proteins display surprisingly different dependenc

118 We used dentin matrix protein (Dmp)-1-mediated Ghr knockout (DMP-GHRKO)

119 to the cuticle via the apical extracellular-matrix protein Dumpy (Dp).

120 Bacterial adherence to extracellular matrix proteins (ECMp) plays important roles during host

121 e tissue fibrosis by producing extracellular matrix proteins, efficiently engulf dead cells.

122 ation of proteins, such as the extracellular matrix protein elastin, have not been reported.

123 study, HMPV VLPs containing viral fusion and matrix proteins elicited epitope-specific TCD8s that wer

124 correct spatiotemporal patterning of enamel matrix protein (EMP) expression is fundamental to orches

125 tif, the molecular mechanism by which enamel matrix proteins (EMPs) assemble into the organic matrix

126 Enamel matrix proteins (EMPs) play a role in enamel formation a

127 n between cancer cells and the extracellular matrix proteins, enhancing cell detachment and promoting

128 Affinity for these extracellular matrix proteins explains the striking ability of NP41 to

129 Here, we report that Tiggrin, a matrix protein expressed in the LG, is a specific regula

130 d kidney cell injury involves an increase in matrix protein expression that is only partly alleviated

131 ant impact on inflammatory and extracellular matrix protein expression.

132 t mimic the nanoscale order of extracellular matrix protein fibers and yield suitable electrical char

133 ons in FBN1, which encodes the extracellular matrix protein fibrillin-1.

134 essed by stromal cells and the extracellular matrix protein, fibrinogen.

135 separately have shown that the extracellular matrix protein fibronectin (FN) contributes to functiona

136 e mechanism of assembly of the extracellular matrix protein fibronectin (FN) into elastic, insoluble

137 antly reduced secretion of the extracellular matrix protein fibronectin (FN).

138 atation, we show here that the extracellular matrix protein fibronectin also contributes to the respo

139 to mimic the binding domain of extracellular matrix protein fibronectin and selectively bind to a sub

140 oteolytic cleavage of the host extracellular matrix protein fibronectin by peritoneal cell-derived ma

141 ntal factor, aggregates of the extracellular matrix protein fibronectin perturb the maturation of oli

142 the detector surface with the extracellular matrix protein fibronectin resulted in cell adherence an

143 heir migration depends on C-cadherin and the matrix protein fibronectin.

144 ray detectors printed with the extracellular matrix protein fibronectin.

145 expansion, accumulation of the extracellular matrix proteins fibronectin and type IV collagen, and lo

146 Here we show that the RSV matrix protein forms dimers in solution and in crystals;

147 Matrin3 is an RNA- and DNA-binding nuclear matrix protein found to be associated with neural and mu

148 us alarmins such as fragmented extracellular matrix proteins found in degenerating discs or cartilage

149 adation of aggrecan and cartilage oligomeric matrix protein from cartilage, fibronectin fragments rat

150 gion of anionic membranes in contrast to the matrix protein from Ebola virus.

151 i requires dimerization of the largest Golgi matrix protein giantin.

152 y impaired dimerization of the largest Golgi matrix protein, giantin.

153 Upon mitotic entry, the Golgi matrix protein GM130 interacts with importin alpha via a

154 s in which adhesion molecules, extracellular matrix proteins, growth factors, and their receptors wor

155 Abs reacting specifically with the influenza matrix protein (IMP)-derived peptide(58-66) displayed by

156 hed primary role of the Pex1/Pex6 complex in matrix protein import and show that peroxisomes in Sacch

157 ation not only facilitates further rounds of matrix protein import but also prevents deleterious PEX5

158 The peroxisomal matrix protein import is facilitated by cycling import r

159 cluding impaired beta-oxidation, inefficient matrix protein import, and decreased growth.

160 ion stabilizes PEX5 and promotes peroxisomal matrix protein import, suggesting that mulibrey nanism i

161 x6 complex, which has an established role in matrix protein import.

162 hat Pex1 and Pex6 play a role in peroxisomal matrix protein import.

163 ulation with growth factors or extracellular matrix proteins in different tumor cells.

164 C was superior amongst several extracellular matrix proteins in enhancing the sphere-forming capacity

165 role of fibronectin and other extracellular matrix proteins in HIV-1 biology.IMPORTANCE Immune tissu

166 rated HMPV VLPs by expressing the fusion and matrix proteins in mammalian cells and tested whether VL

167 that support cooperative functions of enamel matrix proteins in mediating the structural hierarchy of

168 imulation of synthesis of proteins including matrix proteins in podocytes requires integration of the

169 with other membrane transporters or cellular matrix proteins in specialized plasma membrane microdoma

170 ory cytokines, chemokines, and extracellular matrix proteins in the heart.

171 ecular analyses revealed that the removal of matrix proteins in the mutant enamel was drastically del

172 the expression of a subset of extracellular matrix proteins in the skin, including upregulation of e

173 sis involves the production of extracellular matrix proteins in tissues and is often preceded by inju

174 y and promotes the phosphorylation of enamel matrix proteins in vitro and in cells.

175 ated the role of periostin, an extracellular matrix protein, in the pathophysiology of osteoarthritis

176 microenvironment enriched with extracellular matrix proteins including laminin (Ln)-332, produced by

177 endothelial cell integrins bind transitional matrix proteins, including alpha5beta1, alphavbeta3, and

178 e linked by several additional extracellular matrix proteins, including nidogen and perlecan (Figure

179 sulfide (H2S) inhibits high glucose-induced matrix protein increase by activating AMPK in renal cell

180 Tenascin-C, a matrix protein induced upon tissue damage and expressed

181 ctivation by FN, but not other extracellular matrix proteins, induces the release of the granules' co

182 Cartilage oligomeric matrix protein, insulin-like growth factor binding prote

183 nucleocapsid proteins that are important for matrix protein interaction and that are sufficient to di

184 cycling and directed secretion of a specific matrix protein into the fusion-cell interface promote fu

185 s needed to further incorporate other enamel matrix proteins into the system, this study brings us on

186 Oligomerization of the matrix protein is a key step in the process of assembly

187 The VP40 matrix protein is a key structural protein critical for

188 The VP40 matrix protein is a key viral structural protein that is

189 The EBOV VP40 (eVP40) matrix protein is the main driving force for virion asse

190 The destination of peroxisomal matrix proteins is encoded by short peptide sequences, w

191 y of the machinery for import of peroxisomal matrix proteins is that it can accept already folded pro

192 Tenascin-C (TnC), an extracellular matrix protein, is transiently expressed during tissue i

193 ective on how mutations in the extracellular matrix protein laminin cause severe consequences in glia

194 stored the ability to bind the extracellular matrix proteins laminin and cellular fibronectin.

195 kininogen (HK), as well as the extracellular matrix proteins laminin and collagen V.

196 poptosis and the generation of extracellular matrix proteins leading to fibrosis/cirrhosis.

197 p55 processes several imported mitochondrial matrix proteins leading to their stabilization.

198 Because of their remarkably long half-life, matrix proteins, like type I collagen and elastin, are p

199 an spectral changes related to extracellular matrix proteins, lipids, and nucleic acids were tracked

200 presequence peptide degrading enzyme in the matrix.Protein localization plays an important role in t

201 icking of the viral replication machinery, a matrix protein (M) and a glycoprotein, to the plasma mem

202 tural and nonstructural genes, we identified matrix protein (M) as the virus' most potent antihost pr

203 viral ribonucleoprotein (RNP) core with the matrix protein (M) during budding from the plasma membra

204 Oligomerization of the matrix protein (M) is a key step in the process of assem

205 , the nucleoprotein (N), phosphoprotein (P), matrix protein (M), and fusion protein (F) were required

206 For NiV the matrix protein (M), the fusion glycoprotein (F) and, to

207 e all epitopes in the nucleoprotein (NP) and matrix protein (M1) with experimentally identified human

208 orresponding to amino acids 37-47 in the PVM matrix protein (M37-47).

209 , and deposit large amounts of extracellular matrix proteins maintaining the structural integrity of

210 Matrix proteins (MAs) play a key role in the transport o

211 e 3' UTR of MATR3, which encodes the nuclear matrix protein Matrin 3, and mouse Matr3 is strongly exp

212 those areas of platelet extension beyond the matrix protein micropatterns.

213 metalloprotease MMP-10 and the extracellular matrix protein mindin (encoded by Spon2) in the diseased

214 can be caused by the deficiencies of enamel matrix proteins, molecules responsible for the transport

215 Proteomics revealed multiple matrix proteins not previously associated with injured s

216 main-containing protein 5a, an extracellular matrix protein of the brain.

217 In these studies, we identified the matrix protein of the deadly fish novirhabdovirus VHSV a

218 nificantly drive production of extracellular matrix proteins or alpha-smooth muscle actin.

219 HIV-1 matrix protein p17 (p17) has been detected in autoptic b

220 human immune deficiency virus type 1 (HIV-1) matrix protein p17 (p17), although devoid of a signal se

221 in Tg26 tumors, and hypothesized that HIV-1 matrix protein p17 may directly induce RAG1 in B cells.

222 proteins examined, only expression of HIV-1 matrix protein p17 was associated with leukemia/lymphoma

223 The EBOV VP40 (eVP40) and MARV VP40 (mVP40) matrix proteins play a central role in virion assembly a

224 Extracellular matrix proteins play an integral role in modulating vasc

225 The EBOV VP40 matrix protein plays a central role in late stages of vi

226 Periostin, an extracellular matrix protein, plays key role in cell adhesion and moti

227 Most peroxisomal matrix proteins possess a C-terminal targeting signal ty

228 CXCL16 deficiency attenuated extracellular matrix protein production and suppressed bone marrow-der

229 signaling pathways and reduced extracellular matrix protein production, and blocked myofibroblast dif

230 t activation and proliferation and increased matrix protein production.

231 ence that tenascin-C (TNC), an extracellular matrix protein prominent in malignant glioma, increases

232 Netrin-1, a chemorepulsant, laminin-like matrix protein, promotes inflammation by preventing macr

233 A Tyr-10 deletion in the matrix protein provided escape from broad neutralization

234 Vibrio exopolysaccharides (VPS) and the matrix proteins RbmA, Bap1 and RbmC are required for the

235 Pase facilitates recycling of the peroxisome matrix protein receptor PEX5 and is the most commonly af

236 l adhesion molecule L1 and the extracellular matrix protein Reelin play crucial roles in the developi

237 myofibroblasts, which secrete extracellular matrix proteins responsible for the fibrotic scar.

238 We previously found that the extracellular matrix protein secreted protein acidic and rich in cyste

239 It is known that shell matrix proteins (SMPs) play important roles in shell for

240 uces lymphoma cell adhesion to extracellular matrix proteins, subcutaneous tumor size in nude mice, a

241 osis (ie, deposition of excess extracellular matrix proteins such as collagen).

242 MC) activation with pathologic deposition of matrix proteins such as collagen.

243 tion of S. aureus surface proteins with host matrix proteins such as fibrin initiates agglomeration;

244 Fibrogenesis encompasses the deposition of matrix proteins, such as collagen I, by hepatic stellate

245 ated in vitro with immobilized extracellular matrix proteins, such as fibronectin (FN), collagen, and

246 othelial cell interactions with transitional matrix proteins, such as fibronectin, occur early during

247 Collagen and extracellular matrix proteins, such as heat shock protein-47 (markers

248 Basement membrane matrix proteins, such as matrigel, are able to improve t

249 ion, reactive oxygen species generation, and matrix protein synthesis by inhibiting AMP-activated pro

250 yofibroblasts, responsible for extracellular matrix protein synthesis, and the macrophages infiltrati

251 th the tasA gene (encoding the major biofilm matrix protein TasA) and the pgsBCA cluster (responsible

252 m either PDLN or mPIN used the extracellular matrix protein Tenascin-C (TNC) to inhibit T-cell recept

253 Deposition of the extracellular matrix protein tenascin-C is part of the reactive stroma

254 interaction determinants for PopZ, a bipolar matrix protein that anchors the ParB centromere-binding

255 Fibronectin (FN) is an extracellular matrix protein that can be assembled by cells into large

256 Fibronectin (FN) is an extracellular matrix protein that can be assembled by cells into large

257 induced protein (TGFBIp) is an extracellular matrix protein that has a role in a wide range of pathol

258 a unique endothelial specific extracellular matrix protein that has been implicated in angiogenesis

259 ced fibronectin expression, an extracellular matrix protein that is increased in diabetic nephropathy

260 sphoprotein (DSPP) is a dentin extracellular matrix protein that is processed into dentin sialoprotei

261 TIMP3 and vitronectin are 2 extracellular matrix proteins that abnormally accumulate in Notch3(ECD

262 logenin and ameloblastin are 2 extracellular matrix proteins that are essential for the proper develo

263 ralized tissues is governed by extracellular matrix proteins that assemble into a 3D organic matrix d

264 of inflammatory molecules and extracellular matrix proteins that contribute to diseases such as asth

265 development as a self-organizing assembly of matrix proteins that control crystallite habit.

266 roduced fibronectins and other extracellular matrix proteins that copurified with gp120.

267 ses, natural aGPCR ligands are extracellular matrix proteins that dissociate the NTF to reveal the te

268 tile functions and secrete the extracellular matrix proteins that form this fibrous scar.

269 show that starved human fibroblasts secrete matrix proteins that maintain the growth of starved mamm

270 factors including enzymes and extracellular matrix proteins that promote or inhibit hydroxyapatite c

271 igned an approach to target an extracellular matrix protein, the fibronectin extra domain A isoform (

272 (+),K(+)-ATPase alpha-isoforms with cellular matrix proteins, the cytoskeleton, or other membrane pro

273 his peptide interacts with the extracellular matrix protein thrombospondin 4 (TSP4), and antibodies t

274 lasts that excessively deposit extracellular matrix proteins, thus compromising lung architecture and

275 estigate the adsorption properties of enamel matrix proteins to bone grafts after surface coating wit

276 ceived difficulty in modifying extracellular matrix proteins to create aGPCR probes, we developed a s

277 After delivering the matrix proteins to the organelle, these receptors are re

278 enzymes, regulatory proteins, extracellular matrix proteins, transcription factors, and transmembran

279 12-mediated degradation of the extracellular matrix proteins tropoelastin and fibronectin in the tiss

280 r interacting with laminin, an extracellular matrix protein ubiquitously expressed in the respiratory

281 and migration on periostin, an extracellular matrix protein upregulated in asthma by type 2 immunity

282 attern the presentation of the extracellular matrix protein vitronectin, we accomplished reversible d

283 The MARV matrix protein VP40 (mVP40) underlies the inner leaflet

284 arries seven genes, one of which encodes its matrix protein VP40 (mVP40), which regulates the assembl

285 MARV matrix protein VP40 is essential for assembly and releas

286 Ebola virus GP1,2, the Ebola virus matrix protein VP40, and BST2 are at least additive with

287 One of the seven MARV genes encodes the matrix protein VP40, which forms a matrix layer beneath

288 Central to the Ebola life cycle is the matrix protein VP40, which oligomerizes and drives viral

289 e (PS) regulates the assembly of Ebola virus matrix protein VP40.

290 We found that the Ebola virus matrix protein, VP40, and envelope glycoprotein, GP, eac

291 s (wild-type and attenuated VSV with mutated matrix protein [VSVm] versions) that express either the

292 Although the expression of multiple enamel matrix proteins was down-regulated in the mutant amelobl

293 rface of bone grafting materials when enamel matrix proteins were delivered in either a liquid formul

294 d fibronectin (Fn) are two key extracellular matrix proteins, which are known to interact and jointly

295 Laminins are extracellular matrix proteins, widely expressed but also known to be i

296 This strategy labels matrix proteins with high resolution, without compromisi

297 ialized cell, the ameloblast, which secretes matrix proteins with little homology to any other known

298 e impeded because of preceded depositions of matrix proteins within the corneal stroma and the stroma

299 The arenavirus matrix protein Z is highly multifunctional and occurs in

300 The arenavirus matrix protein Z is multifunctional, with at least four