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1 resection of viable disease (GCT other than mature teratoma).
2 est and intrinsic chemotherapy resistance of mature teratomas.
3 1.34 [mean +/- SD]) was higher than that of mature teratoma (1.38 +/- 0.71) and necrosis or scar (1.
4 ely concordant allelic loss patterns between mature teratoma and all of the other germ cell tumor com
5 y analysis, we investigated the clonality of mature teratoma and its relationship to other components
6 the kinetic rate constants K1 and K between mature teratoma and necrosis or scar as follows: K1, 0.1
8 al interpretation nor SUVlean differentiated mature teratoma from necrosis or scar, there were statis
11 hat the stromal cells adjacent to metastatic mature teratoma in postchemotherapy lymph node specimens
12 teratoma of the ovary (six mature) and one a mature teratoma in the mediastinum; five of five tumors
15 nd risk factors for relapse of children with mature teratoma (MT) and immature teratoma (IT) to assis
16 neoplastic cysts (n = 5; two endometriomas); mature teratoma (n = 3); hydrosalpinx (n = 2); fibroma (
17 could easily be differentiated visually from mature teratoma (n = 6) and necrosis or scar (n = 10).
21 toneal lymph nodes that contained metastatic mature teratoma with "fibrosis" to determine the reactiv
22 Our data support the common clonal origin of mature teratoma with other components of malignant mixed
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