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1 have agenesis of >/=1 third molars and that maxillary agenesis was 36% more likely than mandibular a
2 outcome and clinical performance of anterior maxillary all-ceramic implant crowns (ICs) based either
6 how adult endites, the inner branches of the maxillary and labial appendages, are formed at metamorph
8 opeltus giant is a canonical gap gene in the maxillary and labial segments and also plays a gap-like
9 During analysis of CT studies we evaluated maxillary and mandibular alveolar processes for presence
11 tions and less precise axonal sorting of the maxillary and mandibular branches within the trigeminal
12 eural crest cells (NCCs) that develop in the maxillary and mandibular buds of pharyngeal arch 1 (PA1)
13 L) injected into the gingival tissue (GT) of maxillary and mandibular first molars and into the inter
14 of multifocal melanoacanthoma affecting the maxillary and mandibular gingiva and the hard palate.
15 ange in crestal bone levels (AvBL) for 5 x 8 maxillary and mandibular implants after 5.9 years of fol
18 correlated with the dramatic differences in maxillary and mandibular molar phenotypes in Bmp4(f/f);W
19 ects of loss of activin or Bmp4 signaling on maxillary and mandibular molar tooth morphogenesis are m
21 , are expressed in the ectomesenchyme of the maxillary and mandibular processes and have been suggest
22 2, Dlx2, Msx1, Barx1, Foxc2 and Fgf8, in the maxillary and mandibular processes of the mutants, indic
23 sions arise along the sites of fusion of the maxillary and mandibular prominences early in facial dev
26 abnormalities such as mandibular hypoplasia, maxillary and mandibular skeletal deformation, and cleft
30 vent plaque removal during brushing over one maxillary and one mandibular posterior dental sextant fo
31 ome conformation capture assay in developing maxillary and periocular tissues suggest that the DNA re
33 males were, in descending order, mandibular, maxillary, and sphenoid bones, while the sphenoid bone w
35 from four study centers, each with a single maxillary anterior Miller Class I or II recession defect
36 gingiva and underlying alveolar bone in the maxillary anterior region and to establish their associa
37 posterior non-pit and fissure surfaces, (C4) maxillary anterior surfaces, and (C5) mid-dentition surf
43 ome-wide map of candidate enhancers from the maxillary arch (primordium for the upper jaw) of mouse e
44 essential for the expression of Lhx8 in the maxillary arch cells and that Lhx8_enh1 was a direct tar
45 had higher rates of dental anomalies in the maxillary arch than did controls for primary (21% vs. 4%
48 derlying neuroepithelium, the mandibular and maxillary arches, including both their mesenchymal and e
50 vestigated peripheral axons of the zebrafish maxillary barbel (ZMB), an optically clear sensory appen
52 Destruction and invasion of mandibular and maxillary bone frequently occurs and contributes to morb
53 years; mean age: 58 +/- 9.2 years) requiring maxillary bone reconstruction prior to implant placement
55 revealed that 2 lesions were situated in the maxillary bone, 2 in the frontal, and 1 in the zygoma, a
57 We have used a rat model of injury to the maxillary branch (V2) of the trigeminal nerve to produce
58 e subsequently underwent embolization of the maxillary branch of the left ECA using Embozene(R) Micro
60 oth recession defects, oral sites other than maxillary canine and premolar teeth, and Miller Class II
63 phy-based finite element models comprising a maxillary central incisor socket and 4.5 x 13 mm outer-d
64 issing the mandibular second molar and their maxillary central incisors are most susceptible to micro
65 involved making semilunar incisions over the maxillary central incisors that blended into a frenectom
66 Complete root coverage was achieved over the maxillary central incisors that initially presented with
67 ed flap with a frenectomy in a case in which maxillary central incisors were impinged upon by a broad
68 sitioning gingiva for root coverage over the maxillary central incisors while simultaneously performi
69 us surface involvement being highest for the maxillary central incisors, followed by maxillary poster
73 able outcomes after regenerative therapy for maxillary Class III furcation defects are limited to cli
74 Evidence supporting regenerative therapy in maxillary Class III furcation defects in maxillary molar
75 ll have a modified piercing-sucking mandible-maxillary complex that allows them to drain fluids from
81 ere characterised in both the mandibular and maxillary dentition by a loss of the permanent canines,
85 pulsed electrical currents to ophthalmic and maxillary divisions of the right trigeminal nerve and ce
87 hibited a significantly higher percentage of maxillary epithelium in contact with mandibular epitheli
88 l display in the presence of slight vertical maxillary excess and hypermobility of the upper lip.
89 nt of a gummy smile associated with vertical maxillary excess and hypermobility of the upper lip.
95 ogically and clinically for the treatment of maxillary facial or interproximal and mandibular facial
96 ed regenerative therapy for the treatment of maxillary facial, mesial, distal, and mandibular facial
99 e of a lambdoidal junction (formed where the maxillary first arch meets the frontonasal processes) in
100 e, age-dependent expansion of the PDL at the maxillary first molar (M1) furcation area was observed.
101 ed into 4 groups: 50-cN force applied to the maxillary first molar (O), force application plus soft t
103 the palatal gingival tissues adjacent to the maxillary first molars three times per week for 4 weeks.
107 to the cemento-enamel junction (CEJ) of the maxillary fourth premolar (PM4; thin bone over root); 2)
110 We found that, in the turtle, mandibular and maxillary ganglion neuron rostrocaudal segregation and t
117 Noggin(-/-) mice exhibited a solitary median maxillary incisor that developed from a single dental pl
118 cells rescues the development of molars and maxillary incisor, but the rescued teeth exhibit a delay
119 incisive canal and the roots of the central maxillary incisors should be kept in mind during dental-
123 at FPD treatment in posterior mandibular and maxillary jaws with NDIs was as reliable as with SDIs, a
126 two maxillary left second premolars and one maxillary left first molar occurs after sinus-augmentati
132 is case series, loss of pulp vitality of two maxillary left second premolars and one maxillary left f
133 Among treated patients the CoA segment (the maxillary length) and the ANB angle (the antero-posterio
135 mandibular arch-derived structures into more maxillary-like structures, indicating a loss of NCC iden
137 one impressions were taken of mandibular and maxillary master casts, and eight stone replicas of each
138 and Taf1 together in primary mouse embryonic maxillary mesenchymal cells results in up-regulated oste
139 c differentiation in primary mouse embryonic maxillary mesenchymal cells, as seen in Tgfbr2 mutant ce
140 ar development arrested at the bud stage and maxillary molar development arrested at the bud-to-cap t
146 abundantly expressed in the mandibular than maxillary molar mesenchyme in wild-type embryos and that
148 the mandibular molar tooth germs while their maxillary molar tooth germs completed morphogenesis.
149 Runx2(-/-) mutant mice, both mandibular and maxillary molar tooth germs progressed to the early bell
150 ly higher levels of Dkk2 than the developing maxillary molar tooth mesenchyme, these data indicate th
151 opmental arrest at the bud stage but allowed maxillary molars and incisors to develop to mineralized
152 in maxillary Class III furcation defects in maxillary molars is limited to clinical case reports.
153 th-old rats were ovariectomized (OVX) or had maxillary molars removed from one side to induce unilate
155 Finite element (FE) models of the first maxillary molars were constructed from muCT scans to cal
160 facial and interproximal Class I defects in maxillary molars; 2) facial and lingual Class I defects
161 interproximal Class II furcation defects in maxillary molars; 4) facial and lingual Class II furcati
162 ar molars; 5) Class III furcation defects in maxillary molars; 6) Class III furcation defects in mand
164 omeres (tritocerebrum, mandibular neuromere, maxillary neuromere, labial neuromere) of the SEZ at all
165 ar ganglion neurons are located rostrally to maxillary neurons, with some intermingling, supporting p
166 Adults scheduled to receive bone grafting in maxillary, non-molar, single-tooth extraction sites were
169 creened the olfactory sensory neurons of the maxillary palp (MP-OSNs) using a large number of natural
170 stematically examine the role of Acj6 in the maxillary palp and in a major subset of antennal ORNs.
172 eptor (Or) gene expression in the Drosophila maxillary palp, one of the two adult olfactory organs.
173 us investigation has primarily concerned the maxillary palp, the simpler of the fly's two olfactory o
178 These neurons project from the antennae and maxillary palps to the antennal lobe (AL) and from the l
181 mandibular patterning defect resulting in a maxillary phenotype (i.e., homeotic transformation).
185 Experimental gingivitis was induced in one maxillary posterior sextant in nine healthy individuals.
188 furcation defects were surgically created in maxillary premolar teeth in adult, female, mini-pigs and
195 ateral and medial nasal processes and of the maxillary process of the first branchial arch are integr
197 ficantly retarded outgrowth of the nasal and maxillary processes due to reduced proliferation of mese
198 y outgrowth from the oral side of the paired maxillary processes flanking the primitive oral cavity.
200 ls (activates beta-catenin pathway) into the maxillary prominence or by knocking down endogenous WNT1
201 RCAS::WNT11 retrovirus was injected into the maxillary prominence, and the majority of embryos develo
202 which EDNRA is ectopically activated in the maxillary prominence, resulting in a maxillary to mandib
203 aled that WNT11 prevented lengthening of the maxillary prominence, which was due in part to decreased
204 shelves from the oral side of the embryonic maxillary prominences, elevation of the initially vertic
205 bilateral palatal shelves (PS), arising from maxillary prominences, fuse at the midline, forming the
206 elop as outgrowths from the medial nasal and maxillary prominences, respectively, remodel and fuse to
208 e percentage of TLR4-positive neurons in the maxillary region and among the neurons innervating infla
209 R4 was detected in 19% of the neurons in the maxillary region of TG and in 29% of neurons innervating
212 A is an important structure in the posterior maxillary region; the clinician should be aware of its l
217 bone thickness at 3 mm from the CEJ for the maxillary right central incisor was 1.41 mm and for the
218 was subperiosteally injected into the murine maxillary right diastema between the incisor and the fir
219 red-breeder rats underwent extraction of the maxillary right first molar and standard surgical defect
220 for 5 min/day on the occlusal surface of the maxillary right first molar at a very low magnitude of l
222 xillary left first molars (PDSG and PDCimG); maxillary right molars were used as non-ligature control
224 gatures were placed subgingivally around the maxillary second molars and remained there for 2 weeks b
228 atures (size 7-0) were tied around the mouse maxillary second molars on day 0, followed by the inject
237 are the thickness of the lateral wall of the maxillary sinus (TLWMS) and the thickness of the Schneid
238 riptions of sinus involvement other than the maxillary sinus add to the variability of presentation.
239 All bioglass and/or allograft placed in the maxillary sinus after the osteotome technique underwent
240 al findings and mucosal abnormalities of the maxillary sinus among dental patients, using cone-beam c
241 distances from lower margin to the floor of maxillary sinus and alveolar crest in the 1(st) molar an
243 ery (PSAA) is located on the lateral wall of maxillary sinus and may become injured during such surgi
244 s sites with <10 mm between the floor of the maxillary sinus and the alveolar crest were selected.
245 e meatus cannot be used as a surrogate for a maxillary sinus aspirate in children with ABS, although
247 in bone particles harvested intraorally for maxillary sinus augmentation and to assess the clinical
248 ospective record review was performed of all maxillary sinus augmentation cases performed during the
251 indings suggest that the %VB formation after maxillary sinus augmentation is inversely proportional t
254 rforation is the most common complication of maxillary sinus augmentation procedures and has been ass
256 around implants placed in sites treated with maxillary sinus augmentation using anorganic bovine bone
257 eriodontopathogens in individuals undergoing maxillary sinus augmentation with a history of periodont
260 ient with radio- and chemotherapy refractory maxillary sinus carcinoma to gauge the progression of th
264 to 71.1%) of respondents recommended that a maxillary sinus CT scan should be routinely prescribed b
266 of the present study, it was concluded that maxillary sinus elevation with 100% ABB gives predictabl
268 resent study is to investigate the effect of maxillary sinus floor augmentation on sinus membrane thi
269 s in membrane thickness were evaluated in 65 maxillary sinus floor augmentation procedures via a late
272 the concept that the new bone formed in the maxillary sinus lift procedure emanates from the endoste
276 e from the lateral to the medial wall of the maxillary sinus on the outcomes of sinus augmentation pr
283 of the lateral arterial blood supply to the maxillary sinus) were obtained retrospectively from two
288 bullosa was connected with larger volume of maxillary sinuses (right sinus: p=0.005; left sinus: p=0
294 There was higher incidence of bilateral maxillary sinusitis in patients with septal deviation (p
297 s statistically significantly greater in the maxillary teeth, but the difference was not statisticall
299 in the maxillary prominence, resulting in a maxillary to mandibular transformation, suggesting that
300 healthy adults requiring restoration of one maxillary tooth were randomized in a 1:2 ratio to receiv
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