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1 cidification in HG with reduced steady state/maximal oxygen consumption.
2 mass, adiposity, arterial blood pressure, or maximal oxygen consumption.
3 ts performed a 2 h laboratory ride at 72% of maximal oxygen consumption.
4 training was associated with improvements in maximal oxygen consumption.
5 pe natriuretic peptide (NTproBNP), and lower maximal oxygen consumption.
6 0.9 mL/m(2) [95% CI, -6.1 to 4.3]; P = .73), maximal oxygen consumption (1.0 [95% CI, -0.42 to 2.34];
7 asurements, maximal inspiratory pressure, or maximal oxygen consumption among any of the three groups
8                                         Both maximal oxygen consumption and exercise duration were ma
9 etween groups as assessed by peak work rate, maximal oxygen consumption, and rate pressure product.
10  abnormalities, there were no differences in maximal oxygen consumption between the two groups and al
11           Assessed on the basis of basal and maximal oxygen consumption, both populations avail thems
12 ning, the older and younger groups increased maximal oxygen consumption by 17.8% and 20.2%, respectiv
13                                              Maximal oxygen consumption correlated linearly with maxi
14 ; distance walked; and peak and extrapolated maximal oxygen consumption during a 10-meter shuttle wal
15 ns of insulin, glucose, and triacylglycerol; maximal oxygen consumption (f1.gif" BORDER="0">O(2)max)
16                Twelve males exercised at 50% maximal oxygen consumption for 30 min once before and on
17 7.5 watts (-13 to +44 watts, 46%, p < 0.05), maximal oxygen consumption increased 0.16 L/min (-0.17 t
18  with 7 of 9 patients improving >/=30%), and maximal oxygen consumption increased from 13.6+/-2.9 to
19                                              Maximal oxygen consumption increased from 19.4+/-6.4 to
20                   The age-related decline in maximal oxygen consumption is attenuated by habitual aer
21 ants of endurance exercise performance (i.e. maximal oxygen consumption , lactate threshold and exerc
22 re assessed for markers of disease severity (maximal oxygen consumption, left ventricular ejection fr
23 vels of oxygen use as indicated by basal and maximal oxygen consumption levels that are consistent wi
24 ce angiography (MRA), treadmill testing with maximal oxygen consumption measurement, and a 6-min walk
25 pressure of 16 +/- 9 mm Hg (mean +/- SD) and maximal oxygen consumption of 17.4 +/- 4.3 ml/min per kg
26  endurance-trained males, with a mean +/- SD maximal oxygen consumption of 58.2 +/- 5.3 mL . min(-1),
27                                              Maximal oxygen consumption on a cycle ergometer was exam
28 compared with placebo did not improve LVESV, maximal oxygen consumption, or reversibility on SPECT.
29  outflow tract gradients, exercise times and maximal oxygen consumption peak were similar between the
30 as measured by Doppler echocardiography, and maximal oxygen consumption (peak Vo(2)) and percentage o
31 , capillary density was inversely related to maximal oxygen consumption (r = 0.479, p = 0.02).
32  fat-free mass (r = 0.80-0.87); and AEE with maximal oxygen consumption (r = 0.54), fat-free mass (r
33 nfants who developed BPD or died had a lower maximal oxygen consumption rate (mean +/- SEM, 107 +/- 8
34       To analyze the onset of the pathology, maximal oxygen consumption rate of left ventricular perm
35 ings suggest that the age-related decline in maximal oxygen consumption results from a reversible dec
36         Mice (n = 12/group) completed both a maximal oxygen consumption test(VO2max) test and an endu
37 ehabilitation, there was a trend to a higher maximal oxygen consumption (V O(2)max) (13.3 +/- 3.0 ver
38 lues were calculated at approximately 75% of maximal oxygen consumption (V O2).
39  pressure with maximal sniff (Pdimax sniff), maximal oxygen consumption (V O2max), maximal minute ven
40 unning at intensities eliciting 70 and 100 % maximal oxygen consumption (V(O(2),max)).
41 l of 60 exercise sessions starting at 55% of maximal oxygen consumption (VO(2)max) for 30 min/session
42     For endurance sports three main factors--maximal oxygen consumption (.VO(2,max)), the so-called '
43              Relatives with LVE and abnormal maximal oxygen consumption (VO2max) (defined as VO2max <
44           Aerobic capacity was determined as maximal oxygen consumption (VO2max) (ml/kg/min) and anae
45 uring daily activities, muscle strength, and maximal oxygen consumption (VO2max) in 40 postmenopausal
46              Study objectives were to assess maximal oxygen consumption (VO2max) in TX and controls,
47  46) achieved a mean percentage predicted of maximal oxygen consumption (VO2max) of 76.1% +/- 21.1%.
48                                              Maximal oxygen consumption (VO2max) was estimated by mea
49 ed before and after weight loss and included maximal oxygen consumption (VO2max), resting blood press
50  of high-intensity running [76.7 +/- 0.4% of maximal oxygen consumption (VO2max)].
51     Liver transplantation patients had lower maximal oxygen consumption (VO2max/kg) (37.5 +/- 9.3 mL/
52                                              Maximal oxygen consumption was also restricted under the
53                                              Maximal oxygen consumption was higher (P < 0.001) and re
54 ght may be the best predictor of TEE, and 5) maximal oxygen consumption was the strongest marker of A
55                        Exercise duration and maximal oxygen consumption were not related significantl

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