ライフサイエンスコーパス: may be explained by

1 The hypersecretion of insulin may be explained by a loss of interaction between SCHAD and g

2 This may be explained by a temporal integration process in which w

3 , and less Hg in the muscles compared to adult males, which may be explained by accelerated metabolism during gestation a

4 Our results may be explained by an enhanced homing motivation and possibl

5 We speculate that these changes may be explained by an increased solid-like nature of the cyt

6 tion of the trait heritabilities, and additional variations may be explained by associations with rarer variants with lar

7 that BCD inhibits CC-induced inflammatory responses, which may be explained by BCD-mediated attenuation of complement ac

8 roup, this difference was not statistically significant and may be explained by chance.

9 asing incidence of autoimmune diseases in western countries may be explained by changes in early microbial exposure, lead

10 dence of cellulitis is highly seasonal and this seasonality may be explained by changes in the weather, specifically, tem

11 The loss of multi-host parasites from threatened ungulates may be explained by decreased cross-species contact as hosts

12 Initial fluctuations in estimates may be explained by delayed healthcare contact due to the los

13 This effect may be explained by deposition of semiconducting iron oxide p

14 These results may be explained by different functional maturation due to cu

15 hesis by polymer simulations and find that TAD partitioning may be explained by different modes of inter-nucleosomal inte

16 ce in activation of native, not recombinant kappa receptors may be explained by different mouse/human receptor structures

17 Differences in developmental kinetics may be explained by differential regulation of epigenetic mod

18 The exceptional pattern may be explained by females experiencing unobserved neonatal

19 rtex, which is strong in wild-type mice but weak in humans, may be explained by FEZF2-mediated cis-regulatory elements th

20 Variability among studies may be explained by geography and patient factors.

21 d cardiovascular mortality in patients with diabetes, which may be explained by hypoglycemia-induced inflammation.

22 Unexplained cardiac arrest (UCA) may be explained by inherited arrhythmia syndromes.

23 We argue that the pattern diversity and reproducibility may be explained by interpreting the pattern development as a

24 he established association of APOE genotype with CVD events may be explained by isoform-specific functions as well as oth

25 Thus ISE2's evolutionary conservation may be explained by its numerous roles in regulating chloropl

26 t discrepancy between development of structure and function may be explained by life-long synaptic changes in human V1.

27 these melanomas is likely due to tumor heterogeneity, which may be explained by models such as the stochastic, hierarchic

28 ip cannot be ruled out, the previously observed association may be explained by other factors.

29 th that from family studies, a fraction of the heritability may be explained by other genetic variants, such as rare vari

30 The observed "abnormalization" of the vessels may be explained by periodic pruning of angiogenic vascular s

31 This may be explained by phosphorylation of the phosphatase PTEN a

32 Variations in attrition may be explained by program director attitudes, although larg

33 ults indicate that the association with chr5p15.33-Region 2 may be explained by rs36115365, a variant influencing TERT ex

34 es in both reaction center I (RC-I) and RC-II-type bacteria may be explained by selective loss of either RC in the evolut

35 Higher risk of virological failure among PWUD with TDR may be explained by some inappropriate ART prescribing, as we

36 g infected than healthy male locusts, and propose that this may be explained by terminal reproductive effort and a lowere

37 These influences may be explained by the functions of solubilized as well as i

38 tection to tadpoles and the A6 cell line than rXlIFN, which may be explained by the FV3 impairment of IFN-lambda receptor

39 The favorable effects of fiber may be explained by the generation and distribution of one of

40 The grouping of CD8alpha(+) cells may be explained by the greater expression of CCL19, ICAM-1,

41 disease is a possible risk factor for cognitive impairment may be explained by the inflammatory hypothesis.

42 icochemical properties with EcoSSB and that the differences may be explained by the lack of an acidic, disordered C-termi

43 Insensitivity to NLP cytolysins of monocot plants may be explained by the length of the GIPC head group and the

44 I propose that this paradox may be explained by the pervasive use of glycolysis by somati

45 In our case, the HMB-45 negativity may be explained by the rarity of the epithelioid cells, and

46 The discordant results may be explained by the recent discovery that NspA is a human

47 suggest that signalling specificity between these pathways may be explained by the spatial separation of FLS2 and BRI1 w

48 A remarkable improvement with fever may be explained by the temperature-dependent leftward shift

49 This may be explained by the undetectable levels of RNase L as wel

50 bservations suggest that the prevalence of vertebrate uORFs may be explained by their conserved role in repressing CDS tr