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1 genous generation of interstrand cross-links in genomic DNA may contribute to aging, neurodegeneration, and cancer.
2 g NF-kappaB and Nrf2 signaling pathways in the retina which may contribute to ameliorating retinal damage induced by HFD.
3 2 suppresses vasculogenesis in endometriotic lesions, which may contribute to an impaired lesion vascularization and grow
5 ggest that cell autonomous changes in both neurons and glia may contribute to C9orf72-mediated disease, as has been shown
6 nd Meg-01 cells express the MS cation channel Piezo1, which may contribute to Ca(2+) entry and thrombus formation under a
7 Thus, B1 receptor-positive endothelial microvesicles may contribute to chronic inflammation by inducing neutrophil
9 of the environmental cues that regulate FCSC fate decisions may contribute to deciphering the mechanisms underlying the r
10 both aortic smooth muscle cells and adventitial fibroblasts may contribute to development of TAAD and proliferative occlu
11 red in RRMS patients, especially in low-BMI patients, which may contribute to disease progression in these patients.
12 xcitability in the dentate gyrus of Pafah1b1(+/-) mice that may contribute to epilepsy or cognitive impairments associate
13 nous FA, raising the possibility that epigenetic mechanisms may contribute to FA-mediated carcinogenicity.
14 ion of AD-related signaling pathways through this mechanism may contribute to female vulnerability to AD.
15 ired in sensory systems in DS model mice, that such defects may contribute to functional impairment in DS, and that these
16 e loads modulate N2O accumulation in denitrification, which may contribute to further design strategies to control greenh
17 e the anomalous current declines with postnatal age, PIEZO2 may contribute to hair cell development, but it does not unde
18 n, suggesting that NK cells, but not HCV adaptive immunity, may contribute to HCV viral control following RG-101 therapy.
19 riction combined with an elevated thromboxane A2 production may contribute to impaired functional dilator and hyperaemic
20 associated with increased hemolytic activity in ST3081 and may contribute to increased virulence in this clone.
21 Type I IFN-mediated neutrophil activation and NET formation may contribute to inflammatory manifestations observed in pat
23 that the altered response of B cells in tolerant recipients may contribute to long-term stable graft acceptance.
24 transcription in dorsal root ganglion (DRG) neurons, which may contribute to nerve injury-induced neuropathic pain.
25 inappropriate activation of the integrated stress response may contribute to pathogenesis in a subset of neurodegenerati
27 Few data support this standard practice, which may contribute to perioperative fluid overloading.
28 zed that selective exclusion of molecules needed for growth may contribute to regeneration decline.
29 h risk of muscle loss and impaired physical function, which may contribute to sarcopenic obesity.
30 his direct interaction between RNA polymerase and ribosomes may contribute to the coupling of transcription to translatio
31 an early deficiency in beta-cell number in infants with CF may contribute to the development of glucose intolerance in t
32 suggest that the level of expression of restriction factors may contribute to the differential susceptibility of CD4(+) T
34 ure of neuroinflammatory disease and that sulfatide in APCs may contribute to the endogenous pathway of iNKT cell activat
35 The IPS plays a key role in attention orienting and may contribute to the hypervigilance that is a common symptom
36 resolving) and granulocyte-M-CSF (GM-CSF; proinflammatory) may contribute to the inconsistency of FDG vessel wall inflam
37 sulin release and beta cell survival, and their dysfunction may contribute to the loss of functional beta cell mass in di
38 ced mast cell degranulation by PgLPS1690 and PgLPS1435/1449 may contribute to the modulation of disease progression.
39 robial-driven expression of IL-19 by intestinal macrophages may contribute to the pathogenesis of inflammatory bowel dise
40 oliferation and neuroinflammation in the adult hypothalamus may contribute to the pathogenesis of obesity.
41 does not provide a sustained benefit and that Ly6C(low) MPs may contribute to the progressive fibrosis and dysfunction of
42 arent compound's psychotropic and physiological effects and may contribute to the toxicity profile.
43 N-driven serotonergic control of CRF levels in the amygdala may contribute to the transition from moderate to compulsive
44 rrelated to CO2 in daytime, suggesting that human occupants may contribute to their abundance either through direct emiss
45 How LBBB-related effects on LV diastolic function may contribute to those therapeutic failures has not been cla
47 ripheral changes in AMD and their impact on visual function may contribute to understanding AMD pathogenesis.
48 rum 25(OH)D concentration, and that polymorphism rs11185644 may contribute to variation in 25(OH)D dose-response in healt
49 r data suggest that polymorphisms in the CYP2R1 and GC gene may contribute to variation in baseline serum 25(OH)D concent
50 lls, epigenetic downregulation of HLA-E by high-risk HPV E7 may contribute to virus-induced immune evasion during HPV per
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