1 ntin to 4-keto-6-deoxy glucose, resulting in maysin.

2 s mutation has been linked to the absence of maysin.

3 ropanoids and C-glycosyl flavones related to maysin.

4 a (Boddie) in maize (Zea mays L.) because of maysin, a C-glycosyl flavone synthesized in silks via a

5 ntitative trait locus) determining levels of maysin, a C-glycosyl flavone that deters feeding by corn

6 terized pathway, namely the concentration of maysin, a C-glycosyl flavone, in silks of maize, Zea may

7 netic mechanisms underlying the synthesis of maysin and apimaysin and the corresponding effects on co

8 nguished by their B-ring substitutions, with maysin and apimaysin being the di- and monohydroxy B-rin

9                                          The maysin and apimaysin QTLs were significant QTLs for corn

10 n of both p1 and p2 have non-detectable silk maysin and non-browning silks.

11 ted for 55.3% of the phenotypic variance for maysin, and a QTL, pr1, which explained 64.7% of the phe

12       We conclude that both p1 and p2 induce maysin biosynthesis in silk, although the two genes diff

13  of Sm1 and Sm2 described here completes the maysin biosynthetic pathway, providing powerful tools fo

14 e action of this region was dominant for low maysin, but was only expressed in the presence of a func

15                           We determined silk maysin concentrations and restriction fragment length po

16                                              Maysin is a C-glycosyl flavone that, when present in sil

17                                              Maysin is a host-plant resistance factor against the cor

18 hat are deleted for the p1 gene have reduced maysin levels and moderate silk-browning reaction, where

19                                          The maysin QTL did not affect apimaysin synthesis, and the a

20 reater understanding of the genetic basis of maysin synthesis and associated corn earworm resistance

21 thesis, and the apimaysin QTL did not affect maysin synthesis, suggesting that the synthesis of these