ライフサイエンスコーパス: mean arterial pressure

1 hood systolic or diastolic blood pressure or mean arterial pressure.

2 ized this exposure to a 5 mm Hg reduction in mean arterial pressure.

3 The primary outcome was 24-hour mean arterial pressure.

4 required norepinephrine to maintain adequate mean arterial pressure.

5 Our primary outcome was 24-hour mean arterial pressure.

6 l hypertension but without MetS for the same mean arterial pressure.

7 al patients in whom norepinephrine increased mean arterial pressure.

8 lone or when compared with MetS for the same mean arterial pressure.

9 handgrip exercise, paralleling increases in mean arterial pressure.

10 were adjusted for age, body mass index, and mean arterial pressure.

11 prevented the decreases in cardiac index and mean arterial pressure.

12 modynamic variables such as cardiac index or mean arterial pressure.

13 s partially accounted for by tobacco use and mean arterial pressure.

14 ke volume, cardiac output and a reduction in mean arterial pressure.

15 and evoke a pressor reflex known to increase mean arterial pressure.

16 d provides prognostic utility beyond that of mean arterial pressure.

17 stolic BP, diastolic BP, pulse pressure, and mean arterial pressure.

18 se cardiovascular outcomes, independently of mean arterial pressure.

19 ds and a norepinephrine infusion to maintain mean arterial pressure.

20 significantly associated with diastolic and mean arterial pressures.

21 found this correlated with post-brain death mean arterial pressures.

22 , waist circumference (-1.1 to -1.9 cm), and mean arterial pressure (0.0 to -1.1 mm Hg) at 6 months a

23 [95% CI -0.01, 0.03]; p = 0.36; n = 32,961); mean arterial pressure (-0.06 mm Hg [95% CI -0.19, 0.07]

24 the only two groups that gradually increased mean arterial pressure 1.6-fold from 38-39 mm Hg to 52 a

25 it, -8.7, -5.1]; p(group) < 0.0001), similar mean arterial pressure (-1.1 mm Hg [95% confidence limit

26 -1.60 mm Hg; 95% CI: -2.77, -0.43 mm Hg) and mean arterial pressure (-1.64 mm Hg; 95% CI: -3.27, -0.0

27 a blunted increase compared with controls in mean arterial pressure (10+/-1 versus 14+/-1 mm Hg, P=0.

28 g adequate intravascular volume and adequate mean arterial pressure (1C); avoiding use of intravenous

29 phrine by 0.04 +/- 0.02 mug.kg.min increased mean arterial pressure 20 mm Hg in all patients.

30 ur fractures/lung contusion, P), hemorrhage (mean arterial pressure 25-30 mm Hg, H), polytrauma plus

31 output 40% to 48% of baseline), hypotension (mean arterial pressure 27-31 mm Hg), and acidosis (pH 7.

32 amounts of crystalloid to maintain adequate mean arterial pressure (2C) and the avoidance of hetasta

33 Mice underwent 90 minutes of shock (mean arterial pressure 30 mm Hg) and resuscitation via f

34 essure (23+/-2 vs. 10+/-3 mmHg, p=0.006) and mean arterial pressure (37+/-1 vs. 24+/-2 mmHg, p=0.006)

35 o (7.7 +/- 3.1, p = 0.04), and a decrease in mean arterial pressure (4.6 +/- 2.3 mm Hg, p = 0.02).

36 caine, and Mg generated significantly higher mean arterial pressure (48 mm Hg [95% CI, 44-52] vs 33 m

37 e (-17 mm Hg; 95% CI, -25 to -8; p < 0.001), mean arterial pressure (-7 mm Hg; 95% CI, -12 to -1; p =

38 hemodynamic parameters except a decrease in mean arterial pressure (-7 mm Hg; p = 0.041) and in syst

39 dynamic variables were relatively preserved (mean arterial pressure 70 [65-77] mm Hg, cardiac index 3

40 line, patients with RAI presented with lower mean arterial pressure (76 +/- 12 versus 83 +/- 14 mmHg,

41 (23 +/- 8 vs 17 +/- 7; P < .0001) and lower mean arterial pressure (81 +/- 16 vs 85 +/- 15 mm Hg; P

42 CPAP reduced baseline mean arterial pressure (94 +/- 2 vs. 89 +/- 2 mm Hg, P =

43 Endotoxemia had no influence on mean arterial pressure (95 [74-103] mm Hg vs. 92 [78-104

44 5% CI: 0.4, 1.7; p = 0.001), 0.8-mmHg higher mean arterial pressure (95% CI: 0.2, 1.4; p = 0.01), and

45 /spexin produced a 13 +/- 2 mmHg increase in mean arterial pressure, a 38 +/- 8 bpm decrease in heart

46 were treated to normalize central venous and mean arterial pressure, additional management to normali

47 2) a significant (>10%) increase in invasive mean arterial pressure after a cardiovascular interventi

48 In contrast, an elevated mean arterial pressure along with increased central and

49 Saline infusion increased mean arterial pressure and cardiac output.

50 gnancy-induced hypertension, ouabain reduced mean arterial pressure and enhanced placental HSP27 phos

51 Cardiac work was estimated as the product of mean arterial pressure and heart rate.

52 DD and without LVDD, had significantly lower mean arterial pressure and higher Model for End-Stage Li

53 The dynamic information in continuous mean arterial pressure and intracranial pressure monitor

54 eries summary statistics of minute-by-minute mean arterial pressure and intracranial pressure.

55 istic Organ Dysfunction-2 score now includes mean arterial pressure and lactatemia in the cardiovascu

56 An inverse relationship between mean arterial pressure and mortality was identified (p =

57 Low mean arterial pressure and need for high doses of vasopr

58 ular conductance (CVC) was calculated as LDF/mean arterial pressure and normalized to maximal values

59 C) was calculated as laser-Doppler flowmetry/mean arterial pressure and normalized to maximum.

60 on increased cardiac index, whereas reducing mean arterial pressure and peripheral vascular resistanc

61 Mean arterial pressure and PP were continuously recorded

62 ith increased risk for incident CVD, whereas mean arterial pressure and relative wave reflection (cor

63 ally inhibited the effects of vasopressin on mean arterial pressure and significantly reduced the eff

64 s, ethyl gallate and norepinephrine improved mean arterial pressure and stroke work to similar extent

65 Mean arterial pressure and stroke work were significantl

66 lated the 0- to 6-hour time-weighted average mean arterial pressure and used multivariable logistic r

67 s per minute; P<0.01) despite a reduction in mean arterial pressure and was inversely related to puls

68 ion between increasing time-weighted average mean arterial pressures and good neurologic outcome, def

69 ic and diastolic blood pressure, 22 SNPs for mean arterial pressure, and 10 SNPs for pulse pressure)

70 bpm decrease in heart rate with no change in mean arterial pressure, and a marked increase in urine f

71 raction, even after adjustment for sex, age, mean arterial pressure, and BMI.

72 on isolated aortic rings, cardiac function, mean arterial pressure, and both the renal vascular perf

73 These results were adjusted for age, sex, mean arterial pressure, and cardiovascular risk factors.

74 ological variables: central venous pressure, mean arterial pressure, and either central venous oxygen

75 nt CVD with forward pressure wave amplitude, mean arterial pressure, and global reflection coefficien

76 sity lipoprotein cholesterol, triglycerides, mean arterial pressure, and glucose were in the bottom 3

77 as a percentage of total blood volume (TBV), mean arterial pressure, and heart rate, which were recor

78 ral biomarkers, such as WBC, oxygen content, mean arterial pressure, and heart rate, yielded estimati

79 actice, uses shock index as a substitute for mean arterial pressure, and incorporates serum lactate a

80 ose-dependently decreased PP, did not modify mean arterial pressure, and increased SVR.

81 itated to normalize central venous pressure, mean arterial pressure, and lactate clearance of at leas

82 Heart rate, mean arterial pressure, and left ventricular work (2-dim

83 = 17) had larger reductions in diastolic BP, mean arterial pressure, and PWV (-2.24 +/- 1.31 mm Hg, -

84 ardia, a modest but significant elevation in mean arterial pressure, and reductions in mean cerebral

85 itated to normalize central venous pressure, mean arterial pressure, and ScvO2 of at least 70%; and t

86 nt, the mice were monitored for albuminuria, mean arterial pressure, and serum autoantibody levels.

87 seen in the control group without affecting mean arterial pressure, and the second bolus of tetrahyd

88 Heart rate, mean arterial pressure, and total peripheral resistance

89 graphy (EEG) power; (3) a modest decrease in mean arterial pressure; and (4) a progressive shift of t

90 , in null mice, the systolic, diastolic, and mean arterial pressures are higher.

91 Every 10 mmHg drop from baseline in mean arterial pressure associated with a 3% increase in

92 sive rats displayed significant reduction in mean arterial pressure associated with attenuation of bo

93 There was no difference in mean arterial pressure at 1, 24, or 48 hours between gro

94 On average, the 24-hour mean arterial pressure at 12 weeks was lower in the grou

95 uent blood removal/retransfusion to maintain mean arterial pressure at 30 mm Hg).

96 uent blood removal/retransfusion to maintain mean arterial pressure at 30 mm Hg).

97 of the blood volume, subsequent titration of mean arterial pressure at 35 mm Hg), anesthetized and in

98 Mean arterial pressure at 6 hours was 72.2 mm Hg in the

99 rent guidelines, which recommend maintaining mean arterial pressure at 85 to 90mm Hg for a week after

100 ary end point was a response with respect to mean arterial pressure at hour 3 after the start of infu

101 ) at OGTT, maternal height at OGTT, maternal mean arterial pressure at OGTT, maternal smoking and dri

102 itation, norepinephrine titrated to maintain mean arterial pressure at preshock values, mechanical ve

103 norepinephrine infusion titrated to maintain mean arterial pressure at preshock values.

104 repinenephrine infusion titrated to maintain mean arterial pressure at preshock values.

105 E, blood pressure targets were not achieved (mean arterial pressure at study end: NE: 81 mm Hg [76-85

106 ynchronized ventilation elicited the highest mean arterial pressure, best oxygenation, and a normal m

107 The association persisted when adjusted for mean arterial pressure (beta=-0.060+/-0.009 SD/allele, P

108 ower systolic BP (beta=-4.11; P=2.8x10(-4)), mean arterial pressure (beta=-3.50; P=8.9x10(-6)), and r

109 was no significant difference in the 24-hour mean arterial pressure between the control group and the

110 At the baseline mean arterial pressure (BLMAP) close to normal, ARCN sti

111 Advancing age and lower mean arterial pressure, but not the presence of a transt

112 roid BF by 25%+/-7%, heart rate by 19%+/-8%, mean arterial pressure by 22%+/-5% (measured at the midd

113 L-NAME increased mean arterial pressure by approximately 17 mm Hg in both

114 Cocaine increased mean arterial pressure (by 14+/-2 mm Hg [mean+/-SE]), he

115 Mean arterial pressure, cardiac output, arterial blood g

116 Mean arterial pressure, cardiac output, cerebral blood f

117 PP, mean arterial pressure, cardiac output, SVR, and ascites

118 ased NOS3 and GUCY1A3 expression and reduced mean arterial pressure, combined them into a genetic sco

119 system with norepinephrine still maintained mean arterial pressure comparable with the left atrium-a

120 e RDN than the intact group (2-month fall in mean arterial pressure: control-intact, -10 +/- 1 mm Hg;

121 Coronary vascular resistance (CVR; mean arterial pressure/coronary blood velocity) was used

122 Mean arterial pressure decreased similarly during endoto

123 Hg and 170 mm Hg of steady-state changes in mean arterial pressure, defined as static CA.

124 ssation, normalization of serum lactate, and mean arterial pressure did not differ among groups.

125 ment for first trimester body mass index and mean arterial pressure, differences in intima thickness

126 Work rate and mean arterial pressure during exercise were similar in c

127 ficantly enhanced the integrated increase in mean arterial pressure during restraint on the first (80

128 mic information of intracranial pressure and mean arterial pressure during the first 24 hrs increased

129 Higher mean arterial pressure during the initial stages of hemo

130 (ASIC) activation and reflexively increases mean arterial pressure; endomorphin release is also incr

131 ociated with hemodynamic instability (higher mean arterial pressure extrema points frequencies were a

132 Episodes of hypotension were common, with mean arterial pressure falling by a median of 22 mmHg (i

133 If, despite fluid management, mean arterial pressure fell by more than 10 mm Hg from b

134 In response to hemorrhage, mean arterial pressure fell in all groups, with the fall

135 ent protocol, we infused P. aeruginosa until mean arterial pressure first decreased to approximately

136 , HS/CR, or HS/CR+MC-2 (HS = 40% of baseline mean arterial pressure for 60 minutes; CR = return of sh

137 found in food substances, could reverse low mean arterial pressure found in an experimental model of

138 V amplitude, and 1 ms pulse width, restoring mean arterial pressure from 0 to 37 mmHg.

139 In 5-year-old female uni-x and sham sheep, mean arterial pressure, glomerular filtration rate, and

140 included the following physiologic targets: mean arterial pressure greater than 70 mm Hg, cerebral p

141 th good neurologic outcome at a threshold of mean arterial pressure greater than 70 mm Hg.

142 ute and requiring norepinephrine to maintain mean arterial pressure greater than or equal to 65 mm Hg

143 This threshold (mean arterial pressure > 70 mm Hg) had the strongest ass

144 as the first-choice vasopressor to maintain mean arterial pressure >/= 65 mm Hg (1B); epinephrine wh

145 Mean arterial pressure >/=65 mm Hg and central venous pr

146 ature management at 33 degrees C with target mean arterial pressure >/=65 mm Hg is associated with in

147 Hemodynamic goals were mean arterial pressure >70 mm Hg; ScvO2 <70%; central ve

148 on and treatment with vasopressors targeting mean arterial pressure (>/=65 mm Hg) and blood transfusi

149 Maintenance of mean arterial pressure>65 mm Hg has been associated with

150 ts with lower baseline diastolic BP (DBP) or mean arterial pressure had more progression of subcortic

151 3) was associated with incident CVD, whereas mean arterial pressure (hazard ratio, 1.10; 95% confiden

152 Mean arterial pressure, heart rate, and lactate were reg

153 We measured mean arterial pressure, heart rate, central venous press

154 and objective physical variables (including mean arterial pressure, heart rate, respiratory rate, an

155 alization (HR: 23.2; P = 0.01), and baseline mean arterial pressure (HR: 0.92; P = 0.01) were found t

156 continuation showed exaggerated increases in mean arterial pressure in response to air puff or noise

157 ted basal blood pressure and acute change in mean arterial pressure in response to angiotensin II (An

158 difference in the primary outcome of 6-hour mean arterial pressure in septic shock patients receivin

159 effective arterial elastance despite similar mean arterial pressures in control subjects.

160 re observed in CIH and HC rats, although the mean arterial pressure increase was lower after CIH.

161 After infusion of angiotensin II, mean arterial pressure increased by 61.6 mmHg in MD-NOS1

162 After fluid challenge, mean arterial pressure increased from 73 mm Hg (interqua

163 Mean arterial pressure increased rapidly from 75 +/- 2 m

164 Mean arterial pressure increased significantly in MD-NOS

165 Mean arterial pressure initially decreased further under

166 (+) was the only treatment group that raised mean arterial pressure into the permissive range and ret

167 each eye at each of the 8 time points as 2/3(mean arterial pressure-intraocular pressure [IOP]).

168 At 10 to 17 weeks, mean arterial pressure inversely correlated with serum P

169 activity and low (</=40 mm Hg) mean OPP (2/3 mean arterial pressure - IOP) and low (</=50 mm Hg) dias

170 A significant increase in mean arterial pressure is observed in early adulthood in

171 ot be aggressively treated in this period if mean arterial pressure, lactate clearance, and diuresis

172 , hospital location, era, systolic pressure, mean arterial pressure, lactate, bundle compliance, amou

173 Exclusion criteria for both groups were mean arterial pressure less than 60 mm Hg, contraindicat

174 ours, when shock was present, animals with a mean arterial pressure less than 65 mm Hg (n = 6) had si

175 oup in which animals were exsanguinated to a mean arterial pressure level of 40 mm Hg during 30 minut

176 nation from the 30th to the 60th minute to a mean arterial pressure level of 40 mm Hg; or control gro

177 study was a composite of severe hypotension (mean arterial pressure < 60 mm Hg) and bradycardia (hear

178 sion (systolic blood pressure </=90 mm Hg or mean arterial pressure </=65 mm Hg) presenting to the em

179 ly randomized to strict or usual BP control (mean arterial pressure </=92 mmHg or 102-107 mmHg, respe

180 line and randomization to the lower BP goal (mean arterial pressure </=92 mmHg) associated with impro

181 uded: albumin <2.5 g/dL, heart rate >90 bpm, mean arterial pressure <60 mmHg, white blood cell count

182 allowed a reliable detection of 1) invasive mean arterial pressure <65 mm Hg (area under the receive

183 , Wilcoxon test), diastolic BP (P=0.02), and mean arterial pressure (MAP) (P=0.006) during the 3 mont

184 LOD scores were 2.35-2.91 for mean arterial pressure (MAP) and 0.80-1.49 for systolic

185 ntation via beetroot juice (BR) would reduce mean arterial pressure (MAP) and increase hindlimb muscl

186 Mean arterial pressure (MAP) and OPP (systolic, diastoli

187 ug/rat) elicited dose-dependent increases in mean arterial pressure (MAP) and plasma norepinephrine (

188 with systolic BP (SBP), diastolic BP (DBP), mean arterial pressure (MAP) and pulse pressure (PP), we

189 vre evoked significantly larger increases in mean arterial pressure (MAP) and renal sympathetic nerve

190 Moreover, inflammation increased maternal mean arterial pressure (MAP) and was associated with ren

191 ) and diastolic BP (DBP) were measured, with mean arterial pressure (MAP) calculated.

192 The primary outcome measure was Delta mean arterial pressure (MAP) during the first 20 s of ex

193 cus (QTL) on chromosome 1 that was linked to mean arterial pressure (MAP) in the context of sGCalpha1

194 ow levels of estradiol-17beta (E2) increases mean arterial pressure (MAP) in young female Sprague-Daw

195 there was no relationship of MSNA to TPR or mean arterial pressure (MAP) in young women.

196 essment of CA during steady-state changes in mean arterial pressure (MAP) induced by intravenous infu

197 nship of Doppler BP to systolic BP (SBP) and mean arterial pressure (MAP) is uncertain and Doppler me

198 us females; however, i.c.v. leptin increased mean arterial pressure (MAP) only in males.

199 The nocturnal mean arterial pressure (MAP) was compared with the dayti

200 Intracranial pressure (ICP), CBF, and mean arterial pressure (MAP) were measured and, after tw

201 Renal blood flow (RBF), mean arterial pressure (MAP), and heart rate (HR) were c

202 for Endstage Liver Disease (MELD) score, low mean arterial pressure (MAP), and non-SBP infections.

203 udied systolic BP (SBP), diastolic BP (DBP), mean arterial pressure (MAP), and pulse pressure (PP) av

204 with systolic BP (SBP), diastolic BP (DBP), mean arterial pressure (MAP), and pulse pressure (PP), w

205 re (cSBP), central diastolic blood pressure, mean arterial pressure (MAP), augmentation index, pulse

206 erebral artery blood velocity (MCA V(mean)), mean arterial pressure (MAP), cardiac output (CO) and pa

207 se anaesthetised female Sprague-Dawley rats, mean arterial pressure (MAP), heart rate (HR) and lumbar

208 utflow, produced dose-dependent increases in mean arterial pressure (MAP), heart rate (HR), and lumba

209 Mean arterial pressure (MAP), heart rate (HR), BT, motor

210 sociation studies of pulse pressure (PP) and mean arterial pressure (MAP).

211 lly low systolic blood pressure (SBP) and/or mean arterial pressure (MAP).

212 ripheral cardiovascular (heart rate [HR] and mean arterial pressure [MAP]), IOP, and ICP effects were

213 ions in sympathetic nerve activity (SNA) and mean arterial pressure(MAP)with exaggerated sympathetic

214 For mean arterial pressure measurement, arm noninvasive bloo

215 tics of continuous intracranial pressure and mean arterial pressure monitoring during the first 24 hr

216 he first 24 hrs of intracranial pressure and mean arterial pressure monitoring to known baseline risk

217 tics of continuous intracranial pressure and mean arterial pressure monitoring.

218 n also resulted in a significant decrease in mean arterial pressure, MPAP, pulmonary artery occlusive

219 sulted in a significant further reduction of mean arterial pressure, MPAP, pulmonary artery occlusive

220 c expression of Kv1.5 channels), we measured mean arterial pressure, myocardial blood flow, myocardia

221 ratio, 2.71; 95% CI, 1.67-4.39; p < 0.001), mean arterial pressure (odds ratio, 0.979; 95% CI, 0.963

222 morrhagic shock by blood withdrawn until the mean arterial pressure of 30 mm Hg and maintained at thi

223 Hemorrhage animals were bled to a mean arterial pressure of 35 mm Hg.

224 morrhagic shock was induced by phlebotomy to mean arterial pressure of 35-40 mm Hg for 20 mins (~40%

225 igs (35-40 kg) were anesthetized and bled to mean arterial pressure of 35-40 mm Hg for 90 minutes, fo

226 ) on fluid requirement to maintain a minimum mean arterial pressure of 50 mm Hg, and 2) the effect of

227 chemic events rose rapidly below an absolute mean arterial pressure of 60 mmHg.

228 d by a vasopressor requirement to maintain a mean arterial pressure of 65 mm Hg or greater and serum

229 iring high-dose norepinephrine to maintain a mean arterial pressure of 65 mm Hg or higher.

230 4, p = 0.009) compared with animals with an mean arterial pressure of 65-70 mm Hg (n = 4).

231 e mean daily dose of vasopressor to insure a mean arterial pressure of 65-75 mm Hg.

232 mean heart rate of 60+/-5 beats per minute, mean arterial pressure of 78+/-5 mm Hg, ocular perfusion

233 Targeting a mean arterial pressure of 80 to 85 mm Hg, as compared wi

234 iving Sepsis Campaign recommends targeting a mean arterial pressure of at least 65 mm Hg during initi

235 Hypotension was defined as a decrease in the mean arterial pressure of greater than or equal to 15% c

236 0 mL 7.5% NaCl with ALM to maintain a target mean arterial pressure of minimum 50 mm Hg.

237 m creatinine, bilirubin or albumin, baseline mean arterial pressure, or study design, size or time pe

238 e, in patients with higher baseline BP (DBP, mean arterial pressure, or systolic BP), those with decl

239 re (P = .005), pulse pressure (P = .02), and mean arterial pressure (P = .01) when compared with the

240 (p = 0.024), and large, early reductions in mean arterial pressure (p = 0.003) were independent pred

241 , HRP treatment induced progressive falls in mean arterial pressure (P<0.001) in association with dec

242 those in the crystalloid group, had a higher mean arterial pressure (P=0.03) and lower net fluid bala

243 left atrial volume index (P=0.05), and lower mean arterial pressure (P=0.03) were predictors of HF af

244 (P=0.02), central pulse pressure (P<0.0001), mean arterial pressure (P=0.04), and baseline brachial f

245 etion with CHD risk tended to be modified by mean arterial pressure (Pinteraction=0.08) and was modif

246 atin use, blood pressure medication use, and mean arterial pressure, PP quartile was still associated

247 In vehicle-treated sheep, mean arterial pressure progressively declined from 25 to

248 perature were not correlated with changes in mean arterial pressure (r = 0.0002; p = 0.85).

249 ality (p = 0.76) associated with hypotensive mean arterial pressure readings (</=60 mm Hg) were indep

250 In our population, arm noninvasive mean arterial pressure readings were accurate.

251 ability at 2 hours, followed by decreases in mean arterial pressure, renal blood flow (RBF), and rena

252 is study was to examine the effect of RDN on mean arterial pressure, renal function, and the reflex r

253 We measured the changes from baseline mean arterial pressure, renal plasma flow, plasma renin

254 f the alpha2-adrenergic agonist clonidine on mean arterial pressure, renal sympathetic nerve activity

255 norepinephrine required to maintain a target mean arterial pressure; secondary outcomes included hemo

256 ressure and invasive arterial blood pressure mean arterial pressures showed better agreement; acute k

257 Mean arterial pressure, skin blood flow via laser-Dopple

258 nge in body mass index, year 5 and change in mean arterial pressure, starting smoking, and year 5 dia

259 e treatment prevented the further decline in mean arterial pressure, substantially reduced heart rate

260 brainstem regions negatively impacts resting mean arterial pressure, sympathovagal balance, and baror

261 Compared with F+V, F+T decreased mean arterial pressure*, systemic* and pulmonary* vascul

262 septic shock to undergo resuscitation with a mean arterial pressure target of either 80 to 85 mm Hg (

263 ger reduction in systolic blood pressure and mean arterial pressure than did either CPAP or weight lo

264 noninvasive readings was markedly higher for mean arterial pressure than for systolic or diastolic pr

265 r a sustained approximately 5-hr decrease in mean arterial pressure to 60 mm Hg and continued the inf

266 ce index was then calculated as the ratio of mean arterial pressure to regional cerebral blood flow.

267 n be added to norepinephrine to either raise mean arterial pressure to target or to decrease norepine

268 5 vs. 0.40 +/- 0.17%.%; p < .05), and higher mean arterial pressure-to-middle cerebral artery mean fl

269 of proteinuria were repeat transplantation, mean arterial pressure, transplant glomerulopathy, micro

270 rated modestly, but not significantly, lower mean arterial pressure under basal conditions compared t

271 a threshold effect at time-weighted average mean arterial pressure value of 70 mm Hg.

272 (95% CI, 0.01-0.14) and between low and high mean arterial pressure was 0.05% (95% CI, 0.00-0.10).

273 Basal mean arterial pressure was 15 mm Hg higher and glomerula

274 acetaminophen-induced hypotension, the nadir mean arterial pressure was 64 mm Hg (95% CI, 54-74).

275 We found that time-weighted average mean arterial pressure was associated with good neurolog

276 Mean arterial pressure was higher in the Oxyglobin group

277 ce mask was used during the prescribed walk, mean arterial pressure was lower (93 +/- 10 vs. 96 +/- 1

278 mic vascular resistance index (21%), whereas mean arterial pressure was not affected.

279 The 24-hour mean arterial pressure was not inferior on treatment wit

280 Mean arterial pressure was not significantly different i

281 e was negatively related (P<0.0001), whereas mean arterial pressure was positively related (P<0.0001)

282 Mean arterial pressure was reduced to 30 mm Hg for 90 mi

283 Mean arterial pressure was reduced to 30 mm Hg for 90 mi

284 methyl-L-thiocitrulline (1 mumol/kg, IV) and mean arterial pressure was registered.

285 However, mean arterial pressure was significantly lower in both C

286 The effect of vasopressin on mean arterial pressure was unaltered and that on renal v

287 Cutaneous vascular conductance (CVC = flux/mean arterial pressure) was expressed as a change from b

288 Hemodynamic efficacy (mean arterial pressure) was measured at 3 specific condi

289 Changes in mean arterial pressure were closely correlated with decr

290 Leg blood flow and mean arterial pressure were determined, whereas leg vasc

291 diac index, left ventricular dimensions, and mean arterial pressure were measured using bilateral ven

292 The risk scores for blood pressure and mean arterial pressure were not associated with any of t

293 y postdonation, systolic blood pressure, and mean arterial pressure were significantly higher in hype

294 oke volume, cardiac output and reductions in mean arterial pressure were similar between age groups a

295 diolabelled microspheres) and VC (blood flow/mean arterial pressure) were determined during supra-cri

296 tibular otolith system and a decrease in the mean arterial pressure when a person stands up.

297 5 mins from a decrease in cardiac output and mean arterial pressure, whereas treated rats survived un

298 significant increases (P < 0.05; n = 7-8) in mean arterial pressure, which were generally accompanied

299 lepressin was titrated to raise and maintain mean arterial pressure within no less than 10 mm Hg from

300 nge in body mass index, year 5 and change in mean arterial pressure, year 5 and change in heart rate,