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1 ose groups (45 and 30 mg), except for DCE CT mean transit time.
2 ividuals without CCSVI, without any delay in mean transit time.
3 distal pressure multiplied by the hyperemic mean transit time.
4 od flow, relative cerebral blood volume, and mean transit time.
5 al blood volume (6-33% decrease), and tissue mean transit time (10-54% increase) were observed in the
9 striatal necrosis in an 8-month-old infant, mean transit time and cerebral blood volume were low rel
10 aminal lesions, striatal flow was normal but mean transit time and cerebral blood volume were low, co
11 usion coefficient, tissue oxygen saturation, mean transit time, and blood volume fraction in the cort
12 s per group); local brain oxygen saturation, mean transit time, and blood volume fraction were subseq
13 o calculate tissue blood flow, blood volume, mean transit time, and permeability-surface area product
14 asurement of tumor blood flow, blood volume, mean transit time, and permeability-surface area product
16 ime, elevated cerebral blood volume and high mean transit time/cerebral blood flow and cerebral blood
18 ured) had low cerebral blood flow, prolonged mean transit time, elevated cerebral blood volume and hi
21 raction in tumors and normal tissue, and for mean transit time in tumors; however, permeability value
22 monstrated by a 3-fold increase in ribosomal mean transit times in cell-free extracts from hibernator
23 .90) than did those on blood flow and tracer mean transit time maps (r approximately 0.6), likely as
24 as the difference between lesion volumes on mean transit time maps and DW images, divided by DW lesi
25 algorithm identifies hypoperfused tissue in mean transit time maps by simultaneously minimizing the
26 BV), absolute cerebral blood flow (CBF), and mean transit time (MTT) (referenced to an arterial input
27 volume (BV), and lower (>0.30 and >0.39) for mean transit time (MTT) and permeability surface area pr
30 ved brain perfusion: hypoperfusion volume on mean transit time (MTT) map decrease >30% from baseline
31 fraction (PV), distribution volume (DV), and mean transit time (MTT) of gadopentetate dimeglumine.
32 the glomerular filtration rate (GFR) and the mean transit time (MTT) of the tracer for the vascular c
33 f mean CBF, cerebral blood volume (CBV), and mean transit time (MTT) were determined between hemisphe
38 culate tissue blood flow (BF), blood volume, mean transit time (MTT), and vascular permeability-surfa
40 ity, blood volume (BV), blood flow (BF), and mean transit time (MTT), were calculated at the primary
43 ncluding Blood Flow (BF), Blood Volume (BV), Mean Transit Time (MTT)] and permeability parameters [in
44 ed software, it is possible to calculate the mean transit time of room-temperature saline injected do
45 farct volume (SE = 80%; SP = 97%), Increased mean transit time on PCT was predictive of the tissue at
46 , total blood flow, distribution volume, and mean transit time) parameters were calculated by placing
47 o determine tissue blood flow, blood volume, mean transit time, permeability, and hepatic arterial fr
49 sure divided by the inverse of the hyperemic mean transit time provides an index of microcirculatory
51 lower portal flow, distribution volume, and mean transit time than did the background liver (all P <
52 , and positive correlation was found between mean transit time values and disability scales in patien
53 volume (CBV), cerebral blood flow (CBF), and mean transit time were assessed with dynamic susceptibil
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