ライフサイエンスコーパス: mechanical injury

1 tegrity, both in the presence and absence of mechanical injury.

2 sect infestation, manufacturing stresses and mechanical injury.

3 (PTOA) across the knee joint in response to mechanical injury.

4 in and tested for protective effects against mechanical injury.

5 nction of Pmp22 is to protect the nerve from mechanical injury.

6 pon tissue loading and is released following mechanical injury.

7 ficantly regulated at least 2-fold following mechanical injury.

8 Explants were subjected to single-impact mechanical injury.

9 lus region of the hippocampus within days of mechanical injury.

10 hondrocyte survival and cell death following mechanical injury.

11 haviors, rendering axons more sensitive to a mechanical injury.

12 lved in the long-term response of neurons to mechanical injury.

13 genes is one way in which plants respond to mechanical injury.

14 timal hyperplasia and does not require frank mechanical injury.

15 eurons from the effects of neurochemical and mechanical injury.

16 s basalis from the effects of excitotoxic or mechanical injury.

17 and reduces intimal expansion in response to mechanical injury.

18 rphological responses of individual axons to mechanical injury.

19 , are induced in the vasculature after acute mechanical injury.

20 Disruption of the epithelial barrier by mechanical injury activates TGF-beta1, leading to LM-332

21 r of apoptotic cells over that accruing from mechanical injury alone.

22 se of kainate as an agonist, suggesting that mechanical injury alters AMPA receptor desensitization.

23 f iex-1 in vivo, we performed carotid artery mechanical injury and endothelial denudation in low-dens

24 sceptibility of hippocampal and DRG axons to mechanical injury and estimated the elasticity of live a

25 s, in addition to pathogens that induce PCD, mechanical injury and infiltration of leaves with 1 M su

26 car and fibrosis are often the end result of mechanical injury and inflammatory diseases.

27 bly occurs rapidly in response to an in vivo mechanical injury and precedes basal body polarization a

28 hanol inhibition of hepatocyte recovery from mechanical injury and that this impairment occurs indepe

29 hanges in the level of gene expression after mechanical injury are gene specific and time dependent.

30 o how cells protect themselves not only from mechanical injury but also from microbial toxins and por

31 Mechanical injury can also determine spinal cord degener

32 h bleaching phenols, overexposure to UV, and mechanical injury can lead to progressive loss of melano

33 BB integrity, however the mechanism by which mechanical injury can produce a [Ca2+]i increase in brai

34 Mechanical injury caused a significant loss of viable ch

35 eated explants, but not in control explants, mechanical injury caused an immediate reduction in cell

36 Our data suggest that after mechanical injury, chondrocytes distant from the site ma

37 matory corneal neovascularization induced by mechanical injury (debridement), and in vitro in corneal

38 following other forms of CNS damage, such as mechanical injury, excitotoxicity and viral infection.

39 g2576) mice and in brain tissue subjected to mechanical injury expressed vimentin, while those in com

40 Following intra-arterial mechanical injury, FGF2-deficient vessels undergo a norm

41 spasms were similar to vasospasms induced by mechanical injury followed by serotonin, and to those st

42 te matter may be more susceptible to initial mechanical injury forces and that anisotropic diffusion

43 In particular, mechanical injury from the procedure results in extensiv

44 Mechanical injury has been postulated as a risk factor f

45 Indeed, immediately following a mechanical injury, heregulin-alpha activates erbB2 in ce

46 mice repopulated vascular lesions induced by mechanical injury in p27(+/+) recipients, in contrast to

47 , relatively little apoptosis is found after mechanical injury in vitro.

48 We conclude that mechanical injury in vivo and in vitro induces iNOS expr

49 Mechanical injury in vivo results in the expression of t

50 Cell surface biotinylation after mechanical injury indicates that proteolyzed NaChs remai

51 Mechanical injury induced cell death and loss of sGAG in

52 Mechanical injury induces cell death in cartilage and tr

53 ypothesis that alveolar instability causes a mechanical injury, initiating an inflammatory response t

54 ypothesis that alveolar instability causes a mechanical injury, initiating an inflammatory response w

55 and neuronal damage that follows the primary mechanical injury is an important cause of disability in

56 s for lung inflammation and remodeling after mechanical injury is discussed.

57 rate for the first time that ATP released by mechanical injury is one of the signals that triggers ER

58 g from chondrocyte death in the aftermath of mechanical injury is thought to contribute to posttrauma

59 Our findings indicate that mechanical injury leads to suppression of autophagy, pre

60 Following mechanical injury, lubricin messenger RNA (mRNA) levels

61 such as foods, chemicals, friction, thermal/mechanical injury, metals, spices, and oral care product

62 In response to femoral artery mechanical injury, mice with homozygous deletion of p21C

63 rological pathologies, such as autoimmunity, mechanical injury, neurodegeneration, and infection.

64 died (i) the recovery from large sarcolemmal mechanical injuries of the response to electric stimulat

65 We conclude that mechanical injury of brain endothelial cells induces a r

66 aveolin-2 is up-regulated in response to the mechanical injury of differentiated PC12 cells; up-regul

67 We produced a chemical and mechanical injury of the cornea in rats by brief applica

68 igration of adventitial cells in response to mechanical injury of the rat carotid artery.

69 effector cells form the initial response to mechanical injury of the skin.

70 ivation of smooth muscle cells in vivo after mechanical injury of the vessel wall.

71 Here, the effects of mechanical injury on the voltage-dependent magnesium (Mg

72 phytoalexins in solanaceous plants following mechanical injury or pathogen infection.

73 In response to biological and mechanical injury, or in vitro culturing, vascular smoot

74 Our findings indicate that mechanical injury potentiates the catabolic effects of T

75 Finally, efficient repair of colonic mechanical injuries requires villin severing of F-actin,

76 ions of 1.8 mM and 3.8 mM, respectively, but mechanical injury resulted in extensive calcification in

77 This 3D technique is useful for mechanical injury studies and as a drug screening tool,

78 ity under conditions of oxidative stress and mechanical injury, such as those experienced in cardiova

79 GsMTx4 reduced chondrocyte death after mechanical injury, suggesting a possible therapy for red

80 fish are based on invasive surgeries causing mechanical injuries that are limited in size.

81 uries to the brain and ensuing secondary non-mechanical injuries that are not limited to the brain.

82 In response to mechanical injury, there is an immediate induction of ne

83 Mechanical injuries to Arabidopsis roots did not produce

84 Unfavorable TBI outcomes result from primary mechanical injuries to the brain and ensuing secondary n

85 Our findings indicate that mechanical injury to adult human articular cartilage res

86 nges in and functions of autophagy following mechanical injury to cartilage.

87 Mechanical injury to connective tissue causes changes in

88 We found that mechanical injury to mouse skin, which can be caused by

89 Mechanical injury to the adult mammalian spinal cord res

90 Mechanical injury to the adult mammalian spinal cord res

91 Here, we demonstrate that, upon mechanical injury to the adult pallium, axolotls can reg

92 utons into postsynaptic neurons occurs after mechanical injury to the brain.

93 Mechanical injury to the skin results in activation of t

94 ionic homeostasis in cortical cultures, and mechanical injury using an in vitro model of traumatic b

95 In addition, the susceptibility of cells to mechanical injury was not correlated with changes in cel

96 nn cell support for distressed neurons after mechanical injury, we have now asked: do Schwann cells a

97 ve in preventing neointimal thickening after mechanical injury when administered in the absence of an

98 articular cartilage explants 24 hours after mechanical injury with that of uninjured controls by mic

99 ce of MCP-1 in mouse brain after penetrating mechanical injury, with particular focus on early time p