ライフサイエンスコーパス: medial ganglionic eminence

1 suppression of Nkx2-1 leads to a loss of the medial ganglionic eminence.

2 tory interneurons derived from the embryonic medial ganglionic eminence.

3 e telencephalic ventricular zone and not the medial ganglionic eminence.

4 e cortical interneurons that derive from the medial ganglionic eminence.

5 of-function analysis, Gsh2 expression in the medial ganglionic eminence after E10.5 may negatively re

6 interneurons, are generated from the ventral medial ganglionic eminence and dorsal preoptic area base

7 arkers for inhibitory cells derived from the medial ganglionic eminence and few expressed VGAT, found

8 ed radial glial progenitors in the embryonic medial ganglionic eminence and preoptic area preferentia

9 me group, develops domains equivalent to the medial ganglionic eminence and rhombic lip, resembling t

10 upregulation of Dlx1/2 genes in the ventral medial ganglionic eminences and adjacent regions of the

11 tical interneurons that are derived from the medial ganglionic eminence, as most studies have examine

12 t majority arising from either the caudal or medial ganglionic eminences (CGE and MGE).

13 hese data suggest that Satb1 is required for medial ganglionic eminence-derived interneuron different

14 s regulates the differentiation of two major medial ganglionic eminence-derived interneuron populatio

15 essing 5-HT(3A) receptors (5-HT(3A)Rs) and a medial ganglionic eminence-derived subpopulation lacking

16 ived from common precursors generated in the medial ganglionic eminence during embryogenesis.

17 ry enhancer (Dlx5/6ei) in embryonic day 13.5 medial ganglionic eminence (E13.5 MGE).

18 The medial ganglionic eminence exhibited unique patterns of

19 x, future hippocampus as well as lateral and medial ganglionic eminences exhibited a 20-30% reduction

20 as predominantly localised in the developing medial ganglionic eminences, flanking a Fgf8-positive mi

21 Transplanting medial ganglionic eminence interneuron progenitors to in

22 eras; Nkx2.1, which is expressed only in the medial ganglionic eminence, is not.

23 s and fates of cells born in the lateral and medial ganglionic eminences (LGE and MGE) in 13.5-day-ol

24 cification of progenitors in the lateral and medial ganglionic eminences (LGE and MGE).

25 c mice, that the first OLPs originate in the medial ganglionic eminence (MGE) and anterior entopedunc

26 Other precursors--including those from the medial ganglionic eminence (MGE) and OB--fail to generat

27 Progenitor cells in the medial ganglionic eminence (MGE) and preoptic area (PoA)

28 l (subcortical) telencephalon, including the medial ganglionic eminence (MGE) and preoptic area.

29 rth disrupts interneuron neurogenesis in the medial ganglionic eminence (MGE) and, more importantly,

30 Furthermore, Nkx2.1(+) progenitors in the medial ganglionic eminence (MGE) are misspecified such t

31 nsplanted GABAergic precursor cells from the medial ganglionic eminence (MGE) can migrate and differe

32 GABAergic progenitor cells derived from the medial ganglionic eminence (MGE) can reverse mechanical

33 Embryonic medial ganglionic eminence (MGE) cells transplanted into

34 have been described, a system modeling human medial ganglionic eminence (MGE) development, a critical

35 t inhibitory interneuron precursors from the medial ganglionic eminence (MGE) enhances GABAergic sign

36 In the developing telencephalon, the medial ganglionic eminence (MGE) generates many cortical

37 The medial ganglionic eminence (MGE) gives rise to the major

38 llium, lateral ganglionic eminence (LGE) and medial ganglionic eminence (MGE) in the subpallium has b

39 nic precursors of GABAergic neurons from the medial ganglionic eminence (MGE) into adult mouse spinal

40 tation of precursor cells from the embryonic medial ganglionic eminence (MGE) into early postnatal ne

41 phalic GABAergic interneurons from the mouse medial ganglionic eminence (MGE) into the adult mouse sp

42 In the ventral telencephalon, the medial ganglionic eminence (MGE) is a major source of co

43 The medial ganglionic eminence (MGE) is an embryonic forebra

44 Young neurons born in the medial ganglionic eminence (MGE) migrate a long distance

45 er, we report that mosaic elimination in the medial ganglionic eminence (MGE) of Smo, a key effector

46 ron subgroups originate primarily within the medial ganglionic eminence (MGE) of the subcortical tele

47 interneurons targeting cells by lineage from medial ganglionic eminence (MGE) or caudal ganglionic em

48 l interneurons tangentially migrate from the medial ganglionic eminence (MGE) or the adjacent preopti

49 nNOS(+) IvCs and NGCs are both derived from medial ganglionic eminence (MGE) progenitors under contr

50 rons are the two major subtypes generated by medial ganglionic eminence (MGE) progenitors.

51 contrary, it resulted in a partial rescue of medial ganglionic eminence (MGE) properties, including i

52 nd Cdc42 in the ventricular zone (VZ) of the medial ganglionic eminence (MGE) using Olig2-Cre mice ca

53 opment, several cIN subtypes derive from the medial ganglionic eminence (MGE), a transient ventral te

54 a transient embryonic structure known as the medial ganglionic eminence (MGE), but how the remarkable

55 nsplanted neuronal precursors from embryonic medial ganglionic eminence (MGE), but not from lateral g

56 l GABAergic interneuron progenitors from the medial ganglionic eminence (MGE), can overcome the mecha

57 tyric acid (GABA) interneurons, derived from medial ganglionic eminence (MGE), is implicated in disor

58 e-cell RNA sequencing on the mouse embryonic medial ganglionic eminence (MGE), the major birthplace f

59 Cell cycle regulation was examined in the medial ganglionic eminence (MGE), the major source of PV

60 n of GABAergic precursors from the embryonic medial ganglionic eminence (MGE), the source of neocorti

61 ebral cortical interneurons originate in the medial ganglionic eminence (MGE), where the signaling mo

62 ergic neurons, but not by progenitors in the medial ganglionic eminence (MGE), which generate cortica

63 /Ai14 embryos harboring tdTomato-fluorescent medial ganglionic eminence (MGE)-derived cortical GABAer

64 Interestingly, compared with medial ganglionic eminence (MGE)-derived cortical intern

65 Medial ganglionic eminence (MGE)-derived GABAergic corti

66 uired for the differentiation of a subset of medial ganglionic eminence (MGE)-derived neurons, but ar

67 SCs into telencephalic excitatory neurons or medial ganglionic eminence (MGE)-like inhibitory neurons

68 t the directed differentiation of hPSCs into medial ganglionic eminence (MGE)-like progenitors and th

69 gration of immature neurons derived from the medial ganglionic eminence (MGE).

70 d in the embryonic subpallium, including the medial ganglionic eminence (MGE).

71 f the cortical interneurons generated in the medial ganglionic eminence (MGE).

72 of progenitors that primarily resides in the medial ganglionic eminence (MGE).

73 rneurons, including those originating in the medial ganglionic eminence (MGE).

74 we compared interneuronal progenitors in the medial ganglionic eminences (MGEs), lateral ganglionic e

75 ing a role in oligodendrogenesis, within the medial ganglionic eminence of Nkx2.1 mutants, the early

76 Prototypic GPe neurons derive from the medial ganglionic eminence of the embryonic subpallium a

77 0 d, and then patterned to NKX2.1-expressing medial ganglionic eminence progenitors by simple treatme

78 lanted embryonic inhibitory neurons from the medial ganglionic eminence reinstate ocular dominance pl

79 reted here as the homologue of the mammalian medial ganglionic eminence (the adult pallidum in mammal

80 transplantation of cells from the embryonic medial ganglionic eminence (the major origin of cerebral

81 ion of ventral neurons characteristic of the medial ganglionic eminence, the embryonic structure whic

82 In the medial ganglionic eminence, the NKX2-1 transcription fac

83 other hand, disrupting specification of the medial ganglionic eminence through loss of Nkx2.1 homeob

84 e embryonic basal telencephalon (lateral and medial ganglionic eminences) through loss of Dlx1/2 home

85 c interneurons, from their generation in the medial ganglionic eminence up to their settlement in the

86 mice severely impaired proliferation in the medial ganglionic eminence without grossly altering diff