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1 taining glutamatergic neurons in the ventral medial hypothalamus.
2 PAG neurons whose axons project back to the medial hypothalamus.
3 ed defensive rage behavior elicited from the medial hypothalamus.
4 um, infundibulum/median eminence, and dorsal medial hypothalamus.
5 putative GABAergic pathway projecting to the medial hypothalamus.
6 to the suprachiasmatic nucleus and adjacent medial hypothalamus.
7 were equally distributed in the lateral and medial hypothalamus.
8 l limbic cortices were more prevalent in the medial hypothalamus.
9 to suppress defensive rage elicited from the medial hypothalamus.
10 ensive rage behavior elicited from the cat's medial hypothalamus.
11 OCT3 mRNA, but not OCT1 or OCT2 mRNA, in the medial hypothalamus.
12 ce P pathway from the medial amygdala to the medial hypothalamus.
13 in doses of 0.05, 0.5 and 2.5 nmol) into the medial hypothalamus.
14 between IL-1beta and 5-HT2 receptors in the medial hypothalamus.
15 tor agonist (+/-)-DOI hydrochloride into the medial hypothalamus (0.5, 1.0, and 3.0 nmol) facilitated
17 ulation over the rostro-caudal extent of the medial hypothalamus and dorsolateral aspect of the peria
18 ron projects from the medial amygdala to the medial hypothalamus and its functions are mediated by su
19 - and progesterone-dependent signal from the medial hypothalamus and results in heightened sexual mot
20 selectively blocks hissing elicited from the medial hypothalamus and that the suppressive effects of
21 iated over reciprocal pathways that link the medial hypothalamus and the dorsolateral quadrant of the
23 ncluding the arcuate nucleus and the ventral medial hypothalamus, areas implicated in regulation of b
24 ddition, pretreatment with CP96,345 into the medial hypothalamus blocked the suppressive effects of s
25 ctions associated with either the lateral or medial hypothalamus, but not both, can be activated at a
27 of 5-HT(1A) and 5-HT(2) receptors within the medial hypothalamus exert differential modulatory effect
28 indings indicate that NK(1) receptors in the medial hypothalamus facilitate defensive rage elicited f
29 stimulating electrodes were implanted in the medial hypothalamus for elicitation of defensive rage be
30 , cannula-electrodes were implanted into the medial hypothalamus for elicitation of defensive rage be
31 ctrodes were implanted into sites within the medial hypothalamus from which defensive rage behavior c
32 imulating electrodes were implanted into the medial hypothalamus from which defensive rage behavior c
33 imulating electrodes were implanted into the medial hypothalamus from which defensive rage behavior c
34 ed following activation of the region of the medial hypothalamus from which defensive rage behavior i
35 d placement of monopolar electrodes into the medial hypothalamus from which defensive rage could be e
36 oinjected through a cannula-electrode in the medial hypothalamus from which defensive rage had been e
39 pothalamus (LH), and lateral portions of the medial hypothalamus, have widespread projections and inf
42 aspect which receives major inputs from the medial hypothalamus in association with defensive rage b
46 e extracellular fluid compartment within the medial hypothalamus (MH), the OCT blocker, decynium 22 (
47 tested the hypothesis that perfusion of the medial hypothalamus (MH), which includes the DMH, with t
50 ression in the lateral hypothalamus (LH) and medial hypothalamus (MH, including perifornical and dors
52 and function of OCTs in the periventricular medial hypothalamus of male Sprague Dawley rats using re
54 ed by 5-HT(1A) and 5-HT(2C) receptors in the medial hypothalamus on the expression of defensive rage
55 ndings show that activation of IL-1RI in the medial hypothalamus potentiates defensive rage behavior
56 ain-derived cytokine, microinjected into the medial hypothalamus, potentiates defensive rage behavior
57 ron, which also projects from the lateral to medial hypothalamus, serves to suppress defensive rage e
58 st 8-OHDPAT (0.1, 1.0 and 3.0 nmol) into the medial hypothalamus suppressed PAG-elicited hissing in a
59 scimol (0.3-30 pmol), microinjected into the medial hypothalamus, suppressed defensive rage elicited
61 ip between IL-1RI and 5-HT2 receptors in the medial hypothalamus that is consistent with the previous
62 the hypothesis that descending fibers of the medial hypothalamus that supply the dorsal aspect of the
63 , identified large numbers of neurons in the medial hypothalamus that were labeled positively for bot
64 ck-out mice restored 5-HT1A receptors in the medial hypothalamus; this effect was accompanied by elim
65 gdala activates substance P receptors in the medial hypothalamus, thus triggering an inhibitory mecha
66 othesis that the descending pathway from the medial hypothalamus to the dorsal periaqueductal gray (P
67 mediated over a descending pathway from the medial hypothalamus to the dorsolateral midbrain periaqu
68 mediated over a descending pathway from the medial hypothalamus to the dorsolateral quadrant of the
69 of 0.5, 1.0 and 2.0 nmol), directly into the medial hypothalamus upon lateral hypothalamically elicit
70 fects of dual stimulation of the lateral and medial hypothalamus upon response latencies were compare
71 amygdala, lateral hypothalamus (LH), ventral medial hypothalamus (VMH) and arcuate nucleus (ARC).
72 g glutamatergic neurons, or into the ventral medial hypothalamus (VMH), which contains predominantly
73 and the function of 5-HT1A receptors in the medial hypothalamus were significantly reduced in Ad-1AP
74 nt on the same neurons within regions of the medial hypothalamus where IL-1beta and the IL-1beta rece
75 by a pathway from the medial amygdala to the medial hypothalamus which utilizes substance P as a neur
76 ct inhibitory projection from the lateral to medial hypothalamus whose functions are mediated by GABA
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