ライフサイエンスコーパス: medial thalamus

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1 i-motor, parietal and cingulate cortices and medial thalamus.

2 P) is an important source of limbic input to medial thalamus.

3 NAS), caudate-putamen (CP), hippocampus, and medial thalamus.

4 l cortex with motor cortex, hippocampus, and medial thalamus.

5 n involving posterior paramedian pons and/or medial thalamus.

6 pothalamus 9+/-3%, substantia nigra 10+/-2%, medial thalamus 8+/-2%, and amygdala 9+/-3%.

7 Our data are consistent with the central medial thalamus acting as a key hub through which genera

8 LS) in areas serving the medial pain system (medial thalamus, amygdala, caudate nucleus, anterior cin

9 brain activation, primarily in the anterior medial thalamus and inferior frontal gyrus.

10 ensory and association areas, as well as the medial thalamus and superior colliculus.

11 C FC with the PCC/PCu, retrosplenial cortex, medial thalamus, and periaqueductal/periventricular gray

12 of activation within the anterior cingulate, medial thalamus, and visual cortex during delay and trac

13 ose utilization in the orbitofrontal cortex, medial thalamus, anterior and posterior cingulate cortic

14 ior hippocampus, ventral tegmental area, and medial thalamus) as well as in areas related to visuospa

15 iting the CER composed of prefrontal cortex, medial thalamus, auditory, and hippocampal regions.

16 easantness and accounted for activity in the medial thalamus, bilateral anterior insula, ventral stri

17 h of neurites from neurons isolated from the medial thalamus but was permissive for the growth of neu

18 rate in the anterior cingulate, caudate, and medial thalamus compared with controls.

19 The medial thalamus contains mu opioid receptors and sends a

20 recordings from the hippocampus, septum, and medial thalamus demonstrated fast poly-spike activity as

21 alamus [DMP] and dorsolateral nucleus of the medial thalamus [DLM]); (3) restricted regions within th

22 these disturbances might have their basis in medial thalamus dysfunction.

23 establishes a critical role for the ventral medial thalamus in the propagation of this exaggerated b

24 ation of septal area, but not hippocampus or medial thalamus, in the absence of a seizure resulted in

25 Within the ventral posterior medial thalamus, membrane currents modulated by norepine

26 this project was to explore the role of the medial thalamus (MT), including the medial dorsal thalam

27 responses of single neurons in the nuclei of medial thalamus (MT), specifically the mediodorsal thala

28 se, attributable to an effect in the central medial thalamus, occurs at the point of dexmedetomidine

29 observed in lateral thalamus on day 9 and in medial thalamus on day 10 of PTD treatment, a duration o

30 y ischemia of the posterior paramedian pons, medial thalamus, or cerebellum.

31 mygdala, left anterior orbitofrontal cortex, medial thalamus, pregenual and dorsal anterior cingulate

32 SNpr output entrains activity in the ventral medial thalamus (VM) in this frequency range after loss

33 osensory (SI) cortex and the ventroposterior medial thalamus (VPM) before and during the combined adm

34 rges the receptive fields of ventroposterior medial thalamus (VPM) cells, noradrenergic activation de

35 ields of single cells in the ventroposterior medial thalamus (VPM) of urethane-anesthetized rats duri

36 barrel field cortex [BC]), ventral posterior medial thalamus (VPM), and principal nucleus of the trig

37 r effects were observed in ventral posterior medial thalamus (VPm).

38 asal forebrain extending to the anterior and medial thalamus were affected.

39 ctional connectivity (and increased with the medial thalamus), which is implicated in face expression

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