ライフサイエンスコーパス: mediastinal

1 ure rates in patients with and those without mediastinal abnormalities at preoperative PET were compa

2 ung parenchymal, airway, pleural, hilar, and mediastinal abnormalities systematically reviewed initia

3 for enteral nutrition (n = 18), drainage of mediastinal abscess (n = 4), gastric decompression (n =

4 thickness oesophageal segment destroyed by a mediastinal abscess and leading to direct communication

5 Mediastinal activity appears to be constant over time an

6 t computed tomography images showed enlarged mediastinal adenopathy with increased [(18)F]fluorodeoxy

7 vealed a 5-cm right upper lobe mass, without mediastinal adenopathy, and a 6-cm cystic mass in the sp

8 ecoming standard of care for the sampling of mediastinal adenopathy.

9 h EBUS-TBNA requires a detailed knowledge of mediastinal anatomy.

10 n they involve either side of the neck, with mediastinal and distant metastases.

11 jective noise at the level of the trachea on mediastinal and lung parenchymal images (P < .001) and n

12 tion of the pleural cavity rapidly activates mediastinal and pericardial FALCs.

13 solidation (63%), pulmonary nodules (31.4%), mediastinal and/or hilar lymphadenopathy (23%), mass-lik

14 encing of precursor, primary (testicular and mediastinal) and chemoresistant metastatic human GCTs, w

15 pectoral, supraclavicular, internal mammary, mediastinal, and abdominal nodes.

16 Classical Hodgkin lymphoma and primary mediastinal B cell lymphoma (PMBCL) are related lymphoma

17 rminal center B cell-like (GCB), and primary mediastinal B cell lymphoma (PMBL) subgroups of DLBCL.

18 Primary mediastinal B-cell lymphoma (PMBCL) is a subtype of DLBC

19 f Blood, Ceriani et al introduce, in primary mediastinal B-cell lymphoma (PMBCL), a new prognostic fa

20 clerosis Hodgkin lymphoma (NSHL) and primary mediastinal B-cell lymphoma (PMBL) are the common types,

21 Primary mediastinal B-cell lymphoma (PMBL) is a subtype of diffu

22 tivated B-cell-like (ABC) DLBCL, and primary mediastinal B-cell lymphoma (PMBL).

23 79), Burkitt lymphoma (BL; n = 36), primary mediastinal B-cell lymphoma (PMBL; n = 12), B-cell lines

24 Primary mediastinal B-cell lymphoma is a distinct subtype of dif

25 d histology (diffuse large B-cell lymphoma v mediastinal B-cell lymphoma v Burkitt lymphoma or Burkit

26 with diffuse large B-cell lymphoma, primary mediastinal B-cell lymphoma, or transformed follicular l

27 In the mediastinal B-cell lymphoma, the mutation in REL is of g

28 herapy in 51 patients with untreated primary mediastinal B-cell lymphoma.

29 ed for radiotherapy in patients with primary mediastinal B-cell lymphoma.

30 cases were molecularly classified as primary mediastinal B-cell lymphoma.

31 th diffuse large B-cell lymphoma and primary mediastinal B-cell lymphoma.

32 cell lymphoma, Hodgkin lymphoma, and primary mediastinal B-cell lymphoma.

33 Mediastinal B-cell lymphomas present in the mediastinum

34 B-cell lymphomas, and in some follicular and mediastinal B-cell lymphomas.

35 of 6 with gray zone, 1 CR of 6 with primary mediastinal B-cell, and 1 CR of 3 with posttransplant ly

36 edures, 1 patient died of complications of a mediastinal biopsy, and none of the 6 showed metastases.

37 or with residual [18F]FDG activity below the mediastinal blood pool (MBP) uptake.

38 Mediastinal blood pool activity is recommended as the re

39 (68)Ga-DOTATOC uptake was higher in mediastinal blood pool at the 1-h time point (P = 0.018)

40 age I and II (<3 nodal sites, no B symptoms, mediastinal bulk, or extranodal extension) enrolled betw

41 ritoneal, nonfunctional paraganglioma; and a mediastinal, catecholamine-secreting paraganglioma (pheo

42 Air in all mediastinal compartments was also associated with increa

43 er, CT findings of posterior PNM, air in all mediastinal compartments, and concurrent hemothorax are

44 ertrophy of the bronchial arteries along the mediastinal course, diffuse thickening of the walls of n

45 Bulky mediastinal disease greater than one third transthoracic

46 ky mediastinal disease) and those with bulky mediastinal disease or stage III/IV were scanned after 8

47 male sex, low initial hemoglobin, and bulky mediastinal disease predicting for LF.

48 Male sex, low initial hemoglobin, and bulky mediastinal disease were prognostic indicators of LF.

49 Patients with favorable stage I/II (nonbulky mediastinal disease) and those with bulky mediastinal di

50 LDH level at diagnosis, mediastinal disease, and combined BM-positive/CNS-positi

51 ies, such as pulmonary fibrosis, pleural and mediastinal disease, solid lesions, bronchial disease, a

52 vs 21 [14%]), and respiratory, thoracic, and mediastinal disorders (13 [9%] vs 17 [12%]).

53 ssure on the neighboring lung parenchyma and mediastinal displacement.

54 B-cell-like (ABC) DLBCL, as well as primary mediastinal DLBCL.

55 After TAC, enlarged heart mediastinal draining lymph nodes showed a high density o

56 lated with an increase in donor cells in the mediastinal draining lymph nodes; increased lymphatic ve

57 re complicated by extensive subcutaneous and mediastinal emphysema that occurred without any obvious

58 a previous chest X-ray that showed bilateral mediastinal enlargement; for this purpose, enhanced ches

59 rticularly helpful in patients with positive mediastinal findings at preoperative PET.

60 The anterior mediastinal germ cell tumors (GCTs) are the most common

61 ymphoma (PMBL) are the common types, whereas mediastinal gray-zone lymphoma (MGZL) is extremely rare

62 s intermediate between PMBL and NSHL, called mediastinal gray-zone lymphomas, have been described.

63 For patients with stage I or II bulky mediastinal HL, no substantial statistically significant

64 nalysis in patients with stage I or II bulky mediastinal Hodgkin lymphoma (HL).

65 objective noise at the level of the aorta on mediastinal images (P = .507); (b) significantly higher

66 1279 Hodgkin lymphoma patients treated with mediastinal irradiation and quantified the standard inci

67 coronary stenoses in patients who had prior mediastinal irradiation for Hodgkin's disease.

68 vors of Hodgkin's lymphoma (HL) who received mediastinal irradiation have an increased risk of corona

69 ry artery disease are highly prevalent after mediastinal irradiation in young patients.

70 he excess risk of cardiac interventions from mediastinal irradiation.

71 d survivors of HL who survived 5 years after mediastinal irradiation.

72 Blood and mediastinal isolates were characterized as CA-MRSA by pu

73 (103 of 107), Burkitt lymphomas (40 of 40), mediastinal large B lymphomas (7 of 8), and in DLBCLs (1

74 Classical Hodgkin lymphoma (cHL) and mediastinal large B-cell lymphoma (MLBCL) are lymphoid m

75 Primary mediastinal large B-cell lymphoma (MLBCL) shares importa

76 Mediastinal large B-cell lymphoma (MLBL) represents 2% o

77 ssification of children and adolescents with mediastinal large B-cell lymphoma (MLBL), and highlight

78 Primary mediastinal large B-cell lymphoma (PMBCL) is a subtype o

79 The pathogenesis of primary mediastinal large B-cell lymphoma (PMBCL) is incompletel

80 natures to those of diffuse LBCL and primary mediastinal large B-cell lymphoma (PMBL).

81 ment options for relapsed/refractory primary mediastinal large B-cell lymphoma (rrPMBCL) are limited,

82 ling molecules are down-regulated in primary mediastinal large B-cell lymphomas and Hodgkin's lymphom

83 ydatid presents as a fluid-density posterior mediastinal lesion on chest radiograph with destruction

84 est radiograph was suggestive of a posterior mediastinal lesion with fluid density and destruction of

85 f an elderly male with incidental finding of mediastinal lesion, which was initially thought to be an

86 For example, resolution modeling for mediastinal lesions and iterative deconvolution for lung

87 is a safe method for sampling peripheral and mediastinal lesions with high diagnostic yield independe

88 intraperitoneal inoculation, 1 to 8% of the mediastinal LN cells were infected.

89 (4)beta(7)(high) cells also redistributed to mediastinal LN in a manner sensitive to treatment with a

90 dministration of BMDC, respectively, whereas mediastinal LN were targeted by both routes.

91 ns, and COX-2 inhibition markedly suppressed mediastinal LNM.

92 to have prognostic implications despite the mediastinal LNs being histologically negative.

93 ctive value for detecting lymph nodes in any mediastinal location and for patients without lymph node

94 increase diagnostic yield of peripheral and mediastinal lung lesions.

95 sport of live bacteria from the lungs to the mediastinal lymph node (MDLN).

96 cell transfer, the T cells isolated from the mediastinal lymph node (med-LN) of aged animals exhibite

97 tention of virus-specific CD8 T cells in the mediastinal lymph node (MLN) and continuing recruitment

98 atic proliferation were largely found in the mediastinal lymph node (mLN), rather than the airways; h

99 l recruitment mainly occurs in the posterior mediastinal lymph node (pMLN).

100 ium tuberculosis occurs in the lung-draining mediastinal lymph node and requires transport of M. tube

101 After stimulation of mediastinal lymph node and spleen cells with UV-inactiva

102 Mediastinal lymph node and spleen epitope-specific CD8(+

103 significantly enhanced in the lung-draining mediastinal lymph node and spleen, and there is an incre

104 aortic surgery, the patient had undergone a mediastinal lymph node biopsy.

105 d the majority of prion-bearing cells in the mediastinal lymph node by six hours, indicating intranod

106 Mediastinal lymph node cells from Flt3L-treated mice sec

107 The in vitro cytokine secretion of mediastinal lymph node cells was determined using ELISA.

108 igh-affinity ligand CD155 was upregulated in mediastinal lymph node dendritic cells from allergic mic

109 m mass in the right upper lobe with multiple mediastinal lymph node disease ( Fig 1 ).

110 ned in a 1:1 ratio to SABR or lobectomy with mediastinal lymph node dissection or sampling.

111 l-cell lung cancer (NSCLC) is lobectomy with mediastinal lymph node dissection or sampling.

112 He then undergoes right upper lobectomy and mediastinal lymph node dissection, which demonstrate no

113 omography (FDG-PET/CT) imaging for detecting mediastinal lymph node involvement in patients with pote

114 l outcomes, including pathologic evidence of mediastinal lymph node involvement, distant metastasis,

115 Sensitivity for detecting mediastinal lymph node metastases, using pathologic conf

116 with suspected lung cancer, the presence of mediastinal lymph node metastasis is a critical determin

117 The mediastinal lymph node NK cells were activated, expressi

118 h alloantigen-induced expression of IL-10 in mediastinal lymph node or splenic T cells, intragraft ex

119 Lastly, mediastinal lymph node re-stimulation experiments showed

120 mputed tomography screening with and without mediastinal lymph node resection (MLNR) under an Institu

121 n 1 cm by computed tomography scan underwent mediastinal lymph node sampling to rule out N2 disease.

122 as early as 2 days post-IN inoculation; the mediastinal lymph node was an early site of replication

123 Negative regulation of cells in the mediastinal lymph node was minimal compared with that pr

124 airways (bronchoalveolar lavage), lung, and mediastinal lymph node were examined 10 d postinfection

125 lored DC subset present in the lung-draining mediastinal lymph node.

126 lung and enhanced the T(H)2 response in the mediastinal lymph node.

127 d with a decrease in the number of DC in the mediastinal lymph node.

128 sulted in an abolished T(H)2 response in the mediastinal lymph node.

129 berculosis Ag85B-specific CD4 T cells in the mediastinal lymph node.

130 ungs, it decreased trafficking of DCs to the mediastinal lymph node.

131 h occurs earliest in the local lung-draining mediastinal lymph node.

132 Unlike examination of mediastinal lymph nodes (LNs), which depends on surgical

133 lymphocytes rapidly redistribute to regional mediastinal lymph nodes (MedLNs) during influenza infect

134 nd, to a lesser extent, in the lung-draining mediastinal lymph nodes (medLNs) of virus-infected mice.

135 sponses were measured in the lungs, draining mediastinal lymph nodes (MLN), and spleens.

136 ent activation and migration to the draining mediastinal lymph nodes (MLNs) during IV infection.

137 0) levels remained elevated in the lungs and mediastinal lymph nodes (mLNs) throughout the acute LCMV

138 ce, by residual antigen in the lung-draining mediastinal lymph nodes (MLNs).

139 g naive CD4 T cells appear to migrate to the mediastinal lymph nodes along a CD62L-independent, CCR7-

140 uppressed the accumulation of T cells in the mediastinal lymph nodes and lung granulomatous regions w

141 regs rapidly accumulate in the lung-draining mediastinal lymph nodes and lungs.

142 tivation of DN1 T cells was initiated in the mediastinal lymph nodes and showed faster kinetics compa

143 arise in mesenteric, axillary/brachial, and mediastinal lymph nodes and spleen based on differential

144 influenza-specific CD8 T cells in lymphoid (mediastinal lymph nodes and spleen) and nonlymphoid tiss

145 T cells in the mediastinal lymph nodes and the intramyocardial endothel

146 failed to proliferate as extensively in the mediastinal lymph nodes as in mice infected only with BC

147 ere is a large right hilar mass and enlarged mediastinal lymph nodes but no pulmonary emboli.

148 ures yet contributed to TH2 expansion in the mediastinal lymph nodes but not in the lungs.

149 are retained in the peritoneum and draining mediastinal lymph nodes for a prolonged period following

150 Granulomas and mediastinal lymph nodes from active-disease but not late

151 Mediastinal lymph nodes from airway-sensitized Abcg1(-/-

152 in vivo and likewise detect mKATE2(+) DCs in mediastinal lymph nodes from infected mice.

153 rformance of MR imaging in staging hilar and mediastinal lymph nodes in NSCLC on both a per-patient a

154 illus but greatly diminishes their egress to mediastinal lymph nodes independent of neutrophil microb

155 ession of interleukin (IL)-17 transcripts in mediastinal lymph nodes induced by effector cells alone.

156 Therefore, mediastinal lymph nodes may be false-positive on (18)F-F

157 was stratified according to the presence of mediastinal lymph nodes measuring 1 cm or more in the sh

158 xamined the T(H)2 cytokine production in the mediastinal lymph nodes of DEP-exposed CCR2 knockout and

159 -specific CD4(+) T-cell proliferation in the mediastinal lymph nodes of mice.

160 ma-producing CD4(+) T cells in the lungs and mediastinal lymph nodes of the CXCR3-deficient strain wa

161 ic analysis of CD11c(+) dendritic cells from mediastinal lymph nodes of the infected mice showed that

162 mic sites and morphologic characteristics of mediastinal lymph nodes on spiral computed tomography fo

163 MATERIAL/METHODS: Anatomical distribution of mediastinal lymph nodes on spiral CT was reviewed in 39

164 The NKT cells were not observed in mediastinal lymph nodes or granulomatous lesions.

165 In contrast, mediastinal lymph nodes remained nonluminescent througho

166 cells and more IFN-gamma from PBMC, BAL, and mediastinal lymph nodes than monkeys with latent infecti

167 f) of naive CD4 T cells appears to enter the mediastinal lymph nodes through a blood-to-lung-to-lymph

168 lumen and did not need to spread through the mediastinal lymph nodes to cause a systemic infection.

169 r regions of p16 and CDH13 in both tumor and mediastinal lymph nodes was associated with an odds rati

170 ission tomography detected metastases to the mediastinal lymph nodes with accuracies of 93, 81, and 8

171 oscopy to sample peripheral lung lesions and mediastinal lymph nodes with standard bronchoscopic inst

172 es in murine hearts, pericardial AT, spleen, mediastinal lymph nodes, and bone marrow were quantified

173 of infection, is initially restricted to the mediastinal lymph nodes, and does not involve other lymp

174 n early (36 h-4 d) expansion of Tregs in the mediastinal lymph nodes, and later (12-16 d) increases i

175 l distribution and morphological patterns of mediastinal lymph nodes, as demonstrated on spiral CT, c

176 ration (EBUS-TBNA) biopsies of the hilar and mediastinal lymph nodes, but the feasibility and usefuln

177 there was a marked expansion of cells within mediastinal lymph nodes, comprised mainly of innate lymp

178 Such granulomas occur in the lung and the mediastinal lymph nodes, in the heart, and in other vita

179 those in the spleen, bronchoalveolar lavage, mediastinal lymph nodes, or lung.

180 Using this novel approach to study DCs in mediastinal lymph nodes, we observed that most blood-der

181 h2, Th17 cells, and Tregs, in the spleen and mediastinal lymph nodes, with expansion of splenic antig

182 dritic cells (DCs) in lung and lung-draining mediastinal lymph nodes, with lung CD11b(+) DCs displayi

183 in the lungs and the presence of bacteria in mediastinal lymph nodes, with necrosis and inflammation.

184 ease in the size of the right hilar mass and mediastinal lymph nodes.

185 ptoms unexpectedly showed high FDG uptake in mediastinal lymph nodes.

186 ased C. neoformans-specific Th2 cells in the mediastinal lymph nodes.

187 e number of DCs carrying OVA in the lung and mediastinal lymph nodes.

188 ung conventional dendritic cells to draining mediastinal lymph nodes.

189 res across the lung epithelium into draining mediastinal lymph nodes.

190 s in the cervical lymph nodes but not in the mediastinal lymph nodes.

191 ncreased numbers of NK cells in the lung and mediastinal lymph nodes.

192 uickly out of the lung and into the thoracic/mediastinal lymph nodes.

193 charge translocate rapidly from the lung to mediastinal lymph nodes.

194 production in blood but not in the affected mediastinal lymph nodes.

195 the lungs and central memory T cells in the mediastinal lymph nodes.

196 asized to other lobes of the lung and to the mediastinal lymph nodes.

197 pecific CD8+ T cell counts in the spleen and mediastinal lymph nodes.

198 ion of pulmonary dendritic cells (DC) to the mediastinal lymph nodes.

199 RNA expression and impaired DC homing to the mediastinal lymph nodes.

200 ll as several enlarged hilar and ipsilateral mediastinal lymph nodes.

201 hemagglutinin were primarily observed in the mediastinal lymph nodes.

202 h greater regulatory T cell expansion in the mediastinal lymph nodes.

203 Patients with isolated mediastinal lymphadenopathy (IML) are a common presentat

204 and more interlobular septal thickening and mediastinal lymphadenopathy on computed tomography of th

205 rdial delayed enhancement of the septum, and mediastinal lymphadenopathy should raise the suspicion f

206 osed in 5 of 77 patients (6.5%), while hilar/mediastinal lymphadenopathy was found in 25 of 76 patien

207 rdial delayed enhancement of the septum, and mediastinal lymphadenopathy were more often see in those

208 (PET) scan confirmed the lung lesion and the mediastinal lymphadenopathy without distant metastases.

209 ary lesions in the lungs along with necrotic mediastinal lymphadenopathy.

210 pital for examination of bilateral hilar and mediastinal lymphadenopathy.

211 ents were divided into four groups: anterior mediastinal lymphoma (group A, n=16), anterior mediastin

212 differences between prechemotherapy SUVt of mediastinal lymphoma and normal thymus and postchemother

213 De novo mediastinal lymphoma was correctly diagnosed by EBUS-TBN

214 n 100 cases of de novo or suspected relapsed mediastinal lymphoma was investigated by comparing EBUS-

215 nd accuracy of EBUS-TBNA in the diagnosis of mediastinal lymphoma were 89%, 97%, 98%, 83%, and 91%, r

216 diastinal lymphoma (group A, n=16), anterior mediastinal lymphoma with subsequent recurrence (group B

217 Vt of 3.4 or higher is a strong predictor of mediastinal lymphoma.

218 tion of normal thymus or thymic rebound from mediastinal lymphoma.

219 an be successful in the diagnosis of de novo mediastinal lymphomas and is ideally suited in distingui

220 Emicro-Myc/DNMT3B7 mediastinal lymphomas have more chromosomal rearrangemen

221 NMT3B7 expression increases the frequency of mediastinal lymphomas in Emicro-Myc animals.

222 Recently, mediastinal lymphomas with features intermediate between

223 ibitor A20, in Hodgkin lymphomas and primary mediastinal lymphomas.

224 images demonstrated the presence of a large mediastinal mass (11x8 cm) located in the anterior media

225 or and transverse dimensions of the anterior mediastinal mass or thymus on axial CT images and measur

226 otic systems, involving the thoracic cavity (mediastinal mass resections, lobectomies, and esophagect

227 Of the 9263 individuals, 71 had a mediastinal mass seen at baseline screening (prevalence

228 1 126 annual repeat screenings, only one new mediastinal mass was identified (incidence of 0.01%).

229 rocyte sedimentation rate, B symptoms, large mediastinal mass, extranodal disease, and 3 or more lymp

230 ximum standardized uptake values of anterior mediastinal mass, thymus (SUVt), and bone marrow at the

231 3 years (range, 14 to 59 years), and 46% had mediastinal masses >/=10 cm.

232 All documented mediastinal masses among the 9263 baseline and 11 126 an

233 Mediastinal masses found in the context of CT screening

234 This suggests a long average duration for mediastinal masses in asymptomatic people.

235 sions in a posttreatment status including 30 mediastinal masses in lymphoma.

236 The prevalence and incidence of mediastinal masses were then determined.

237 ted thoracoscopic surgery, pectus repair and mediastinal masses will be discussed.

238 ): 22 volunteers and nine patients (two with mediastinal masses, seven with pulmonary arterial hypert

239 nts are usually young and present with large mediastinal masses.

240 In response to mediastinal nerve stimulation, most IC neurons became ex

241 Risk factors were female sex (RR = 3.1), mediastinal NHL disease (RR = 5.2), and breast irradiati

242 uisition and optimization, identification of mediastinal nodal and vascular structures, EBUS-TBNA sam

243 and colorectal cancers and of CT in lung and mediastinal nodal disease points to future tailored use

244 non-small-cell lung cancer with ipsilateral mediastinal nodal metastases (N2) have shown the feasibi

245 nths postoperatively and required a negative mediastinal node biopsy, no excessive vitamin intake, no

246 re biopsies of the lung mass and ipsilateral mediastinal nodes confirmed a poorly differentiated non-

247 F was seen in cases of mildly (18)F-FDG-avid mediastinal nodes in lung cancer and small liver metasta

248 , and cisplatin for relapse in the lungs and mediastinal nodes with a rising AFP level starting in Ja

249 (absence of (18)F-FDG-avid foci in nonhilar mediastinal nodes), symmetry (difference between left an

250 evidence of testicular, retroperitoneal, or mediastinal non-seminomatous germ cell tumours based on

251 e prognostic factors in both groups; primary mediastinal nonseminoma (group A) and elevations of alph

252 nd were particularly prevalent among primary mediastinal nonseminomas (72%).

253 Five (24%) of 21 primary mediastinal nonseminomatous GCTs are continuously diseas

254 Mediastinal nonseminomatous GCTs carry a poor prognosis

255 perforation or anastomotic leak with limited mediastinal or pleural contamination.

256 safe and effective when performed along with mediastinal or pleural drainage.

257 s; (c) similar visual perception of noise on mediastinal (P = .132) and lung (P = .366) images, mainl

258 scopy and surgery); 13 patients had positive mediastinal PET findings, and 77 had negative mediastina

259 ediastinal PET findings, and 77 had negative mediastinal PET findings.

260 gnosis (benign vs malignant) on the basis of mediastinal pleural thickening (sensitivity, 81%; specif

261 e interpretation, the diagnostic accuracy of mediastinal pleural thickening, shrinking lung (hemithor

262 ations, performing substantially better than mediastinal pleural thickness and shrinking lung, and mi

263 e finding of frequent TP53 alterations among mediastinal primary nonseminomas may explain the more fr

264 Mediastinal primary site and two or more lines of prior

265 later therapy, platinum-refractory disease, mediastinal primary tumor site, nonseminoma histology, i

266 Mediastinal primary, pulmonary metastases, age, or doses

267 llaborative Group (IGCCCG) prognostic group, mediastinal primary, pulmonary metastases, and smoking a

268 Recently, the recognition that mediastinal radiation is associated with significant lon

269 ns for TI included porcelain aorta, previous mediastinal radiation, chest wall deformity, and potenti

270 lude a combination of immunochemotherapy and mediastinal radiation.

271 efficacy and challenge the need for routine mediastinal radiation.

272 D for patients receiving a MHD of 20 Gy from mediastinal radiotherapy, compared with patients not tre

273 e has resulted in routine consolidation with mediastinal radiotherapy, which has potentially serious

274 apy, compared with patients not treated with mediastinal radiotherapy.

275 Global innervation assessed with heart-to-mediastinal ratio and washout rates was preserved in all

276 ons including cardiopulmonary resuscitation, mediastinal reexploration, placement on extracorporeal m

277 t commonly found in sacrococcygeal, gonadal, mediastinal, retroperitoneal, cervicofacial and intracra

278 Mean counts in mediastinal ROI were computed from a fixed volume in 3 d

279 method that incorporates various cardiac and mediastinal segmentation schemes in which upper and lowe

280 ed inflammatory and atelectatic changes with mediastinal shift to the right.

281 The ratio between myocardial and mediastinal signal at 15 and 125 min and extrapolated at

282 The mediastinal signal was measured and fitted to a linear m

283 On MVA, primary mediastinal site (P < .001), two or more lines of prior

284 aphy-guided transbronchial needle aspiration mediastinal staging (EBUS group) in 62 patients (37.3%)

285 S may allow near-complete minimally invasive mediastinal staging in patients with suspected lung canc

286 plus EBUS may be an alternative approach for mediastinal staging in patients with suspected lung canc

287 NA) is an established technique for invasive mediastinal staging of non-small cell lung cancer (NSCLC

288 nded as a first-line diagnostic modality for mediastinal staging.

289 th parameters allowing for identification of mediastinal structures and adrenal glands is still much

290 pneumonectomy, proximal to or involved with mediastinal structures, abutting the chest wall, or recu

291 nition of EBUS/computed tomography images of mediastinal structures.

292 was identified and removed in four patients (mediastinal teratoma, thymoma, thymic carcinoma and thyr

293 human B-cell lymphomas (follicular (FL) and mediastinal) that changes serine (Ser)525 (TCA) to proli

294 graphy (PET/CT) in the management of primary mediastinal (thymic) large B-cell lymphoma (PMBCL).

295 It is within this mediastinal tissue that T cells develop and are extensiv

296 edge of benign conditions that might mimic a mediastinal vascular pathology is important for therapeu

297 PS if a solid portion was detectable in the mediastinal window setting (nonmeasurable, < 50%, or > 5

298 ially, images were displayed with a standard mediastinal window setting (window width, W = 400 HU; wi

299 nclusion Detection of a solid portion in the mediastinal window setting allows subsolid nodules to be

300 When a solid portion was measurable in the mediastinal window, the specificity for adenocarcinoma i