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1 conversion of the isoflavone formononetin to medicarpin.
2 f the antimicrobial isoflavonoid phytoalexin medicarpin.
3 for use in studies to selectively eliminate medicarpin accumulation to assess the relative importanc
5 response through 48 h for compounds such as medicarpin and daidzin; and a lesser delayed and protrac
8 to specifically hydroxylate the phytoalexins medicarpin and maackiain, converting them to less fungit
9 nthesis (chalcone synthase), a key enzyme in medicarpin biosynthesis (isoflavone reductase) and a key
10 sm, and indicate differential mechanisms for medicarpin biosynthesis depending on the nature of elici
11 induced along with other enzymes involved in medicarpin biosynthesis, methylates the A-ring 7-hydroxy
12 ciated with endoplasmic reticulum membranes; medicarpin biosynthetic enzymes have been localized to t
13 alfalfa root isoflavonoids formononetin and medicarpin but not by two triterpenoids present in alfal
14 resulted in accumulation of the phytoalexin medicarpin, coordinated increases in isoflavonoid precur
15 emical mechanisms underlying accumulation of medicarpin differ depending on the nature of the stimulu
17 es involved in the production of phytoalexin medicarpin in M. truncatula upon infection with P. pachy
18 ula accumulated the isoflavonoid phytoalexin medicarpin in response to yeast elicitor or methyl jasmo
19 ulation to assess the relative importance of medicarpin in the antifungal defense mechanisms of alfal
20 n of formononetin (4'-O-methyl daidzein) and medicarpin in the leaves than does elicitation or infect
21 ylated isoflavonoids such as the phytoalexin medicarpin in vivo, whereas biochemical studies indicate
27 NA construct readily metabolized infiltrated medicarpin to 1a-hydroxymedicarpin, indicating high leve
28 Enhanced accumulation of the phytoalexin medicarpin was observed in P. medicaginis-infected leave
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