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1 1 malignant FNA, 5 oncocytic/Hurthle cell, 2 medullary, 1 follicular, and 4 metastases from underlyin
2 ced V-ATPase apical membrane accumulation in medullary A-ICs but not in cortical A-ICs or other IC su
3                         Our study shows that medullary A-ICs respond to luminal adenosine through ADO
4 nce of the NKCC2 transporter responsible for medullary accumulation of ammonium.
5 nsepithelial resistance, and defective renal medullary accumulation of sodium and other osmolytes.
6 -pituitary-adrenal axis, sympathetic adrenal-medullary activation and catecholamine levels, the infla
7 001), cortical ADC (r = 0.43, P = .003), and medullary ADC (r = 0.35, P = .01).
8       Reconstructions reveal that many trans-medullary-afferents (TmAs) connect the eye with each LGM
9  progression, was associated with aggressive medullary and anaplastic carcinomas, and its expression
10  other, more rare subtypes of thyroid cancer-medullary and anaplastic-are ideally treated by physicia
11 or of total renal NKCC2 protein abundance in medullary and cortical thick ascending limb.
12 tic disorders, cyst-dependent obstruction of medullary and cortical tubules initiates a process culmi
13  of the sympathetic nervous system, expanded medullary and extramedullary hematopoiesis.
14 n of SLAMF7-CAR T cells led to resolution of medullary and extramedullary myeloma manifestations in a
15 eatment options for patients with hereditary medullary and nonmedullary thyroid cancers related to mu
16 t due to the accumulation of ammonium in the medullary and papillary interstitium.
17 rons in the medial aspect of the superficial medullary and spinal dorsal horn from the trigeminal sub
18                   The latter is reflected in medullary and supramedullary control.
19 -expressing NK cells, including conventional medullary and thymic NK cells, whereas TRAIL(+) Eomes(-)
20 llular scaffolds with vascular, cortical and medullary architecture, a collecting system and ureters.
21 d into the preBotzinger complex (preBotC), a medullary area that is critical for breathing.
22 CR7, how other T cell subsets gain access to medullary areas during their normal development is not c
23 eses that in conditions of MeCP2 deficiency, medullary astrocytes are unable to produce/release appro
24 (ii) MeCP2 deficiency impairs the ability of medullary astrocytes to sense changes in PCO2/[H(+) ].
25 tude of CO2 -induced [Ca(2+) ]i responses in medullary astrocytes was markedly reduced in conditions
26 ice with normal angiopoietin-Tie2 signaling, medullary AVR exhibited an unusual hybrid endothelial ph
27  adrenal cortex, and the sympathetic-adrenal-medullary axis, which controls stress-induced catecholam
28 some also prevented the secondary decline in medullary blood flow and ischemia that develops 2 hours
29 ctive role in renal IR injury by maintaining medullary blood flow and that a genetic deficiency in th
30                                              Medullary bone (MB), an estrogen-dependent reproductive
31 hat progressively increases from the cortico-medullary boundary to the inner medullary tip.
32 ning REM sleep is located in the pontine and medullary brainstem and includes ascending and descendin
33 c databases revealed ANTXR1 amplification in medullary breast carcinoma and overexpression in estroge
34 atory muscle, and in patients with defective medullary breathing pattern generators.
35                                              Medullary, but not cortical, ADCT values stayed increase
36 nce of Wnt7b signaling, the periureteric bud medullary capillaries displayed narrower lumens lined wi
37 ing and the ureteric bud epithelium in renal medullary capillary development.
38 ndocrine glands-like C cells of the thyroid (medullary carcinoma), the parasympathetic and sympatheti
39  we test whether these rostral ventrolateral medullary catecholaminergic (RVLM-CA) neurons use glutam
40  which bone tissue progressively invades the medullary cavity in the mid-diaphysis.
41  of the limb bones are solid without an open medullary cavity, for buoyancy control in water.
42 st PSB-0739 in primary cultures of rat inner medullary CD cells potentiated the expression of AQP2 an
43                           GPC protects inner medullary cells against the perturbing effects of high N
44 ine release was measured from bovine adrenal medullary chromaffin cell (CC) cultures maintained over
45 ine and norepinephrine released from adrenal medullary chromaffin cells and norepinephrine released l
46                       Mice lacking the inner medullary collecting duct (IMCD) urea transporter A1 (UT
47 n regulates NO production in the renal inner medullary collecting duct (IMCD), the segment with the g
48  3D cell culture model that uses mouse inner-medullary collecting duct (mIMCD3) cells to generate epi
49 hibition of p38MAPK activity in murine inner medullary collecting duct 3 (mIMCD3) cells decreased exp
50 (PI3K-C2alpha) in renal tubule-derived inner medullary collecting duct 3 cells and show that PI3K-C2a
51 ar to cells lacking PC2, NEK8-depleted inner medullary collecting duct cells exhibited a defective re
52  genetic knockdown of TRIP13 in murine inner medullary collecting duct cells in the presence of hydro
53 retinal pigmented epithelial and mouse inner medullary collecting duct cells-3.
54 s from the proximal tubule through the inner medullary collecting duct of rat kidneys.
55 ely integrated into the aquaporin 2-positive medullary collecting duct when microinjected into the ki
56      ENaC was appropriately increased in the medullary collecting duct, suggesting a localized inhibi
57 ubule and decreased slightly in the cortical/medullary collecting duct, whereas BK channel abundance
58 nts with the highest expression in the inner medullary collecting duct.
59      We employed proteomic analysis of inner medullary collecting ducts (IMCD) from rats fed with a p
60 n vitro microperfusion of cortical and outer medullary collecting ducts, was unaffected in mutant mic
61 de use of primarily cultured rat renal inner medullary collecting-duct cells and microarray analysis
62 artment kidney phantom (70% cortical and 30% medullary compartment), a sphere, and an ellipsoid of eq
63 TECs and in addition affects the size of the medullary compartment, TEC-specific HIPK2 deletion only
64 t are connected to or separated from a major medullary compartment.
65 ion-mediated cross-talk in generation of the medullary compartment.
66                  The microenvironment of the medullary cords is positioned to control these key proce
67 at yellow fluorescent protein(+) ASCs in the medullary cords migrated along myelomonocytic cells and
68 s sensed higher concentrations of S1P in the medullary cords than in the T cell zone and that the S1P
69 h environments were observed in the expanded medullary cords.
70 omplete SCI, we created bilaterally complete medullary corticospinal tract lesions in adult mice, eli
71 utosomal dominant polycystic kidney disease, medullary cystic kidney disease, diabetic nephropathy, o
72 These findings demonstrate that cortical and medullary DCs play specialized roles in their respective
73 he immune defense against pyelonephritis, as medullary DCs were less CX3CR1 dependent than cortical D
74 contrast, Foxp3(+) nTreg cell development is medullary dependent, with mTECs fostering the generation
75 F-kappaB in the thymus and are necessary for medullary development.
76 equirement for cross-talk with thymocytes in medullary development.
77 tricted to interneurons in lamina IIi of the medullary dorsal horn, where they constitute 1/3 of tota
78 ters by projection neurons in the spinal and medullary dorsal horn.
79               Mechanisms promoting thymocyte medullary entry and interactions with APCs are incomplet
80 microscopy to demonstrate that CCR4 promotes medullary entry of the earliest post-positive selection
81 vation with 5-thioglucose stimulated adrenal medullary epinephrine (Epi) release (3,153%) and feeding
82 fluenced the ability of Aire to regulate the medullary epithelial cell transcriptome and so was cruci
83 alysis of Aire-sufficient and Aire-deficient medullary epithelial cells (mTECs).
84  with increased frequencies of AIRE-positive medullary epithelial cells and CD11c-positive dendritic
85 expression of a large set of genes in thymic medullary epithelial cells, thereby controlling the repe
86 d CD40-CD40L pathways is required for normal medullary epithelium and for maintenance of self-toleran
87 -) and that transitioned into a multilaminar medullary epithelium forming neurotubules with adluminal
88 ssion of tissue-specific genes in the thymic medullary epithelium.
89 al source for the tumor and its multilaminar medullary epithelium.
90 ng youth were maintained as a result of less medullary expansion and cortical trabecularization.
91 youth were gradually lost because of greater medullary expansion and cortical trabecularization.
92                          Here we examine the medullary expression of sst2a with particular reference
93 was significant correlation between eGFR and medullary FA (r = 0.65, P < .001), cortical ADC (r = 0.4
94                             In group B, mean medullary FA was significantly lower in patients whose r
95 owed a 14-24 fold increase from baseline for medullary GRP78, sXBP-1, and CHOP.
96                                              Medullary hyperosmolarity is protected from washout by c
97                                              Medullary hypoxia due to intrarenal blood flow redistrib
98                                              Medullary hypoxia-inducible factor gene expression decre
99 ll mice was markedly increased, resulting in medullary hypoxia.
100 ependent protein phosphorylation in purified medullary ICs that were isolated from mice.
101 hat adenosine activates V-ATPase in isolated medullary ICs.
102 cal deficit that was attributable to lateral medullary infarction.
103                               Proliferating, medullary, interstitial myofibroblasts strongly expresse
104 e mice, an effect accompanied by a hypotonic medullary interstitium and impaired countercurrent multi
105             The hyperosmolality in the renal medullary interstitium is of major importance as a drivi
106 al reabsorption of water into the hypertonic medullary interstitium mediated by collecting ducts.
107 rapid accumulation of fluid and cysts in the medullary interstitium, loss of medullary vascular bundl
108 s canonical Wnt signaling in the surrounding medullary interstitium, where the capillaries reside.
109 ith predominant involvement of glomeruli and medullary interstitium.
110 /-) mice exhibit only ~20 small, unconnected medullary islets.
111 ormally induces clearance of the renal outer medullary K(+) channel (ROMK) from the cell surface.
112 ne-independent activation of the renal outer medullary K(+) channel and ENaC, to which angiotensin II
113 iets induced upregulation of the renal outer medullary K(+) channel in AS(-/-) mice, whereas upregula
114 +) and NeuN(+) cells within the multilayered medullary layer approximate expression patterns similar
115                    All patients showed a new medullary lesion on brain magnetic resonance imaging.
116 ystagmus had cerebellar peduncle and lateral medullary lesions.
117 of their role has been limited to spinal and medullary loci.
118 s to positions adjacent to both cortical and medullary lymphatic sinuses where the T cells exhibited
119 ered a program of PGE that is common between medullary (m) and cortical TEC, further elaborated in mT
120 vant MF59 localizes in subcapsular sinus and medullary macrophage compartments of mouse draining LNs,
121               CD169(+) SIGNR1(+) subcapsular medullary macrophages are the primary cells to take up G
122 the development and functional importance of medullary microenvironments during self-tolerant T-cell
123  thymus, interactions with both cortical and medullary microenvironments regulate the development of
124 of thymocyte precursors through cortical and medullary microenvironments, enabling specialized stroma
125 ire are involved in the regulation of thymus medullary microenvironments.
126 indicate that a functional compromise of the medullary mTEC(high) compartment may link alloimmunity t
127  chromophobe (n = 5), papillary (n = 5), and medullary (n = 2) RCC and unclassified RCC (uRCC, n = 23
128 mmunoblotting revealed downregulation of the medullary Na(+)-K(+)-2Cl(-) cotransporter NKCC2 in these
129 ation between chronic hyperaldosteronism and medullary nephrocalcinosis has rarely been made, with on
130  a long- standing history in whom associated medullary nephrocalcinosis was established.
131  increased risk of kidney stone formation or medullary nephrocalcinosis, namely 46% compared with 6%
132 s a causal factor in the etiopathogenesis of medullary nephrocalcinosis.
133  identification of a discrete JAG1(+) thymic medullary niche enriched for DC-lineage cells expressing
134 in multiple cortical areas, and thalamic and medullary nuclei.
135 6-fold specifically in these plastic, caudal medullary nuclei.
136 urn, the PAG has strong access to the caudal medullary nucleus retroambiguus (NRA).
137  to mammalian water balance and depends on a medullary osmotic gradient generated by a countercurrent
138 cal oxygenation and perfusion, but decreased medullary oxygenation and perfusion.
139 3 to 36.3 +/- 3.5 mm Hg (p < 0.001), whereas medullary oxygenation decreased from 29.6 +/- 4.7 to 13.
140 though sleep-active GABAergic neurons in the medullary parafacial zone (PZ) are needed for normal SWS
141                     Stenotic kidney cortical/medullary perfusion and RBF were measured using contrast
142 perfusion was not significantly changed, but medullary perfusion decreased (671 BPU [500-900 BPU] to
143                                              Medullary perfusion in atherosclerotic RAS patients was
144                      Single kidney cortical, medullary perfusion, and renal blood flow were measured
145 mic accumulation of mature thymocytes within medullary perivascular spaces and reduced numbers of rec
146 n AC6(-/-) mice seem to be restricted to the medullary portion of the thick ascending limb.
147 ition of the potassium-excretory renal outer medullary potassium (ROMK) channel have also been implic
148 me is caused by mutations in the renal outer medullary potassium (ROMK) channel, but the molecular me
149 elial sodium channel (ENaC), the renal outer medullary potassium channel (ROMK), and other transport
150                              The renal outer medullary potassium channel (ROMK, or Kir1.1, encoded by
151 A, SUR1B, SUR2A, SUR2B, or ROMK (renal outer medullary potassium channel).
152 epithelial sodium channel-alpha, renal outer medullary potassium channel, and Na(+), K(+)-ATPase in t
153  on functional expression of the renal outer medullary potassium channel.
154 at changes in the electrical excitability of medullary pre-sympathetic neurones are the main causal m
155 tability of respiratory rhythm recorded from medullary preparations decreased with age but was higher
156 on of both AQP2 and pAQP2 in the renal inner medullary principal cells appeared more dispersed, and t
157 cement of electrodes less than 5 mm from the medullary pyramids resulted in treatment effect arcing i
158  as well as axonal loss, at the level of the medullary pyramids.
159 ventional regional estimates of cortical and medullary R2* levels were measured.
160 ional tissue hypoxia rather than cortical or medullary R2* values used to assess renal BOLD MR imagin
161 ing the nucleus of the solitary tract (NTS), medullary raphe and retrotrapezoid nucleus (RTN).
162 ons of ATP or a P2-receptor blocker into the medullary raphe had no effect on cardiorespiratory activ
163 lamic nucleus [PaMP]) and autonomic (rostral medullary raphe pallidus [RPa]) responses were targeted
164 if CO2/H(+)-sensitive neurons in the NTS and medullary raphe respond to ATP, and whether purinergic s
165 inase drivers targeted different portions of medullary raphe serotonergic, tryptophan hydroxylase 2 (
166                                              Medullary raphe transcriptome comparisons revealed lower
167 m several respiratory centres, including the medullary raphe.
168  (vertical phloem and inter-connected radial medullary rays [MR]).
169 g in the pre-Botzinger complex (pre-BotC), a medullary region generating the inspiratory phase of bre
170                   These mice lacked a thymic medullary region, exhibited thymocyte retention, had a p
171 cluding the rostral and caudal ventrolateral medullary regions (RVLM and CVLM, respectively).
172 of spindle-shaped cells in both cortical and medullary regions of the kidney.
173 ) has previously been identified as an extra-medullary reservoir for normal hematopoietic stem cells
174 nimals with MeCP2 removed from specific pons medullary respiratory circuits, we performed whole-body
175 nctional connection between the amygdala and medullary respiratory network in humans.
176 esults indicate that MeCP2 expression in the medullary respiratory network is sufficient for normal r
177 e from lumbar locomotor CPG circuitry to the medullary respiratory networks that is able to depolariz
178 lterations in the electrical excitability of medullary respiratory neurones and their central modulat
179 eir spinal projections, several parts of the medullary reticular formation as well as the spinally pr
180                                Lhx3-positive medullary reticular formation neurons express Fos follow
181                                       In the medullary reticular formation of the mouse, we identifie
182                       CTb injection into the medullary RF labels cells and axonal endings predominant
183 lox) kidneys showed more cell death in outer medullary S3 segments at 2 hours but less tubular damage
184 ressive increase of neuronal excitability in medullary slices containing the pre-BotC produces mixed-
185 nd hypoglossal (XII) nerve motor activity in medullary slices from neonatal mice in conditions where
186 holographic photolysis of caged glutamate in medullary slices from neonatal mice.
187                  We used rhythmically active medullary slices from wild-type (WT) and Lmx1b(f/f/p) ne
188  providing evidence for an independence from medullary support by the earliest stages after positive
189 ein, we evaluated the involvement of ventral medullary sympatho-excitatory catecholaminergic C1 neuro
190  heart rate variability (sympathetic adrenal medullary system), EEG event-related potentials (nocicep
191 s of p53 primarily disrupts the integrity of medullary TEC (mTEC) niche, a defect that spreads to the
192 a crucial transcription factor implicated in medullary TEC function.
193 ression of thymotropic chemokines, decreased medullary TEC to cortical TEC ratios, and altered thymic
194                                    In mature medullary TEC, AIRE-driven pGE upregulates non-TRA codin
195  adult mice, we found that both cortical and medullary TECs (cTECs and mTECs) proliferated more activ
196 el antigen specifically expressed in Aire(+) medullary TECs (mTECs) induced efficient deletion via di
197 antigens or presenting a neo-self-antigen by medullary TECs, displaying decreased negative selection-
198 The proximal convoluted tubule (PCT) and the medullary thick ascending limb (mTAL) in the kidney cons
199 tion of BMAL1 expression was observed in the medullary thick ascending limb.
200 (2+)]i oscillations were markedly reduced in medullary thick ascending limbs isolated from P2Y2 recep
201 thymocytes, and expression of its ligands by medullary thymic dendritic cells (DCs).
202 tical role in T cell tolerance by regulating medullary thymic epithelial cell (mTEC) development.
203 o-step terminal differentiation model of the medullary thymic epithelial cell (mTEC) lineage from imm
204 hough the molecular mediators that stimulate medullary thymic epithelial cell (mTEC) maturation are p
205 sting of a branching structure that contains medullary thymic epithelial cell (mTEC) networks to supp
206  reduction of autoimmune receptor-expressing medullary thymic epithelial cells (Aire1 mTEC) and a dec
207 ism, we have previously reported that mature medullary thymic epithelial cells (mTEC(high)) expressin
208 eral self-antigens (TRA), which is in mature medullary thymic epithelial cells (mTEC(high)) partly co
209 ize the developmental pathways that generate medullary thymic epithelial cells (mTEC) from their imma
210                               In the thymus, medullary thymic epithelial cells (mTEC) regulate T cell
211 in developing T cells is highly dependent on medullary thymic epithelial cells (mTEC), and mTEC devel
212    CD70 was expressed on Aire(-) and Aire(+) medullary thymic epithelial cells (mTECs) and on dendrit
213 ulate transcription of a battery of genes in medullary thymic epithelial cells (mTECs) and, consequen
214                                              Medullary thymic epithelial cells (mTECs) are critical i
215          In this study, we reveal a role for medullary thymic epithelial cells (mTECs) during iNKT ce
216                                              Medullary thymic epithelial cells (mTECs) eliminate self
217 of tissue-restricted self antigens (TRAs) in medullary thymic epithelial cells (mTECs) is essential f
218  roles for bone marrow (BM)-derived APCs and medullary thymic epithelial cells (mTECs) on the convent
219       We show that bone marrow (BM) APCs and medullary thymic epithelial cells (mTECs) played nonover
220  plethora of tissue-restricted Ags (TRAs) by medullary thymic epithelial cells (mTECs) plays an essen
221                                           In medullary thymic epithelial cells (mTECs), the Autoimmun
222 neages--cortical thymic epithelial cells and medullary thymic epithelial cells (mTECs)--are yet to be
223 g-presenting, autoimmune regulator (AIRE)(+) medullary thymic epithelial cells (mTECs).
224         Autoimmunity is largely prevented by medullary thymic epithelial cells (TECs) through their e
225 (+) T cells preferentially damaged recipient medullary thymic epithelial cells and impaired negative
226                           First, we increase medullary thymic epithelial cells by using mice lacking
227 on is enriched, particularly in neonates, in medullary thymic epithelial cells expressing the autoimm
228 lls that colocalize with dendritic cells and medullary thymic epithelial cells in the thymic medulla.
229 r of surface MHC-self peptide complexes from medullary thymic epithelial cells to thymic DC.
230 ng T cells and autoimmune regulator-positive medullary thymic epithelial cells, a key process for cen
231 nity by promoting self-antigen expression in medullary thymic epithelial cells, such that developing
232                                       Unlike medullary thymic epithelial cells, which express and pre
233 ngly autoimmune regulator-expressing, mature medullary thymic epithelial cells, which play a pivotal
234 a transcription factor which is expressed in medullary thymic epithelial cells.
235 xpression, particularly in keratinocytes and medullary thymic epithelial cells.
236 ssues of patients with APECED, especially in medullary thymic epithelial cells.
237 ssion of MPO mRNA predominantly localized to medullary thymic epithelial cells.
238 cells, cortical thymic epithelial cells, and medullary thymic epithelial cells.
239 n of Bim during early/cortical, but not late/medullary, thymic development controls the agonist selec
240 s, as well as efficient interactions between medullary thymocytes and DCs.
241 ative selection and functional maturation of medullary thymocytes as well as the production of regula
242 ytes exhibit a stochastic migration, whereas medullary thymocytes show confined migratory behavior.
243  were located in the medulla and conditioned medullary thymocytes.
244                                     Familial medullary thyroid cancer (MTC) and its precursor, C cell
245                                              Medullary thyroid cancer (MTC) can be caused by germline
246 ity of nonendocrine manifestations preceding medullary thyroid cancer (MTC) for early diagnosis of mu
247 onstrated clinical activity in patients with medullary thyroid cancer (MTC) in phase I.
248 tory differentiated thyroid cancer (DTC) and medullary thyroid cancer (MTC) in the past 10 years.
249 oring the prospects of a cure for persistent medullary thyroid cancer (MTC) stratified by basal calci
250  tumors including small cell lung cancer and medullary thyroid cancer (MTC).
251 ttee on Cancer (AJCC) TNM staging system for medullary thyroid cancer (MTC).
252 -degree relatives with histologically proven medullary thyroid cancer and phaeochromocytoma.
253                                              Medullary thyroid cancer arises from calcitonin-producin
254 their antiproliferative efficacy against the medullary thyroid cancer cell line TT.
255                            The prevention of medullary thyroid cancer in patients with multiple endoc
256 ycaemia, pancreatitis, pancreatic cancer, or medullary thyroid cancer reported between GLP-1 receptor
257          The discovery of a locally advanced medullary thyroid cancer that is not amenable to surgery
258 ment and treatment of patients with advanced medullary thyroid cancer with emphasis on current target
259 s cabozantinib (FDA-approved for progressive medullary thyroid cancer) and PF-04217903 block their ac
260  from a genome-wide association study of non-medullary thyroid cancer, including in total 3,001 patie
261                 Knockdown of RET by shRNA in medullary thyroid cancer-derived cells stimulated expres
262 ctivation of ATF4 during the pathogenesis of medullary thyroid cancer.
263 eted medical therapies are now available for medullary thyroid cancer.
264 tient (0.6%) died from incidentally detected medullary thyroid cancer.
265 MET in addition to VEGFR and is approved for medullary thyroid cancer.
266 nts that are approved for differentiated and medullary thyroid cancers have prolonged progression-fre
267 t breast cancers, non-Hodgkin lymphomas, and medullary thyroid cancers represent novel indications fo
268                                     One of 5 medullary thyroid cancers was positive with the agonist,
269 t frequently mutated malignant subtypes were medullary thyroid carcinoma (9/12, 75%) and PTC (14/30,
270 nase, are associated with the development of medullary thyroid carcinoma (MTC) and pathogenesis of mu
271                                              Medullary thyroid carcinoma (MTC) is a neuroendocrine ca
272                                              Medullary thyroid carcinoma (MTC) is a neuroendocrine tu
273 efines these 2 phenotypes is the presence of medullary thyroid carcinoma (MTC).
274 d peptides labeled with (111)In or (131)I in medullary thyroid carcinoma (MTC).
275 (4q32A>C) in a large pedigree displaying non-medullary thyroid carcinoma (NMTC).
276                                              Medullary thyroid carcinoma accounts for 2% to 5% of thy
277  explain the increased expression of CAIX in medullary thyroid carcinoma and provide a rationale for
278 ilar expression pattern was recapitulated in medullary thyroid carcinoma cells in vivo, consistent wi
279                                              Medullary thyroid carcinoma is a relatively rare tumor w
280 onal activation of mutated RET gene in human medullary thyroid carcinoma TT cells.
281 ce of acute pancreatitis, pancreatic cancer, medullary thyroid carcinoma, and serious adverse events
282 ogene and virtually all of them will develop medullary thyroid carcinoma.
283 ne in the TT cell line, derived from a human medullary thyroid carcinoma.
284 ic carcinomas, and its expression pattern in medullary thyroid carcinomas suggested contribution of b
285 ressed in various human cancers (e.g., lung, medullary thyroid, pancreatic, colon, and gastrointestin
286  the cortico-medullary boundary to the inner medullary tip.
287 normal sheep, resting levels of cortical and medullary tissue PO2 were 29.5 +/- 4.4 and 29.1 +/- 4.3
288     In one model, approximately one-third of medullary Tph2(+) neurons are silenced by postnatal (P)
289 in the other, approximately three-fourths of medullary Tph2(+) neurons, also with some Tph2(low or ne
290  the mechanisms underlying the modulation of medullary trigeminovascular (Sp5C) neurons have not been
291 ng autophagic defects in epithelial cells of medullary tubules.
292 t majority of thyroid cancers are of the non-medullary type.
293 cysts in the medullary interstitium, loss of medullary vascular bundles, and decreased urine concentr
294 exity of the tidal volume signal (related to medullary ventilatory command), (3) autonomic function,
295                     We measured cortical and medullary volumes, perfusion, and RBF using multidetecto
296 ogenous adrenal gland hormones (cortical and medullary) was associated with 3- to 10-fold higher conc
297 ation of the prohemocytes located within the medullary zone and the secondary lobes of the lymph glan
298                                 The central, medullary zone which contain undifferentiated hematopoie
299 ich normally consists of an immature center (medullary zone) where cells remain undifferentiated, and
300 known to be required to maintain an immature medullary zone.

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