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1 ctivation of ATF4 during the pathogenesis of medullary thyroid cancer.
2 eted medical therapies are now available for medullary thyroid cancer.
3 n life for RET mutation carriers at risk for medullary thyroid cancer.
4 tient (0.6%) died from incidentally detected medullary thyroid cancer.
5 es has been advocated for early detection of medullary thyroid cancer.
6 on in a family with hereditary renal and non-medullary thyroid cancer.
7 dic counterpart, but is less aggressive than medullary thyroid cancer.
8 (ab)2 with AHSCR in patients with metastatic medullary thyroid cancer.
9 patients with rapidly progressing metastatic medullary thyroid cancer.
10 MET in addition to VEGFR and is approved for medullary thyroid cancer.
11 ers without clinical disease before age 6 in medullary thyroid cancer and MEN type IIA, and as soon a
14 s cabozantinib (FDA-approved for progressive medullary thyroid cancer) and PF-04217903 block their ac
16 size to the uveal tract, even after decades; medullary thyroid cancer can be part of multiple endocri
21 nts that are approved for differentiated and medullary thyroid cancers have prolonged progression-fre
23 from a genome-wide association study of non-medullary thyroid cancer, including in total 3,001 patie
30 ity of nonendocrine manifestations preceding medullary thyroid cancer (MTC) for early diagnosis of mu
32 tory differentiated thyroid cancer (DTC) and medullary thyroid cancer (MTC) in the past 10 years.
35 s institutional study aimed at quantifying a medullary thyroid cancer (MTC) patient's risk of lung, l
37 oring the prospects of a cure for persistent medullary thyroid cancer (MTC) stratified by basal calci
38 ll provide an update of important studies in medullary thyroid cancer (MTC) with an emphasis on targe
39 sess the targeting of established and occult medullary thyroid cancer (MTC) with radiolabeled monoclo
40 f cabozantinib, which included patients with medullary thyroid cancer (MTC), led to expansion of an M
48 ged as a promising target for the therapy of medullary thyroid cancers (MTC) and of a subset of papil
49 an arise from parafollicular cells (familial medullary thyroid cancer) or from follicular cells (fami
50 lorectal, 9 lung, 7 pancreas, 6 breast and 4 medullary thyroid cancer patients), mostly in very advan
51 ts (8 colorectal, 3 lung, 1 pancreatic and 1 medullary thyroid cancer) received RAIT with 131I-NP-4 F
52 ycaemia, pancreatitis, pancreatic cancer, or medullary thyroid cancer reported between GLP-1 receptor
53 t breast cancers, non-Hodgkin lymphomas, and medullary thyroid cancers represent novel indications fo
56 tients having minimal disease, as well as in medullary thyroid cancer, where cytotoxic tumor doses mi
57 ment and treatment of patients with advanced medullary thyroid cancer with emphasis on current target
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