ライフサイエンスコーパス: megakaryocytic leukemia

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1 nsient myeloproliferative disorder and acute megakaryocytic leukemia.

2 e A (AURKA) is a therapeutic target in acute megakaryocytic leukemia.

3 not only lymphoid leukemia but also erythro-megakaryocytic leukemia.

4 7.5 years, respectively; P <.001), with more megakaryocytic leukemia (70% v 6%; P <.001).

5 Acute megakaryocytic leukemia (AMegL) is a rare subtype of acu

6 ty to develop leukemia, in particular, acute megakaryocytic leukemia (AMkL) associated with somatic G

7 ion of mature megakaryocyte markers in acute megakaryocytic leukemia (AMKL) blasts and displayed pote

8 ptional regulation of the CBS gene in the DS megakaryocytic leukemia (AMkL) cell line, CMK, character

9 Children with Down syndrome (DS) with acute megakaryocytic leukemia (AMkL) have very high survival r

10 ts in Down syndrome (DS) children with acute megakaryocytic leukemia (AMkL) were 4.4-fold (P < .001)

11 ient myeloproliferative disease (TMD), acute megakaryocytic leukemia (AMKL), and acute lymphoid leuke

12 ncreased in ML-DS compared with non-DS acute megakaryocytic leukemias (AMKLs).

13 esin mutations are highly prevalent in acute megakaryocytic leukemia associated with Down syndrome (D

14 We used a bcr-abl-deficient human megakaryocytic leukemia cell line MO7E and an isogenic M

15 However, MB-02 cells, a human megakaryocytic leukemia cell line which can be infected

16 Erythroleukemic and megakaryocytic leukemia cell lines (n = 5) bound little

17 CD34(+), CD61(+) cells, blood platelets, and megakaryocytic leukemia cell lines all expressed the CXC

18 eration, and autocrine production of IL-3 by megakaryocytic leukemia cell lines and bone marrow-deriv

19 scriptional regulation of IL-3 expression in megakaryocytic leukemia cell lines is similar, but not i

20 ecule probes that induce polyploidization of megakaryocytic leukemia cells and serve as perturbagens

21 lanoma cells, lymphoblastoid cell lines, and megakaryocytic leukemia cells by reverse transcriptase p

22 We used DAMI human megakaryocytic leukemia cells to study transmembrane ion

23 pharyngeal carcinoma) cells, and M07e (human megakaryocytic leukemia) cells were infected with vCMVp-

24 To characterize acute megakaryocytic leukemia (FAB M7 AML), we identified 37 p

25 and a 500-fold increased risk of developing megakaryocytic leukemia; however, the specific effects o

26 nsient myeloproliferative disorder and acute megakaryocytic leukemia in children with Down syndrome a

27 ormalities and underlies the pathogenesis of megakaryocytic leukemia in Down syndrome.

28 a fusion partner in t(1;22)-associated acute megakaryocytic leukemia, is also essential for maintaini

29 ted in Mks lacking Mkl1, which is mutated in megakaryocytic leukemia, is via elevated GEF-H1 expressi

30 and the aberrant erythroid phenotype of the megakaryocytic leukemias of DS.

31 elp elucidate the mechanism of t(1,22) acute megakaryocytic leukemia pathogenesis, a conditional alle

32 e uniform detection of GATA1 mutations in DS megakaryocytic leukemia suggested the potential role of

33 restricted to newborns with trisomy 21, is a megakaryocytic leukemia that although lethal in some is

34 In a subset of human megakaryocytic leukemias, the transcription factor GATA1

35 re congenital malformations, including acute megakaryocytic leukemia, transient myeloproliferative di

36 ose being the rare but uniformly fatal acute megakaryocytic leukemia type 7.

37 nique occurrence of myelodysplasia and acute megakaryocytic leukemia type 7.

38 n to the presence of few mutations was acute megakaryocytic leukemias, with the majority of these leu

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