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1 he existence of developmental differences in megakaryocytopoiesis.
2 to the pathogenesis of disorders of neonatal megakaryocytopoiesis.
3 opoiesis/monocytopoiesis, lymphopoiesis, and megakaryocytopoiesis.
4 ked stimulus to drive the observed excessive megakaryocytopoiesis.
5 strate that TRIB3 is a negative modulator of megakaryocytopoiesis.
6 , functioning as a thermostat to "fine-tune" megakaryocytopoiesis.
7 is presumably caused by an up-regulation in megakaryocytopoiesis.
8 nd may contribute to the local regulation of megakaryocytopoiesis.
9 ietic progenitor cell development and during megakaryocytopoiesis.
10 The lack of B-Raf also impairs megakaryocytopoiesis.
11 ikely to affect cell cycle regulation during megakaryocytopoiesis.
12 ansduction protein 14-3-3xi and to influence megakaryocytopoiesis.
13 tudied the effects of ITP plasma on in vitro megakaryocytopoiesis.
14 d to directly determine the role of Raf-1 in megakaryocytopoiesis.
15 ly and indirectly to play a critical role in megakaryocytopoiesis.
16 ly to regulate lineage-specific genes during megakaryocytopoiesis.
17 ay play important roles in the regulation of megakaryocytopoiesis.
18 level, selective gene transcription early in megakaryocytopoiesis.
19 red delivery of platelets despite stimulated megakaryocytopoiesis.
20 RF-dependent and SRF-independent activity in megakaryocytopoiesis.
21 d SDF-1alpha may modulate several aspects of megakaryocytopoiesis.
22 and is the primary physiologic regulator of megakaryocytopoiesis.
23 poietic factor involved in the regulation of megakaryocytopoiesis.
25 In studying the role of Tie-2 and Ang-1 in megakaryocytopoiesis, 3 alternatively spliced species of
26 (CD31) knockout (KO) mice exhibit excessive megakaryocytopoiesis accompanied by increased numbers of
27 t that B-Raf plays a cell-autonomous role in megakaryocytopoiesis and a permissive role in myeloid pr
29 ukopenia because of significant increases in megakaryocytopoiesis and concomitant blockage of erythro
30 high level of TPO overexpression stimulates megakaryocytopoiesis and myelopoiesis leading to thrombo
31 control of membrane remodeling, critical for megakaryocytopoiesis and normal platelet production and
32 ted regulatory effects of miRNAs relevant to megakaryocytopoiesis and platelet biology by analyzing e
35 y have evidence of underlying impaired fetal megakaryocytopoiesis and platelet production following p
36 hematopoietic growth factor that stimulates megakaryocytopoiesis and platelet production in vivo and
37 sk of bleeding, but its biological effect on megakaryocytopoiesis and platelet production is unknown.
38 rovide evidence for a novel pathway by which megakaryocytopoiesis and platelet production may be regu
41 delineate the expression of microRNAs during megakaryocytopoiesis and suggest a regulatory role of mi
42 suggest that VIP may have direct effects on megakaryocytopoiesis and support our earlier observation
43 ine kinase phosphatase PTP-RO is involved in megakaryocytopoiesis and that its function is mediated b
44 o, it has provided both unique insights into megakaryocytopoiesis and the means to stimulate platelet
48 c tail sequestered signaling proteins during megakaryocytopoiesis and, as such, became a critical reg
50 gs demonstrate that Raf-1 is dispensable for megakaryocytopoiesis, and for thrombopoietin-induced ERK
52 shown that rhIL-11-induced murine and human megakaryocytopoiesis are not mediated by thrombopoietin
53 Although the growth factors that regulate megakaryocytopoiesis are well known, the molecular deter
54 our understanding of normal versus malignant megakaryocytopoiesis, as well as aberrant mitosis in ane
55 hinted that TRIB3 could be also involved in megakaryocytopoiesis but its role in this process has so
56 pond to Mpl ligand, the pivotal regulator of megakaryocytopoiesis, by increasing their expression of
58 hrombopoietic cytokines regulating, in part, megakaryocytopoiesis during states of acute thrombocytop
59 creased PLT survival, but intact bone marrow megakaryocytopoiesis, endogenous IL-11 levels were signi
60 ed in the hepatic sinusoids support in vitro megakaryocytopoiesis from murine hematopoietic stem cell
65 reports have defined the in vivo changes in megakaryocytopoiesis in response to a single injection o
66 of hematopoietic stem cell self-renewal and megakaryocytopoiesis in the bone marrow microenvironment
67 Recombinant human interleukin-11 stimulates megakaryocytopoiesis in vitro and platelet production in
72 ess the role of P-selectin and E-selectin in megakaryocytopoiesis, in vitro assays were performed in
77 o further investigate the role of Lyn during megakaryocytopoiesis, Lyn-deficient mice (lyn(-/-)) were
79 growth and development factor (rHu-MGDF) on megakaryocytopoiesis, platelet function, and thrombogene
80 The effects of thrombopoietic stimulation on megakaryocytopoiesis, platelet production, and platelet
81 effects of PEG-rHuMGDF on pharmacokinetics, megakaryocytopoiesis, platelet production, and platelet
82 h likely compensates for the impaired marrow megakaryocytopoiesis, resulting in normal peripheral pla
83 these cytokines play a critical role in the megakaryocytopoiesis seen in patients who develop reacti
84 fication of TRIB3 as a negative regulator of megakaryocytopoiesis suggests that in-vivo this gene cou
85 thrombopoietin (TPO) and its contribution to megakaryocytopoiesis, the exact mechanisms and sites of
86 ated bioenergetic pathway governing terminal megakaryocytopoiesis; these observations also define a m
87 ligand for c-mpl and the major regulator of megakaryocytopoiesis, TPO deficient mice were generated
88 mportance of stathmin down-regulation during megakaryocytopoiesis, we used a lentiviral-mediated gene
90 s exhibited excessive splenic extramedullary megakaryocytopoiesis, which likely compensates for the i
91 miR-150 and miR-155 play divergent roles in megakaryocytopoiesis, with the former promoting developm
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