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1 tion as a means to prevent pregnancy-induced megaloblastic anemia.
2 parable with those observed in patients with megaloblastic anemia.
3 en the S-phase accumulation and apoptosis in megaloblastic anemia.
4 thesis of thymidylate in the pathogenesis of megaloblastic anemia.
5  the metabolic disorder, thiamine-responsive megaloblastic anemia.
6 es explanations for the clinical findings in megaloblastic anemia.
7           Imerslund-Grasbeck syndrome (I-GS, megaloblastic anemia 1) is an autosomal recessive disord
8  of vitamin B12 (a phenotype associated with megaloblastic anemia 1, MGA1; OMIM 261100; refs.
9 f folate or vitamin B(12) (cobalamin) causes megaloblastic anemia, a disease characterized by pancyto
10 Mutations in THTR-1 cause thiamin-responsive megaloblastic anemia, a tissue-specific disease associat
11                          Thiamine-responsive megaloblastic anemia, also known as "TRMA" or "Rogers sy
12                Vitamin B12 deficiency causes megaloblastic anemia and neurologic disorder in humans.
13 r de novo dTMP biosynthesis can lead to both megaloblastic anemia and SCID in MTHFD1 deficiency.
14 TRMA) is characterized by diabetes mellitus, megaloblastic anemia and sensorineural deafness.
15 inborn error of metabolism, characterized by megaloblastic anemia and/or pancytopenia, severe cerebra
16       B12 deficiency is the leading cause of megaloblastic anemia, and although more common in the el
17             The proband presented with SCID, megaloblastic anemia, and neurologic abnormalities, but
18 c disease associated with diabetes mellitus, megaloblastic anemia, and sensorineural deafness.
19 data) for risk of deficiency on the basis of megaloblastic anemia as a hematologic indicator in perso
20 ary levels of the vitamin folate can lead to megaloblastic anemia, birth defects, impaired cognitive
21 normal erythropoiesis is a common feature of megaloblastic anemias, congenital dyserythropoietic anem
22       "Cyclin D1-only" mice developed severe megaloblastic anemia, "cyclin D2-only" mice presented ne
23          A mouse model of thiamin-responsive megaloblastic anemia (diabetes mellitus, deafness, megal
24 essive disorder defined by the occurrence of megaloblastic anemia, diabetes mellitus, and sensorineur
25                          Hereditary juvenile megaloblastic anemia due to vitamin B12 (cobalamin) defi
26 paired purine nucleotide metabolism, whereas megaloblastic anemia has been associated with impaired d
27 dies of impaired DNA synthesis and repair in megaloblastic anemia have concerned mainly the decreased
28         Clinical and experimental studies of megaloblastic anemia have demonstrated an impairment of
29 wed not only as a nutrient needed to prevent megaloblastic anemia in pregnancy but also as a vitamin
30 sm, the degree of uracil misincorporation in megaloblastic anemia is sufficient to increase the stead
31 blastic anemia (diabetes mellitus, deafness, megaloblastic anemia) lacking functional Slc19a2 has bee
32 -vitamin B12 receptor, results in hereditary megaloblastic anemia (MGA1), owing to vitamin B12 malabs
33                 Although rarely resulting in megaloblastic anemia, mild deficiency may be associated
34 amage, accounting for the pathophysiology of megaloblastic anemia observed in vitamin B12 and folate
35 However, in patients with thiamin-responsive megaloblastic anemia, plasma thiamin levels are within n
36 and symptoms of vitamin B12 deficiency, e.g. megaloblastic anemia, precise evaluation and treatment i
37 Cubulin mutations cause a hereditary form of megaloblastic anemia secondary to vitamin B(12) deficien
38                           Thiamin-responsive megaloblastic anemia syndrome (TRMA) is characterized by
39       Pernicious anemia is a common cause of megaloblastic anemia throughout the world and especially
40 isincorporated into the DNA of patients with megaloblastic anemia to levels detectable by nonradioact
41 lasts from patients with thiamine-responsive megaloblastic anemia (TRMA) syndrome with diabetes and d
42 y of the syndrome called thiamine-responsive megaloblastic anemia (TRMA) with diabetes and deafness.
43 iency disorders, such as thiamine-responsive megaloblastic anemia (TRMA), which is associated with sp
44  parents had diabetes mellitus, deafness, or megaloblastic anemia, which raised the possibility that
45 linical vitamin B12 deficiency can result in megaloblastic anemia, which results from the inhibition
46                             They suffer from megaloblastic anemia with or without some degree of neur

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