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1 ned with a selective MC3-R agonist (gamma(2)-melanocyte-stimulating hormone).
2 ormone = adrenocorticotropic hormone > gamma-melanocyte-stimulating hormone).
3 asopressin, agouti-related protein and alpha-melanocyte stimulating hormone.
4 mentation does not appear to be regulated by melanocyte stimulating hormone.
5 table levels of adrenocorticotropin or alpha-melanocyte-stimulating hormone.
6 ed how these responses are affected by alpha-melanocyte-stimulating hormone.
7 ction but this response was reduced by alpha-melanocyte-stimulating hormone.
8 e levels were increased in response to alpha-melanocyte-stimulating hormone.
9 mulation of tyrosinase gene transcription by melanocyte-stimulating hormone.
10 on stimulation with its natural ligand alpha-melanocyte-stimulating hormone.
11 d the positive effects of [Nle, D-Phe]-alpha-melanocyte-stimulating hormone.
12 ucagon and proopiomelanocortin-derived alpha-melanocyte-stimulating hormone.
14 h a selective MC3R agonist ([D-Trp(8)]-gamma-melanocyte-stimulating hormone; 10 microg i.v.) produced
15 des alpha-melanocyte-stimulating hormone and melanocyte-stimulating hormone 11-13 (KP-D-V) on the inv
16 nt in the aqueous humor (10-9 M), as well as melanocyte-stimulating hormone 11-13 (KP-D-V) reduced th
17 (188)Re-(Arg(11))[Cys(3,4,10),d-Phe(7)]alpha-melanocyte-stimulating hormone(3-13) (CCMSH) in the B16/
18 rahydrobiopterin resulting in a stable alpha-melanocyte stimulating hormone/6(R)-L-erythro 5,6,7,8 te
19 cid, and niacinamide) and stimulators (alpha-melanocyte-stimulating hormone, 8-methoxypsoralen, and 3
20 bitor of rac1 abrogated the ability of alpha-melanocyte stimulating hormone, a peptide hormone known
22 f the melanotropin peptides alpha-MSH (alpha-melanocyte-stimulating hormone, Ac-Ser-Tyr-Met-Glu- His-
23 his study provides further evidence of alpha-melanocyte stimulating hormone acting to "protect" melan
24 similar in these two species homologs (alpha-melanocyte-stimulating hormone = adrenocorticotropic hor
25 For this purpose antibodies against alpha-melanocyte stimulating hormone, adrenocorticotropin, pro
27 eceptor its and adrenocorticotropic hormone, melanocyte stimulating hormone, agouti protein ligands (
29 ts of two melanocortin system ligands, alpha melanocyte stimulating hormone (alpha-MSH) and agouti-re
30 The regulated release of anorexigenic alpha-melanocyte stimulating hormone (alpha-MSH) and orexigeni
32 ic hormone (ACTH), beta-endorphin, and alpha-melanocyte stimulating hormone (alpha-MSH) are synthesiz
33 n the ARC, IRS-2 was co-localized with alpha melanocyte stimulating hormone (alpha-MSH) as well as ne
34 orms act as competitive antagonists of alpha-melanocyte stimulating hormone (alpha-MSH) at melanocort
35 ne whether the neuroprotective peptide alpha-melanocyte stimulating hormone (alpha-MSH) attenuates GA
37 ulated transcript (CART) coexists with alpha-melanocyte stimulating hormone (alpha-MSH) in the arcuat
38 leus (ARC), and on opioid peptides and alpha-melanocyte stimulating hormone (alpha-MSH) in the parave
41 lanocortin (POMC)-derived neuropeptide alpha-melanocyte stimulating hormone (alpha-MSH) is known to m
43 compared with four other radiolabeled alpha-melanocyte stimulating hormone (alpha-MSH) peptide analo
44 sp-conjugated and X-Ala-Asp-conjugated alpha-melanocyte stimulating hormone (alpha-MSH) peptides.
45 w (99m)Tc-labeled Arg-X-Asp-conjugated alpha-melanocyte stimulating hormone (alpha-MSH) peptides.
46 a prevention based on using analogs of alpha-melanocyte stimulating hormone (alpha-MSH) that function
49 age chemoattractant protein-1 (MCP-1), alpha-melanocyte stimulating hormone (alpha-MSH), and peroxiso
50 ReCCMSH(Arg(11)), a cyclic analogue of alpha-melanocyte stimulating hormone (alpha-MSH), exhibited hi
51 tor (CRF) might be mediated via MC4-R. alpha-Melanocyte stimulating hormone (alpha-MSH), the MC4-R ag
52 ing the production of the MC4R ligand, alpha-melanocyte stimulating hormone (alpha-MSH), to regulate
53 C4-R agonists, melanotan-II (MT-II) or alpha-melanocyte stimulating hormone (alpha-MSH), were unilate
57 ls was blocked by co-administration of alpha-melanocyte stimulating hormone (alpha-MSH, 20 ng), a hor
58 tivity of leptin, the leptin receptor, alpha-melanocyte stimulating hormones (alpha-MSH) and the mela
59 er the functional IL-1beta antagonist, alpha-melanocyte-stimulating hormone (alpha-MSH(1-13)), would
60 ibution, and clearance kinetics of the alpha-melanocyte-stimulating hormone (alpha-MSH) analog 1,4,7,
62 eport in vitro and in vivo data of new alpha-melanocyte-stimulating hormone (alpha-MSH) analogues whi
63 NPY) and anorexigenic peptides such as alpha-melanocyte-stimulating hormone (alpha-MSH) and anorexige
64 n, resulting in selective increases of alpha-melanocyte-stimulating hormone (alpha-Msh) and carboxy-c
65 ein-coupled receptor (GPCR) that binds alpha-melanocyte-stimulating hormone (alpha-MSH) and has a cen
67 he melanocortin system, which includes alpha-melanocyte-stimulating hormone (alpha-MSH) and its endog
68 ese proteins antagonize the effects of alpha-melanocyte-stimulating hormone (alpha-MSH) and other mel
69 ng effects of the anorexigenic agonist alpha-melanocyte-stimulating hormone (alpha-MSH) and the orexi
72 ntake and PK2 increased the release of alpha-melanocyte-stimulating hormone (alpha-MSH) from ex vivo
73 recordings revealed that both NPY and alpha-melanocyte-stimulating hormone (alpha-MSH) inhibit and s
77 the appetite-suppressing neuropeptide alpha-melanocyte-stimulating hormone (alpha-MSH) is reduced, y
78 TH) of pars distalis corticotropes and alpha-melanocyte-stimulating hormone (alpha-MSH) of pars inter
79 s study aimed at revealing the role of alpha-melanocyte-stimulating hormone (alpha-MSH) on basophil f
81 melanocortin receptor agonists such as alpha-melanocyte-stimulating hormone (alpha-MSH) or antagonist
82 phage that displayed up to 5 copies of alpha-melanocyte-stimulating hormone (alpha-MSH) peptide analo
83 he radiolabeled lactam bridge-cyclized alpha-melanocyte-stimulating hormone (alpha-MSH) peptide on it
84 e whether (99m)Tc- and (111)In-labeled alpha-melanocyte-stimulating hormone (alpha-MSH) peptides coul
85 ) neurons and the POMC-derived peptide alpha-melanocyte-stimulating hormone (alpha-MSH) promote satie
87 type 1 receptor (MC1R), also known as alpha-melanocyte-stimulating hormone (alpha-MSH) receptor, is
88 ion (suntanning) requires induction of alpha-melanocyte-stimulating hormone (alpha-MSH) secretion by
89 Activation of MC1R in melanocytes by alpha-melanocyte-stimulating hormone (alpha-MSH) stimulates cA
92 prolylcarboxypeptidase (PRCP) degrades alpha-melanocyte-stimulating hormone (alpha-MSH) to an inactiv
93 t color by antagonizing the binding of alpha-melanocyte-stimulating hormone (alpha-MSH) to the alpha-
94 us of the hypothalamus (PVN), to block alpha-melanocyte-stimulating hormone (alpha-MSH) type 3 and 4
95 1R, inhibiting the alpha-melanocortin (alpha-melanocyte-stimulating hormone (alpha-MSH))-induced incr
96 elanocortin 4 (MC-4) receptors such as alpha-melanocyte-stimulating hormone (alpha-MSH), a product of
97 in the regulation of feeding by using alpha-melanocyte-stimulating hormone (alpha-MSH), an endogenou
98 -tetraacetic acid), a cyclic analog of alpha-melanocyte-stimulating hormone (alpha-MSH), has the pote
99 nd regulating release of products like alpha-melanocyte-stimulating hormone (alpha-MSH), neuropeptide
100 re potent appetite stimulants, whereas alpha-melanocyte-stimulating hormone (alpha-MSH), neurotensin,
101 ither a melanocortin receptor agonist, alpha-melanocyte-stimulating hormone (alpha-MSH), or antagonis
102 (NDP-MSH), a highly potent analogue of alpha-melanocyte-stimulating hormone (alpha-MSH), possesses na
103 designed to examine the evidence that alpha-melanocyte-stimulating hormone (alpha-MSH), which is tho
104 group of Lys(10) of the cyclic lactam alpha-melanocyte-stimulating hormone (alpha-MSH)-derived Pro(6
105 both direct and indirect regulation by alpha melanocyte-stimulating hormone (alpha-MSH)-synthesizing
112 ocked by 2 different IL-1 antagonists, alpha melanocyte stimulating hormone (alphaMSH) and interleuki
115 melanoma cells synthesize and release alpha-melanocyte stimulating hormone (alphaMSH, the ligand for
116 havior of these cells is influenced by alpha-melanocyte-stimulating hormone (alphaMSH) and melanin-co
118 erestingly, hypothalamic Pomc mRNA and alpha-melanocyte-stimulating hormone (alphaMSH) peptide levels
120 the rise in cAMP levels in response to alpha-melanocyte-stimulating hormone, an MC4R agonist, by bloc
122 ions did not affect the ability of the alpha-melanocyte stimulating hormone analogue [Nle4,D-Phe7] me
123 valuated the safety and efficacy of an alpha-melanocyte-stimulating hormone analogue, afamelanotide,
125 dendrite formation in B16F1 cells, and alpha-melanocyte stimulating hormone and ultraviolet light sti
126 ac1 mediates the well-known ability of alpha-melanocyte stimulating hormone and ultraviolet light to
127 We used known melanogenic stimulators (alpha-melanocyte-stimulating hormone and 3,4-dihydroxyphenylal
128 lanocortin peptides alpha-, beta-, and gamma-melanocyte-stimulating hormone and ACTH are full agonist
130 e proopiomelanocortin-derived peptides alpha-melanocyte-stimulating hormone and beta-endorphin, the g
131 enic neuropeptides pro-opiomelanocortinalpha-melanocyte-stimulating hormone and cocaine-amphetamine-r
132 and anorexigenic (pro-opiomelanocortinalpha-melanocyte-stimulating hormone and cocaine-amphetamine-r
135 ontraction in melanocytes, mediated by alpha-melanocyte-stimulating hormone and melanin-concentrating
136 and of the anti-inflammatory peptides alpha-melanocyte-stimulating hormone and melanocyte-stimulatin
137 -derived macrophages, we observed that alpha-melanocyte-stimulating hormone and selective MC1-R agoni
138 wn that a cell-permeable peptide, a hormone (melanocyte stimulating hormone), and a blood-clotting ag
139 tra of the model peptides (bradykinin, alpha-melanocyte stimulating hormone, and melittin) change sig
140 omelanocortin (POMC)-derived peptides, alpha-melanocyte-stimulating hormone, and adrenocorticotropic
141 rphin A-17, a decrease in the level of alpha-melanocyte-stimulating hormone, and an alteration in the
142 encodes both the anorexigenic peptide alpha-melanocyte-stimulating hormone, and the opioid peptide b
143 including arbutin, hydroquinone, kojic acid, melanocyte-stimulating hormone, and thymidine dimers) as
144 adrenocorticotropin, alpha-, beta- and gamma-melanocyte stimulating hormone; and the endogenous opioi
145 to investigate the mechanism by which alpha-melanocyte-stimulating hormone antagonizes proinflammato
146 and that the anti-invasive actions of alpha-melanocyte stimulating hormone are consistent with its w
148 stochastic carcinogenesis and identify alpha-melanocyte-stimulating hormone as a potential attenuatin
149 Additionally, our results identify alpha-melanocyte-stimulating hormone as a potential mediator o
150 s through the blockade of signaling by alpha-melanocyte-stimulating hormone at central nervous system
152 Notably, the density of anorexigenic alpha-melanocyte-stimulating hormone axons was reduced in adul
153 s of novel, disulfide-constrained human beta-melanocyte stimulating hormone (beta-MSH)-derived peptid
154 rupts the dibasic cleavage site between beta melanocyte-stimulating hormone (beta-MSH) and beta-endor
156 ptides include adrenocorticotrophic hormone, melanocyte stimulating hormone, beta-lipotrophin, and th
157 e precursor for adrenocorticotropic hormone, melanocyte-stimulating hormones, beta-lipotropic hormone
158 prevented the inhibitory actions of gamma(2)-melanocyte stimulating hormone both in vitro and in vivo
159 by the more selective MC3-R agonist gamma(2)-melanocyte stimulating hormone but not by the selective
160 n S-91 mouse melanoma cells respond to alpha-melanocyte-stimulating hormone) by demonstrating a marke
162 ear run-off transcription assays showed that melanocyte-stimulating hormone caused a slow increase in
163 mers with different molecular sizes, [d-Trp]-melanocyte-stimulating hormone, [d-Ala]-deltorphin, [d-P
164 At concentrations in excess of 10-9 M alpha-melanocyte-stimulating hormone decreased nitric oxide pr
165 cell surface, but it does not restore alpha-melanocyte-stimulating hormone-dependent cAMP signaling.
166 Tc(CO)3-labeled lactam bridge-cyclized alpha-melanocyte stimulating hormone derivative, betaAlaNleCyc
167 hat inhibits the binding and action of alpha-melanocyte-stimulating hormone derived from proopiomelan
168 ow report that treatment of melanocytes with melanocyte-stimulating hormone down-regulates expression
169 melanoma cells were exposed continuously to melanocyte-stimulating hormone for 6 d, a large but tran
172 rly to the previously described cyclic gamma-melanocyte-stimulating hormone (gamma-MSH)-derived hMC3R
177 minase-containing neurons) but not others (a-melanocyte-stimulating hormone), indicating specificity
178 te was unaffected by treatment of cells with melanocyte-stimulating hormone, indicating that the horm
180 ment with cycloheximide had no effect on the melanocyte-stimulating hormone-induced increase in tyros
183 (dynorphin A-17, beta-endorphin, and alpha- melanocyte-stimulating hormone) involved in the control
184 oduction by melanocytes in response to alpha-melanocyte-stimulating hormone is associated with such a
187 n of the fibroblast growth factor (FGFR) and melanocyte stimulating hormone (MC1R) receptors stimulat
188 grin expression and ask to what extent alpha-melanocyte stimulating hormone might protect cells from
189 two intercellular signaling molecules, alpha-melanocyte stimulating hormone (MSH) and agouti signal p
190 uces activation of regulatory T cells, alpha-melanocyte stimulating hormone (MSH) and transforming gr
191 uggested a critical role for the MC1R ligand melanocyte stimulating hormone (MSH) in this response, a
192 ignaling either by the agonistic MC1R ligand melanocyte stimulating hormone (MSH) or by pharmacologic
194 , from the absence of induction by leptin of melanocyte stimulating hormone (MSH) secretion in the hy
195 gmentation in mammals is stimulated by alpha-melanocyte stimulating hormone (MSH), which binds to the
198 s agonists such as alpha-, beta-, and gamma2-melanocyte stimulating hormones (MSH) and adrenocorticot
199 genous agonists [alpha-, beta-, and gamma(2)-melanocyte stimulating hormones (MSH) and adrenocorticot
200 ptor agonists including alpha-, beta-, gamma-melanocyte stimulating hormones (MSH) and adrenocorticot
201 he intermediate lobe melanotropes, producing melanocyte-stimulating hormone (MSH alpha), have remaine
202 two intercellular signaling molecules, alpha-melanocyte-stimulating hormone (MSH) and agouti signal p
203 r MMS increases the response of S91 cells to melanocyte-stimulating hormone (MSH) and increases the b
204 the selective MC3R agonist [d-Trp(8)]-gamma-melanocyte-stimulating hormone (MSH) and of the recessiv
206 mes in melanocytes, whereas treatment with a-melanocyte-stimulating hormone (MSH) induced exocytosis
207 zed rats, unilateral microinjection of alpha-melanocyte-stimulating hormone (MSH) into the medullary
212 scopic visualization and characterization of melanocyte-stimulating hormone (MSH) receptors of melano
216 e and functionality of ACTH, alpha- and beta-melanocyte-stimulating hormone (MSH), and beta-endorphin
217 ion throughout the cytoplasm is triggered by melanocyte-stimulating hormone (MSH), with both of these
219 omplete loss of both [Nle(4)-d-Phe(7)]-alpha-melanocyte stimulating hormone (NDP-MSH) binding and NDP
220 activity studies of [Nle(4), D-Phe(7)]-alpha-melanocyte stimulating hormone (NDP-MSH) identified D-Ph
221 uti-related protein (AGRP)/[Nle4,DPhe7]alpha-melanocyte stimulating hormone (NDP-MSH) ligands is repo
222 rotective effect of [Nle(4), D-Phe(7)]-alpha-melanocyte stimulating hormone (NDP-MSH), a potent non-s
224 g assays for agonist, [Nle(4)-d-Phe(7)]alpha-melanocyte-stimulating hormone (NDP-alpha-MSH) or forsko
225 human MC4R activation by [Nle4, d-Phe7]alpha-melanocyte-stimulating hormone (NDP-MSH), by first defin
226 oximity to the bound ligand [Nle(4),D-Phe(7)]melanocyte-stimulating hormone (NDP-MSH), thereby differ
227 neuropeptide glutamic acid-isoleucine/alpha-melanocyte-stimulating hormone (NEI/alphaMSH) peptides.
228 were carried out to determine the effect of melanocyte-stimulating hormone on tyrosinase gene transc
230 grown in the presence of 10-11-10-9 M alpha-melanocyte-stimulating hormone prior to stimulation.
231 gh actions on TRH neurons, addition of alpha-melanocyte-stimulating hormone produced a 3.5-fold incre
232 ultures prepared from 7B2 null mice restored melanocyte-stimulating hormone production, substantiatin
233 the expression of proopiomelanocortin/alpha-melanocyte-stimulating hormone, provoked by C75 and reve
235 e mutation in a classical pigmentation gene, melanocyte stimulating hormone receptor (MC1R), is stron
236 e skin, is a high-affinity antagonist of the melanocyte-stimulating hormone receptor (MC1-R), thus ex
237 aberrantly antagonizes the MC4-R, a related melanocyte-stimulating hormone receptor expressed in hyp
240 alpha-melanocyte--stimulating hormone to the melanocyte-stimulating--hormone receptor (MSHR), the pro
241 ologies concerning cells which express alpha-melanocyte-stimulating hormone receptors and utilize the
243 icotropin-releasing factor, leptin and alpha-melanocyte stimulating hormone regulate cytokine balance
246 ature just before light onset, whereas alpha-melanocyte stimulating hormone release, especially at th
247 ively increases beta-endorphin but not alpha-melanocyte-stimulating hormone release in the hypothalam
249 y, whereas receptor binding with the agonist melanocyte-stimulating hormone resulted in an increased
250 h the melanocortin analog [Nle, D-Phe]-alpha-melanocyte-stimulating hormone (starting 3 or 6 hrs afte
251 ndothelin 1, hepatocyte growth factor, alpha-melanocyte stimulating hormone, stem cell factor, and fi
252 Neuropeptide Y (NPY) inhibited and alpha-melanocyte-stimulating hormone stimulated ARC glucose-ex
253 rease in tyrosinase mRNA occurred 60 h after melanocyte-stimulating hormone stimulation and was follo
254 tial glands of rodents is regulated by alpha-melanocyte stimulating hormone, the major agonist for me
255 ith the regulation of melanogenesis by alpha-melanocyte-stimulating hormone through melanocortin 1 re
256 e stimulating hormone analogue [Nle4,D-Phe7] melanocyte stimulating hormone to bind or activate the c
257 tern immunoblotting and the ability of alpha-melanocyte stimulating hormone to oppose the actions of
260 of NF-kappaB DNA binding activity with alpha-melanocyte-stimulating hormone was detected 2 h after ce
261 n cAMP in response to stimulation with alpha-melanocyte-stimulating hormone was measured in HEK-293 c
262 vating cyclic adenosine monophosphate, alpha-melanocyte-stimulating hormone was not found to have any
263 appear to be physiological targets of alpha-melanocyte-stimulating hormone, which inhibits food inta
264 d tumorigenicity assays indicates that alpha-melanocyte-stimulating hormone, which is overproduced by
265 onses to the cAMP agonists cholera toxin and melanocyte-stimulating hormone, which normally promote m
266 ch aims to maintain high urine output; alpha-melanocyte-stimulating hormone, with anti-inflammatory a
267 and in a subset of cells coexpressing alpha-melanocyte-stimulating hormone within the pituitary pars
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